共查询到20条相似文献，搜索用时 93 毫秒
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患者男性,50岁。因右腰腹部间断性疼痛4年余,加重3天入院。MRI示右侧肾脏形态基本正常,右侧肾盂处见团块样异常信号,大小3.7 cm×3.4 cm,右侧肾盏扩张,右侧输尿管上段扩张,内见低信号的充盈缺损。 相似文献
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我们收治1例下腔静脉—右髂外静脉后输尿管,兹报导如下。女性,38岁,右腰酸隐痛半年,无发热,无尿路刺激症状和血尿。B超发现右肾积水。入院后排泄性尿路造影示右肾功能轻度减退,肾盂肾盏扩张积水,上段输尿管呈倒置“丁”型扭曲扩张,扩张之尾部转向内侧并逐渐变细,造影剂中断于第3腰椎下缘水平(附图),右侧输尿管逆行插管18cm受阻.摄片见输尿管导管 相似文献
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例1,患者女性,52岁,因无明显诱因出现右腰部不适、活动时加重半年入院.查体:一般情况好,BP:120/80 mg,右侧肾区扣击痛(±),未扪及明显肿块.B超:右肾切面形态稍大,下段背侧稍隆起,其内可见多个形态不规则的囊性暗区,内见多条强光带分隔.CT示:⑴右肾盂旁囊肿;⑵右肾下极皮质囊肿.既往有高血压病史,术中见右肾下极及肾盂输尿管旁各有1囊肿,剔除囊肿送病检.术后给予抗炎对症治疗,未行特殊处理,患者痊愈出院.随访7年未见复发或转移. 相似文献
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例1,男,20岁,因右侧腰部酸痛一年入院,入院前一月B超检查右肾积水,左肾显示不清,腹平片显示右肾影15×10cm,未见到左肾影。静脉肾盂造影显示右肾中度积水。左肾未显影。膀胱镜检及逆行造影:输尿管间嵴左侧平坦,左输尿管开口缺如。同位素肾图:右肾正常曲线,左肾无功能。诊断右侧先天性弧立肾。例2 男,34岁,因右腰部酸胀痛半年,B超检查右肾轻度积水,左肾不显影。入院后摄泌尿系平片,左侧无肾影。右侧肾影大于正常。静脉肾盂造影:右肾盂肾盏轻度扩张,左侧肾区未显影。膀胱镜检查:左侧无输尿管开口,右侧输尿管开口正常。CT片显示右肾影 相似文献
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Properties of chemoreceptors of tongue of rat 总被引：14，自引：0，他引：14
BEIDLER LM 《Journal of neurophysiology》1953,16(6):595-607
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A further analysis of already published data supports the position that retardates of low ability level less frequently have retarded siblings, retarded parents, and parents low in occupational level than do retardates higher in ability level. The analysis supports the position that there are two types of retarded individuals, persons retarded as a result of gene or chromosomal anomalies, brain injury, etc., who more frequently occur in the lower-level retardate group, and persons whose retardation represents polygenic segregation, who more frequently occur in the higher-level group. 相似文献
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Eighteen families in which both parents had refractions within the range of +4·0 D to −4·0 D and axial lengths seen in emmetropia (22·3-26·0 mm) showed coefficients of correlation of the order 0·5 indicative of polygenic inheritance. Such coefficients were seen for axial length (0·407) and for the cornea (0·487), but not for the lens (which is known to be yoked to the axial length). No such coefficients were seen in 19 families in which one of the parents had axial length outside the emmetropic range (nine families with long axes and 10 with short axes).
The pattern of polygenic inheritance for emmetropia (completely correlated optical components) and errors of refraction up to 4·0 D (inadequately correlated components: correlation ametropia) follows that seen in stature and other measurable characters. In contrast the high refractive errors with their abnormal axial lengths (component ametropia) are—like the extremes in stature—pathological anomalies with monofactorial inheritance.
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