79例性病流行病学分析

一、资料来源和方法 1.全部性病疫情资料来源于我县皮肤病性病防治门诊。 2.报告病种为1986年卫生部制定的性病监测试行方案规定的8种疾病。 3.诊断标准参照1991年卫生部制定的《性病诊断标准与治疗方案》。二、结果 1.患者来源:本文收集到的病例为1999年在皮肤病性病防治门诊就诊的所有性病患者,病人均为外     

喜闻《性病防治手册》出版发行

欣喜卫生部卫生防痊司编的《性病防治手册》出版了,这是性病防治工作中的一件大事.     

1994～2003年泰安市性病流行病学分析

我们对泰安市1994～2003年的STD疫情进行回顾性分析，现将结果总结如下：资料来源病例资料来源于各级性病防治机构及综合性医院预防保健科报米的疫情报告卡。病种为《中华人民共和国传染病防治法》和《性病防治管理办法》规定的病种：结果流行趋势：1994～2003年全市累计发现各种性病患者15256例，性病疫情呈增长态势，前9年年均增长51．28％，其中淋病年均增长42．25％，非淋菌性尿道炎增长159．33％，尖锐湿疣增长41．89％。这三种病增长幅度最大，梅毒、生殖器疱疹增长比较平稳。全市性病总数自2002年开始出现负增长(表1)。     

甘肃省性病监测中心门诊性病1223例分析

我中心性病门诊依据卫生部防疫司编《性病防治手册》的诊断标准和治疗方案，１９９１～１９９５年诊治性病１２２３例，现统计分析如下：１、资料来源：省性病中心门诊性病疫情报告。１２２３例患者的流行病学特点见表１—８。表１甘肃省性病监测中心门诊１９９１—１９９...     

α-2b干扰素加电灼治疗尖锐湿疣的疗效观察

我科采用α 2ｂ干扰素皮损内注射加ＧＸ -ＩＩＩ型多功能治疗仪电灼治疗尖锐湿疣 ,有效地提高了一次性治愈率 ,降低了复发 ,现报告如下。临床资料　本组 80例均符合卫生部防疫司编《性病防治手册》规定的诊断标准 ,排除以下情况 :(1)对α 2ｂ干扰素过敏者及不适宜使用的患者 ;(     

百色市近20年性病流行特点分析

百色市近20年来性病的发病人数急剧增长。为了解其流行趋势和特点,为有关部门制定更有效的防治措施提供依据,我们将1985年1月1日~2004年12月31日百色市各医疗保健机构(性病监测点)报告的性病患者的流行病学资料进行整理。现分析如下:1资料来源和方法1.1资料来源资料来自百色市12个县(区)各性病监测单位1985年至2004年性病疫情季、年报表。病种为卫生部规定为重点防治的7种(艾滋病未包含在内)性病。诊断标准按卫生部《性病诊断与治疗方案》为依据。1.2方法对20年来百色市的性病疫情不同病种的发病情况、构成情况以及流行病学特点进行统计分析…     

甘肃省2000年性病流行病学分析

1　资料来源病例资料来源于 2 0 0 0年甘肃省 14个地 (市、自治州 )、甘肃矿区卫生防疫站上报季度及年度报表。人口资料来自甘肃省统计局。报告性病病种由《中华人民共和国传染病防治法》及卫生部《性病防治管理办法》规定监测报告的 8种性病。2　 2 0 0 0年甘肃省性病流行情况     

云南省文山州2000-2009年性病流行病学分析

为了解文山州性病流行状况和趋势,并为制定防治措施提供依据,现将2000-2009年全州性病流行状况报道如下.资料和方法 病种为卫生部规定上报的8种性病;对象为2000- 2009年州皮肤病防治所和所辖8县皮肤病性病门诊确诊的各种性病患者.执行卫生部制定的《性病诊断标准与治疗》.     

寿宁县1992～1998年STD流行病学监测分析

寿宁县地处闽东山区 ,自 1985年发现首例淋病以来 ,疫情呈逐年急剧上升趋势 ,严重危害人们的身心健康。为观察性传播疾病 (ＳＴＤ)的流行动态 ,我们于 1992～ 1998年根据《性病防治管理办法》确定的病种开展了ＳＴＤ流行病学监测。现将监测结果分析如下 :1　监测内容与方法1 1　疫情报告　成立县性病防治领导小组 ,设立县性病防治中心 ,建立健全三级ＳＴＤ疫报网 ,要求县辖区内所有医疗机构的临床医生发现ＳＴＤ患者 ,应做好登记 ,填写传染病报告卡和ＳＴＤ个案调查表 ,并及时报县性病防治中心。1 2　重点人群监测　在县辖区内确定以下重点…     

海南省澄迈县一九九五年性病防治情况

1995年澄迈县皮防所性病专科门诊报告性传播疾病(STD)389例。 资料来源 我所性病专科门诊发现病例,其诊断标准按卫生部防疫司颁布的《性病诊断标准与治疗方案》。 结果 (一)流行情况 1995年共报告STD389例。年发病率为86.4/10万,比1994年307例,增加26.71％。 (二)病种构成 报告STD四种,淋病245例(其中儿童淋病3例)占62.98％;梅毒47例(其中儿童梅毒1例,隐性早期梅毒1     

Dermatology in private practice in France in 2000

OBJECTIVES: Data regarding French dermatological practice are scarce. Our objective was to identify the skin disorders most commonly diagnosed by office-based dermatologists. We also documented the severity of these skin disorders, as reflected by the repercussions on patient's everyday life, and the way physicians managed patients. DESIGN: We carried out a one-day survey of visits to a randomly selected sample of 900 French office-based dermatologists. The randomization was stratified according to the five French different dialing area codes. RESULTS: Office-based dermatologists saw 6411 patients with 7839 skin disorders during the survey. The daily number of visits to French dermatologists was estimated at 47 000 and the annual number between 12 and 14 millions. Office-based dermatologists mostly managed warts, acne, nevus, dermatitis, malignancies and pre-malignancies, fungal infection and psoriasis. Repercussions on patients'everyday life were assessed by physicians as important or very important in 28 p. 100 of cases. Half of the patients received topical treatment, 20.5 p. 100 a systemic drug and 40 p. 100 a minor surgical procedure (including cryotherapy). CONCLUSION: Although dermatologists frequently see benign skin disorders such as warts or nevus, more severe diseases represent an important part of their activity.     

Pentoxifylline in reactions in leprosy

A study was conducted to assess the response of reactions in leprosy to pentoxifylline therapy. Ten cases were studied; 8 cases had type 2 reaction and 2 cases had type 1 reaction. Pentoxifylline was given orally 400 mg three times daily. In patients with type 2 reaction, good response was observed within one week. There was near complete regression of ENL lesions within one month. Cases with type 1 reaction did not respond to pentoxifylline.     

Leukocyte migration in vivo and in vitro in patients with psoriasis

Leukocyte migration in vivo was studied with a skin chamber technique in 21 patients with active psoriasis vulgaris and 18 with cleared psoriasis vulgaris. Measuring over 24 h, no difference was found between healthy volunteers and most patients with active psoriasis, although a subgroup of patients with long-lasting relapses showed subnormal migration values. In patients with cleared psoriasis on the other hand the in vivo leukocyte migration values were increased. In addition, leukocyte migration in vitro under agarose was studied, but no difference was found between healthy controls and patients with psoriasis, active or cleared.     

Distribution of fodrin in the keratinocyte in vivo and in vitro

Distribution of fodrin in the keratinocyte, both in vivo and in vitro, was examined by immunofluorescence microscopy. In the rat epidermis in vivo, fodrin was localized in the cell periphery of the spinous layer of all the skins studied. In only the basal layer of the thick skin, however, fodrin was seen intensely in the cytoplasm. As in vitro keratinocytes, a mouse cell line (Pam 212) cultured in low (0.06 mM) as well as standard (1.87 mM) Ca2+ was examined. In low Ca2+, fodrin was observed throughout the cytoplasm without marked accumulation irrespective of the cell density. The cytoplasmic labeling in low Ca2+ looked filamentous and became aggregated when cells were treated with cytochalasin B; at least some of the aggregates coexisted with those of F-actin. In contrast, fodrin distribution was not affected with colchicine. On the other hand, in standard Ca2+, the protein became concentrated along the cell periphery and less conspicuous in the cytoplasm as the cells reached confluency. When cells were transferred from low to standard Ca2+, the distribution of fodrin changed accordingly within 180 min. The present results indicate that fodrin in the keratinocyte is likely to be associated with actin filaments and that it takes two different ways of distribution both in vivo and in vitro. The peripheral and the cytoplasmic labeling of in vivo and in vitro cells are likely to correspond. It may be that fodrin changes its localization according to the cell's proliferative activity.