晶状体超声乳化人工晶状体植入治疗葡萄膜炎并发白内障

目的 探讨葡萄膜炎并发白内障晶状体超声乳化吸出及人工晶状体植入术的疗效.方法 对46例（52眼）葡萄膜炎并发白内障行晶状体超声乳化吸出及人工晶状体植入术,其中2眼联合小梁切除术.结果 术后48眼（92.31%）视力较术前提高.术后6月视力≥0.5者43眼（82.69%）,0.3～0.4者3眼（5.77%）,0.1～0.2者3眼（5.77%）,＜0.1者3眼（5.77%）.结论 晶状体超声乳化吸出术治疗葡萄膜炎并发白内障,术前充分准备,术中、术后处理得当,则术后炎症反应轻,视力恢复好.     

超声乳化人工晶状体植入治疗葡萄膜炎并发白内障临床观察

目的探讨葡萄膜炎并发白内障晶状体超声乳化吸出及人工晶状体植入术的疗效。方法对38例38眼葡萄膜炎并发白内障患者行晶状体超声乳化吸出及人工晶状体植入术。结果 38眼术后裸眼视力均有不同程度的提高。术后3月≥0.5者31眼（80.77%）,0.3～0.4者3眼（7.69%）,0.1～0.2者2眼（5.77%）,〈0.1者2眼（5.77%）。结论晶状体超声乳化吸出术治疗葡萄膜炎并发白内障,术前充分准备,术中、术后处理得当,则术后炎症反应轻,视力恢复好。     

葡萄膜炎并发白内障患者行超声乳化吸除联合人工晶状体植入术的预后分析

目的研究葡萄膜炎并发白内障患者行超声乳化吸除联合人工晶状体植入术后的疗效及影响其预后的因素。方法回顾性分析我科2004年1月至2009年12月期间诊断为葡萄膜炎并发白内障患者28例(32只眼),行超声乳化吸除联合后房型人工晶状体植入术,评估术后患眼的视力以及眼部并发症。术后随访3～24个月,平均10.2个月。结果大多数患眼(87.5%)视力有不同程度的提高,其中24只眼(75%)矫正视力≥0.5。术后发生后囊膜混浊7只眼、葡萄膜炎复发3只眼及黄斑水肿2只眼。患者术后视力欠佳与术前黄斑病变存在相关性。结论葡萄膜炎并发白内障患者行超声乳化吸除联合人工晶状体植入术后可获得较好的视力恢复,黄斑病变是导致患者预后不良的原因。     

超声乳化白内障吸除治疗葡萄膜炎并发白内障

目的观察超声乳化白内障吸除人工晶状体植入术治疗葡萄膜炎并发白内障的临床效果。方法对25例（29只眼）葡萄膜炎并发白内障行超声乳化白内障患者吸除人工晶状体植入术,同时用非切开方式对粘连性小瞳孔进行扩张。结果白内障术后患者视力均得到提高,术后6个月最佳矫正视力≥0．5者24只眼占82.8％,术后恢复生理性小圆瞳孔25只眼占86．2％。结论对葡萄膜炎并发白内障施行超声乳化白内障吸除人工晶状体植入术可提高视力,恢复生理性圆瞳孔。     

表面麻醉下白内障超声乳化人工晶状体植入治疗葡萄膜炎并发白内障

目的 :探讨葡萄膜炎并发白内障超声乳化摘除及人工晶状体植入术的疗效。方法 :对 3 8例 42只眼葡萄膜炎并发白内障患者行超声乳化摘除及人工晶状体植入术 ,其中 4只眼联合小梁切除术 ,3只眼联合周边虹膜切除术。结果 :术后 3 9只眼 (92 86% )视力较术前提高。术后 6个月裸眼视力≥ 0 5者 3 3只眼 (78 57% ) ,0 3～ 0 5者 3只眼 (7 14 % ) ,0 1～ 0 3者4只眼 (9 52 % ) ,<0 1者 2只眼 (4 76% )。结论 :超声乳化白内障摘除术治疗葡萄膜炎并发白内障 ,术后炎症反应轻 ,视力恢复好。     

白内障超声乳化治疗葡萄膜炎并发白内障临床分析

目的：探讨白内障超声乳化治疗葡萄膜炎并发白内障的临床效果。方法：对20例20眼葡萄膜炎并发白内障患者行白内障超声乳化摘除及人工晶状体植入术,其中3眼联合周边虹膜切除术。结果：术后2wk18眼（90%）视力较前提高。术后2wk矫正视力〉0.5者10眼（50%）,0.3～0.5者5眼（25%）,0.1～0.2者2眼（10%）,〈0.1者1眼（5%）,视力无变化者2眼（10%）。结论：超声乳化治疗葡萄膜炎并发白内障,虽然手术较复杂,但整体效果良好。     

高度近视白内障晶状体超声乳化人工晶状体植入术的临床观察

目的 观察晶状体超声乳化吸出联合折叠式人工晶状体植入术治疗白内障合并高度近视的临床效果.方法 对27例(47眼)白内障合并高度近视行晶状体超声乳化吸出,低度数丙烯酸酯折叠式人工晶状体植入术,观察术中、术后并发症,术后视力及屈光状态,术后随访>6月.结果 术中有后囊破裂1眼(2.13%),角膜水肿7眼(14.89%),葡萄膜炎症1眼(2.13%).术后1月-3月矫正视力<0.1者1眼(2.13%),0.1-0.4者12眼(25.53%),0.5-0.8者30眼(63.83%),1.0-1.5者4眼(8.51%).晶状体后囊浑浊6眼(12.77%).观察2年无视网膜脱离者.结论 晶状体超声.乳化吸出,低度数丙烯酸酯折叠式人工晶状体植入治疗白内障合并高度近视安全并且效果良好.     

顽固性葡萄膜炎并发性白内障的手术治疗

目的 探讨顽固性葡萄膜炎并发性白内障的手术方法和效果.方法 采用晶状体超声乳化后房型人工晶状体植入术治疗顽固性葡萄膜炎并发性白内障12例(16眼).术前单独使用皮质类固醇或需要联合使用免疫抑制剂对葡萄膜炎患者治疗.术后随访6～12月,平均8.5月.结果 手术后视力均有提高,术后最佳矫正视力<0.1者2眼(12.50%),0.1～0.4者3眼(18.75%),0.5～0.9者7眼(43.75%),≥1.0者4眼(25.00%).无严重并发症发生.结论 顽固性葡萄膜炎并发性白内障经过规范治疗进行白内障人工晶状体手术是安全有效的.     

糖尿病患者白内障超声乳化摘出联合人工晶状体植入术临床分析

目的 探讨糖尿病患者进行白内障超声乳化摘出联合人工晶状体植入术的临床效果.方法 对158例(193眼)糖尿病患者施行白内障超声乳化摘出联合人工晶状体植入术,术后观察视力及并发症情况.结果 术后视力0.01～0.1者26眼(13.47%),0.12～0.3者37眼(19.17%),0.4～0.5者50眼(25.90%),0.6-1.0者以上80 眼(41.46%);术中后囊膜破裂7眼,术后角膜水肿95眼,经治疗3～6 d消失.结论 糖尿病白内障患者行超声乳化摘出联合人工晶状体植入术是安全有效的.     

非超声乳化小切口白内障摘出联合人工晶状体植入术治疗葡萄膜炎并发性白内障

目的探讨非超声乳化小切口白内障摘出联合人工晶状体植入术治疗葡萄膜炎并发性白内障的疗效。方法对36例40眼葡萄膜炎并发性白内障施行非超声乳化小切口白内障摘出联合人工晶状体植入术,观察手术前后视力变化及并发症。结果40眼全部植入人工晶状体;术后随访1个月,视力≥0.6者5眼,0.5～0.6者23眼,0.3～0.5者10眼,≤0.3者2眼。20眼出现不同程度炎性反应,经5d-3周治疗均消失。结论非超声乳化小切口白内障摘出联合人工晶状体植入术治疗葡萄膜炎并发性白内障疗效好。     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.