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151.
Krimer LS; Herman MM; Saunders RC; Boyd JC; Hyde TM; Carter JM; Kleinman JE; Weinberger DR 《Cerebral cortex (New York, N.Y. : 1991)》1997,7(8):732-739
The entorhinal cortex (ERC) has been implicated in schizophrenia by a
number of studies. There is anatomical observation of neuronal heterotopias
in the rostral ERC, which is consistent with a hypothesis of
neurodevelopmental abnormalities in this disease. In view of the
significant cytoarchitectonic variation of the ERC throughout its
rostro-caudal extent, we performed a detailed subareal analysis of the
rostral two-thirds of the entorhinal cortex (ERCr) in 14 postmortem
schizophrenic brains and 14 matched controls (mean ages of 48 and 47
respectively). This systematic evaluation included both a qualitative
microscopic analysis of morphogenetic anomalies that would be consistent
with neurodevelopmental pathology and quantitative measurements of total
neuronal number, average neuronal density, laminar volume and laminar depth
from the cortical surface in cytoarchitectonically matched subareas of
schizophrenic and control brains. Parcellation of the entire ERC on the
basis of cytoarchitectonic criteria identified five distinct regions,
similar to those described in the macaque, except that in the human brain
three of the regions were further divisible into two or three subareas,
yielding nine distinct cellular compartments. Five rostral areas, prorhinal
(Pr), lateral (28L), intermediate rostral and caudal (281r and 281c), and
sulcal (28S), comprise the ERCr. Gross and microscopic examination of these
subdivisions throughout the ERCr failed to reveal laminar disorganization
in any of the schizophrenic brains. The brains also did not differ
significantly with respect to total neuronal number, total volume and
neuronal density per laminar and subareal subdivision, or laminar thickness
per entorhinal subarea. However, neuronal number and density were reduced
by 12-18% in Pr and 28L, suggesting that mild quantitative abnormalities
may exist in the ERCr and might possibly be revealed in a larger sample of
schizophrenic brains. We have failed to confirm previous reports of laminar
disorganization in the ERCr in brains of patients with schizophrenia; to
the extent that this region is implicated in schizophrenia, the structural
changes are likely to consist of more subtle cellular disturbances.
相似文献
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153.
Endomyocardial fibrosis: electron-beam CT features 总被引:1,自引:0,他引:1
Mousseaux E; Hernigou A; Azencot M; Sapoval M; Auguste M; Papo T; Chauvaud S; Gaux JC 《Radiology》1996,198(3):755
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María J. Calderon Pilar Manrique Rosario Gonzalez JC Garcia-Monco I. Zabalza 《Journal of the European Academy of Dermatology and Venereology》1995,5(1):51-53
Calcifying epithelioma of Malherbe, pilomatricoma, is a benign neoplasm of the skin, originating from primitive cells of the hair matrix, Myotonic dystrophy (Steinert's disease) is an uncommon hereditary disease characterized by myotonia, muscle wasting, cataracts, testicular atrophy and frontal baldness. We present the case of a woman with pilomatricomas and myotonic dystrophy and then we review the relationship between these two disorders. 相似文献
158.
Women worried about their familial breast cancer risk--a study on genetic advice in general practice
de Bock GH; Perk DC; Oosterwijk JC; Hageman GC; Kievit J; Springer MP 《Family practice》1997,14(1):40-43
AIMS: To ascertain whether women who consulted their GP because they
perceived themselves as at increased risk of familial breast cancer were
indeed at increased risk, and to evaluate potential strategies for
assessing genetic risk of breast cancer in general practice. METHODS:
Sixty-seven out of 81 women who had consulted their GP for advice about
their possible increased risk of developing breast cancer due to breast
cancer in the family were interviewed. Familial breast cancer risk was
assessed by a clinical geneticist. This assessment was compared with two
recent guidelines for referral for genetic counselling. RESULTS: More than
half (52%; n = 35) the women had a relative risk of two and over for
developing breast cancer, while another half of these 35 (25%; n = 17) had
a relative risk of three and over. All the women (n = 17) with a relative
risk of three and over were identified by means of the two current
guidelines for referral for genetic counselling, while more than half of
the women (61%; n = 11) with a relative risk between two and three were
identified. CONCLUSIONS: More than half the women concerned about their
familial risk of breast cancer are indeed at increased risk of breast
cancer. Current guidelines correctly identify women at high risk. However,
doubts about the health gain and feasibility of referral warrant caution,
and need further investigation.
相似文献
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