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排序方式: 共有2063条查询结果,搜索用时 0 毫秒
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Isabel Tol Elspeth Cumber Daphne Nakakande Silvana Wijaya Catherine Turberfield Abdul Badran Safia Siddiqui Prakhar Srivastava Bethany Chung Molly Dineen Cariosa Devlin Claire Worrall Rebecca Green Emily Bennett Elizabeth Golding Ashling Lillis Ami Sabharwal Andrew S. Protheroe Robert A. Watson 《European journal of cancer care》2020,29(3)
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Debra Lynch Kelly David Buchbinder Rafael F. Duarte Jeffrey J. Auletta Neel Bhatt Michael Byrne Zachariah DeFilipp Melissa Gabriel Anuj Mahindra Maxim Norkin Helene Schoemans Ami J. Shah Ibrahim Ahmed Yoshiko Atsuta Grzegorz W. Basak Sara Beattie Sita Bhella Christopher Bredeson Bronwen E. Shaw 《Biology of blood and marrow transplantation》2018,24(2):228-241
Hematopoietic cell transplantation (HCT) is a potentially curative treatment for children and adults with malignant and nonmalignant diseases. Despite increasing survival rates, long-term morbidity after HCT is substantial. Neurocognitive dysfunction is a serious cause of morbidity, yet little is known about neurocognitive dysfunction after HCT. To address this gap, collaborative efforts of the Center for International Blood and Marrow Transplant Research and the European Society for Blood and Marrow Transplantation undertook an expert review of neurocognitive dysfunction after HCT. In this review we define what constitutes neurocognitive dysfunction, characterize its risk factors and sequelae, describe tools and methods to assess neurocognitive function in HCT recipients, and discuss possible interventions for HCT patients with this condition. This review aims to help clinicians understand the scope of this health-related problem, highlight its impact on well-being of survivors, and help determine factors that may improve identification of patients at risk for declines in cognitive functioning after HCT. In particular, we review strategies for preventing and treating neurocognitive dysfunction in HCT patients. Finally, we highlight the need for well-designed studies to develop and test interventions aimed at preventing and improving neurocognitive dysfunction and its sequelae after HCT. 相似文献
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Ryungsa Kim MD PhD Ami Kawai Pharm D Megumi Wakisaka RN Yuri Funaoka LPN Yui Nishida RN Naomi Yasuda RT Koji Arihiro MD PhD 《The breast journal》2018,24(4):628-632
Advances in surgical and anesthetic techniques have allowed for outpatient treatment of breast cancer. We evaluated the feasibility, safety, efficacy, and surgical outcomes of outpatient surgery in 370 patients with breast cancer who underwent breast‐conserving surgery (BCS)/axillar lymph node (ALN) management. There were no deaths or severe intraoperative complications, but 41 complications were observed and disease recurrence occurred in 18 patients. The cumulative overall survival rate was 95.2%. Outpatient surgery was well tolerated, feasible, and safe in patients receiving BCS/ALN management. 相似文献
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TLR4 stimulation and corticosteroid interactively induce osteonecrosis of the femoral head in rat 下载免费PDF全文
Shunichiro Okazaki Satoshi Nagoya Hiroshi Matsumoto Keisuke Mizuo Junya Shimizu Satoshi Watanabe Hiromasa Inoue Toshihiko Yamashita 《Journal of orthopaedic research》2016,34(2):342-345
We previously reported that a toll‐like receptor 4 signaling contributes to the development of osteonecrosis of the femoral head. Also, oxidative stress is suggested to be one of the possible pathogenesis of osteonecrosis of the femoral head. A recent study showed that toll‐like receptor 4 signaling leads to oxidative stress. The aim of the present study was to evaluate whether toll‐like receptor 4 stimulation and subsequent corticosteroid treatment lead to the development of osteonecrosis of the femoral head in rat, and oxidative stress is associated with it. Male Wistar rats were randomly divided into four treatment groups: Saline + Saline, Saline + Methylprednisolone, Lipopolysaccharide + Saline, Lipopolysaccharide + Methylprednisolone. Osteonecrosis of the femoral head at 14 days after the treatment was observed in 1 of 10 Lipopolysaccharide + Saline, and 5 of 10 Lipopolysaccharide + Methylprednisolone treated rats. However, it was not observed at all in the Saline + Saline and Saline + Methylprednisolone treated groups. Glutathione peroxidase activity in the liver at 1 day after the treatment was significantly increased when treated with lipopolysaccharide. However, methylprednisolone treatment reduced the activity. On the other hand, glutathione peroxidase activity in the femur did not change in any intergroup. In conclusion, the present study showed that toll‐like receptor 4 stimulation by lipopolysaccharide administration strengthen incidence of corticosteroid‐induced osteonecrosis of the femoral head, however, concomitant oxidative stress via toll‐like receptor 4 signaling may not contribute to the development of osteonecrosis of the femoral head in rats. © 2015 Orthopaedic Research Society. Published by Wiley Periodicals, Inc. J Orthop Res 34:342–345, 2016. 相似文献