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1.
目的 探讨尘螨阳性婴幼儿首次喘息后反复喘息发作的危险因素。方法 选取2014年8月至2015年2月间住院的首次喘息发作婴幼儿共1 236例,其中尘螨阳性387例,出院后随访1年,随访1年内再发喘息3次及3次以上的患儿设定为反复喘息组(n=67),随访期间未再发生喘息的患儿设定为对照组(n=84)。采用单因素分析和多因素logistic逐步回归分析,探讨尘螨阳性的婴幼儿反复喘息发作的危险因素。结果 单因素分析显示,入院时年龄、入院前喘息时间、肺炎支原体感染率、流感病毒感染率与反复喘息发作相关联。多因素logistic逐步回归分析显示,入院时年龄较大(OR=2.21,P=0.04)、合并肺炎支原体感染(OR=3.54,P=0.001)为反复喘息发作的独立危险因素。结论 尘螨阳性的婴幼儿,特别是幼儿,若首次喘息时合并有肺炎支原体感染,则反复喘息发作的风险明显升高。  相似文献   

2.
目的 探讨乌鲁木齐地区喘息患儿发生支气管哮喘(哮喘)的危险因素.方法 对2008年1 -12月在新疆医科大学第五附属医院门诊及住院的300例喘息患儿的临床资料进行统计.用统一的调查表调查其年龄、性别、湿疹、变应性鼻炎、食物过敏、家族过敏史/哮喘史、运动相关性喘息等.出院后通过门诊或电话进行随访.采用 Logistic回归分析方法对各因素与哮喘发生的关系及相关程度进行分析.结果 随访2a,275例获得随访;25例失访.275例喘息患儿在随访期内86例(31.2%)发生哮喘.Logistic回归分析发现湿疹、变应性鼻炎、家族过敏史/哮喘史、运动相关性喘息、反复下呼吸道感染( LRTI)、外周血嗜酸性粒细胞(EOS)增高与喘息患儿发生哮喘有关(湿疹:OR=2.376,95% CI0.098~0.935,P=0.039;变应性鼻炎:OR=1.052,95% CI2.267 ~14.283,P =0.024;家族过敏史/哮喘史:OR=1.886,95%CI1.004~3.542,P =0.048;运动相关性喘息:OR=1.881,95% CI2.267 ~18.983,P =0.001;LRTI:OR=5.341,95% CI1.676~ 10.983,P =0.016;外周血EOS增高:OR=3.915,95% CI1.459~ 10.501,P=0.002).结论 个人过敏史(湿疹和变应性鼻炎)、家族过敏史/哮喘史、运动相关性喘息、LRTI、外周血EOS增高是乌鲁木齐地区喘息患儿发生哮喘的危险因素.  相似文献   

3.
Chen YZ  Ma Y  Wang HY  Wang HJ  Zhao J  Cao L  Li S  Wong GW  Zhong NS  Fok TF  Lai CK 《中华儿科杂志》2003,41(7):538-541
目的 了解个人过敏原阳性与喘息及气道高反应性的关系。方法 在北京、广州及香港三城市中采用整群抽样的方法,在9~11岁在校学龄儿童中,应用国际间儿童哮喘与过敏性疾病研究的第二阶段研究方案进行研究,内容包括(1)家长书面问卷(共收集问卷10902份),(2)儿童皮肤过敏原点刺试验(3478例),(3)乙酰甲胆碱支气管激发试验(608例)。结果 近期喘息(在12个月内有发作)发生率:北京3.8%、广州3.4%、香港5.8%。特应性(即≥1种过敏原阳性)阳性率北京23.9%、广州30.8%、香港41.2%。乙酰甲胆碱支气管激发试验阳性率:北京33.2%、广州45.8%、香港30.7%。多因素logistic回归分析显示,屋尘螨P[相对危险度(OR)=4.48;95%可信限(CI):3.02—6.66]、猫毛(OR=2.59;95%CI:1.67~4.02)、粉尘螨F(OR=2.41;95%CI:1.65~3.51)及混合草花粉过敏(OR=2.85;95%CI:1.24~6.50)是近期喘息显著相关的危险因素;特应性(OR=1.29;95%CI:0.74~2.24)与近期喘息无显著相关性。特应性(OR=2.53;95%CI:1.07~5.97)、猫毛(OR:3.01;95%CI:1.39~6.52)及粉尘螨F(OR=3.67;95%CI:1.93~6.97)是气道高反应性显著相关的危险因素。结论 屋尘螨P、粉尘螨F、猫毛、混合草花粉是9-ll岁组儿童近期喘息的危险因素,而特应性不是近期喘息的独立危险因素。特应性、猫毛、粉尘螨F是气道高反应性的危险因素。  相似文献   

4.
目的 系统评价阿奇霉素(AZM)辅助治疗儿童毛细支气管炎的有效性。方法 采用RevMan 5.3软件,对截止2019年2月17日国内外发表的关于AZM用于辅助治疗毛细支气管炎的临床随机对照试验进行Meta分析。结果 共纳入14篇文献,干预组667例,对照组651例。合并效应量结果显示,AZM辅助治疗不能缩短毛细支气管炎患儿的住院时间(MD=-0.29,95% CI:-0.62~0.04,P=0.08)及用氧时间(MD=-0.33,95% CI:-0.73~0.07,P=0.10);可以缩短患儿喘息(MD=-1.00,95% CI:-1.72~-0.28,P=0.007)及咳嗽缓解时间(MD=-0.48,95% CI:-0.67~-0.29,P < 0.00001)。菌群分析结果提示,AZM能有效减少患儿鼻咽部肺炎链球菌(OR=0.24,95% CI:0.11~0.54,P=0.0006)、嗜血杆菌属(OR=0.28,95% CI:0.14~0.55,P=0.0002)和卡他莫拉菌(OR=0.21,95% CI:0.11~0.40,P < 0.00001)的检出率。结论 AZM辅助治疗儿童毛细支气管炎一定程度上有助于缩短患儿咳嗽及喘息缓解时间,但对住院及用氧时间的影响不显著。  相似文献   

5.
目的:了解儿童重症社区获得性肺炎病毒病原谱,探讨儿童重症社区获得性肺炎的危险因素。方法:收集2007年9月至2008年8月1096例社区获得性肺炎患儿的气道抽吸物标本,其中重症社区获得性肺炎100例。采用RT-PCR、PCR或巢式PCR方法对呼吸道病毒进行核酸检测。应用logistic回归法对患儿的临床相关资料进行单因素和多因素分析,以调查重症社区获得性肺炎的危险因素。结果:100例儿童重症社区获得性肺炎标本中,病毒总检出例数为82例(82%),其中RSV检出率最高(37%),其次为HBoV(25%)和HRV (18%)。2种及2种以上病毒协同感染32例(32%)。Logistic回归分析显示,合并基础疾病及RSV感染为儿童重症社区获得性肺炎发病的危险因素(分别OR=6.623,P<0.01;OR=1.672,P<0.05),月龄为保护因素(OR=0.475,P<0.01)。结论:RSV是儿童重症社区获得性肺炎最常见病毒病原;合并基础疾病及RSV感染是儿童重症社区获得性肺炎发病的危险因素,月龄为保护因素。  相似文献   

6.
儿童腹股沟疝发病危险因素的病例对照研究   总被引:2,自引:1,他引:1  
目的:儿童腹股沟疝为出生缺陷中常见疾病类型,目前关于其发病危险因素的报道尚不多见。该研究旨在探讨儿童腹股沟疝发生的主要危险因素。方法:采用频数匹配的病例对照研究方法,对132例0~6岁腹股沟疝患儿和132例0~6岁对照儿童的情况进行问卷调查,用χ2检验和logistic回归对所收集资料进行单因素及多因素分析,计算OR值(比值比)及OR值的95%CI(可信区间)。结果:儿童腹股沟疝与儿童啼哭不安史(OR=3.701,95%CI:1.724~7.945)、母亲孕前1年和孕期头3个月腌制品摄入史(OR=2.534,95%CI:1.279~5.021)、母亲孕前1年和孕期头3个月贫血史(OR=3.761,95%CI:1.497~9.450) 及儿童腹股沟疝家族史(OR=13.505,95%CI:5.825~31.307)有关。结论: 儿童啼哭不安史、腹股沟疝家族史、母亲孕前1年和孕期头3个月贫血史及腌制品摄入史是儿童腹股沟疝发生的危险因素。  相似文献   

7.
目的 探讨中国儿童哮喘的主要危险因素,为哮喘的防治提供参考依据。方法 系统收集中国知网、万方数据库、中国生物医学文献数据库、维普中文科技期刊全文数据库、Web of Science和PubMed等数据库从建库至2017年9月有关中国儿童哮喘危险因素的研究。采用Stata 12.0软件进行Meta分析。结果 共纳入24篇病例对照研究,其中病例组5 309例,对照组6 404例。Meta分析显示,家族哮喘史(OR=5.246,95% CI:3.435~8.011)、家族过敏史(OR=4.627,95% CI:2.450~8.738)、特应性体质(OR=4.659,95% CI:2.511~8.644)、变应性鼻炎(OR=11.510,95% CI:6.769~19.574)、湿疹/皮炎史(OR=4.919,95% CI:3.514~6.886)、患儿过敏史(OR=4.732,95% CI:2.802~7.989)、食物过敏史(OR=5.890,95% CI:3.412~10.166)、药物过敏史(OR=4.664,95% CI:2.637~8.252)、家中有霉斑(OR=2.483,95% CI:1.671~3.690)、家中种花草(OR=1.748,95% CI:1.383~2.209)、房屋装修史(OR=2.823,95% CI:2.206~3.935)、剖宫产(OR=1.894,95% CI:1.166~3.077)是儿童哮喘的危险因素,母乳喂养是儿童哮喘的保护因素(OR=0.508,95% CI:0.396~0.653)。结论 中国儿童哮喘的发生与多种因素有关,其中家族哮喘史、家族过敏史、个体特应性体质、过敏史、过敏合并症、剖宫产出生及不良的家庭环境因素可增加儿童哮喘的患病风险,而母乳喂养可降低儿童哮喘的患病风险。  相似文献   

8.
目的探讨年幼儿童反复喘息发作的转归及不同表型的危险因素。方法选择2013年10月至2014年5月间纳入研究的≤5岁反复喘息的年幼儿童,在时隔3年后进行电话随访。调查患儿入组以来的喘息发作情况、鼻炎、湿疹及父母哮喘史等。根据患儿喘息起始及缓解年龄,将接受调查的患儿分为一过性喘息、持续性喘息和晚发性喘息组,比较三组患儿出生史、鼻炎、湿疹、父母哮喘史、过敏相关指标、喘息起始年龄等的差异。结果在入组的100例≤5岁的年幼儿童中随访到85例,男性66例、女性19例,随访时平均年龄(6.82±1.20)岁。其中14例一过性喘息组患儿的喘息起始年龄早于54例持续性与17例晚发性喘息组患儿,差异有统计学意义(P0.05)。与入组时相比,三组患儿近一年的喘息次数均显著减少,差异有统计学意义(P0.05)。既往鼻炎、近一年伴发鼻炎在三组间的差异均有统计学意义(P0.05),持续性和晚发性喘息组的发生率较高。血清总Ig E、吸入性过敏原检测阳性率以及外周血嗜酸性粒细胞(EOS)百分比在一过性喘息、持续性喘息、晚发性喘息患儿中依次升高,差异均有统计学意义(P0.05)。结论年幼儿童随着年龄增加,其喘息发作显著减少,喘息的转归与过敏、鼻炎、起病年龄等显著相关。  相似文献   

9.
目的探讨年幼儿童反复喘息发作的转归及不同表型的危险因素。方法选择2013年10月至2014年5月间纳入研究的≤5岁反复喘息的年幼儿童,在时隔3年后进行电话随访。调查患儿入组以来的喘息发作情况、鼻炎、湿疹及父母哮喘史等。根据患儿喘息起始及缓解年龄,将接受调查的患儿分为一过性喘息、持续性喘息和晚发性喘息组,比较三组患儿出生史、鼻炎、湿疹、父母哮喘史、过敏相关指标、喘息起始年龄等的差异。结果在入组的100例≤5岁的年幼儿童中随访到85例,男性66例、女性19例,随访时平均年龄(6.82±1.20)岁。其中14例一过性喘息组患儿的喘息起始年龄早于54例持续性与17例晚发性喘息组患儿,差异有统计学意义(P0.05)。与入组时相比,三组患儿近一年的喘息次数均显著减少,差异有统计学意义(P0.05)。既往鼻炎、近一年伴发鼻炎在三组间的差异均有统计学意义(P0.05),持续性和晚发性喘息组的发生率较高。血清总Ig E、吸入性过敏原检测阳性率以及外周血嗜酸性粒细胞(EOS)百分比在一过性喘息、持续性喘息、晚发性喘息患儿中依次升高,差异均有统计学意义(P0.05)。结论年幼儿童随着年龄增加,其喘息发作显著减少,喘息的转归与过敏、鼻炎、起病年龄等显著相关。  相似文献   

10.
婴儿期食物过敏的预后研究   总被引:5,自引:0,他引:5  
Wang NR  Li HQ 《中华儿科杂志》2005,43(10):777-781
目的了解婴儿期食物过敏(FA)的预后及影响因素。方法对119例患儿进行回顾性研究,采用Kaplan-Meier法计算食物耐受的累积概率,非条件Logistic回归模型分析食物耐受形成的预测因素和其他过敏性疾病发生的影响因素。结果鸡蛋、牛奶耐受的累计概率在诊断后1年分别为31%、42%;2年为62%、63%;3年为80%、77%;4年后均可达100%。牛奶、鸡蛋过敏患儿的皮肤点刺试验阳性强度是持续牛奶、鸡蛋敏感的预测因素(OR=2.535,95%CI:1.159~5.543;OR=2.654,95%CI:1.302~5.410,P均〈0.05)。本组13例患儿发生其他FA,危险因素是持续鸡蛋敏感(OR=6.109,95%CI:1.818~20.527,P〈0.05);4例发生变态反应性鼻炎,15例发生支气管哮喘,危险因素是持续鸡蛋敏感和呼吸道过敏症状(OR=3.596,95%CI:1.429~9.045;OR:4.235,95%CI:1.152~15.563,P均〈0.05)。结论至少75%的鸡蛋或牛奶过敏患儿在诊断后3年内可获耐受。10.9%、12.6%和3.4%的FA儿童发生其他FA、支气管哮喘和变态反应性鼻炎。加强持续FA高危儿的筛查和管理,可能有助于改善FA的预后。  相似文献   

11.
婴幼儿哮喘的危险因素   总被引:1,自引:0,他引:1  
目的寻找婴幼儿喘息、儿童哮喘的危险及保护因素。方法1个月~3岁肺炎患儿82例、毛细支气管炎65例、婴幼儿哮喘76例,107例正常儿童作为对照。采用向家长书面问卷调查方法询问各组婴儿既往病史、家族及个人过敏史、怀孕出生及婴幼儿喂养情况、生活条件等。结果父母任何一方有喘息史、父母任何一方有其他过敏史、父母任何一方吸烟、湿疹等个人过敏史任何一种、父(母)亲有哮喘史及过敏史、兄弟姐妹有哮喘史及过敏史、个人有过敏性鼻炎、个人有药物过敏、母妊娠期间早孕反应重、母亲是素食者、母亲文化程度低是婴幼儿哮喘的危险因素;而自然分娩、婴幼儿期补充VitD和钙剂、生后冬季户外活动时间长、每天平均睡眠时间长、住所有上下水设备是婴幼儿哮喘的保护因素。结论个人过敏史和家族过敏史为婴幼儿哮喘的主要危险因素。  相似文献   

12.
Early identification of wheezing children with an increased risk of recurrent wheezing or subsequent asthma is important. The aim of the study was to determine the role of markers of eosinophil activation, along with other parameters, in the prediction of recurrent wheezing and allergic sensitization in children with early and severe wheezing. We examined 105 children without atopic dermatitis, hospitalized for wheezing during the first year of life. At a 20-mo follow-up, 101 of the children were assessed for the occurrence of recurrent wheezing (at least 3 episodes, including 1 in the previous 6 mo) and allergic sensitization (positive skin-prick test). By univariate analysis, levels of eosinophil counts at the time of hospitalization (p = 0.005, OR = 18.9), age in months (p < 0.0001, OR = 1.5), respiratory syncytial virus (RSV)-negative disease (p < 0.0001, OR = 8.8), parental atopy (p = 0.006, OR = 3.3) and male sex (0.02, OR = 2.7) were all predictive factors for recurrent wheezing at follow-up. With all parameters included in a multiple regression analysis, RSV-negative disease was not a predictive factor for recurrent wheezing. A simple model including eosinophil counts > or = 0.1 x 10(9)/L and age had a predictive accuracy of 79%, with only a 6% chance of a child being wrongly predicted as symptomatic. Urinary protein X (U-EPX) was not a predictive factor for recurrent wheezing. When included in a multiple logistic regression analysis, a level of U-EPX > or = 100 microg/mmol creatinine was the only parameter with a positive predictive value for allergic sensitization (p = 0.007, OR = 18.9), whereas age, parental allergy or parental asthma were not. CONCLUSION: Children with severe wheezing during the first year of life and subsequent recurrent wheezing are characterized by a normal or high eosinophil count in response to viral infections.  相似文献   

13.
Objectiveto identify possible risk factors associated with wheezing in infants (12-15 months-old) in the state of Mato Grosso, Brazil.Methodsthis was a cross-sectional study performed by applying a standardized written questionnaire from the international study on wheezing in infants (Estudio Internacional de Sibilancia en Lactantes - EISL), phase 3. Parents and/or guardians of infants were interviewed at primary health care clinics or at home from August of 2009 to November of 2010. Factors associated to wheezing were studied using bivariate and multivariate analysis (using the Statistical Package for Social Sciences [SPSS] v.18.0), and expressed as odds ratios (OR) and 95% confidence intervals (95% CI).Resultsthe written questionnaire was answered by 1,060 parents and/or guardians. The risk factors for wheezing were: history of asthma in the family [mother (OR = 1.62; 95% CI = 1.07-2.43); father (OR = 1.98; 95% CI = 1.22-3.23); siblings (OR = 2.13; 95% CI = 1.18-3.87)]; history of previous pneumonia (OR = 10.80; 95% CI = 4.52-25.77); having had more than six upper respiratory tract infections (URTIs) (OR = 2.95; 95% CI = 2.11-4.14); having had first URTI before the third month of life (OR = 1.50; 95% CI = 1.04-2.17); living in a moderately polluted area (OR = 1.59; 95% CI = 1.08-2.33); paracetamol use for URTI (OR = 2.13; 95% CI = 1.54-2.95); and antibiotic use for skin infection (OR = 2.29; 95% CI = 1.18-4.46).Conclusionsthe study of risk factors for wheezing in the first year of life is important to help physicians identify young children at high risk of developing asthma and to improve public health prevention strategies in order to reduce the morbidity of wheezing in childhood.  相似文献   

14.
AIM: In this study we aimed to detect the prevalence and risk factors of asthma and allergic diseases in children aged between 7 and 14 years old at rural and urban areas of Bolu, Turkey. METHODS: Questionnaire of International Study of Asthma and Allergies in Childhood (ISAAC) phase one and questionnaire including questions about family, demographic, socio-economic characteristics of children were applied to 931 schoolchildren who were selected by randomized sampling. RESULTS: In children, the prevalence of diseases and symptoms were as follows: wheeze ever: 15.5%, asthma: 5.6%, nasal symptoms ever: 41.4%, allergic rhinitis: 23.2%, itchy rash ever: 5.9% and eczema: 5.0%. In multivariate regression analysis, presence of allergic disease in the family was risk factor for wheezing (OR=1.74, 95% CI=1.19-2.76), asthma (OR=2.19, CI=1.06-4.52), allergic rhinitis (OR=2.68, CI=1.80-3.98) and eczema (OR=2.33, CI=1.17-4.65); living in shanties was risk factor for allergic rhinitis (OR=5.26, CI=2.1-13.16); a monthly income below $300 was risk factor for asthma (OR=2.54, CI=1.06-6.08). CONCLUSION: It was detected that the prevalence of allergic rhinitis and its symptoms was more common in schoolchildren living in Bolu. Presence of allergic disease in fathers or mothers and low socio-economic level increase the risk of asthma and other allergic diseases in children.  相似文献   

15.
The Prevalence And Risk Factors of Allergies in Turkey (PARFAIT) study was planned to evaluate prevalence and risk factors of asthma and allergic diseases and also to find out which geographical variables and/or climatic conditions play a role determining the prevalence of allergic diseases in Turkish school children. Study was planned as cross-sectional questionnaire-based. About 25,843 questionnaires from 14 centers were appropriate for analysis. Parental history of allergy, having an atopic sibling and other atopic disease in index case was significant risk factors for all allergic diseases. Breast feeding decreased the risk of current asthma (OR: 0.92, CI: 0.86-0.99) and wheezing (OR: 0.93, CI: 0.87-0.99) but not allergic rhinitis and eczema. Respiratory infection in the past was an important risk factor for the occurrence of allergic diseases especially for asthma which was increased 4.53-fold. Children exposed to household smoke were significantly at higher risk of asthma, wheezing, and allergic rhinitis (OR: 1.20, CI: 1.08-1.33; OR: 1.21, CI: 1.09-1.34; and OR: 1.32, CI: 1.21-1.43, respectively). All allergic diseases were increased in those children living in areas which have altitude of below 1000 m and mean yearly atmospheric pressure above 1000 mb. The study has suggested that household and country-specific environmental factors are associated with asthma, wheezing, allergic rhinitis, and eczema risk during childhood in Turkey.  相似文献   

16.
目的探讨婴幼儿喘息发作与肺炎支原体(MP)感染之间的关系。方法选取228例下呼吸道感染婴幼儿分为初次喘息组(65例)、反复喘息组(83例)和无喘息组(80例)。收集患儿入院当天或次日空腹血清,采用ELISA法检测MP-Ig M,化学发光法测定血清总免疫球蛋白E(TIg E),欧蒙印迹法检测血清常见过敏原特异性免疫球蛋白E(s Ig E),并且收集患儿特应性体质表现及过敏性疾病家族史临床资料。结果初次喘息组和反复喘息组患儿MP感染阳性率及血清TIg E水平高于无喘息组(P0.05);反复喘息组患儿s Ig E检测阳性率显著高于初次喘息组和无喘息组(P0.05),且这部分患儿特应性体质表现及过敏性疾病家族史与发病密切相关。结论 MP感染与婴幼儿喘息密切相关,MP是诱发婴幼儿喘息发作的主要病原体之一,过敏原、特应性体质和过敏性疾病家族史是婴幼儿反复喘息的主要危险因素。  相似文献   

17.
目的探讨早产儿发生支气管肺发育不良(BPD)的危险因素及远期随访结局。方法 以2012年1月至2013年12月在复旦大学附属儿科医院新生儿科病房住院的胎龄≤32周、出生体重≤1 500 g及生后7 d内入院的BPD早产儿为BPD组,同期入住我院的非BPD早产儿中选取与BPD组等同样本量的病例为对照组。采集与BPD发生的母亲和新生儿因素行单因素分析和多因素分析。同时统计BPD早产儿生后1岁内和~2岁的支气管炎、肺炎、喘息发作次数和住院次数等指标。结果 BPD组和对照组均纳入了156例早产儿。单因素分析显示,BPD组母亲年龄(P=0.046)、先兆子(P=0.025)和阴道产(P<0.001)比例显著高于对照组;BPD组出生胎龄、出生体重显著低于对照组 (P均<0.001 )。BPD组1 和5 min Apgar 评分,败血症 ≥72 h、动脉导管未闭(PDA)、早产儿视网膜病变、应用肺表面活性物质、呼吸机相关性肺炎、机械通气≥7 d的比例均与对照组差异有统计学意义。多因素Logistic回归分析显示胎龄(OR= 0.46,95%CI:0.37~0.58)、机械通气≥7 d(OR=9.47,95%CI:3.70~24.27)、PDA(OR=2.21,95%CI:1.18~4.12)、先兆子(OR=4.91,95%CI:1.26~19.15 )是发生BPD的危险因素。BPD组1岁以内支气管炎、喘息的发生率高于对照组,再入院率两组差异无统计学意义;BPD组生后~2岁较生后1岁以内肺炎的发生率显著下降,支气管炎、喘息的发生率及再入院率差异无统计学意义。结论 低出生胎龄、机械通气≥7 d、PDA、先兆子是发生BPD的危险因素;BPD早产儿在生后1年以内下呼吸道感染的发生率增高。  相似文献   

18.
目的 探讨儿童厌食症的危险因素,以降低儿童厌食症的患病率。方法 采用问卷调查方式和病例对照研究方法收集150例厌食症儿童 (病例组)和150例正常儿童 (对照组)的一般资料,采用单因素分析和多因素logistic逐步回归分析研究儿童厌食症的危险因素。结果 单因素分析显示,病例组和对照组在添加辅食月龄、喂养方式、儿童是否喜欢肉食、是否喜欢蔬菜、是否喜欢咸食、是否常进食零食和/或饮料、是否边吃边玩、家长是否要求儿童按时进食等方面的差异有统计学意义 (P < 0.05)。多因素logistic回归分析显示,添加辅食时间晚 (OR=5.408)、常进食零食和/或饮料 (OR=11.813)、喜欢边吃边玩 (OR=6.654)是儿童厌食症的主要危险因素;而喜欢肉食 (OR=0.093)、喜欢蔬菜 (OR=0.272)以及家长要求儿童按时进食 (OR=0.079)是儿童厌食症的保护因素。结论 适时添加辅食、合理膳食、培养儿童正确的饮食和生活习惯可以减少儿童厌食症的发生。  相似文献   

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