Arthroscopic treatment of calcifying tendonitis of subscapularis and supraspinatus tendon: a case report

Reports concerning the surgical treatment of calcifying tendinits of the subscapularis tendon are rare. We present the case of a symptomatic calcifying tendonitis involving the subscapularis and supraspinatus tendons. The patient was treated with an arthroscopic removal of the calcific deposits. One year after the surgical procedure the patient was completely pain free, had full range-of-motion and negative supraspinatus and subscapularis signs on manual muscle testing. The overall constant score was 93 points. Radiographic evaluation revealed a complete removal of the calcific deposits immediately after the procedure without recurrence after 1 year. We conclude that a combined arthroscopic removal of the calcific deposits of the subscapularis and supraspinatus tendons can lead to an excellent clinical outcome without compromising the functional integrity of the rotator cuff tendons. No potential conflict of interest declared by both authors.     

MR arthrography in calcific tendinitis of the shoulder: diagnostic performance and pitfalls

The purpose was to assess the diagnostic performance of MR arthrography to diagnose calcific tendinitis of the shoulder and to assess the reasons for diagnostic errors. Standard MR arthrograms of 22 patients with calcific tendinitis and 61 controls were retrospectively analyzed by two independent and blinded radiologists. All cases were consecutively collected from a database. Conventional radiographs were available in all cases serving as gold standard. The supraspinatus was involved in 16, the infraspinatus in four and the subscapularis in two patients. All diagnostic errors were analyzed by two additional readers. Reader 1 correctly detected 12 of the 22 shoulders with and 42 of the 61 shoulders without calcific tendinitis (sensitivity 0.55, specificity 0.66). The corresponding values for reader 2 were 13 of 22 and 40 of 61 cases (sensitivity 0.59, specificity 0.69). Inter-rater agreement (kappa-value) was 0.42. Small size of the calcific deposits and isointensity compared to the surrounding tissue were the most important reasons for false negative results. Normal hypointense areas within the supraspinatus tendon substance and attachment were the main reason for false positive results. In conclusion, MR arthrography is insufficient in the diagnosis of calcific tendinitis. Normal hypointense parts of the rotator cuff may mimic calcific deposits and calcifications may not be detected when they are isointense compared to the rotator cuff. Therefore, MR imaging should not be interpreted without corresponding radiographs.     

Acute calcific tendinitis of tibialis posterior tendon (TPT): A rare site of involvement

Calcific tendinitis is a benign inflammatory disorder characterised by deposition of calcium hydroxyapatite crystals in tendons and is commonly seen in tendons around shoulder joint but can also sometimes be encountered in other sites including the hip, wrist, elbow, hand, neck and foot. Calcific tendinitis can clinically mimic many other pathologic entities like infection, gout, myositis ossificans or avulsion fracture which frequently leads to misdiagnosis and inappropriate management. Imaging plays an important role in recognizing and evaluating calcific tendinitis and in differentiating this entity from other pathologic conditions. Calcific tendinitis is largely a self-limiting process which usually responds to conservative treatment although in refractory cases invasive options like steroid injection, needle aspiration or surgery can be considered. Early and accurate diagnosis can prevent needless tests and interventions which the patient may be subjected. We report a case of acute calcific tendinitis involving a rare site (tibialis posterior tendon) with its multimodality imaging appearance.     

Calcific tendinitis of the long head of the biceps brachii distal to the glenohumeral joint: plain film radiographic findings

Calcific tendinitis is a painful condition related to deposition of hydroxyapatite crystals; it favors large joints. The shoulder, specifically the tendons of the rotator cuff and the insertion of the long head of the biceps on the superior glenoid rim, is a well-recognized location for this abnormality. The purpose of this article is to describe a second site of calcific tendinitis of the biceps, distal to the joint and corresponding to the junction of the tendon and muscle. Radiographs in 119 cases of calcific tendinitis of the shoulder, obtained between 1980 and 1988, were reviewed. Twenty had calcific tendinitis in the region of the tendon of the long head of the biceps (nine at the glenoid insertion and 11 adjacent to the humeral shaft). All 11 patients with calcific tendinitis at the more distal site had a small, homogeneous deposit adjacent to the proximal humeral shaft. The densities in these 11 cases followed the normal course of the tendon of the long head of the biceps and were therefore medial to the proximal humeral shaft on the internal rotation view, lateral to the proximal humeral shaft on the external rotation view, and anterior to the proximal humeral shaft on the axillary projection. The major differential diagnosis of calcific tendinitis of the tendon of the long head of the biceps is loose bodies trapped in the biceps tendon sheath. Although the position of the soft-tissue densities in these two entities is similar, loose bodies have an appearance of bone, and their source (degenerative arthritis or recurrent dislocations) is usually apparent. A site of calcific tendinitis distal to the glenohumeral joint that is detectable on plain films is reviewed. Accurate diagnosis depends on understanding the anatomy of the tendon of the long head of the biceps brachii. The clinical charts of the 11 patients also are summarized, with emphasis on the association between the roentgen finding and bicipital tendinitis and impingement syndrome.     

Echo-guided percutaneous treatment of chronic calcific tendinitis of the shoulder

PURPOSE: We report on our personal technique and the results of US-guided percutaneous treatment of chronic calcific tendinitis. MATERIAL AND METHODS: January 1997 to March 1999, seventy patients with known chronic calcific supraspinatus tendinitis were submitted to the US-guided treatment. All patients had undergone plain radiography, US, and physical and psychiatric examination. Plain radiography and aspiration biopsy demonstrated hard and radiopaque calcification in 59 patients and soft and faintly milky calcification in 11 cases; calcification diameter ranged 6-30 mm. US showed tendon thickening, with bulging of the outer tendon surface; 10 patients also had moderate dilatation of the subacromial bursa. Psychiatric examination revealed chronic pain exacerbated at night, which was always associated with motion impairment. The selection criteria for treatment were calcification diameter > 6 mm, integrity of the tendon, and chronic pain. After superficial planes were anesthetized, a 16 G needle was positioned inside the calcification under US guidance and the calcific deposits were fragmented and aspirated. Then, 0.5-1 mL triamcinolone acetonide (40 mg) was injected in the soft tissues or subacromial bursa. RESULTS: Pain resolution and recovery of the full range of motion were seen in 42 patients (60%), and mild functional impairment was seen in 7 cases (10%), while 2 patients (2%) were unchanged. Post-treatment plain radiography showed calcification disappearance in 41 patients (58.5%) and debulking in 29 (41.5%); the calcifications were significantly debulked (> 60%) in 27 patients (38.5%). However, calcification diameter was substantially unchanged in 2 patients (3%) and there remained tendon bulging; in these patients clinical symptoms did not improve. No rotator cuff tears or new tendon calcifications were found in any of our patients even at 19-28 months' follow-up. DISCUSSION: The US-guided technique always allowed easy location of calcific deposits and complete aspiration of all soft calcifications. Splintering of hard calcifications helped migration of residual deposits to vascularized soft tissues, which accelerated the--frequently complete--resorption process. We privileged extensive and prolonged fragmentation of the calcifications using a single needle, versus the technique using a second needle, saline lavage and aspiration of residual deposits. CONCLUSION: US-guided percutaneous treatment with aspiration and splintering of chronic calcific supraspinatus tendinitis is a conservative, simple, well-tolerated procedure which can be considered the method of choice after the failure of medical treatment.     

Acute retropharyngeal calcific tendinitis: a case report with unusual location of calcification

Retropharyngeal calcific tendinitis is an inflammatory process caused by calcium hydroxyapatite crystal deposition in the longus colli tendon of the prevertebral space, and it may mimic a retropharyngeal infection or abscess. The diagnosis of retropharyngeal calcific tendinitis will be made radiologically by the detection of calcifications anterior to C1–C3 and prevertebral soft tissue swelling. We present a case of acute retropharyngeal calcific tendinitis with an unusual location of calcification anterior to the C5–C6 disc.     

Calcific tendinitis of the gluteus maximus tendon (Gluteus maximus tendinitis)

Seven cases of calcific tendinitis of the gluteus maximus tendon are presented. Awareness of the precise anatomic location of the calcific deposit is essential for the accurate diagnosis of this uncommon site of tendinitis. Clinically, the presenting complaint is that of pain. In some instances, however, the patients are asymptomatic and the calcification is an incidental finding.     

Surgical treatment of Achilles tendinitis by decompression of the retrocalcaneal bursa and the superior calcaneal tuberosity

BACKGROUND: Initial nonoperative treatment of pain at the Achilles tendon, often referred to as "tendinitis," is not always successful. HYPOTHESIS: Surgical treatment is effective for patients with insertional tendinitis unrelieved by nonoperative measures. STUDY DESIGN: Retrospective cohort study. METHODS: Thirty-five patients (41 feet) who had painful Achilles tendon syndrome unrelieved by 6 months of nonoperative measures were treated surgically. The technique consisted of a single incision along the lateral border of the Achilles tendon. The dissection exposed the retrocalcaneal bursa and fat pad, which were completely excised along with any scarred and thickened paratenon. A partial calcaneal exostectomy of the tubercle was performed. RESULTS: At a minimum follow-up of 20 months (average, 39), the patients' pain scores (rated from 0 to 6) improved from 4.7 (SD, 1.1) preoperatively to 1.5 (SD, 1.3); 90% had complete or significant relief of symptoms, 10% felt improved, and none felt unchanged or worse. CONCLUSIONS: Surgical treatment of chronic Achilles tendon pain with resection of the prominent tuberosity, complete debridement of the bursa, excision of thickened, scarred paratenon, and removal of accessible calcific deposits within the tendon is an effective treatment.     

Semimembranosus tendinitis: an overlooked cause of medial knee pain

A significant number of patients presenting with nontraumatic chronic medial knee pain have been found, after careful examination, to have chronic semimembranosus tendinitis. Of 115 patients diagnosed and treated for semimembranosus tendinitis over a 5 year period, 10 patients were refractory to conservative treatment. The purpose of this paper is to discuss the symptomatology and diagnosis of semi-membranous tendinitis, and to relate the treatment that these 10 patients underwent, i.e., surgical exploration, drilling of the insertion site, and semitendinosus tendon transfer. Most cases of semitendinosus tendinitis respond to conservative therapy; however, if surgical treatment is considered, arthroscopy should probably be performed at the time of surgery. In this way, it is possible to determine the presence of significant intraarticular abnormalities that may have a causal relationship with the onset of semimembranosus tendinitis.     

Extracorporeal shock wave treatment for shoulder calcific tendonitis: a systematic review

The treatment of patients with calcific tendonitis is typically conservative, including physical therapy, iontophoresis, deep friction, local or systemic application of noninflammatory drugs, needle irrigation–aspiration of calcium deposit, and subacromial bursal steroid injection. If the pain becomes chronic or intermittent after several months of conservative treatment, arthroscopic and open procedures are available to curette the calcium deposit, and additional subacromial decompression can be performed if necessary. As an alternative, minimally invasive extracorporeal shock wave therapy (ESWT) has been postulated to be an effective treatment option for treating calcific tendinitis of the shoulder, before surgery. Herein we discuss the indications, mechanism of therapeutic effect, efficacy of treatment, and complications after ESWT application.     

Acute calcific tendinitis of the flexor pollicis longus in an 8-year-old boy

Calcific tendinitis is a common source of musculoskeletal pain in adults; however, it is rarely encountered in children. Calcific tendinitis is the most commonly encountered manifestation of hydroxyapatite deposition disease, in which calcium hydroxyapatite crystal deposition occurs in tendons. It may cause acute or chronic pain, or may be entirely asymptomatic. We describe a case of acute calcific tendinitis of the flexor pollicis longus tendon in an 8-year-old boy, who initially presented to our department for workup of a mass felt along the volar aspect of the right wrist.     

Symptomatic calcific tendinitis at unusual sites.

Three cases of symptomatic calcific tendinitis at unusual sites (in the vastus lateralis tendon, the rectus femoris tendon and the flexor carpi ulnaris tendon) are presented. In two, the plain radiographs were nondiagnostic, and the diagnosis was made on the basis of the findings in computed tomography (CT) scans. The value of CT in examining atypical patients is demonstrated.     

Calcific tendinitis of the neck

When a calcific deposit in the shoulder, hip or elbow ruptures, it results in severe pain and tenderness. Few clinicians are aware that the longus colli muscle of the neck is also subject to acute tendinitis. When it occurs, the pain and spasm mimic infectious spondylitis or meningitis. Four patients are presented with acute tendinitis of the longus colli muscle and the classic radiographic findings of soft-tissue swelling and amorphous calcium deposits in the tendon.     

Arthroscopic treatment of chronic calcific tendinitis with complete removal and rotator cuff tendon repair

Relatively large calcific materials on radiographs of shoulders with persistent symptoms after extended periods of conservative treatment are candidates for operative treatment. But complete removal of calcific materials sometimes leaves a large defect in the rotator cuff tendon, and tendon repair might be essential if defects are large. We evaluated the clinical results of complete removal of calcific deposits with or without repair of the rotator cuff tendon in 35 consecutive patients. Eighteen patients underwent calcific material removal, which resulted in a complete tear in the rotator cuff tendon, and suture anchor repair. And the other 17 patients received either side-to-side repair or simple debridement. Clinical outcomes improved at a median 31 (range 24–45) months after surgery, and pain relief was achieved within 6 months of surgery in 30 of 35. However, ten patients developed a secondary stiff shoulder. Repair with or without suture anchor after complete removal of calcific material provides good clinical results and earlier pain relief when it was compared to previous literatures of minimal removal technique.     

Imaging the long head of bicepstendon—a pictorial essay emphasizing magnetic resonance

Lesions of the long head of the bicepstendon (BT) are seen in association with tears of the rotator-cuff, particularly lesions of the subscapularis tendon and the rotator-interval. The frequency of positive MR-findings at the BT is approximately 25%. The pathologic alterations include complete medial luxation, subluxation and entrapment by the subscapularis tendon, tendinitis or tendovaginitis and lesions at the origin of the tendon at the superior labrum (superior labrum anterior to posterior (SLAP)--lesions). The imaging signs of BT pathology on MR include an abnormal course and position of the tendon, alterations in shape and changes in signal-intensity (SI), obliteration and thickening of the tendon-sheath. In long standing intra-articular BT rupture, neo-insertion of the tendon in the bicipital sulcus may ensue. The long BT needs to be visualized in transaxial, coronal oblique and sagittal oblique projections. Besides serving as stabilizer of the long head of biceps muscle, the BT is also an important stabilizer of the anterior joint capsule.     

Calcific Tendinitis: A Pictorial Review

Calcific tendinitis is caused by the pathologic deposition of calcium hydroxyapatite crystals in tendons and is a common cause of joint pain. The disease typically affects the shoulder and hip, with characteristic imaging findings; however, any joint can be involved. Occasionally, calcific tendinitis can mimic aggressive disorders, such as infection and neoplasm, especially on magnetic resonance imaging. Radiologists should be familiar with the imaging findings to distinguish calcific tendinitis from more aggressive processes. Image-guided percutaneous needle aspiration and steroid injection of calcific tendinitis are useful techniques performed by the radiologist for the treatment of symptomatic cases. Familiarity with these procedures and their imaging appearance is an important aspect in the management of this common disease.     

Calcifying tendinitis of the rotator cuff with cortical bone erosion

Calcifying tendinitis occurs most commonly in the rotator cuff tendons, particularly involving the supraspinatus tendon insertion, and is often asymptomatic. Cortical erosion secondary to calcifying tendinitis has been reported in multiple locations, including in the rotator cuff tendons. We present a pathologically proven case of symptomatic calcifying tendinitis involving the infraspinatus tendon with cortical erosion with correlative radiographic, CT, and MR findings. The importance of considering this diagnosis when evaluating lytic lesions of the humerus and the imaging differential diagnosis of calcifying tendinitis and cortical erosion are discussed.An erratum to this article can be found at     

Calcific tendinitis of the gluteus medius tendon with bone marrow edema mimicking metastatic disease

A case of calcific tendinitis of the gluteus medius is presented. This report describes a patient with a history of breast cancer who had the combination of amorphous calcifications in the gluteus medius tendon and the MR finding of conspicuous bone marrow edema in the adjacent greater trochanter, prompting concern for metastatic disease. We present images from radiography, bone scanning, CT, and MR imaging. The unusual combination of findings in these studies should be considered conclusive for calcific tendinitis, and should not be confused with malignancy.     

Partial Achilles Tendon Rupture Following Corticosteroid Injection

Corticosteroid injections are sometimes used to treat Achilles tendinitis after conservative measures such as anti-inflammatory medications, rest, ice massage, and immobilization have failed. Some research suggests that corticosteroid injections can lead to tendon rupture, though other investigations have not shown a correlation. A case report detailing partial Achilles tendon rupture after corticosteroid injection renews concern about the risks of this treatment.