晶状体超声乳化人工晶状体植入治疗葡萄膜炎并发白内障

目的 探讨葡萄膜炎并发白内障晶状体超声乳化吸出及人工晶状体植入术的疗效.方法 对46例（52眼）葡萄膜炎并发白内障行晶状体超声乳化吸出及人工晶状体植入术,其中2眼联合小梁切除术.结果 术后48眼（92.31%）视力较术前提高.术后6月视力≥0.5者43眼（82.69%）,0.3～0.4者3眼（5.77%）,0.1～0.2者3眼（5.77%）,＜0.1者3眼（5.77%）.结论 晶状体超声乳化吸出术治疗葡萄膜炎并发白内障,术前充分准备,术中、术后处理得当,则术后炎症反应轻,视力恢复好.     

超声乳化人工晶状体植入治疗葡萄膜炎并发白内障临床观察

目的探讨葡萄膜炎并发白内障晶状体超声乳化吸出及人工晶状体植入术的疗效。方法对38例38眼葡萄膜炎并发白内障患者行晶状体超声乳化吸出及人工晶状体植入术。结果 38眼术后裸眼视力均有不同程度的提高。术后3月≥0.5者31眼（80.77%）,0.3～0.4者3眼（7.69%）,0.1～0.2者2眼（5.77%）,〈0.1者2眼（5.77%）。结论晶状体超声乳化吸出术治疗葡萄膜炎并发白内障,术前充分准备,术中、术后处理得当,则术后炎症反应轻,视力恢复好。     

葡萄膜炎并发白内障超声乳化人工晶状体植入

目的：评价超声乳化人工晶状体植入术对葡萄膜炎并发白内障的效果。方法：对28例32眼葡萄膜炎并发白内障施行了超声乳化吸出和后房人工晶状体植入术，对术后视力和并发症作了分析。结果：超声乳化人工晶状植入术后31眼视力提高，术后最常见的并发症为后囊浑浊。结论：超声乳化人工晶状植入术对于葡萄膜炎并发白内障是一种安全可靠的手术。     

表面麻醉下白内障超声乳化人工晶状体植入治疗葡萄膜炎并发白内障

目的 :探讨葡萄膜炎并发白内障超声乳化摘除及人工晶状体植入术的疗效。方法 :对 3 8例 42只眼葡萄膜炎并发白内障患者行超声乳化摘除及人工晶状体植入术 ,其中 4只眼联合小梁切除术 ,3只眼联合周边虹膜切除术。结果 :术后 3 9只眼 (92 86% )视力较术前提高。术后 6个月裸眼视力≥ 0 5者 3 3只眼 (78 57% ) ,0 3～ 0 5者 3只眼 (7 14 % ) ,0 1～ 0 3者4只眼 (9 52 % ) ,<0 1者 2只眼 (4 76% )。结论 :超声乳化白内障摘除术治疗葡萄膜炎并发白内障 ,术后炎症反应轻 ,视力恢复好。     

青光眼术后晶状体超声乳化术的探讨

目的探讨抗青光眼术后白内障行晶状体超声乳化吸出术的手术技巧，并评价手术效果。方法对26例（32眼）青光眼术后的白内障行颞侧透明角膜切口晶状体超声乳化吸出折叠人工晶状体植入术，术后随访4-21月，观察视力、滤过泡、眼压和并发症的情况。结果有30眼（93．75％）视力有不同程度的提高；2眼术后视力无提高者系青光眼性视神经萎缩者。术后矫正视力低于0．05者3眼（9．38％），0．05～0．25者8眼（24．99％），0．3～1．0者21眼（65．63％）。所有病例术后滤过泡均无影响，术后眼压在正常范围。并发症主要为角膜水肿和虹膜炎症反应。结论青光眼术后白内障采用颞侧透明角膜切口行晶状体超声乳化吸出折叠人工晶状体植入术效果良好，并发症少，可提高视力并保持滤过泡的功能。     

葡萄膜炎并发白内障乳化吸出人工晶状体植入

目的 探讨晶状体超声乳化吸出联合人工晶状体植入术在葡萄膜炎并发白内障治疗中的应用。方法 对36例(44眼)葡萄膜炎并发白内障术后，观察其疗效及并发症。结果 所有患者术后视力均有不同程度的提高，其中27眼(61．36％)术后最佳矫正视力≥0．5。术中发生前房积血2眼(4.55％)，后囊破裂1眼(2.27％)；术后瞳孔均能维持圆形居中。术后并发症：早期角膜水肿14眼(31．82％)；前房轻度炎症反应39眼(88.64％)；1周后出现成形性前房渗出1眼(2．27％)；后囊浑浊7眼(15．91％)。结论 超声乳化品状体摘出联合人工晶状体植入术治疗葡萄膜炎并发白内障是安全有效的。     

超声乳化白内障吸除治疗葡萄膜炎并发白内障

目的观察超声乳化白内障吸除人工晶状体植入术治疗葡萄膜炎并发白内障的临床效果。方法对25例（29只眼）葡萄膜炎并发白内障行超声乳化白内障患者吸除人工晶状体植入术，同时用非切开方式对粘连性小瞳孔进行扩张。结果白内障术后患者视力均得到提高，术后6个月最佳矫正视力≥0．5者24只眼占82.8％，术后恢复生理性小圆瞳孔25只眼占86．2％。结论对葡萄膜炎并发白内障施行超声乳化白内障吸除人工晶状体植入术可提高视力，恢复生理性圆瞳孔。     

葡萄膜炎并发白内障术中植入肝素修饰人工晶状体

目的评价葡萄膜炎并发白内障术中植入肝素表面修饰的PMMA人工晶状体的效果。方法98例（146眼）葡萄膜炎并发白内障行超声乳化吸出术,术中分别植入肝素修饰的PMMA人工晶状体56例（98眼）（肝素组）和未修饰PMMA人工晶状体42例（48眼）（对照组）。对两组术后视力、眼前段反应、后囊浑浊情况进行回顾性对比分析。结果矫正视力≥0．5者,肝素组86眼（87．76％）,对照组27眼（56．25％）,（P〈0．05）。眼前段反应：人工晶状体表面纤维素样渗出、房水细胞,均在术后1周时差异最显著（P〈0．05）,虹膜后粘连在6个月后两组差异最显著（P〈0．05）。后囊浑浊发生率6个月后对照组明显高于肝素组,分别为54．17％和12。24％（P〈0．05）。结论葡萄膜炎并发白内障术中植入肝素修饰人工晶状体能显著减轻术后眼前段的炎症反应,降低后囊浑浊的发生率。     

白内障超声乳化人工晶状体植入临床分析

目的探讨白内障超声乳化及人工晶状体植入术的效果、手术方法及并发症的防治。方法对473例(495眼)白内障行超声乳化吸出及人工晶状体植入术进行观察和分析。结果术后1周矫正视力1．0以上为147眼(29．70％)，术后3月矫正视力1．0以上者341眼(68．90％)，0．5以上者476眼(96．16％)。主要并发症术中为后囊破裂和虹膜损伤，术后为角膜内皮水肿。结论超声乳化白内障吸出联合后房型人工晶状体植入术具有术后早期反应少及术后视力提高迅速的优点。     

超声乳化吸出治疗晶状体半脱位白内障

目的探讨超声乳化白内障吸出联合人工晶状体植入术在晶状体半脱位白内障手术中的应用。方法对15例（15眼）行超声乳化白内障吸出联合人工晶状体植入术。结果术后视力0．4～0．8者9眼,0．1～0．3者5眼,0．08以下者1眼。结论超声乳化白内障吸出联合人工状体植入术,是晶状体半脱位白内障的有效的手术方法。     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.