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1.
洁治与派丽奥局部应用对种植体周围炎治疗的疗效比较   总被引:1,自引:0,他引:1  
目的:观察种植体周围洁治、派丽奥药物局部应用和二者联合治疗对种植体周围炎的临床治疗效果。方法:选择15个患种植体周围炎的病人,且每个病人至少有2个种植体,随机将这些病人分为洁治组、药物组、洁治 药物组,在基线期、治疗后4周、12周检查种植体周围组织的相关变化。结果:三种治疗方法均可使菌斑指数、龈沟出血指数和探诊深度等指标明显改善,但洁治 药物治疗的疗效持久。结论:种植体周围洁治与留置派丽奥药物治疗方法均具有明显的治疗作用,二者结合应用效果更好。  相似文献   

2.
洪兵  梅予锋 《口腔医学》2015,35(3):205-207
目的:观察局部洁治配合使用盐酸米诺环素或碘甘油对种植体周围炎的临床效果。方法 选择门诊60例种植体周围炎病人, 常规进行种植体龈上洁治、龈下刮治后, 随机分为盐酸米诺环素软膏组30例和2%碘甘油组30例, 分别于治疗前、治疗后的1、3、6周检查种植体周围菌斑指数(PLI)、出血指数(SBI)、探诊深度(PD)的指标。结果 使用盐酸米诺环素软膏和2%碘甘油治疗,可使菌斑指数、龈沟出血指数和探诊深度等指标明显改善,但使用盐酸米诺环素软膏治疗的效果更持久。结论 局部使用盐酸米诺环素软膏治疗种植体周围炎有明显的效果。  相似文献   

3.
透明质酸钠治疗种植体周粘膜炎的临床研究   总被引:2,自引:0,他引:2  
目的:评价透明质酸钠治疗种植体周粘膜炎的临床效果。方法:对28例种植体周粘膜炎患牙,分别行碳纤维头超声洁治及局部应用透明质酸钠治疗,定期复查,观察牙周各项指标变化。结果:应用碳纤维头超声洁治及透明质酸钠治疗后,种植体周牙龈出血指数及菌斑指数均有显著降低,牙周探诊深度呈下降趋势。结论:透明质酸钠对种植体周粘膜炎有良好的治疗作用。  相似文献   

4.
目的:探讨和评估盐酸米诺环素软膏在治疗种植体周围炎的临床疗效。方法:选择种植体周围炎患者20名(共23枚种植体),X线示种植体周围骨质吸收范围为种植体1/3~1/2。所有患者进行龈上洁治、龈下刮治,局部给予盐酸米诺环素软膏,一次/周。观察治疗前,治疗后第1~6周种植体周围的菌斑指数(PLI)、出血指数(SBI)和探诊深度(PD)等指标的变化。结果:除治疗后第2周,一枚种植体发生脱落外,6周后,其余种植体稳定性均良好,检测菌斑指数、探诊出血指数和探诊深度有明显改善,X线示未见明显继续骨质吸收。结论:局部应用盐酸米诺环素软膏对种植体周围炎有一定的治疗效果,远期效果应进一步观察。  相似文献   

5.
25例种植体周围黏膜炎治疗的临床研究   总被引:1,自引:0,他引:1  
目的:观察口腔卫生指导配合碳纤维工作头洁治治疗种植体周围黏膜炎的临床疗效。方法:25例种植体周围黏膜炎患者,用碳纤维工作头对种植体周围进行洁治,同时对患者进行口腔卫生指导以控制菌斑。记录前后种植体周围改良菌斑指数(mPLI)、改良龈沟出血指数(mSBI)、探诊深度(PD)等相关指标。结果:用碳纤维工作头洁治种植体周围和口腔卫生指导可使mPLI、mSBI、PD等指标明显改善。结论:口腔卫生指导和碳纤维工作头洁治术对种植体周围黏膜炎的临床治疗是有效的。  相似文献   

6.
超声洁治和局部用甲硝唑治疗轻、中度种植体周围炎   总被引:17,自引:0,他引:17  
目的:评价用碳纤维工作头的超声洁牙机和25%甲硝唑凝胶治疗种植体周围炎的疗效。方法:将27颗患轻中度种植体周围炎的种植牙随机分为洁牙机治疗组和甲硝唑治疗组,在基线、1、2、6和12周时检查种植体的临床和生物学指标。结果: 两种治疗方法均可使菌斑指数、龈沟出血指数、龋下微生物的酶活性检查等指标改善。治疗期间未发现任何不良反应。结论;碳纤维头超声洁治和局部用25%甲硝唑凝胶是治疗牙种植体周围炎的安全有效的方法。  相似文献   

7.
目的:分析不同表面形态种植体周围炎症的病变程度,探讨种植体表面形态与种植体周围炎病变程度的相关性。方法:分别将螺纹圆柱形种植体和非螺纹圆柱形种植体植入Beagle犬下颌,并建立犬种植体周围炎动物模型;3个月后测定各组种植体周探诊深度、观察种植体周围的骨缺损程度、组织切片分析其炎症程度。结果:临床检查见非螺纹圆柱形种植体周缺损深度大于螺纹圆柱形种植体组,差异具有统计学意义(P〈0.05);螺纹圆柱形种植体周探诊深度小于非螺纹圆柱形种植体周探诊深度,差异具有统计学意义(P〈0.05);骨组织形态学检查表明螺纹圆柱形组种植体颈部牙槽骨吸收不如非螺纹圆柱形种植体组明显。结论:种植体表面形态与实验性种植体周围炎病变程度存在一定相关性,非螺纹圆柱形种植体比螺纹圆柱形种植体的周围炎更明显。  相似文献   

8.
35例种植体周围炎临床治疗效果观察   总被引:5,自引:2,他引:3  
目的:针对种植体周围炎的致病相关因素采取系统治疗后,进行临床疗效评价。方法:35枚种植体根据临床检查指标确诊为种植体周围炎或种植体周围黏膜炎,采取对症治疗,局部以双氧水、生理盐水交替冲洗,龈袋内注入派力奥等方法,从病因上和症状上进行控制。记录用药前、用药后6周的菌斑指数,探诊出血指数,改良出血指数,探诊深度,牙龈乳头指数,并进行统计学分析。结果:探诊出血指数和改良出血指数有明显改变(P〈0.05),菌斑指数,探诊深度,牙龈乳头指数,无明显统计学改变(P〉0.05),种植体边缘骨吸收趋于稳定。结论:对症治疗对早期种植体周围炎有良好治疗效果。  相似文献   

9.
目的探讨比较局部使用盐酸米诺环素软膏和口服奥硝唑治疗种植体周围黏膜炎的临床疗效。方法选择种植体周围黏膜炎患者24例,对种植体进行龈上洁治、龈下刮治后,随机分为盐酸米诺环素软膏组和口服奥硝唑组,分别于基线、治疗后4周和治疗后8周行临床牙周指数检测。结果在治疗后4周和8周时,2组种植体周围改良菌斑指数、改良龈沟出血指数、探诊深度均明显下降,与基线时相比较,差异均有统计学意义(P〈0.05)。在治疗后4周和治疗后8周,盐酸米诺环素治疗组种植体周围改良菌斑指数、改良龈沟出血指数、探诊深度均低于口服奥硝唑组,且组间差异均有统计学意义(P〈0.05)。结论对于种植体周围黏膜炎,局部使用盐酸米诺环素软膏治疗或口服奥硝唑治疗均有效果;使用盐酸米诺环素软膏治疗更加有效。  相似文献   

10.
种植体周围炎是指发生于种植体周围软硬组织的炎症性损害,并伴有支持骨的丧失。种植体周围炎的危险因素包括菌斑、牙周病史、吸烟、糖尿病等。临床诊断标准包括探诊出血、种植体周袋内有溢脓、探诊深度≥5 mm以及影像学显示边缘骨丧失≥2 mm。种植体周围炎的治疗包括机械清创联合药物治疗的非手术治疗,以及切除性和再生性手术等手术治疗。种植体周维护治疗可以保护种植体周围组织的健康和提高种植成功率。因此,要重视种植体周的维护,预防种植体周围炎的发生。  相似文献   

11.
目的 :探讨半导体激光在种植体周围炎治疗中的作用,为临床应用提供实验依据。方法 :6只实验用比格犬共植入36枚种植体,建立种植体周围炎动物模型后测量种植体周临床指标作为基线数据。然后将36枚罹患种植体周围炎的种植体随机分为3组,A组采用常规刮治,B组采用常规刮治+盐酸米诺环素软膏(派丽奥),C组为常规刮治+盐酸米诺环素软膏(派丽奥)+半导体激光照射。治疗4周后通过大体观察、临床有效率和组织学切片进行组间比较。结果:建模时3组各项检查指标差异均无统计学意义(P>0.05)。治疗4周后C组的临床有效率最高,B组次之,A组最低(91.67%>75.00%>41.67%),其中C组PLI和SBI指标均优于B组[(0.25±0.45)vs(0.67±0.49),(0.33±0.49)vs(0.75±0.45),P<0.05]。组织学观察发现C组种植体颈部新生骨组织较多,垂直向骨缺损最小。结论 :半导体激光在种植体周围炎治疗中可有效减少牙龈出血和病原菌量并能促进新骨形成,可作为种植体周围炎治疗的辅助手段。  相似文献   

12.
目的建立即刻种植过程中种植体周围炎动物实验模型,利用Micro-CT观察种植体周围炎发展过程中的骨缺损特性。方法在12只哈尔滨长耳白兔双侧下颌第一前牙即刻植入种植体,4周后,采用丝线拴结法在左下颌第一前牙诱发种植体周围炎,右下颌第一前牙作为对照,通过Micro-CT观察不同时期种植体周围炎导致的骨缺损状况。结果丝线拴结法可以较好地诱发出即刻种植体周围炎,随着种植体周围炎的逐渐发展,种植体与骨之间无法达到良好结合,骨缺损逐渐产生并不断扩大,最后形成典型的“V”形骨缺损。结论建立的即刻种植体周围炎动物模型简便有效,可为种植体周围炎的相关研究提供重要参考。  相似文献   

13.
目的 探讨种植体周围炎发生的相关影响因素,为临床预防和控制提供依据。方法 采用病例对照研究收集相关资料,病例为2010年1月—2013年6月期间完成的种植修复后确诊种植体周围炎新发病例(A组)52例,对照为同期完成种植修复种植体周围健康者(B组)95例。采用SPSS16.0软件包中的非条件logistic回归分析,明确种植体周围炎发生密切相关的患者自身和手术修复相关因素。结果 单因素分析结果中,有意义的变量包括年龄、文化程度、牙周病、吸烟、糖尿病、定期洁牙并使用种植专门护理产品、上部结构修复方式(P<0.05)。多因素回归分析表明,吸烟、糖尿病、牙周疾病、种植部位是种植体周围炎的危险因素;定期洁牙是种植体周围炎的保护因素。结论 影响种植体周围炎发生的危险因素多为患者自身因素,建议严格掌握种植手术适应证,对高风险患者加强卫生宣教,术后注意随访,尽早发现、治疗种植体周围炎。  相似文献   

14.
目的:建立并评价FTA-DNA直接提取法在种植体周围炎和牙周炎细菌DNA扩增中的应用效果。方法:选取14例种植体周围炎患者和26例种植体周围炎患者,用传统法、煮沸法和FTA-DNA直接提取法分别提取DNA,同时进行16S-rDNA扩增,凝胶电泳检测,比较3种DNA提取方法的差异。结果:种植体周围炎和牙周炎之间差异无显著性,3种方法提取的DNA在PCR扩增后均有条带,但FTA-DNA直接提取法成功率高于试剂盒法和煮沸法,存在统计学差异;且FTA-DNA直接提取法在提取的步骤、时间、费用上有较高的优势。结论:FTA-DNA直接提取法可快速有效地从种植体周围炎和牙周炎临床标本中提取并保存细菌DNA,有望用于种植体周围炎和牙周炎的研究及临床诊断。  相似文献   

15.
目的 观察轻、中、重度种植体周围炎的重复非手术治疗效果。 方法 选择种植修复完成2年以上的种植体周围炎病例29例、植体31颗,按病情严重程度分为轻、中、重度三组,每组分别有种植体周围炎植体12、10、9颗,进行种植体周袋内超声清创+甘氨酸喷砂+盐酸米诺环素凝胶袋内注射+复方氯己定含漱联合治疗,共治疗两次。分别记录三组治疗前(T0)、治疗第1次后1个月(T1)、重复治疗第2次后1个月(T2)3个时间点的种植体周探诊深度(PD)、探诊出血 (BOP)位点阳性率、探诊溢脓位点阳性率,比较各个时间点的临床参数变化。结果 经过两次治疗,轻、中、重度组PD均值减少,轻度组T0 vs.T1,P>0.05,T0 vs.T2, P<0.05;中重度组T0 vs.T1,P<0.05,T0 vs.T2, P<0.01 。三组在治疗两次后种植体周探诊出血率均不断降低,轻、中度组T0 vs.T1, P<0.05,T0 vs.T2, P<0.01,重度组治疗前后三个时间点没统计学差异。溢脓位点阳性率随炎症程度加重而升高,轻度组没有溢脓位点,中度和重度组治疗后溢脓位点阳性率不断下降(T0 vs.T1,P<0.05, T0 vs.T2,P<0.01)。经治疗,中、重度组T1、T2时仍有种植体周探诊深袋、探诊出血点和溢脓点。结论 非手术方法治疗轻、中、重度种植体周围炎能显著改善其种植体周软组织炎症,重复进行非手术治疗是有必要的。轻度种植体周围炎经两次非手术治疗,能基本控制软组织炎症;中重度种植体周围炎经两次非手术治疗,不能完全控制软组织炎症。  相似文献   

16.
PURPOSE: Tissue reactions to 4 different implant surfaces were evaluated in regard to the development and progression of ligature-induced peri-implantitis. MATERIALS AND METHODS: In 6 male mongrel dogs, a total of 36 dental implants with different surfaces (9 titanium plasma-sprayed, 9 hydroxyapatite-coated, 9 acid-etched, and 9 commercially pure titanium) were placed 3 months after mandibular premolar extraction. After 3 months with optimal plaque control, abutment connection was performed. Forty-five days later, cotton ligatures were placed around the implants to induce peri-implantitis. At baseline and 20, 40, and 60 days after placement, the presence of plaque, peri-implant mucosal redness, bleeding on probing, probing depth, clinical attachment loss, mobility, vertical bone loss, and horizontal bone loss were assessed. RESULTS: The results did not show significant differences among the surfaces for any parameter during the study (P > .05). All surfaces were equally susceptible to ligature-induced peri-implantitis over time (P < .001). Correlation analysis revealed a statistically significant relationship between width of keratinized tissue and vertical bone loss (r2 = 0.81; P = .014) and between mobility and vertical bone loss (r2 = 0.66; P = .04), both for the titanium plasma-sprayed surface. DISCUSSION AND CONCLUSIONS: The present data suggest that all surfaces were equally susceptible to experimental peri-implantitis after a 60-day period.  相似文献   

17.
Objective: Not much information exists on post-treatment pain related to peri-implantitis. The purpose of this study was to evaluate intensity and quality of pain after non-surgical and surgical treatment of peri-implantitis.

Material and methods: A total of 30 patients with a diagnosis of peri-implantitis were included in the study. The patients registered pain using a VAS scale after non-surgical and surgical treatment of peri-implantitis. The data were registered for one week after each treatment. The patients also recorded quality of pain and if analgesics were taken. Factors included in the study were number of implants, severity of peri-implantitis (millimetre bone loss at most severely affected implant), implant localization, smoking and gender.

Results: Statistically significant difference in intensity of pain was found between day zero and day one for both non-surgical and surgical treatment of peri-implantitis (p?Conclusion: Levels of pain are found to be low to moderate for most patients after treatment of peri-implantitis. The pain was most pronounced on the first two days post-treatment. Throbbing/soreness and numbness were the most frequently reported quality of pain.  相似文献   

18.
目的 探讨种植体周围炎与健康种植体黏膜下微生物生态环境。方法 选取2017年9月—2019年1月于北京口腔医院治疗种植体周围炎、接受种植术并已随访1年以上的患者,按照是否发生种植体周围炎分为种植体周围炎组(n=43,51颗种植体)与健康组(n=59,63颗种植体)。检测2组患者探诊深度、骨吸收情况、探诊出血情况、改良菌斑指数(modified plaque index,mPLI)、改良出血指数(modified bleeding index,mSBI)。收集2组患者黏膜下菌斑,使用微生物基因提取试剂盒提取细菌DNA,经Illumina公司的MiSeq测序平台测序。采用SPSS 23.0软件包对数据进行统计学分析。结果 种植体周围炎组平均最大探诊深度显著大于健康组(P<0.05);mPLI 2级[56.86%(29/51)]显著高于健康组[0.00%(0/63)](P<0.05);mSBI 2级[60.78%(31/51)]显著高于健康组[0.00%(0/63)](P<0.05)。种植体周围炎组Simpson指数显著高于健康组,Shannon指数显著低于健康组(P<0.05)。种植体周围炎组优势菌属中占比最多的为新月形单胞菌属(16.35%)、依次为假单胞菌属(11.20%)、梭杆菌属(10.05%)、链球菌属(7.63%)、密螺旋体菌属(6.84%)。结论 与健康种植体相比,种植体周围炎黏膜下微生物具有高物种丰度、低物种均匀度,其优势菌属中的假单胞菌属、新月形单胞菌属、梭杆菌属、链球菌属、密螺旋体菌属占比较高。  相似文献   

19.
BACKGROUND: Progressive peri-implant bone losses, which are accompanied by inflammatory lesions in the soft tissues, are referred to as peri-implantitis. The aim of this study was to compare the effects of photodynamic therapy (PDT) and conventional technique on microbial reduction in ligature-induced peri-implantitis in dogs. METHODS: Eighteen third premolars from nine Labrador retriever dogs were extracted and the implants were submerged. After osseointegration, peri-implantitis was induced. After 4 months, ligature was removed and natural bacterial plaque was allowed to form for another 4 months. The animals were then randomly divided into two groups. In the conventional group, they were treated using mucoperiosteal flaps for scaling the implant surface and chlorexidine (conventional) irrigation. In the PDT group, only mucoperiosteal scaling was carried out before photodynamic therapy. Inside the peri-implant pocket, a paste-based azulene photosensitizer was placed and then a GaAlAs low-power laser (lambda=660 nm, P=40 mW, E=7.2 J for 3 minutes) was used. Microbiological samples were obtained before and immediately after treatment. Before treatment, one implant was removed and analyzed by scanning electron microscopy to validate the contamination. RESULTS: The results of this study showed that Prevotella sp., Fusobacterium sp., and S. Beta-haemolyticus were significantly reduced for both groups. After treatment, no significant differences were observed between the groups. CONCLUSION: These findings suggest that photodynamic therapy is a non-invasive method that could be used to reduce microorganisms in peri-implantitis.  相似文献   

20.
Objectives: Some cases of retrograde peri-implantitis arise from adjacent natural teeth that have peri-radicular infection. The present study was designed to investigate the incidence of retrograde peri-implantitis from adjacent teeth with endodontic treatment.
Materials and methods: One hundred and twenty-eight patients of ages ranging from of 24–61 years were recruited for this study. A total of 128 ITI SLA implants with adjacent teeth that had received endodontic treatment at least 1 week before were placed in 128 patients. The date of endodontic therapy and the pulp status of the adjacent tooth before endodontic therapy were recorded. The distance between the implant and the adjacent tooth was determined using a radiograph. The stability of all implants was tested by OSSTELL and recorded at implant placement, after 4 and 12 weeks.
Results: The incidence of retrograde peri-implantitis was 7.8%. The duration from endodontic-treated adjacent teeth to implant placement was 12.15±10.1 weeks, and the distance between the implant and the adjacent teeth was 2.99±1.4 mm. Distance and time were found to be related to retrograde peri-implantitis ( P <0.05). The stability of implants with retrograde peri-implantitis was less than that of the normal implants, but the difference was not significant ( P >0.05).
Conclusions: The incidence of retrograde peri-implantitis may reduce by increasing the distance between the implant and adjacent tooth, and/or the duration from endodontically treated adjacent tooth-to-implant placement. Although preliminary, these data might orient the practitioner to avoid retrograde peri-implantitis.  相似文献   

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