Brugada法和Griffith法在宽QRS心动过速诊断中的价值

目的 :探讨 Brugada法和 Griffith法在宽 QRS心动过速 (WRT)鉴别诊断中的价值。方法 :对 61例 WRT〔其中室性心动过速 (VT) 34例 ,室上性心动过速 (SVT) 2 7例〕心电图采用Brugada法及 Griffith法进行回顾性分析。结果 :Brugada法诊断 VT的敏感性、特异性、准确性分别为 91 .2 %、85.2 %、88.5% ,略高于 Griffith法的 82 .4%、77.8%、80 .3%。结论 :Brugada法和Griffith法对器质性心脏病所致的 VT及 SVT伴室内差异传导或原有单侧束支传导阻滞者诊断的符合率高 ,但对左束支阻滞型特发性 VT、SVT伴双分支阻滞 (右束支阻滞加左前分支阻滞 )及预激综合征伴旁路前传型 SVT诊断符合率低 ,易误诊     

Brugada法联合Steurer法在宽QRS波心动过速鉴别诊断中的价值

为评价Brugada法联合Steurer法在宽QRS波心动过速 (WRT)鉴别诊断中的应用价值及存在的缺陷。对 1 0 1例WRT[室性心动过速 (VT) 5 8例 ,室上性心动过速 (SVT) 43例 ]进行分析。结果 :Brugada法诊断VT灵敏度、特异度、准确性分别为 85 .7%、89.5 %、87.1 % ;联合Steurer法后灵敏度、特异度、准确性分别升至 91 .5 %、90 .5 %、91 .1 %。进一步分析显示 :Brugada法对器质性原因所致VT、右束支阻滞型 (RBBB)特发性VT(IVT)、SVT伴室内差异性传导 (AC)或原有单侧束支阻滞 (BBB)者诊断符合率高 ( 95 .8%～ 1 0 0 .0 % ) ;对左束支阻滞型 (LBBB)特发性VT、SVT伴原有双支阻滞、心肌坏死或心肌梗死伴宽QRS波SVT及预激综合征伴旁道前传型SVT(WPW SVT)诊断的符合率低 ( 0～ 5 0 .0 % )。联合Steurer法可使WPW伴旁道前传型SVT得以明确诊断 ,但对前三者无鉴别意义 ,故不适合在前三者中应用。结论 :Brugada法联合Steurer法能提高WPW伴旁道前传型SVT的鉴别能力 ,是目前鉴别WRT的重要方法。     

形态心电图特征在宽QRS波心动过速鉴别诊断中的价值

目的探讨形态心电图特征在宽QRS波心动过速（WCT）鉴别诊断中的价值。方法对广西医科大学第一附属医院2005年1月-2008年6月住院并经心内电生理检查确诊WCT 96例患者的心电图,分别应用Brugada标准及形态心电图特征进行回顾分析。结果 Brugada标准诊断室性心动过速（VT）的敏感性高（92.4%）,但特异性较低（53.3%）;而形态心电图特征可应用于WCT的鉴别诊断,30例WCT心电图QRS波形态符合典型束支传导阻滞图形,诊断为室上性心动过速（SVT）伴室内差异性传导或束支传导阻滞;66例WCT心电图QRS波形态不符合典型的束支传导阻滞表现,诊断为VT。结论形态心电图特征在WCT鉴别诊断中的应用价值更高、更简便。     

有关室性心动过速诊断标准的价值评估

目的 评价Brugada标准、Steurer标准、Verecke标准以及这3种标准中各个单项标准对诊断室性心动过速(VT)的价值及其存在的缺陷.方法 对120例VT者发作时常规12导联同步心电图进行分析.计算以上各种标准对诊断VT的敏感性以及上述3种标准对各种器质性心脏病所致的VT及无器质性心脏病的特发性VT(IVT)的敏感性.结果 3种标准中的各个单项标准对诊断VT的敏感性较低,均小于60%;而多项组合上述3种标准对诊断VT的敏感性明显提高,依次分别为:96.7%、74.2%及86.7%.另外,3种标准对器质性心脏病所致VT的敏感性较高,分别为99.1%、81.3%及96.7%,而Brugada标准对右束支传导阻滞(RBBB)型的IVT的敏感性较高(100%),对左束支传导阻滞(LBBB)型的IVT的敏感性较低(40%),而Steurer标准及Verecke标准对IVT的敏感性均较低(0%～12.5%).结论 心电图单项标准对诊断VT敏感性较低,不能依据心电图某单一标准来诊断VT.多项组合的Brugada标准是诊断VT最敏感的方法,但对LBBB型的IVT敏感性较低.诊断时应密切结合临床资料以及其它诊断标准综合考虑.     

Griffith法和Brugada法诊断宽QRS性室上性心动过速

为了解Griffith法和Brugada法诊断宽QRS性室上性心动过速(SVT)的价值,选择34例心电图表现为宽QRS心动过速者,其中SVT25例,室性心动过速(VT)9例,均经心脏电生理检查证实,采用上述两法进行了比较和综合分析.结果发现,Griffith法诊断SVT的敏感性、特异性和假阴性率分别为76%、77.8%和24%;而Brugada法的则分别为80%、88.9%和20%;两者合用时分别为84%、88.9%和16%.4例SVT为右侧旁路前传者均不符合诊断标准.认为,Griffith法和Brugada法或二者合用对SVT合并原有束支阻滞或室内差异性传导者有较高的诊断价值,而对预激旁路前传的SVT诊断价值低.     

室性心动过速的诊断与鉴别诊断

室性心动过速 (VT)是一种严重的心律失常 ,与并发室内差传的室上速 (SVT)同为宽大畸形 QRS心动过速 ,发作时心率极快 ,均可反复发作。本文作者报道两者的心电图表现特点。1　材料与方法　从我院 1995 - 0 1～ 1999- 0 8门诊与住院患者的 94例 (VT)与 80例 (SVT)心电图 (ECG) ,观测两组心动过速发作时、发作前后的 QRS波形态、宽度、心电轴有无偏移及有无房室分离等。2　结果　 VT组男 5 4例 ,女 40例 ,阵发性 VT 5 2例 ,非阵发性 VT 16例 ,特发性 VT 18例 ,双向尖扭转型 8例 ,QRS呈右束支传导阻滞 (RBBB) 2 8例 ,呈左束支传导…     

从完全性左束支传导阻滞型心动过速中检出室性心动过速的心电图标准

QRS波增宽型心动过速中,鉴别室上性心动过速(SVT)和室性心动过速(VT)甚为困难。然而诊断错误导致延误治疗,甚至危及生命。完全性左束支传导阻滞(CLBBB)型心动过速的鉴别诊断的资料更少,本文对鉴别SVT和VT的4项心电图标准进行评价。作者回顾分析118例CLBBB型心动过速患者的12导联心电图,各例V_1导联均以S波为主且QRS波时限>120ms。5例颈动脉窦按压后体表心电图表明为心房颤动(1例)或窦性心动过速(4例),其余     

Griffith法Brugada法诊断宽QRS性室上性心动过速

为了解Griffith法和Brugada法诊断宽QRS性室上性心动过速（SVT）的价值，选择34例心电图表现为宽QRS心动过速者，其中SVT25例，室性心动过速（VT）9例，均经心脏电生理检查证实，采用上述两法进行了比较和综合分析。结果发现，Griffith法诊断SVT的敏感性、特异性和假阴性率分别为76％、77．8％和24％；而Brugada法的则分别为80％、88．9％和20％；两者合用时分别为84％、88．9％和16％。4例SVT为右侧旁路前传者均不符合诊断标准。认为，Griffith法和Brusada法或二者合用对SVT合并原有束支阻滞或室内差异性传导者有较高的诊断价值，而对预激旁路前传的SVT诊断价值低。     

鉴别宽QRS心动过速心电图指标对原有束支传导阻滞患者的应用价值

目的　探讨鉴别宽 QRS心动过速心电图指标对原有束支传导阻滞患者的应用价值。方法　以非选择性、连续性 42 0例完全性束支传导阻滞窦性心律患者为研究对象 ,分析以往文献报道鉴别宽 QRS心动过速心电图标准中的 QRS形态指标的特异性。结果　 12个分析指标中 4个指标特异性 >90 % :右束支传导阻滞 V1 呈三相型 (Rsr′,r SR′,RSR′) ;右束支传导阻滞 V6呈 QS或 QR型 ;左束支传导阻滞 V6有 q或 Q波 ;V1 ～ V6无 RS波。其它 8个指标特异性在 45 %～ 87%范围内。结论　有利于室性心动过速诊断心电图标准中的 QRS形态指标对鉴别原有束支传导阻滞的室上性心动过速患者存在局限性     

66例室性心动过速心电图分析

室性心动过速（VT）与室上性心动过速（SVT）伴室内差异性传导或束支传导阻滞的鉴别是体表心电图讨论的热点。有作者研究的Wellens方法和Brugada方案等一直具有很大的实用性。我们参考了这些经典方法，结合自己的经验，对66例确诊为VT患者的12导联心电图进行回顾性分析。     

Regelm??ige Tachykardien mit breitem Kammerkomplex

Differential diagnosis of regular tachycardia with broad QRS complex can be challenging in daily practice. There are four different arrhythmias that have to be taken into account when being confronted with a broad QRS complex tachycardia: (1) ventricular tachycardia (VT); (2) supraventricular tachycardia (SVT) with bundle branch block (BBB); (3) SVT with AV conduction over an accessory AV pathway; (4) paced ventricular rhythm. Due to potentially fatal consequences, the correct diagnosis is important in view of both the acute treatment and the long-term therapy. Since SVT with accessory conduction is rare and a paced ventricular rhythm can be identified easily by stimulation artifacts, in most cases, a VT has to be differentiated from an SVT with BBB. Several ECG criteria can be helpful: (1) QRS complex duration > 140 ms in right BBB tachycardia or > 160 ms in left BBB tachycardia; (2) ventricular fusion beats; (3)“Northwest” QRS axis; (4) ventriculoatrial dissociation; (5) absence of an RS complex or RS interval > 100 ms in leads V₁-V₆; (6) a positive or negative concordant R wave progression pattern in leads V₁-V₆; (7) absence of an initial R wave or an S wave in lead V₁ in right BBB tachycardia; (8) absence of an R wave or an R/S ratio < 1 in lead V₆ in right BBB tachycardia; (9) absence or delay of the initial negative forces in lead V₁ in left BBB pattern (R wave duration > 30 ms in V₁; interval between onset of R wave and Nadir of S wave > 60 ms in V₁); (10) presence of Q wave. Any of these variables favor VT. However, none of the criteria has both a sufficient sensitivity and specificity when utilized on its own. Therefore, various diagnostic algorithms have been proposed using a number of the above criteria consecutively. By doing so, the specificity and sensitivity of correctly identifying a VT or an SVT with BBB can be raised to > 95%.     

Application of a new algorithm in the differential diagnosis of wide QRS complex tachycardia.

AIMS: The Brugada criteria proposed to distinguish between regular, monomorphic wide QRS complex tachycardias (WCT) caused by supraventricular (SVT) and ventricular tachycardia (VT) have been reported to have a better sensitivity and specificity than the traditional criteria. By incorporating two new criteria, a new, simplified algorithm was devised and compared with the Brugada criteria. METHODS AND RESULTS: A total of 453 WCTs (331 VTs, 105 SVTs, 17 pre-excited tachycardias) from 287 consecutive patients with a proven electrophysiological (EP) diagnosis were prospectively analysed by two of the authors blinded to the EP diagnosis. The following criteria were analysed: (i) presence of AV dissociation; (ii) presence of an initial R wave in lead aVR; (iii) whether the morphology of the WCT correspond to bundle branch or fascicular block; (iv) estimation of initial (v(i)) and terminal (v(t)) ventricular activation velocity ratio (v(i)/v(t)) by measuring the voltage change on the ECG tracing during the initial 40 ms (v(i)) and the terminal 40 ms (v(t)) of the same bi- or multiphasic QRS complex. A v(i)/v(t) >1 was suggestive of SVT and a v(i)/v(t) 相似文献   

Wide complex tachycardias. Differential diagnosis and management.

The majority of wide complex tachycardias are secondary to VT. The differential diagnosis of wide complex tachycardia also includes SVT with aberrancy or underlying bundle branch block and antegrade SVT conduction over an accessory pathway (antidromic SVT). VT is usually the result of reentry and most commonly arises in an area of diseased myocardium in the setting of previous myocardial infarction or cardiomyopathy. VT, however, can also occur in patients with structurally normal hearts. Criteria useful in diagnosis of wide complex tachycardia include clinical criteria (presence of structural heart disease or a history of previous myocardial infarction) and electrocardiographic criteria (the presence of capture or fusion beats, relation of atrial or ventricular activity, QRS duration and axis, and morphology). The acute management of wide complex tachycardia includes cardioversion and intravenous pharmacologic therapy. Almost all patients with VT require chronic therapy, although in rare patients treatment of acute precipitating factors may be sufficient. While pharmacologic therapy has been the mainstay of treatment for these patients, there have been many exciting advances using surgical, device, and ablative therapies.     

RBBB tachycardia with north-west axis. What is the mechanism?

RBBB tachycardia with NW axis is considered to be VT unless proved otherwise. However underlying conduction system disease can produce electrocardiographic patterns suggestive of bundle branch block with extreme left axis which can cause difficulty in differentiating VT from SVT as in this case.     

aVR 导联在心动过速鉴别诊断中的应用

在传统的心电图诊断中 aVR 导联一直没有受到足够的重视，但是近几年的研究显示aVR 导联在诸多方面都发挥着重要作用，尤其是在鉴别室性心动过速和室上性心动过速方面有着重要的作用，本文对此加以简述。     

室性心动过速合并室上性心动过速的射频消融

目的报道7例室性心动过速（VT）合并室上性心动过速（sVT）的射频消融。方法7例患者男6例,女1例,平均年龄（21±9）岁。阵发性心动过速病史（3．7±2．0）年。术中心房和心室刺激诱发VT和SVT,并进行消融。结果7例患者心房或心室刺激能反复诱发和终止VT合并SVT。法洛四联症矫治术后右心室VT合并三尖瓣环峡部依赖性心房扑动（AFL）1例,其余6例均为维拉帕米敏感性左心室特发性室速（ILVT）,分别合并AFL1例,左后间隔旁路参与的顺向型房室折返性心动过速（AVRT）1例,冠状静脉窦口慢旁路参与的顺向型AVRT1例,慢慢型房室结折返性心动过速（AVNRT）1例,左侧游离壁旁路参与的顺向型AVRT2例。7例患者的两种心动过速均成功消融,所有患者消融术后随访2年,无一例VT或SVT复发。结论VT合并SVT并不少见,消融术中应放置必需的心腔内电极导管,完成详细电生理检查,避免漏诊。一次消融应根除两种疾病。     

Performance of a dual-chamber implantable defibrillator algorithm for discrimination of ventricular from supraventricular tachycardia.

BACKGROUND: Inappropriate therapies remain a major problem in patients with implantable cardioverter defibrillators (ICDs). Decreasing the proportion of inappropriate therapies is a major objective. With the addition of atrial detection and advanced algorithms, dual-chamber ICDs are designed to offer better discrimination of ventricular (VT) and supraventricular (SVT) arrhythmias. The present multicentre, open study aimed to evaluate the performance of a dual-chamber detection algorithm, the Atrial View algorithm, incorporated in a dual-chamber ICD, the Ventak AV (Guidant Inc., St. Paul, Minnesota, USA). METHODS AND RESULTS: Fifty-one patients (45 males, 62+/-11 years, ejection fraction 42+/-15%) with standard indications received a Ventak AV ICD which analyzes, within the VT zone RR stability, tachycardia onset, atrial rate and AV relationship. Predischarge enhanced-detection algorithms were prospectively programmed: stability 24 ms, onset 9%, atrial fibrillation threshold 200 beats/min, and Vrate>Arate. An additional sustained rate duration criterion was programmed at least at 30 s. ICDs were interrogated every 3 months or when patients received shocks. A blinded review of electrograms for arrhythmia diagnosis and appropriateness of therapy was performed by 2 experts. Over the follow-up period (12+/-3.6 months), a total of 400 tachycardia episodes was recorded within the VT zone. After the review of stored electrograms, 237 (59%) true positive, 143 (36%) true negative, 17 (4%) false positive and 3 (1%) false negative episodes were diagnosed. Considering the 3 VTs incorrectly detected by the detection algorithms, therapy was delivered in 2 cases after sustained rate duration and 1 VT reverted spontaneously. Inappropriate therapy occurred in 17 cases. All but 1 were related to SVT with 1:1 atrioventricular relationship. Finally, on a per episode basis, the detection algorithm sensitivity was 99% and specificity was 89%. CONCLUSIONS: Programming of detection criteria based on stability, onset, atrial fibrillation rate threshold and Vrate>Arate allows a 99% sensitivity and an 89% specificity in Guidant ICDs. Discrimination of SVT with 1:1 atrioventricular relationship, however, remains a challenge for which new algorithms have to be designed.     

Electrocardiographic differentiation of typical atrioventricular node reentrant tachycardia from atrioventricular reciprocating tachycardia mediated by concealed accessory pathway in children

The value of the electrocardiogram (ECG) in children with supraventricular tachycardia (SVT) is unclear. The noninvasive differentiation of typical atrioventricular node reentrant tachycardia (AVNRT) and atrioventricular reciprocating tachycardia (AVRT) mediated by concealed accessory pathway conduction is clinically important, as it helps in counseling and potentially facilitates ablation procedures. One hundred forty-eight ECGs showing narrow QRS complex SVT were obtained from children before successful radiofrequency catheter ablation. An initial 102 ECGs were analyzed by 3 blinded observers to assess the utility of various electrocardiographic findings. No electrocardiographic criteria were found to discriminate between SVT mechanisms on 1- to 3-channel Holter/event recorder tracings (n = 32); their interpretation mainly (55%) resulted in an incorrect SVT diagnosis. On 12-lead ECGs (n = 70), the 2 arrhythmias were accurately diagnosed in 76% of patients; 5 findings were found to be discriminators of tachycardia mechanism. Predictors of AVRT were visible P waves in 74% of cases (sensitivity 92%; specificity 64%), RP intervals of > or =100 ms in 91% (sensitivity 84%; specificity 91%), and ST-segment depression of > or =2 mm in 73% of cases (sensitivity 52%; specificity 82%). Pseudo r' waves in lead V(1) and pseudo S waves in the inferior leads during tachycardia predicted AVNRT in 100% of cases (sensitivity 55% and 20%, respectively; specificity 100% for both). Based on these results, we developed a new diagnostic 12-lead electrocardiographic algorithm for pseudo r'/S waves, RP duration, and ST-segment depression during tachycardia. Two observers tested the algorithm in 46 (21 AVNRT; 25 AVRT) additional cases; they correctly diagnosed the SVT mechanism in 91% and 87%, respectively. Thus, the stepwise use of diagnostically relevant 12-lead electrocardiographic parameters helps to more accurately differentiate mechanisms of reentrant SVT.