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Felipe S Ferreira Samuel V Brito Samuel C Ribeiro Ant?nio AF Saraiva Waltécio O Almeida R?mulo RN Alves 《BMC complementary and alternative medicine》2009,9(1):17-8
Background
Human communities consistently develop a detailed knowledge of the therapeutical and medicinal properties of the local flora and fauna, and these folk remedies often substitute medicines produced by the pharmaceutical industry. Animals (and their derived products) are essential ingredients in the preparation of many traditional remedies. The present work prepared an inventory of the animals sold in public markets in the cities of Crato and Juazeiro do Norte, Ceará State, Brazil. 相似文献114.
Morley KI Medland SE Ferreira MA Lynskey MT Montgomery GW Heath AC Madden PA Martin NG 《Behavior genetics》2006,36(1):87-99
Many twin studies have identified sex differences in the influence of genetic and environmental factors on smoking behaviors. We explore the evidence for sex differences for smoking initiation and cigarette consumption in a sample of Australian twin families, and extend these models to incorporate sex differences in linkage analyses for these traits. We further examine the impact of including or excluding non-smokers in genetic analyses of tobacco consumption. Accounting for sex differences improved linkage results in some instances. We identified one region suggestive of linkage on chromosome 11p12. This locus, as well as another region identified on chromosome 6p12, replicates regions identified in previous studies. 相似文献
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Authors – Thongudomporn U, Chongsuvivatwong V, Geater AF Objectives – To investigate to what extent maximum bite force contributes to alveolar bone morphology parameters, i.e. alveolar thickness, shape and arch width. Design – An observational cross‐sectional survey. Setting and Sample Population – One hundred and fifty one 12‐ to 14‐year‐old students from a secondary school in Hatyai City, Songkhla Province, Thailand. Material and Methods – Height, weight and maximum bite force of each subject were recorded. Alveolar bone morphology parameters were measured from study models. Results – Maximum bite force moderately correlated with alveolar thickness and shape (r = 0.31–0.44, p < 0.001), but weakly correlated with arch width (r = 0.03–0.05, p > 0.05). After adjusting for gender and body mass index (BMI), the maximum bite force significantly determined alveolar thickness and shape (p < 0.001), accounting for 10–20% of the variations. Boys were associated with larger posterior arch width (p < 0.01), where BMI was not associated with alveolar bone morphology parameters (p > 0.01) after Bonferroni correction for multiple testing. Conclusion – Maximum bite force had a selective influence on alveolar thickness and shape, but not on arch width. 相似文献
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Sarah AF Henkel Judy H Squires Mary Ayers Armando Ganoza Patrick Mckiernan James E Squires 《World journal of hepatology》2019,11(5):450-463
BACKGROUND Progressive familial intrahepatic cholestasis(PFIC) refers to a disparate group of autosomal recessive disorders that are linked by the inability to appropriately form and excrete bile from hepatocytes, resulting in a hepatocellular form of cholestasis. While the diagnosis of such disorders had historically been based on pattern recognition of unremitting cholestasis without other identified molecular or anatomic cause, recent scientific advancements have uncovered multiple specific responsible proteins. The variety of identified defects has resulted in an ever-broadening phenotypic spectrum, ranging from traditional benign recurrent jaundice to progressive cholestasis and end-stage liver disease.AIM To review current data on defects in bile acid homeostasis, explore the expanding knowledge base of genetic based diseases in this field, and report disease characteristics and management.METHODS We conducted a systemic review according to PRISMA guidelines. We performed a Medline/PubMed search in February-March 2019 for relevant articles relating to the understanding, diagnosis, and management of bile acid homeostasis with a focus on the family of diseases collectively known as PFIC. English only articles were accessed in full. The manual search included references of retrieved articles.We extracted data on disease characteristics, associations with other diseases, and treatment. Data was summarized and presented in text, figure, and table format.RESULTS Genetic-based liver disease resulting in the inability to properly form and secrete bile constitute an important cause of morbidity and mortality in children and increasingly in adults. A growing number of PFIC have been described based on an expanded understanding of biliary transport mechanism defects and the development of a common phenotype.CONCLUSION We present a summary of current advances made in a number of areas relevant to both the classically described FIC1(ATP8 B1), BSEP(ABCB11), and MDR3(ABCB4) transporter deficiencies, as well as more recently described gene mutations--TJP2(TJP2), FXR(NR1 H4), MYO5 B(MYO5 B), and others which expand the etiology and understanding of PFIC-related cholestatic diseases and bile transport. 相似文献
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Methodological recommendations for cognition trials in bipolar disorder by the International Society for Bipolar Disorders Targeting Cognition Task Force
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Campbell SE Filzen TW Bezzant SM Beall DP Burton MP Sanders TG Parsons TW 《Skeletal radiology》2003,32(4):231-235
This report describes a primary periosteal location of non-Hodgkin's lymphoma, without nodal disease, and without adjacent intramedullary disease at presentation. The clinical and imaging appearance of periosteal lymphoma simulates other neoplastic osseous surface tumors more than that of lymphoma in other locations. Consideration of this rare presentation of non-Hodgkin's lymphoma in the differential diagnosis of periosteal bone lesions can be helpful to ensure proper diagnosis and treatment. 相似文献
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A molecular basis for hemoglobin-H disease in American blacks 总被引:4,自引:0,他引:4
Phillips JA d; Scott AF; Smith KD; Young KE; Lightbody KL; Jiji RM; Kazazian HH Jr 《Blood》1979,54(6):1439-1445
We have applied gene counting and restriction endonuclease mapping techniques to the study of two American black families in which there were one or more cases of HbH disease. We found deletions of three of the four normal alpha-globin genes in individuals with HbH disease. In two of these individuals, the chromosome containing the single alpha gene could have originated by crossing over between mispaired alpha genes, resulting in a deletion of about 4.2 kilobases (kb). 相似文献