狼疮肾炎血性腹水

病历摘要 患者女性,28岁,未婚,因颜面红斑、水肿1年,腹胀、尿少2个月,加重伴腹痛、发热2 d,于2003年12月9日入院.1年前无明显诱因出现颜面红斑、乏力,继之逐渐出现颜面、双下肢水肿,无关节疼痛、怕光、口腔溃疡及发热.曾在福州某医院查尿蛋白3+,ANA阳性,ds-DNA阳性,肾功能正常,诊断系统性红斑狼疮,狼疮肾炎,给予泼尼松60 mg/d治疗,症状缓解后出院.     

一例单克隆IgG3κ沉积的增生性肾小球肾炎合并活动性丙型肝炎临床分析

病例资料 患者,女,77岁,因"反复双下肢水肿2个月,加重1周"于2019年9月18日入院.患者2月前无明显诱因出现双下肢水肿,晨轻暮重,双下肢皮温正常,无胀痛,无间歇性跛行,无关节疼痛,当地医院查24h尿蛋白定量1.4g,血肌酐79 μmol//L ,丙肝病毒RNA定量1 .31 × 106 IU/mL ,建议患者行...     

糖尿病合并纤维样肾小球病1例

病例女,60岁,因"血糖升高18年,发现肌酐升高3月"入院。18年前于我院诊断为"2型糖尿病",长期使用胰岛素控制血糖,血糖控制良好。入院前3月,感冒后出现颜面及双下肢水肿,查血肌酐260.0μmol/L。入院前1周再次复查血肌酐320.1μmol/L,尿蛋白(+++)。近期小便量无明显变化。既往有银屑病史20年,反复治疗无效。高血压病史8年,最高达     

脂蛋白肾病一例

患者：男,47岁,因颜面及双下肢水肿5个月于2002年6月13日入院。5个月前无明显诱因出现颜面及双踝关节以下浮肿,1周后发展至双下肢浮肿,伴腹胀,尿量逐渐减少。4个月前在当地就医发现尿蛋白( ),潜血( ),24h尿蛋白定量5．75g。血压170／120mmHg,白蛋白27g／L,胆固醇10．48mmol／L,尿素氮9．39mmol／L,诊断为原发性肾病综合征,     

对强的松敏感的C1q肾病1例报告并文献复习

Ｃ１ｑ肾病是临床上比较少见的一种原发性肾小球疾病，大多数对口服激素治疗不敏感。笔者近遇１例Ｃ１ｑ肾病，且对强的松高度敏感，报道如下。 病 例 患者，男性，３３岁，农民，住院号１６１８７８，因“间断颜面、双下肢水肿５年，加重半月”于２００１年１月４日第二次入院。患者于５年前因“颜面、下肢水肿”入住本院，入院时化验尿蛋白＋＋＋，２４ ｈ尿蛋白定量３．２４ ｇ，诊为“原发性肾病综合征”。同时Ｂ超、ＣＴ提示“双肾多发错构瘤”。予强的松６０ｍｇ／ｄ口服，一周后尿蛋白即转阴，水肿消退，好转出院。出院后病情稳定，…     

Ⅳ+Ⅴ型狼疮肾炎并发强直性脊柱炎一例

我们遇到1例Ⅳ+Ⅴ型狼疮肾炎(LN)并发强直性脊柱炎(AS)患者,报告如下. 患者,女,33岁,1年前无诱因出现双下肢水肿,四肢小关节肿痛及腰痛,伴晨僵,服"欣克洛"后,好转,仍反复水肿.10 d前水肿加重,门诊查尿蛋白3+,Scr 197.3μmol/L,于2010年4月18日入院.查体:BP 150/100 mm Hg,面部蝶形红斑,眼睑水肿,Schober试验阳性,Patrick 试验双侧阳性,双下肢水肿.辅助检查:尿蛋白3+.血常规WBC 2.11×109/L,Hb 82 g/L,Plt 85×109/L.BUN 27.01mmol/L,Scr 227.4 μmol/L,血风湿因子(RF)9.75 U/ml,ASO 53.1 U/L,抗SSA、抗SSB、抗Sm、抗RNP、抗ds-DNA 均阳性,ANA 1∶640,HIA-B27阳性.24 h尿蛋白5.59 g(2200 ml).     

甲泼尼龙相关性重症药疹一例

<正>病例资料患者:女,61岁,2014年2月起间断出现颜面及双下肢水肿,查尿蛋白(++)、尿隐血(+),血清白蛋白30.4 g/L、总胆固醇7.11 mmol/L、三酰甘油2.59 mmol/L,诊断为"成人原发性肾病综合征",给予护肾、利尿等治疗3个月余,症状无缓解,2015年6月入住我科,拒绝肾活检,6月29日起给予甲泼尼龙(甲基强的松龙)60 mg静滴(1次/日),辅以补钙     

PLA2R相关性膜性肾病合并肾透明细胞癌一例

临床资料 患者,男,40岁,因"双下肢水肿2个月余"于2019年4月30日入院.入院体格检查:血压 160/82 mmHg (1 mmHg=0 .133 kPa) ,全身皮肤黏膜色泽正常,心、肺、腹无特殊,双下肢中度凹陷性水肿.入院后查尿潜血(+ +),尿蛋白(+ + +), 24h尿蛋白定量7g,血红蛋白119g/L...     

尿毒症并发剥脱性食管炎一例

患者,男,27岁,因双下肢水肿、Scr升高2年,呕血、胸骨后疼痛3 h入院.患者2年前因双下肢水肿,Scr升高,在我院肾内科诊断为慢性肾功能不全.入院前3 h进食梨后,呕吐暗红色血液约30 ml,内含灰白色管状组织,约1.5 cm×10 cm,伴胸骨后疼痛及烧灼感.查体:BP 190/120 mm Hg,慢性病容,贫血貌,颜面及双眼睑水肿,睑结膜苍白,心率103次/min,上腹压痛,双下肢水肿.     

银屑病相关IgA肾病并发肾脏淀粉样变一例

我科收治1例银屑病相关IgA肾病并发肾脏淀粉样变患者,报告如下.患者男,48岁,因水肿3个月,加重1周于2010年9月入院.患银屑病20余年,未系统治疗.查体:颜面水肿,舌体稍大,躯干、四肢薄膜现象及Auspitz征阳性,腹部膨隆,移动性浊音阳性,双下肢水肿.实验室检查:尿蛋白3+,尿蛋白量8.81 g/d,尿κ轻链103.2 mg/d,λ轻链492 mg/d,血总蛋白28.6 g/L,白蛋白10.7 g/L,球蛋白17.9 g/L,BUN 7.59 mmol/L,Scr 87.5 μmol/L,三酰甘油2.1 mmol/L,胆固醇9.78 mmol/L.     

Neurohistology of the sinus tarsi and sinus tarsi syndrome

The purpose of this study was to clarify the neural anatomy of the sinus tarsi. The nerve endings of the synovium in the sinus tarsi were examined. The synovial membrane in the sinus tarsi was excised in 20 patients with sinus tarsi syndrome (20 feet) and in 2 feet from subjects without symptoms (controls). In 15 of the 20 patients and the two controls, the excised synovial membrane was studied histologically with staining by a modified Gairns gold chloride method. Numerous neural elements were observed in the sinus tarsi in all examined synovium. There were abundant free nerve endings and three types of mechanoreceptors: Pacinian corpuscles, Golgi corpuscles, and Ruffini corpuscles. Macroscopic observation and histological examination, using hematoxylin and eosin, in the other 5 patients revealed chronic synovitis in the sinus tarsi. Our findings suggest that the sinus tarsi is not only a talocalcaneal joint space but a source of nociceptive and proprioceptive information on the movement of the foot and ankle. Sinus tarsi syndrome may result from disorders of nociception and proprioception in the foot. Received for publication on Dec. 14, 1998; accepted on Feb. 23, 1999     

Frontal sinus fracture following osteoplastic frontal sinus obliteration

A patient who sustained frontal sinus fracture and who earlier had undergone an osteoplastic fat obliteration procedure is described. The literature is reviewed and recommendations are made for management of this and other cases of frontal sinus fractures with posterior table involvement.     

Calculocutaneous sinus

Extrusion of a urinary calculus is a rare occurrence, and to date only 7 cases have been previously documented. Three patients are described, all of whom had a persistent discharging calculocutaneous sinus as a manifestation of the extruded calculus. The 3 cases had similar features of presentation with a perinephric abscess, a staghorn calculus within a nonfunctioning kidney, and a classic radiologic feature of a portion of calculus clearly distinct from the main bulk of the stone.     

Functional endoscopic sinus surgery for isolated sphenoid sinus disease

Background. This article reviews 12 cases of isolated sphenoid sinus disease: chronic inflammatory sinusitis (7), mucoceles (2), aspergillus lesions (2), and isolated polyp (1). Methods. Criteria for diagnosis were based on clinical symptoms, nasal endoscopic evaluation, and computed tomography (CT). Magnetic resonance imaging was used only in cases of bone erosion and when patients presented with vision problems. All patients were treated by functional endoscopic sphenoidotomy. Any postoperative complications were noted. Conclusion. The reported good results, on the basis of regression of functional symptoms and with nasal endoscopic and CT evaluation, suggest that intranasal sphenoidotomy under endoscopic control is a safe and effective method of treatment of nonmalignant isolated sphenoid disease. The mean follow-up is 26 months. © 1994 John Wiley & Sons, Inc.