外伤性硬膜下积液与慢性硬膜下血肿

目的探讨硬膜下积液与慢性硬膜下血肿关系。方法回顾2001年1月至2010年1月本院165例外伤性硬膜下积液的病人。结果自发性演变成慢性硬膜下血肿18例,发生率10.9%。结论外伤性硬膜下积液演变成慢性硬膜下血肿发生率低。对转化成CSDH后症状明显加重者,主张早期手术治疗,对症状不明显者,特别是CSDH呈低密度或等密度、体积较小,继续行保守治疗。     

慢性硬膜下血肿伴对侧硬膜下积液45例临床分析

近年来。硬膜下积液转变成慢性硬膜下血肿报道渐渐增多，而一侧慢性硬膜下血肿（CSDH）合并对侧硬膜下积液（SFC）的病例鲜有报道。本院自2004年1月至2008年12月共收治45例一侧慢性硬膜下血肿（CSDH）合并对侧硬膜下积液病例，现报道如下。     

慢性硬膜下血肿钻孔引流210例分析

慢性硬膜下血肿(CSDH)在颅脑损伤中并不少见.本院自1987年8月至2002年12月期间,共收治(CSDH)病人210例,占同期收治颅脑损伤病人的4%左右.现报告如下.     

外伤性硬膜下积液演变血肿34例临床分析

目的:探讨外伤性硬膜下积液( TSE)演变慢性硬膜下血肿(CSDH)的机制、临床特点.方法:对我科34例外伤性硬膜下积液演变为慢性硬膜下血肿患者进行回顾性分析.结果:34例演变为慢性硬膜下血肿的时间为伤后35-155天,演变为血肿后症均加重,28例患者经钻空引流术均治愈.结论:TSE是CSDH形成的因素之一,中老年多见,对TSE患者应行动态观察CT或MRI,早期发现CSDH.     

外伤性硬膜下积液转化为慢性硬膜下血肿分析

随着影像学技术的发展和普及．头部外伤后硬膜下积液（TSE）逐步演变为慢性硬膜下血肿（CSDH）的报道越来越多，并逐渐已被人们所认识和重视。本院白1996年1月至2006年1月．收治TSE患者60例，发现10例演变为CS-DH。现报道如下。     

118例老年慢性硬膜下血肿的护理

慢性硬膜下血肿（CSDH）以颅内压增高、偏瘫及智能障碍为三主征。如果慢性硬膜下血肿能及时确诊，予以适当治疗，预后多属良好，本文通过总结本院神经外科2000年3月至2005年4月．收治的118例老年慢性硬膜下血肿病人的观察与护理上经验。现报告如下。     

应用微创术治疗慢性硬膜下血肿

目的 探讨应用YL-1型微创颅内血肿穿刺针治疗慢性硬膜下血肿（CSDH）疗效和治疗方法。方法 回顾性分析微创引流术治疗126例CSDH的临床资料。结果 治愈率95．2％（120／126），复发率6．3％（8／126），并发症发生率4．8％（6／126）。结论 应用YL-1型微创颅内血肿穿刺针治疗慢性CSDH是简便、安全的，且疗效满意，并发症少。     

老年慢性硬膜下血肿35例诊治分析

慢性硬膜下血肿（Chronic subdural hematoma CSDH）是一种常见的颅内疾病，好发于老年人及小儿，占颅内血肿的10％：硬膜下血肿的25％，其中双侧血肿的发生率高达14．8％。随着老年人口的增多及CT的普及，老年慢性硬膜下血肿发病呈增高趋势。本院自2004年3月至2005年9月间，共收治老年CSDH患35例，均行手术治疗，取得满意的疗效。现报道如下。     

锥颅双管法闭式冲洗引流治疗慢性硬膜下血肿

慢性硬膜下血肿（Chronic Subdural Hematomas CSDH）约占颅脑损伤病例的1％,占颅内血肿的10％,高龄患者多发,是神经外科常见病和多发病。钻孔慢性硬膜下血肿缓慢外引流术是公认的最有效的治疗方法．但CSDH术后脑膨胀缓慢、硬膜下积液、颅内积气和术后复发是目前CSDH治疗后常见难题。本院采用锥颅双管法闭式冲洗引流术治疗35例慢性硬膜下血肿,较好地解决了术后颅内积气、积液和复发等问题,现报告如下。     

钻孔位置在慢性硬膜下血肿钻孔冲洗引流术中的作用

钻孔冲洗引流术是目前普遍认可的治疗慢性硬膜下血肿(chronic subdural hematoma，CSDH)的首选方法，具有简单有效、损伤小等优点，但也可发生多种并发症。我院1992年～2003年应用钻孔冲洗引流治疗CSDH 122例，死亡1例，出现急性硬膜外血肿1例，余120例无并发症治愈。作者认为术中适宜调整钻孔位置是预防CSDH钻孔冲洗引流术后并发症的关键。     

Percutaneous subdural tapping and subdural peritoneal drainage for the treatment of subdural hematoma

Different therapeutic approaches may be used in the treatment of chronic subdural hematoma because it is more fluid. Age-dependent characteristics of the calvarium allow for different treatment in children and adults. Treatment options are discussed.     

Mixed bacterial subdural empyema complicating subdural peritoneal shunt

Bilateral subdural empyema occurred in a nine-month-old boy with a subdural-peritoneal shunt. Because of the mixed composition of bacterial growths, representative of faecal flora, it is assumed that the infection originated from perforation of the bowel by the distal tip of the shunt catheter, although no abnormal abdominal signs or symptoms were noted.     

Traumatic subdural hydroma developing into chronic subdural hematoma

Objective: To probe the incidence, pathogenesis and clinical characteristics of traumatic subdural hydroma (TSH) developing into chronic subdural hematoma(CSDH). Methods: We retrospectively analyzed the clinical data of 32 patients with TSH developing into CSDH and reviewed related literature. Results: 16.7 % of TSH developed into CSDH in this study. The time of evolution was from 22 to 100 days after head injury. All the patients were cured with hematoma drainage. Conclusions : TSH is one of the origins of CSDH. The clinical characteristics of TSH developing into CSDH follow that the ages of the patients are polarized, that the evolution often happens in the patients with small chronic hydromas and being treated conservatively, that the patients are usually injured deceleratedly and that the accompanying cerebral damage is often very mild.     

Posterior fossa subdural hygroma with supratentorial chronic subdural haematoma

Posterior fossa subdural hygromas are very rare. They tend to occur following direct occipital trauma. We present an unusual case of complex subdural hygroma of the posterior fossa, which was associated with a supratentorial chronic subdural haematoma. This developed after an apparently minor injury to the head. The unusual features of our case are discussed. We also review the literature and discuss the natural history and pathogenesis of subdural hygroma.     

Symptomatic subacute subdural haematoma following spontaneous acute subdural haematoma

Summary Two patients with non-traumatic acute subdural haematoma were observed, initially without surgical intervention. Eleven days after the onset, each patient developed hemiparesis and an increase in severity of headache. Serial computed tomography scans demonstrated that the initial hyperdense haematomas became hypodense with a definite increase in volume. The term symptomatic subacute subdural haematoma was proposed to properly define this pathological process, which necessitated removal of the haematoma in the subacute stage. The mechanism of increase in the volume of the haematoma is discussed.     

Accidental subdural catheterisation

A case of accidental subdural catheterisation is presented, and its course is compared with previously reported subdural injections of local anaesthetic agents. The importance of obtaining proof of the site of malposition of an epidural catheter is stressed.     

Spontaneous evolution of posttraumatic subdural hygroma into chronic subdural haematoma

Summary Thirteen of 145 patients with post-traumatic subdural hygroma (SDHy) developed chronic subdural haematoma (CSDH) at the involved site over a period of 6 years. CSDHs were found at the site of SDHys with no history of further head injury at a mean interval of 56 days. It appeared that these 13 patients did not have any distinguishing clinical features early on. Old age and brain atrophy on CT scans do not seem to be reasonable causative factors in the evolution of SDHy into CSDH. Initial enlargement of subdural accumulations at an early stage of SDHy and a subsequent increase in density at a later stage may point to the development of CSDH from SDHy in some instances. Ten of these 13 CSDH cases underwent surgical drainage, and the remaining 3 cases received no specific management. All resolved completely. The prognosis was good in all patients. The possible mechanism for the evolution of SDHy into CSDH is discussed.