脊髓发育不良和隐性骶椎裂致神经源性膀胱功能障碍的尿动力学表现

为了解脊髓发育不良和隐性骶椎裂对泌尿系统的影响，对３４例有泌尿系统症状的患者行尿动力学检查和辅助检查。结果显示有反射性膀胱２１例（６１．７％），其中逼尿肌外括约肌协同失调１３例，排尿困难为主要表现；逼尿肌外括约肌协同正常８例，急迫性尿失禁为主要表现。无反射性膀胱１３例（３８．５％），排尿困难、尿潴留为主要表现。１２例膀胱颈口开放患者９例有尿失禁，最大尿道闭合压为３．１７±１．４０ｋＰａ（１ｋＰａ＝１０．２０ｃｍＨ２Ｏ），２２例膀胱颈口闭合患者最大尿道闭合压为７．７７±３．５０ｋＰａ（Ｐ＜０．０１）。１５例ＩＶＵ显示有上尿路损害的患者排尿期膀胱压力为平均８．０１±４．３０ｋＰａ，１９例无上尿路损害的患者排尿期膀胱压力平均为３．０６±１．２０ｋＰａ，（Ｐ＜０．０１）。综合分析显示患者临床症状和尿动力学表现与脊髓损伤平面无对应关系。     

尿流动力学检查对脊髓拴系诊治的临床意义

目的讨论尿流动力学检查在脊髓拴系患者治疗中的重要性.方法对38例脊髓拴系患者行尿流动力学检查、静脉肾盂造影和排泄性膀胱尿道造影,了解患者泌尿系损害情况.结果上尿路损害组最大逼尿肌压(7.45±1.22kPa)明显高于上尿路组无损害组(4.25±1.40kPa),膀胱顺应性(2.6±0.6ml/cmH2O)低于下尿路无损害组(6.7±0.8ml/cmH2O).逼尿肌与括约肌协调组上尿路损害的发生率(16.7%)明显低于逼尿肌与括约肌不协调组(52.1%).结论逼尿肌压升高、膀胱顺应性下降、逼尿肌与括约肌不协调是上尿路损害的高危因素,尿流动力检查可为临床治疗提供指导.     

脊髓拴系综合征并发上尿路扩张患者的尿动力学特征

目的：探讨脊髓拴系综合征(TCS)并发上尿路扩张患者尿动力学特征。方法：对23例TCS并发上尿路扩张患者(扩张组)和26例TCS未并发上尿路扩张患者(对照组)进行尿动力学测定。结果：扩张组患者逼尿肌无收缩伴膀胱低顺应性。其剩余尿量、排尿前逼尿肌压力、最大膀胱压测定容量、逼尿肌漏尿点压、逼尿肌无收缩和逼尿肌漏尿点压大于3．92kPa发生率均显著高于对照组;而膀胱顺应性和神经源性逼尿肌功能过度活跃发生率显著低于对照组。扩张组相对安全容量与对照组之间的差异无统计学意义,而相对危险容量显著大于对照组。结论：TCS并发上尿路扩张患者膀胱多为无反射性膀胱。对本病患者应尽早并定期进行尿动力学检查,及时消除相对危险容量,防止上尿路损害的发生。     

前列腺增生症急性尿潴留患者尿流动力学分析

目的 研究前列腺增生症(BPH)急性尿潴留(AUR)患者尿流动力学特点.方法 143例BPH AUR 患者,留置尿管1周后行尿流动力学检查.患者根据检查结果 分为无逼尿肌过度活动(none detrusor overactive,NDO)组(75例)和逼尿肌过度活动组(detrusor overactive,DO)组(68例);比较两组年龄、国际前列腺症状评分(IPSS)、前列腺体积及尿流动力学参数.结果 比较两组患者的年龄、IPSS评分中储尿期症状评分、排尿期症状评分和前列腺体积,差异均无统计学意义(P>0.05).NDO组中无逼尿肌收缩患者22例(22/143,15.4%),NDO组患者(不包括无逼尿肌收缩患者22例)最大逼尿肌压力及线性被动尿道阻力关系梗阻程度分级均小于DO组,差异具统计学意义(P<0.05);所有NDO组患者初始感觉时膀胱容量、顺应值和测压容积均大于DO组,差异也均具统计学意义(P<0.05).结论 BPH患者出现AUR,应行尿流动力学检查以明确膀胱功能状态,这有利于治疗方案的选择.对于存在DO的患者早期手术解除膀胱出口梗阻,有利于膀胱功能的恢复和降低上尿路损害风险.     

不同α受体阻滞剂联合M受体阻滞剂治疗BPH下尿路症状的尿流动力学研究

目的:比较多沙唑嗪联合托特罗定与坦索罗辛联合托特罗定治疗良性前列腺增生(BPH)下尿路症状(LUTS)的尿流动力学变化的研究。方法:选取诊断明确的BPH患者50例,采用随机非双盲方法平均分为A、B两组,A组患者服用多沙唑嗪联合酒石酸托特罗定缓释剂、B组患者服用坦索罗辛联合酒石酸托特罗定缓释剂联合治疗12周,于治疗前后对潴尿期与排尿期的相关尿流动力学指标,如最大尿流率(Qmax)、膀胱剩余尿量、最大膀胱容量、排尿开始时膀胱压、膀胱顺应性等进行比较。结果:两组在治疗前的各项指标无统计学意义。服药治疗12周后,两组Qmax、排尿开始时膀胱压、膀胱顺应性均较治疗前有明显改善。而剩余尿量和最大膀胱容量较治疗前无明显变化。治疗后两组间比较,A组Qmax、排尿开始时膀胱压、膀胱顺应性均优于B组(P0.05),而两治疗组之间的剩余尿量及最大膀胱容量差异无统计学意义(P0.05)。结论:α受体阻滞剂联合M受体阻滞剂治疗BPH能明显提高Qmax,降低排尿开始时膀胱压力,改善膀胱顺应性,且多沙唑嗪联合应用M受体阻滞剂优于坦索罗辛联合应用M受体阻滞剂。     

脊髓损伤的尿流动力学检查

尿流动力学(Urodynamic)是近年来逐渐形成并获得迅速发展的泌尿外科检查新技术,现已成为各类排尿障碍患者重要的检查手段,对脊髓损伤患者。更是一项必不可少的检测手段,通过尿流动力学各项检查,可动态地了解下尿路功能,并在此基础上,对诊断和治疗提供更多的客观指标和依据。 尿流动力学是运用流体力学,电生理及神经生理学等学科的原理和方法,研究泌尿系统有关尿液输送,贮存和排出功能的新学科。通过检测膀胱容积压力变化,尿道压力分布测定,肌电图,尿流率等项检查,可较全面完整地反映出下尿路功能动态改变。1膀胱压力容积测定(Cystometry) …     

脊髓栓系综合征对上尿路的影响及其机制

目的 探讨脊髓栓系综合征(TCS)对上尿路的影响及其机制. 方法经脊髓MRI确诊的TCS患者40例.男21例,女19例.平均年龄23岁.病程1～40年.行尿液分析及中段尿培养、血肌酐(SCr)测定、泌尿系B超、IVU、膀胱造影及尿动力学检查,评价TCS对上尿路的影响.将有无SCr升高、肾积水、膀胱输尿管反流(VUR)界定为是否有上尿路损害. 结果 40例患者中有尿路感染17例(42.5%),SCr异常升高[(251.6±98.5)μmol/16例.B超检查(29例)示双肾积水、双侧输尿管上段扩张12例(41.4%).IVU检查(30例)示肾、输尿管扩张、积水10例(33.3%),膀胱形态异常22例(73.3%),有较多憩室及小梁形成.逆行造影(22例)示VUR 17例(77.3%)27侧.31例(31/35)有残余尿(261.8±232.4)ml.33例膀胱压力容积测定示储尿期最大逼尿肌压(41.2±20.9)cm Hz0(1 cm H2O=0.098 kPa),顺应性(22.3±18.8)ml/cm H20.33例行压力一流率及肌电图测定示排尿期出现逼尿肌-外括约肌协同失调者16例(48.5%),逼尿肌无反射16例(48.5%),逼尿肌反射减弱13例(39.4%).16例成人行静态尿道压力描记,最大尿道闭合压(76.1±33.1)cm H2O.20例有上尿路损害者与20例无上尿路损害者中逼尿肌顺应性分别为(9.4±7.8)和(19.3±15.8)ml/cm H2O、储尿期最大逼尿肌压分别为(43.1±21.2)和(24.0±11.9)cm H2O、残余尿量分别为(189.0±138.0)和(47.8±36.8)ml、最大尿道闭合压分别为(86.2±32.4)和(46.8±20.8)cm H2O、发生逼尿肌收缩力受损分别为20/20和9/13、发生逼尿肌一外括约肌协同失调分别为13/20和3/13,2组比较差异均有统计学意义(P<0.05).17例VUR者中16例发生尿路感染,5例无VUR者仅1例发生尿路感染,二者比较差异有统计学意义(P=0.003).17例尿路感染者中15例发生上尿路损害,23例无尿路感染者5例发生上尿路损害,二者比较差异有统计学意义(P=0.000). 结论 TCS患者逼尿肌顺应性降低、储尿期逼尿肌压升高、残余尿量增多、最大尿道闭合压升高、逼尿肌收缩力受损、逼尿肌一外括约肌协同失调是上尿路损害的危险因素.     

逼尿肌活动低下在下尿路症状中的临床特征及诊疗策略分析

目的:探讨逼尿肌活动低下(DU)在下尿路症状(LUTS)患者中的流行病学现状、临床特征及诊疗策略。方法:回顾性分析了我院1 019例排除神经源性膀胱及解剖结构异常的LUTS患者的尿流动力学检查结果及随访资料,探讨DU在LUTS中的流行病学及临床诊治特点,并对106例DU患者治疗前后行尿流动力学检查,结合文献进行临床分析。结果:在就诊患者中,储尿期症状最为多见,在男性患者中,排尿期症状稍多于储尿期症状,而女性患者储尿期症状明显多于排尿期症状。男性患者中膀胱出口梗阻(BOO)的患者为57.9%,而女性患者中压力性尿失禁(SUI)患者达到了43.3%。27.4%男性及23.2%女性诊断为DU,男性及女性患病率差异无统计学意义。DU患者与非DU患者的临床表现无明显差异,均以LUTS为主要表现。DU可能合并有逼尿肌过度活动或BOO。DU随着年龄的增加,患病率逐渐增加。106例DU患者经治疗后行尿动力学检查发现逼尿肌收缩力有一定提高,治疗前后逼尿肌肌力差异有统计学意义。结论:DU是LUTS患者的常见病因,并有可能同时合并有逼尿肌过度活动或BOO。目前针对DU患者的治疗有一定效果。     

尿流动力学检查的护理

尿流动力学检查的护理中国康复研究中心戴凤君尿流动力学是借助流体力学及电生理、神经生理方法研究尿液在尿路中输送、贮存、排泄等功能的新学科。通过检测膀胱贮尿期容积压力变化，排尿期尿流率，尿道压力分布测定，膀胱和尿道有关肌肉电生理活动与神经生理情况，借以反...     

小儿神经源性膀胱与上尿路损害

目的：探讨脊髓发育不良致神经源性膀胱造成上尿路损害的尿动力学危险因素,方法：对32例脊髓发育不良患儿行尿动力学和影像学检查。结果：18例（56％）有肾和输尿管积水,其中4例伴IV－V级膀胱输尿管返液,19例逼尿肌漏尿点压力大于等于40cmH2O(1cmH2O=0.098kPa)且膀胱顺应性小于等于40ml/cmH2O的患儿中,17例合并上尿路损害,13例逼尿肌漏尿点压力＜40cmH2O且膀胱顺应性＞40ml/c mH2O的患儿中,仅1例有双侧输尿管扩张,差别有非常显著性意义（P＜0．001）,结论：对脊髓发育不良致神经源性膀胱的患儿应行尿动力学检查,了解逼尿肌漏尿点压力和膀胱顺应性,确定与上尿路损害有关的危险因素,有助于避免上尿路进一步损害。     

Value of routine evaluation of the voiding phase when performing urodynamic testing in women with lower urinary tract symptoms

PURPOSE: Lower urinary tract symptoms in women are often evaluated by cystometrography. We only assessed the bladder response to filling and not the impact of abnormal voiding, which is known to cause lower urinary tract symptoms. We determined the prevalence of voiding abnormalities in women with lower urinary tract symptoms and compared cystometrography to cystometrography plus voiding pressure flow study for evaluating this condition. We also determined whether storage or voiding symptoms predicted abnormal voiding. MATERIALS AND METHODS: We reviewed the records of 134 women who underwent video urodynamics with cystometrography and voiding pressure flow study to evaluate lower urinary tract symptoms. Patients with a history of neurological disease, grade 4 pelvic prolapse or a primary complaint of stress incontinence were excluded from study. All participants completed an American Urological Association symptom index and scores were subclassified as total, storage and voiding. A diagnosis was made in each case based on cystometrography findings, while any additional diagnoses when applicable were based on the voiding pressure flow study. Symptom scores were compared in women in whom the voiding study did and did not add information. RESULTS: Mean patient age was 53.1 years (range 19 to 90). Voiding studies added information in 44 cases (33%), including dysfunctional voiding in 16, obstruction due to a moderate cystocele in 6, primary bladder neck obstruction in 6, external-detrusor sphincter dyssynergia as the initial presentation of neurological disease in 5, obstruction after incontinence surgery in 3, urethral stricture in 3, post-void contraction mimicking symptoms in 2, impaired contractility in 2 and an obstructing urethral diverticulum in 1. A total of 32 patients (24%) did not void during the study. Those with voiding abnormalities had higher total and voiding but similar storage symptom scores (23.1 versus 18.5, 12.3 versus 8.0 and 10.8 versus 10.5 points, p = 0.0008, 0.0001 and 0.58, respectively). CONCLUSIONS: Women with lower urinary tract symptoms may have voiding abnormalities that are missed by cystometrography only. Voiding studies are useful for properly diagnosing and treating such cases. Women with abnormal voiding seem to have more severe voiding symptoms than those without such abnormalities. Occult neurological disease may also be identified in patients with lower urinary tract symptoms and voiding abnormalities.     

Sphincteric urinary incontinence: relationship of vesical leak point pressure,urethral mobility and severity of incontinence

PURPOSE: We examined the relationships among urethral hypermobility, intrinsic sphincter deficiency and incontinence in women. MATERIALS AND METHODS: A total of 65 consecutive women with stress urinary incontinence and 28 with lower urinary tract symptoms not associated with stress urinary incontinence were evaluated with videourodynamics, 24-hour voiding diaries and pad tests, vesical leak point pressure measurement and the cotton swab test. RESULTS: A total of 93 patients with a mean age +/- SD of 63 +/- 13 years were studied, including 65 who presented with stress urinary incontinence and 28 who presented with lower urinary tract symptoms without stress urinary incontinence. The incidence of urethral hypermobility was 32% in the stress urinary incontinence group and 36% in the lower urinary tract symptoms group (p = 0.46). When stress urinary incontinence cases were stratified according to a vesical leak point pressure of 0 to 60, 60 to 90 and greater than 90 cm. H2O, urethral hypermobility was noted in 25%, 31% and 41%, respectively, a difference that was not statistically significant (p = 0.6). Overall incontinent patients with and without urethral hypermobility had the same median number of incontinence episodes (5, range 1 to 13 versus 7, range 1 to 15, p = 0.39) and median pad weight (39.5 range 1 to 693 gm. versus 33.5, range 1 to 751, p = 0.19). When patients with intrinsic sphincter deficiency, defined as vesical leak point pressure less than 60 cm. H2O, were divided into those with and without urethral hypermobility, there were no differences in the mean number of incontinence episodes (9.4 +/- 3 versus 6 +/- 3.6, p = 0.17) or median pad weight (90 gm., range 10 to 348 versus 86, range 30 to 81, p = 0.76). The degree of change in the urethral angle did not correlate with vesical leak point pressure (r = 0.16, p = 0.24) or with pad weight (r = -0.23, p = 0.1). CONCLUSIONS: Urethral hypermobility was equally common in this group of women with lower urinary tract symptoms and stress urinary incontinence. Intrinsic sphincteric deficiency and urethral hypermobility may coexist and they do not define discrete classes of patients with stress urinary incontinence. Urethral hypermobility did not appear to have an independent effect on the frequency or severity of incontinence. Patients with stress urinary incontinence can still be characterized by vesical leak point pressure and change in the urethral angle, although these variables do not always define discrete classes.     

排尿功能障碍致上尿路损害的机制及防治策略

排尿功能障碍导致的上尿路损害在泌尿外科临床常见,严重威胁患者生命。尿动力学研究发现3．92kPa（1cmH2O=0．098kPa）是上尿路损害的膀胱压临界值,达到或超过安全压仍继续储尿是导致上尿路损害的最重要原因。因此,无论采用何种治疗手段,将膀胱压降至安全压以下,在安全容量前排空膀胱是保护上尿路的根本原则。     

非神经源性逼尿肌膀胱颈协同失调

报告５例原发性膀胱颈功能障碍患者表现为排尿时膀胱颈开放不良，病变为逼尿肌膀胱颈协同失调，可命名为非神经源性逼尿肌膀胱颈协同失调。提示其诊断标准：（１）有下尿路功能性梗阻；（２）排尿时膀胱颈开放不良，膀胱内压－膀胱颈内压梯度异常；（３）无影响膀胱尿道功能的神经系疾患。同时对此症的发病机理和治疗选择进行了讨论。     

Learned voiding dysfunction (non-neurogenic, neurogenic bladder) among adults

Data concerning learned voiding dysfunction (Hinman syndrome; non-neurogenic, neurogenic bladder) in adults are scarce. The present study was conducted to assess the pre-valence and clinical characteristics of this dysfunction among adults referred for evaluation of lower urinary tract symptoms. Learned voiding dysfunction was suggested by a characteristic clinical history and intermittent "free" uroflow pattern and by the absence of any detectable neurological abnormality or anatomic urethral obstruction. A definitive diagnosis was made by the demonstration of typical external urethral sphincter contractions during micturition by EMG or fluoroscopy. A urodynamic database of 1,015 consecutive adults was reviewed. Twenty-one (2%) patients (age, 24-76 years) met our strict criteria of learned voiding dysfunction. Obstructive symptoms were the most common presenting symptoms, followed by frequency, nocturia, and urgency. Eight (35%) patients had recurrent urinary tract infections, seven of these being women. None of the patients had any clinically significant upper urinary tract damage. First sensation volume was significantly lower in women than in men. Both detrusor pressure at maximum flow and maximum detrusor pressure during voiding were found to be significantly higher in men than in women. Further differentiation between adult women and men failed to reveal any other clinically significant differences. In conclusion, by strict video-urodynamic criteria, 2% of our patients had learned voiding dysfunction. Other patients, with presumed learned voiding dysfunction, who did not undergo video-urodynamics were not included in the present series. Thus, the prevalence of learned voiding dysfunction among adults referred for evaluation of lower urinary tract symptoms is likely to be even higher.     

Vesicourethral dysfunction of diabetic patients

In order to evaluate vesicourethral dysfunction in diabetic patients, urodynamic studies, IVP and urinalysis were performed on 173 diabetic patients (male 78, female 95) and 17 nondiabetic control cases. In addition to the classical findings as increased volume at the first desire to void and decreased maximum vesical pressure, diabetic patients showed varieties of vesicourethral dysfunctions such as overactive bladder (14.5%), low compliance bladder (11.0%) and loss of detrusor-external sphincter coordination (31.7%). Vesicourethral function of diabetics was classified in following 5 types by analysing the volume at first desire to void, volume at maximum desire to void, maximum vesical pressure, residual urine volume and bladder compliance. 1. Type 1, normal vesical function, 13 cases. 2. Type 2, vesical dysfunction with minimal residual urine, 49 cases. 3. Type 3, vesical dysfunction with residual urine, 66 cases. 4. Type 4, low compliance bladder, 20 cases. 5. Type 5, overactive bladder, 25 cases. Pyuria was observed in 59.8%, hydronephrosis was found in 10.9% and ectasia of lower ureter was found in 17.8% of diabetic patients. The highest incidence of pyuria and abnormality of the upper urinary tract were noted in Type 4 and followed by Type 3 and by Type 2 in decreasing order. Extent of pyuria and ectasis of the upper urinary tract showed statistically significant correlation with residual urine volume and detrusor-external sphincter coordination. When vesicourethral function was compensated by abdominal strain, the volume of residual urine is not elevated, but when the mechanism of compensation is lost or in the absence of detrusor-external sphincter coordination results in gradual accumulation of residual urine. In cases with long standing chronic urinary tract infection may results in fibrosis of the bladder wall with low compliance bladder. Fibrotic obstruction of uretero-vesical junction can cause hydroureteronephrosis and followed by renal function impairment. As vesical damage become irreversible at this end stage, proper management during early stage is crucial for management of diabetic patients. Cholinergic agent were effective to reduce residual urine volume in Type 3. alpha-blocking agent were effective to reduce residual urine volume in Type 3 and some cases of Type 4. In cases in which medication therapy failed to reduce residual urine, the clean intermittent catheterization was successful in control of urinary tract infection and upper urinary tract ectasis. Transurethral resection of the prostate and the bladder neck is indicated in the male patients with a large amount of residual urine in Type 3 and 4.     

Video urodynamics using transrectal ultrasonography for lower urinary tract symptoms in women

AIMS: To understand the properties of lower urinary tract disorders in women, we evaluated 60 female patients with lower urinary tract disorders or symptoms of recurrent cystitis by free uroflowmetry and video urodynamics using transrectal ultrasonography (VU-TRUS). METHODS: Results of urodynamic studies or symptoms of stress incontinence were used to divide 60 women into 7 normal controls and 53 with voiding dysfunctions. RESULTS: In normal controls, VU-TRUS showed that the mean posterior urethrovesical angle and anteroposterior diameter of the proximal urethra at maximum flow was 151.4 degrees and 4.9 mm, respectively. In patients with voiding dysfunction, VU-TRUS during voiding revealed various urethral abnormalities, including 16 detrusor sphincter dyssynergia, 4 detrusor bladder neck dyssynergia, and 13 insufficient opening of the entire urethra. VU-TRUS also showed pelvic floor abnormalities, including 24 urethral hypermobilities (group 1) and 11 cystoceles (group 2). Eighteen patients had neither urethral hypermobility nor cystocele (group 3). Major pressure-flow abnormalities in the 53 patients with voiding dysfunctions were weak detrusor (72%) and/or bladder outlet obstruction (25%). There were no significant differences in the distribution of the pressure-flow abnormalities among the three groups. However, the mean values of abdominal pressure at maximum flow of group 1 (20.9 cm H(2)O) and group 2 (17.9 cm H(2)O) were significantly higher than that of group 3 (6.3 cm H(2)O; each P < 0.05). The mean values of residual urine volume of group 2 (60.8 mL) and group 3 (77.6 mL) were significantly higher than that of group 1 (23.5 mL; each P < 0.05). CONCLUSIONS: The symptoms of women with lower urinary tract disorders were frequently accompanied by urethral and/or pelvic floor abnormalities during voiding that were clearly detected by VU-TRUS. VU-TRUS is useful for objective evaluation of female lower urinary tract symptoms.     

梨状腹综合征的尿动力学表现(附二例报道)

目的 探讨梨状腹综合征(PBS)患儿尿动力学表现及临床意义。方法 报告2例男性PBS患儿(年龄分别为12岁和8岁)尿动力学检查结果及其指导临床治疗的意义。结果 2例患儿均表现为下尿路梗阻，剩余尿增多，膀胱容量增大，尿道高压，其中1例表现不稳定膀胱。1例行膀胱颈口电切，尿路梗阻症状缓解。结论 PBS患儿尿动力学主要表现为下尿路梗阻，剩余尿增多。对于梨状腹综合征患儿需及时行尿动力学检查，了解下尿路功能状况，采取相应的治疗措施如膀胱颈口电切等方法解除梗阻，防止上尿路损害发展。     

Long-term chronic complications from Stamey endoscopic bladder neck suspension: a case series

Purpose/objective Long-term complications from anti-incontinence surgical procedures are rarely reported. We report on delayed presentation of complications relating to the synthetic bolster placed for the Stamey bladder neck suspension. Materials and methods: Patients undergoing re-operative surgery following prior Stamey endoscopic bladder neck suspension were selected from a surgical database. Four women with lower urinary tract and/or vaginal symptoms following prior Stamey endoscopic bladder neck suspension were identified. All patients had undergone removal of the bolster material by a single surgeon (ESR) at re-operation. Preoperative, operative, and postoperative inpatient and outpatient records were reviewed. Results: Patients presented with a variety of symptoms including urinary incontinence, recurrent cystitis, vaginitis, and urinary frequency at 9, 11, 11, and 12 years after Stamey bladder neck suspension. In addition, two patients presented with recurrent, intermittent bloody vaginal discharge and two patients complained of recurrent urinary tract infections and irritative voiding symptoms. All patients underwent transvaginal excision of the Dacron bolster. Three patients also underwent placement of an autologous pubovaginal sling for symptomatic recurrent stress urinary incontinence. At a mean follow-up of 30 months all four patients were improved. There was no recurrence of vaginal discharge or urinary tract infections. Irritative voiding symptoms resolved. Conclusions: Delayed complications from surgically implanted synthetic materials can present many years after initial implantation. The clinical findings are often subtle and require a high degree of suspicion. Vaginal discharge and irritative urinary symptoms in patients with even a remote history of Stamey bladder neck suspension should prompt a thorough vaginal exam and cystoscopy. Excision of the bolsters can be performed and is usually followed by symptomatic improvement.