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乳房原发性鳞状细胞癌极为罕见,文献中有确切证据者迄今仅21例。本文报道两例。病例1,44岁,经产妇。1981年4月入院。主诉左乳有一黄豆大小肿块两年。近二月来明显增大,偶有乳头溢液。体格检查,左乳中内象限触及5.5×4.5cm 肿块,质坚有轻 相似文献
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患者男,59岁。1985年曾患肺结核,经二年抗结核治疗痊愈。以后有间歇干咳,痰中不带血。1989年8月咳出肉样组织一块(未送检)。1990年12月晨起又咳出花生米大小肉样组织一块,并有数口鲜血,无其他不适。一周内做纤维支气管镜检查无异常发现。将咳出组织送检,大小:2cm×1cm×1cm, 相似文献
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Krukenters瘤是指卵巢内见含有粘液的印戒细胞、管状腺样结构和间质增生构成的转移癌。其原发癌多来自胃肠道,也可以来自胆囊、胰腺及乳腺等处。据统计70%来自胃癌,15%来自结肠癌,5%来自乳腺癌。近4年来我们遇到7例Krukenterg瘤,有5例原发癌在胃,1例在结肠,1例在直 相似文献
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本文报导浆细胞样淋巴细胞性淋巴瘤11例,按Stain等分类可以分为浆细胞性、浆细胞样及多形性三种亚型。部分病例可找到胞浆内及核内球形小体、PAS染色阳性。 相似文献
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在长期应用左心辅助心泵或全人工心脏时,气压驱动管穿经皮肤而长久存在,气压驱动管的周围组织易于感染。为了预防入口处沿气压驱动管的感染,本实验应用了管周植被绒毛和在管周设置皮下圆盘。结果证明此装置在预防沿气压驱动管的感染中是简单而有效的。 相似文献
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Xu Jing-bai徐惊伯 Zhang Bai-gen张柏根 Gu Xiao-ping顾小平and Zheng Zhong-hua郑忠华Departments of Radiology Surgery Pathot og''y Ren Ji HospitalShanghai Second Medical. University Shanghai 《中华医学杂志(英文版)》1988,101(7):478-480
Twenty patients with jugular phlebectasia hadthe chief symptom of remittent bulging of the neck
(unilateral 19, and bilateral l). Phlebography was
performed in 18 patients and showed the lesion was
mainly of two types: cystic and cylindrical. Those
of external and anterior jugular veins were excised
and those of the internal jugular veins were treated
by plication. Pathologic studies on 11 excised veins
revealed thin venous wall and that smooth muscle
fibers were replaced by fibrous tissues. Etiologically,
congenital hypoplasia of the venous wall led to the
impedance of venous flow secondary to the particular
anatomical position at the entry of the thoracic cage.
Jugular phlebography demonstrated clearly the size,
shape and position of these changes. We found that
plication is effective in the treatment of internal ju-
gular phlcbectasia and is worth recommending. 相似文献
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In the present study we observed dynamically and systemically the changes of plasma somatostatin and glucagon in the peripheral and portal vein, and the changes of pancreatic immunopathology in the course of development of cirrhosis induced by CCl_4 and after portacaval shunt (PCS) in the cirrhotic rats as well as investigated their causes and correlationship. The results showed that hyperglucagonemia was caused by spontaneous portosystemic shunting and surgically induced portacaval anastomosis. Moreover, there was much higher level of glucagon in the portal vein with corresponding increase of A cells in PCS rats than those in the controls, indicating that another cause for elevation of glucagon was hypersecretion of pancreatic A cells. Our data demonstrated that both deterioration of liver function and portosystemic shunting might not be responsible for the elevated level of somatostatin in the cirrhotic rats with PCS. However, there was a closed positive correlation between plasma glucagon and somatostatin. Thus it was concluded that hyperglucagonemia stimulated the release of somatostatin. In view of the fact the elevated level of glucagon was much higher than that of somatostatin, there was probably a relative lack of somatostatin in cirrhosis with portal hypertension. 相似文献
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