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121.
122.
Prediction of subtle left ventricular systolic dysfunction in homozygous and heterozygous familial hypercholesterolemia: Genetic analyses and speckle tracking echocardiography study 下载免费PDF全文
123.
Evaluation of Pulmonary Artery Stiffness in Patients with Obstructive Sleep Apnea Syndrome 下载免费PDF全文
124.
Mustafa Ahmed M.D. Houng Le M.D. Juan M. Aranda Jr. M.D. Charles T. Klodell M.D. 《Journal of cardiac surgery》2012,27(5):639-642
Abstract As the number of heart failure patients supported with left ventricular assist devices (LVAD) increases, the frequency of elective, noncardiac surgery in this patient population will similarly rise. We retrospectively analyzed our LVAD patient database and identified six patients who underwent elective, noncardiac surgery while on LVAD support. These cases are discussed, with an emphasis on the anesthetic and perioperative considerations. These patients have an acceptable risk profile for elective surgery and should be treated similarly to their age‐matched counterparts not on LVAD support. (J Card Surg 2012;27:639‐642) 相似文献
125.
Mustafa I. Ahmed M.D. Gulay Gok M.D. Burcu Yuzbas M.D. Joshua Burkhart M.D. Maximiliano German Amado Escañuela M.D. Oluseun O. Alli M.D. Navin C. Nanda M.D. 《Echocardiography (Mount Kisco, N.Y.)》2014,31(6):779-782
We describe the incremental value of live/real time three‐dimensional (3D) echocardiography over the 2D technique in the assessment of ruptured right sinus of Valsalva into the right ventricle associated with an adjacent perimembranous ventricular septal defect in an elderly patient. 相似文献
126.
Mufide Oncel Seval Akbulut Turkan Toka Ozer Aysel Kiyici Mustafa Keles Beyhan Baltaci 《Renal failure》2016,38(7):1071-1075
Background: Cytokines are essential mediators of immune response. Chronic renal failure patients suffer from chronic inflammation that results from factors such as impaired renal function, accumulation of uremic toxins and bio incompatibility of dialyzer membranes. These patients are also at increased risk of cardiovascular diseases. We have evaluated cytokines, adipocytokines and inflammatory markers in patients with chronic renal failure undergoing hemodialysis (HD) and continuous ambulatory peritoneal dialysis (CAPD).Material and methods: We have determined serum tumor necrosis factor-α (TNF-α), interleukin-6 (IL-6), high-sensitivity C-reactive protein (hsCRP), leptin and ghrelin levels of chronic renal failure patients treated with either HD (n?=?20) or CAPD (n?=?20). TNF-α, IL-6, ghrelin and leptin measurements were performed by commercially available kits based on enzyme-linked immunosorbent assay (ELISA) method. hsCRP levels were determined by turbidimetric methods.Results: Serum TNF-α and IL-6 levels of patients on HD were significantly higher than those of the ones on CAPD (p?<?0.05). Ghrelin, leptin and hsCRP concentrations were similar in both groups.Conclusions: We can conclude that cytokine production is more obvious in HD process. 相似文献
127.
128.
Harun Kilic M.D. Mustafa M. Balcı M.D. Murat N. Aksoy M.D. Esra Bilgin M.D. Kevser G. Gülsoy M.D. Ekrem Yeter M.D. Sadik Acikel M.D. Ramazan Akdemir M.D. 《Echocardiography (Mount Kisco, N.Y.)》2010,27(6):687-690
Purpose: To evaluate whether patent foramen ovale (PFO) is a contributing factor to hypoxia in patients with chronic obstructive pulmonary disease (COPD). Methods: Twenty‐one patients over 40 years of age with mild COPD (Forced expiratory volume (FEV1)/Forced Vital Capacity (FVC): > 50%) who had hypoxia (PO2 < 80 mmHg, SaO2 < 95%) that could not be explained by COPD alone were included in this study. Arterial oxygen pressures (PO2) and arterial oxygen saturations (SaO2) were recorded from laboratory evaluations of arterial blood gases. Respiratory function tests were performed to analyze the degree of COPD. Standard and contrast echocardiography was used to calculate pulmonary artery pressure (PAP) levels and to determine patients with a PFO. Results: The mean age of the patients was 64 ± 12 years. Four patients (19%) had a PFO. The mean PO2, mean SaO2, and mean PAP levels were 57.4 ± 6.8 mmHg, 90 ± 3.2%, and 33.8 ± 5.4 mmHg, respectively, in patients without PFO. The mean PO2, mean SaO2, and mean PAP levels were 46.5 ± 13.7 mmHg, 79.3 ± 12.8%, and 42.5 ± 6.5 mmHg, respectively, in patients with PFO. There were no statistically significant differences noted between the two groups in the PO2 levels (P = 0.172) and SaO2 levels (P = 0.065). A comparison of the PAP levels revealed a statistically significant difference between the two groups, with values that were more elevated in the PFO group than in the non‐PFO group (P = 0.031). Conclusion: This study demonstrated that PFO is not a contributing factor to deep hypoxia in COPD patients with lower PO2 and SaO2 levels; however, higher PAP levels were detected in patients with a PFO. Further studies involving a larger number of patients are needed to be conclusive. (Echocardiography 2010;27:687‐690) 相似文献
129.
Histopathology of the alimentary canal of Chirruh snowtrout, Scizothorax esocinus (Heckel), naturally infected with the acanthocephalan parasite, Pomphorhynchus kashmiriensis was studied by light microscopy. The proboscis and bulb was found to be deeply penetrated into the host tissues. Macroscopic
examination revealed over secretion of mucous and shedding of host tissues at the host parasite interface and white fibrous
nodules on the external surface of infected intestine, which was an indication for the presence of parasite. The major changes
in parasite induced histopathology were at the site of attachment to the host’s intestine which includes destruction of villi
and epithelial linings. Increased cellular infiltrations at the site of attachment may be a consequence of host’s defence
involving cell mediated immunity. In the areas of trunk contact with the host tissue, compression/absence of intestinal folds
and loss of columnar appearance of epithelial cells were evident. 相似文献
130.
Fahrettin Yildiz Alpaslan Terzi Sacid Coban Hakim Celik Nurten Aksoy Muharrem Bitiren Hale Çakir Mustafa K Ozdogan 《Journal of gastroenterology and hepatology》2009,24(11):1781-1785
Background and Aim: The aim of this study was to determine whether resveratrol could prevent intestinal tissue injury induced by ischemia–reperfusion (I/R).
Methods: Intestinal I/R was induced in rats' intestines by 60-min occlusion of the superior mesenteric artery, followed by a 60-min reperfusion. Thirty rats were divided into three groups as follows: sham (group 1), control (group 2), and the treatment groups (group 3). The rats in the treatment group received resveratrol both before ischemia and before reperfusion. In all groups, serum aspartate aminotransferase, alanine aminotransferase, and lactate dehydrogenase levels were determined. Total antioxidant capacity (TAC), catalase, total oxidative status (TOS), oxidative stress index (OSI), and myeloperoxidase (MPO) in the intestinal tissue were measured. Intestinal tissue histopathology was also evaluated by light microscopy.
Results: The levels of liver enzymes in group 3 were significantly lower than those in group 2 ( P < 0.05). TAC in the intestinal tissue was significantly higher in group 3 than in group 2 ( P < 0.05). TOS, OSI, and MPO in the intestinal tissue were significantly lower in group 3 than in group 2 ( P < 0.05 for all). Histological tissue damage was milder in the resveratrol treatment group than in the control group.
Conclusions: The results of this study indicated that resveratrol treatment limits the oxidative injury of the small intestine induced by I/R in rats. However, more precise investigations are required to evaluate the antioxidative effect of resveratrol on small intestine tissue damage in clinical and experimental models. 相似文献
Methods: Intestinal I/R was induced in rats' intestines by 60-min occlusion of the superior mesenteric artery, followed by a 60-min reperfusion. Thirty rats were divided into three groups as follows: sham (group 1), control (group 2), and the treatment groups (group 3). The rats in the treatment group received resveratrol both before ischemia and before reperfusion. In all groups, serum aspartate aminotransferase, alanine aminotransferase, and lactate dehydrogenase levels were determined. Total antioxidant capacity (TAC), catalase, total oxidative status (TOS), oxidative stress index (OSI), and myeloperoxidase (MPO) in the intestinal tissue were measured. Intestinal tissue histopathology was also evaluated by light microscopy.
Results: The levels of liver enzymes in group 3 were significantly lower than those in group 2 ( P < 0.05). TAC in the intestinal tissue was significantly higher in group 3 than in group 2 ( P < 0.05). TOS, OSI, and MPO in the intestinal tissue were significantly lower in group 3 than in group 2 ( P < 0.05 for all). Histological tissue damage was milder in the resveratrol treatment group than in the control group.
Conclusions: The results of this study indicated that resveratrol treatment limits the oxidative injury of the small intestine induced by I/R in rats. However, more precise investigations are required to evaluate the antioxidative effect of resveratrol on small intestine tissue damage in clinical and experimental models. 相似文献