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Nobuaki Sakamoto Huanhuan Hu Akiko Nanri Tetsuya Mizoue Masafumi Eguchi Takeshi Kochi Tohru Nakagawa Toru Honda Shuichiro Yamamoto Takayuki Ogasawara Naoko Sasaki Akiko Nishihara Teppei Imai Toshiaki Miyamoto Makoto Yamamoto Hiroko Okazaki Kentaro Tomita Akihiko Uehara Ai Hori Makiko Shimizu Taizo Murakami Keisuke Kuwahara Ami Fukunaga Isamu Kabe Tomofumi Sone Seitaro Dohi 《Journal of diabetes investigation.》2020,11(3):719-725
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Michael Mlynash Marion S. Buckwalter Ami Okada Anna Finley Caulfield Chitra Venkatasubramanian Irina Eyngorn Marcel M. Verbeek Christine A. C. Wijman 《Neurocritical care》2013,19(2):161-166
Background
In comatose post-cardiac arrest patients, a serum neuron-specific enolase (NSE) level of >33 μg/L within 72 h was identified as a reliable marker for poor outcome in a large Dutch study (PROPAC), and this level was subsequently adopted in an American Academy of Neurology practice parameter. Later studies reported that NSE >33 μg/L is not a reliable predictor of poor prognosis. To test whether different clinical laboratories contribute to this variability, we compared NSE levels from the laboratory used in the PROPAC study (DLM-Nijmegen) with those of our hospital’s laboratory (ARUP) using paired blood samples.Methods
We prospectively enrolled cardiac arrest patients who remained comatose after resuscitation. During the first 3 days, paired blood samples for serum NSE were drawn at a median of 10 min apart. After standard preparation for each lab, one sample was sent to ARUP laboratories and the other to DLM-Nijmegen.Results
Fifty-four paired serum samples from 33 patients were included. Although the serum NSE measurements correlated well between laboratories (R = 0.91), the results from ARUP were approximately 30 % lower than those from DLM-Nijmegen. Therapeutic hypothermia did not affect this relationship. Two patients had favorable outcomes after hypothermia despite NSE levels measured by DLM-Nijmegen as >33 μg/L.Conclusions
Absolute serum NSE levels of comatose cardiac arrest patients differ between laboratories. Any specific absolute cut-off levels proposed to prognosticate poor outcome should not be used without detailed data on how neurologic outcomes correspond to a particular laboratory’s method, and even then only in conjunction with other prognostic variables. 相似文献26.
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Ami Inui Shunji Nakano Shinji Yoshioka Tomohiro Goto Daisuke Hamada Yoshiteru Kawasaki Hiroshi Egawa Natsuo Yasui 《European journal of orthopaedic surgery & traumatology : orthopedie traumatologie》2013,23(7):791-795
Introduction
Bone cyst formation in hips increases as osteoarthritis worsens. Although bone cysts in hips have been described in many studies, their etiology remains unclear and under debate. The purpose of this study was to investigate the communication between a bone cyst and the joint space, as well as the relationship between the severity of osteoarthritis and the formation of subchondral bone cysts in dysplastic hips.Method
We studied bone cysts from 150 dysplastic hips in 97 patients by computed tomography (CT) and plain radiography. We investigated the distribution of the bone cysts and the presence or absence of a communication path between the cysts and the joint space by three-dimensional (3D) CT.Result
Of the 150 hips, 94 acetabula and 55 femoral heads were found to contain cysts. Of the 94 hips containing acetabular cysts, 89 and 5 hips showed black lines and gray lines connecting the cyst and the joint space, respectively, on 3D-CT. The rate of cyst presentation in the hip increased as the joint space became narrower. The number of hips that possessed cysts in the anterior and/or middle portion was significantly higher than that in the posterior portions.Conclusion
Bone cysts in dysplastic osteoarthritic hips were found to communicate with the joint space in all cases. This suggests that the formation and enlargement of the cysts in dysplastic hips may be greatly influenced by the joint fluid. Cyst formation was initially observed in the anterior acetabulum, gradually progressing to involve the entire joint, including the posterior acetabulum and the femoral head, with worsening of the osteoarthritis. 相似文献30.