共查询到20条相似文献,搜索用时 78 毫秒
1.
2.
3.
小儿心肌炎的心率变异性 总被引:1,自引:0,他引:1
目的:探讨小儿心肌炎的心率变异性。方法:采用ECG-LA2.OGold型心电综合分析系统,描记肢体导联采样5分钟,应用时域及功率谱分析法,通过计算机计算出全部R-R间期的标准差(sDNN),极低频(VLF)低频(LF)高频(HF)。结果:以SDNN<27ms为阳性诊断标准,将57例小儿心肌炎病儿分为两组,一组SDNN<27ms共17例,病情较重,另一组SDNN>27ms共41例,病情较轻。结论:病情较重的心肌炎患儿短程HRV降低SDNN<27ms。 相似文献
4.
稳定型心绞痛与不稳定型心绞痛患者心率变异性对比分析 总被引:1,自引:0,他引:1
目的 通过稳定型心绞痛(SA0)不稳定型心绞痛(UA)患的心率变异性(HRV)对比分析,探讨HRV在冠心病SA和UA患中的影响。方法 68例心绞痛患随机分为SA组(35例),UA组(33例),正常人32例列入正常对照组;对三组的24h动态心电图进行HRV的时域和频域结果分析比较。结果 冠心病SA组,UA组与正常组对照HRV有明显下降,冠心病UA组与SA组比较HRV多数数据有明显下降。结论 临床上检测HRV对判断冠心病心肌缺血的损害程度可能有重要的临床意义。 相似文献
5.
目的:探讨心率变异性指标与冠心病患者冠脉病变程度的关系。方法:选取冠心病患者84例(观察组),同时选取健康志愿者90例作为对照组,均给予24 h动态心电图监测。结果:观察组NN间期的标准差(SDNN)、RR间期的均值标准差(SDANN)、相邻RR间期差值的均方根(RMSSD)、相邻RR间期差值>50 ms的心搏数所占百分比(PMN50)和高频功率(HF)分别为(95.54±20.12) ms、(94.48±21.15) ms、(29.84±10.06) ms、(7.46±2.31)%和(0.85±0.20) Hz,明显低于对照组(P<0.05),而低频功率(LF)为(137.74±21.16) Hz,明显高于对照组(P<0.05);冠状动脉病变≥3支患者SDNN、SDANN、RMSSD、PMN50和HF分别为(84.31±19.64) ms、(89.42±18.82) ms、(23.38±7.15) ms、(5.40±1.90)%和(0.70±0.21) Hz,明显低于冠状动脉病变数为1支和2支患者(P<0.05),而LF为(150.54±21.15) Hz,明显高于冠状动脉病变数为1支和2支患者(P<0.05);观察组男女患者SDNN、SDANN、RMSSD、PMN50、HF和LF比较差异无统计学意义(P>0.05);Gensini评分与SDNN、SDANN、RMSSD、PMN50、HF呈负相关(r=-0.511、-0.487、-0.543、-0.512和-0.507, P<0.05),与LF呈正相关(r=0.577, P<0.05)。结论:动态心电图在冠心病患者冠脉病变程度评价中有一定应用价值,其心率变异性指标与冠状动脉病变程度有一定关系。 相似文献
6.
7.
8.
9.
10.
海洛因成瘾者的心率变异性研究 总被引:5,自引:0,他引:5
利用功率谱估计的方法 ,对 15例海洛因成瘾者和 15例正常人的 HRV信号进行分析。分析结果表明 ,在海洛因成瘾者和正常人心率变异性信号功率谱间存在高频谱峰位置的显著差异 ,即海洛因成瘾者 HRV信号功率谱的高频谱峰位于 0 .437± 0 .0 6 4Hz,而正常人的 HRV信号功率谱的高频谱峰位于 0 .32 5± 0 .0 5 2 Hz 相似文献
11.
S. S. Zaguskina S. L. Zaguskin Y. V. Gurov 《Bulletin of experimental biology and medicine》2008,146(2):196-199
Circahoralian oscillations of various indices describing variations of interpulse, respiratory, and R-R intervals were recorded in normal subjects, in hypertensive patients, and in patients rehabilitating after cardiac infarction.
The parameters of these rhythms reflect changes in the status of autonomic nervous system, correlate with pathological disturbances
in circadian rhythm, and accompany the sleep-wake and wake-sleep transitions. In assessment of circadian rhythm parameters,
the most informative are the plots of changes in Pierson correlation coefficients of interpulse and R-R intervals. The 5-min ECG segments recorded at various phases of circahoralian rhythm are subjected to circahoralian variations
and can distort diagnostics.
Translated from Byulleten' Eksperimental'noi Biologii i Meditsiny, Vol. 146, No. 8, pp. 161–164, August, 2008 相似文献
12.
呼吸对心率变异性的影响 总被引:4,自引:0,他引:4
呼吸是心率变异性(HRV)的一个重要影响因素。简要介绍了呼吸影响HRV的机制,综述了呼吸频率、呼吸深度和呼吸模式对HRV各种参数的影响,以及病理条件下呼吸对HRV影响的变化。指出了用HRV判断心脏生理状态应考虑到呼吸的影响。 相似文献
13.
14.
目的通过检测轻度急性饮酒前后心率变异性指标,评价酒精对于自主神经活动影响的性别差异。方法20例健康志愿者.其中男性10例.年龄(20.1±0.6)岁:女性10例.年龄(19.6±0.8)岁。根据受试者体质量计算乙醇溶液饮用量(0.27g/kg)。分别在饮酒前和饮酒后15min、45min共3次记录心电信号.提取RR间期时间序列,计算心率变异性(HRV)评价指标,包括时域参数RR间期的平均值(RRI)、全部RR间期标准差(SDRR)、全程相邻RR间期之差的均方根(RMSSD)、相邻RR间期之差大于50ms的心搏动数占总心搏动数的比例(pNN50),频域参数低频频段(LF)、高频频段(HF)和LF/HF,Poincar6图参数宽度(短轴,SDl)、长度(长轴,SD2)和SDl/SD2。结果时域参数、频域参数和P0inca诺参数存急性饮酒后降低。对于RMSSD和SDRR参数.仅在女性受试者中观测到饮酒45min后降低.差异均具有统计学意义(P〈0.05)。对于LF和SDl参数在饮酒后15min即观测到在男性受试者中降低,差异均具有统计学意义(P〈0.05)。结论少量的急性饮酒降低了心率变异程度.酒精抑制了副交感神经的活动度.女性比男性对酒精的敏感程度更高.男性自主种经活动对酒精影响的响应时间更短。 相似文献
15.
心率变异性作为判断心脏自主神经状态的无创性指标常被用来定量分析自主神经的功能状态,其时域及频域降低与恶性心律失常、心功能不全,乃至心源性猝死等有关。人类的岛叶是控制交感神经和副交感神经介导的心血管调节最重要的皮层区域,多数学者在临床实践中发现,岛叶病变会导致心血管功能紊乱及自主神经功能状态的变化,因此岛叶与心脏功能之间的联系倍受重视。 相似文献
16.
The present study examined the effects of caffeine, as typically ingested through coffee, on ambulatory systolic and diastolic blood pressure (BP), heart rate, and mood. Normotensive coffee drinkers wore a BP monitor for two 24-hr periods, consuming decaffeinated coffee. Each cup was supplemented with 125 mg caffeine or cornstarch. Systolic and diastolic BPs were elevated on the day caffeine was consumed (maximum, 3.6 and 5.6 mm Hg, respectively), most notably shortly after ingestion. Heart rate was higher overnight following caffeine consumption. Negative Affectivity was also increased by caffeine, but Positive Affectivity and tiredness were unaffected. 相似文献
17.
18.
Cortical thickness,resting state heart rate,and heart rate variability in female adolescents
下载免费PDF全文
![点击此处可从《Psychophysiology》网站下载免费的PDF全文](/ch/ext_images/free.gif)
Julian Koenig Peter Parzer Corinna Reichl Ayaka Ando Julian F. Thayer Romuald Brunner Michael Kaess 《Psychophysiology》2018,55(5)
Resting state heart rate variability (HRV) is a psychophysiological marker that has gained increasing research interest, in particular in developmental neuroscience. HRV has been shown to be associated with mental and physical health, beyond simple measures of heart rate (HR) and shows inter‐ and intraindividual variance across aging. Recently, three studies reported on a positive correlation between resting state HRV and cortical thickness in selected regions of interest (ROIs) in adult samples. Structural thickness, HRV, and HR change during the sensitive period of adolescence. Previously, no study has addressed the structural concomitants of resting HR and HRV in adolescents. Cortical thickness (3‐T MRI), HR, and HRV were recorded in 20 healthy, female adolescents (mean age: 15.92 years; SD = 1.06; range: 14–17). In line with existing research in adults, cortical thickness in a number of ROIs was associated with resting state HRV but not HR. The comparison of regression analyses using the Bayes factor revealed evidence for a correlation between HRV and cortical thickness of the bilateral rostral anterior cingulate cortex. However, unlike in adults, greater cortical thickness was associated with reduced HRV in female adolescents. Analyses on HR showed no superior model fit. Results suggest that greater HRV might be beneficial for cortical development during adolescence (cortical thinning). On the other hand, cortical development might determine changes in autonomic nervous system function in adolescents. Future studies are needed to replicate these findings in larger samples including boys and to test these hypotheses in longitudinal designs. 相似文献
19.
Sleep apnea in heart failure increases heart rate variability and sympathetic dominance 总被引:1,自引:0,他引:1
AIMS: Sleep disordered breathing (SDB) is common in heart failure and ventilation is known to influence heart rate. Our aims were to assess the influence of SDB on heart rate variability (HRV) and to determine whether central sleep apnea (CSA) and obstructive sleep apnea (OSA) produced different patterns of HRV. METHODS AND RESULTS: Overnight polysomnography was performed in 21 patients with heart failure and SDB. Two 10-minute segments each of SDB and stable breathing from each patient were visually identified and ECG signal exported for HRV analysis. SDB increased total power (TP) with very low frequency (VLF) power accounting for the greatest increase (1.89+/-0.54 vs 2.96+/-0.46 ms2, P <0.001); LF/HF ratio increased during SDB (1.2+/-1.0 vs 2.7+/-2.1, P <0.001). Compared to OSA, CSA was associated with lower absolute LF (2.10+/-0.47 vs 2.52+/-0.55 ms2, P = 0.049) and HF power (1.69+/-0.41 vs 2.34+/-0.58 ms2, P = 0.004), increased VLF% (78.9%+/-13.4% vs 60.9%+/-19.2%, P = 0.008), decreased HF% (6.9%+/-7.8% vs 16.0%+/-11.7%, P = 0.046) with a trend to higher LF/HF ratio. CONCLUSIONS: SDB increases HRV in the setting of increased sympathetic dominance. HRV in CSA and OSA have unique HRV patterns which are likely to reflect the different pathophysiological mechanisms involved. 相似文献
20.
目的探讨心率变异性生物反馈(HRV BF)干预高血压前期患者心血管应激反应的效果。方法比较高血压前期者与正常血压者应激反应上血压变化的差异;将36位高血压前期受试者随机分为心率变异性生物反馈(HRV BF)组、慢腹式呼吸(SAB)组和空白对照组。干预组受试者接受每次30 min,每周2次,持续2个月共15次的训练;空白对照组不予干预。实验间隔期嘱受试者每天进行不少于20 min的家庭训练。所有受试者在实验开始前及实验结束后均接受连续2 d同一时间点进行的两种应激测试。结果高血压前期者较正常血压者在应对心算应激时收缩压和舒张压反应性更大。干预2个月后,HRV BF和SAB组收缩压和舒张压平均降压幅度分别为15.4/10.2 mmHg、11.6/7.4 mmHg;空白对照组干预后血压无明显变化。HRV BF能显著降低受试者心算应激时收缩压反应性,较SAB更为明显。结论高血压前期者应激反应性较高,HRV BF训练可以降低受试者应对心理应激时心血管的反应,并降低高血压前期者血压。 相似文献