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排序方式: 共有173条查询结果,搜索用时 15 毫秒
81.
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R. P. Evstigneeva N. V. Sergeeva T. E. Rudakova G. S. Shaimardanova P. M. Kochergin 《Pharmaceutical Chemistry Journal》1992,26(5):423-425
Translated from Khimiko-farmatsevticheskii Zhurnal, Vol. 26, No. 5, pp. 48–50, May, 1992. 相似文献
84.
R. P. Evstigneeva N. V. Sergeeva T. E. Rudakova S. V. Shorshnev 《Pharmaceutical Chemistry Journal》1991,25(8):550-552
Translated from Khimiko-farmatsevticheskii Zhurnal, Vol. 25, No. 8, pp. 36–38, August, 1991. 相似文献
85.
86.
S. F. Rudakova V. K. Ivanov I. A. Rudakov 《Bulletin of experimental biology and medicine》1974,78(6):1411-1413
In two independent series of experiments, in each of which 16 monolayer cultures of guinea pig spleen cells were set up (four at each time), the distribution of the colonies formed by their diameter was studied 6, 7, 10, and 12 days after explanation. At all times studied the distribution of the colonies corresponded to the normal law of distribution; this points to homogeneity of the colony-forming cell population in the hematopoietic organs of guinea pigs as regards their ability to form colonies in monolayer cultures. 相似文献
87.
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Galstian GM Vasil'ev SA Galuziak VS Likhacheva EA Pliushch OP Rudakova VE Riazanova IB Sakhibov IaD Togonidze DK Khorobrykh LS 《Terapevticheski? arkhiv》2005,77(12):33-39
AIM: To ascertain risk factors of thromboembolism of the pulmonary artery (TPA) in Willebrand's disease (WD). MATERIAL AND METHODS: We made a retrospective analysis of hospitalizations of WD patients for 10 years. We analysed causes of the patients' admission, interventions, registered maximal levels of factor VIII (FVIII) and Willebrand's factor (FW) in which the interventions were made, cases of TPA. RESULTS: Thirty four patients with WD were hospitalized 45 times. Three patients were treated conservatively because of gastrointestinal bleeding, the rest patients received surgical therapy. All the patients were given FVIII concentrates, cryoprecipitate, fresh-frozen plasma. In the course of the treatment, FW and FVIII levels were determined in 38% cases, FW--in 23%, FVIII--in 27%, coagulation was studied in 12% without test for FVIII and FW levels. Maximal concentration of FW was 72.1 +/- 11.8%, FVIII--125 +/- 15.8%. TPA developed in 2 (4.4%) of 45 patients. In both cases we observed a marked rise of plasmic concentration of FVIII due to therapy (250 and 240%). CONCLUSION: In patients with WD thromboembolic complications risk factors are age, obesity, surgical interventions, immobilization, etc. Simultaneous administration of several drugs containing FW and FVIII was also among the risk factors. Overdosage of FVIII is one of the causes of thrombotic complications in WD. FW and FVIII correlations in FVIII preparations must be considered. Prophylactic heparin therapy is recommended in patients with a high risk of thrombotic complications upon achievement of normal hemostasis. 相似文献
89.
Brown KA; Janjua AH; Karbani G; Parry G; Noble A; Crockford G; Bishop DT; Newton VE; Markham AF; Mueller RF 《Human molecular genetics》1996,5(1):169-173
Autosomal recessive non-syndromal hearing impairment (NSRD) is genetically
heterogeneous. Five loci have been identified to date which map to
chromosomes 13 (DFNB1), 11 (DFNB2), 17 (DFNB3), 7 (DFNB4) and 14 (DFBN5).
We report definite linkage of NSRD to the locus DFNB1 in a single family of
27 families studied of Pakistani origin. Haplotype analysis of markers in
the pericentromeric region of chromosome 13q revealed a recombination event
which maps DFNB1 proximal to the marker D13S175 and in the vicinity of
D13S143.
相似文献
90.