表面麻醉下白内障摘除人工晶状体植入术

目的进一步探讨表面麻醉下小切口非超声乳化白内障摘除人工晶状体植入术的安全性及有效忡。方法随机抽取220例(256眼)行小切口非超声乳化白内障摘除及人工晶状体植入术患者分为2组。表面麻醉组110例(126 眼),球后麻醉组110例(130眼),比较2组的手术效果及并发症。结果表向麻醉组手术时间明显短于球后麻醉组(P<0.01),而且并发症更少,术后1d及1周视力对比无显著性差异(P>0.05)。结论表面麻醉下小切口非超声乳化白内障摘除人工晶状体植入术是安全有效的,而且并发症少,简便易行。     

表面麻醉下小切口非超声乳化白内障摘除及人工晶状体植入

目的:探讨表面麻醉下行小切口非超声乳化白内障囊外摘除及人工晶状体植入术的安全性、有效性及适应证。方法:对150例152眼白内障患者在表面麻醉下行隧道小切口非超声乳化白内障囊外摘除及人工晶状体植入术并观察其麻醉效果及术后视力。结果:所有患者在表面麻醉下均能很好地配合手术顺利完成。无1例改用其他麻醉方法。平均手术时间为24min。术后第2d视力≥0.2者147眼(97%);视力≥0.5者103眼(68%);视力≥0.8者20眼(13%)。结论:表面麻醉下小切口非超声乳化白内障摘除及人工晶状体植入是安全经济、实用、效果好、相对来说易掌握,特别适用于经济欠发达地区成熟、过熟、大核、硬核白内障多的情况。     

表面麻醉下小切口非超声乳化白内障摘除及人工晶状体植入术

目的:探讨表面麻醉下行小切口非超声乳化白内障摘除及人工晶状体植入术的安全性及有效性。方法:对79例86眼白内障患者在表面麻醉下行小切口非超声乳化白内障摘除及人工晶状体植入术并观察其麻醉效果及术后视力。结果:其中64例71眼无疼痛,15例15眼胀痛,术中追加一次表面麻醉。平均手术时间为18min,术后1d及1wk视力≥0.5者分别占83%和92%。结论:表面麻醉小切口非超声乳化白内障摘除及人工晶状体植入术具有操作简单,手术时间短,术后视力恢复快且稳定,手术成本低,适合我国国情,有利于规模化手术。     

微小切口非超声乳化白内障摘除联合折叠式人工晶状体植入术临床观察

目的 观察微小切口非超声乳化白内障摘除联合折叠式人工晶状体植入术手术效果.方法 抽选107例（107眼）行微小切口非超声乳化白内障摘除联合折叠式人工晶状体植入术,112例（112眼）行小切口非超声乳化白内障摘除联合硬片人工晶状体植入术,比较术后1周视力.结果 术后1周视力大于0.3者分别为88.78％及66.96％.结论 微小切口非超声乳化白内障摘除联合折叠式人工晶状体植入术术后1周视力优于小切口非超声乳化白内障摘除联合硬片人工晶状体植入术,术后近期（1周）效果与超声乳化近期（1周）效果相近,在缺少设备、缺少投入的基层医院值得推广使用.     

改良小切口非超声乳化白内障摘除人工晶状体植入术

目的:探讨改良小切口非超声乳化白内障摘除人工晶状体植入术的手术效果及在基层防盲治盲中的应用。方法:对2008-09/11278例278眼行改良小切口非超声乳化白内障摘除人工晶状体植入术。结果:278例278眼患者术后视力明显提高,脱盲率100%。结论:改良小切口非超声乳化白内障摘除人工晶状体植入术是一种安全、可靠、经济的手术方法,适合于在基层防盲治盲中推广应用     

表面麻醉下小切口非超声乳化白内障摘除及人工晶状体植入术

目的：探讨表面麻醉下行小切口非超声乳化白内障摘除人工晶状体植入术的安全性及有效性。方法：对90例（102只眼）白内障患者在表面麻醉下行小切口非超声乳化白内障摘除及人工晶状体植入术并观察其麻醉效果及术后视力。结果：所有患者在表麻下均能很好地配合手术顺利完成。无一例改用其它麻醉方法。平均手术时间为20.04分钟。术后1天及1周视力≥0.5者，分别占67.65％和77.27％。结论：表面麻醉小切口非超声乳化白内障摘除及人工晶状体植入术是安全有效的。     

表面麻醉下小切口非超声乳化白内障摘除术

目的评价在爱尔凯因麻醉下行小切口非超声乳化白内障摘除及人工晶状体植入术的安全性及优越性.方法在爱尔凯因表麻下对116例(120只眼)白内障患者行小切口非超声乳化白内障摘除及人工晶状体植入术,并观察其麻醉效果及视力.结果在表麻下全部患者均可很好地配合手术完成,无一例更换麻醉方法.平均手术时间13.51分钟,术后第1天视力大于0.5者占60.83%,术后1周大于0.5者占75.83%.结论在爱尔凯因表麻下行小切口非超声乳化白内障摘除及人工晶状体植入术是安全优越的.     

改良隧道切口非超声乳化白内障摘除联合折叠晶状体植入术临床应用观察

目的:观察改良隧道切口非超声乳化白内障摘除联合折叠人工晶状体植入术的临床效果和特点。方法:118例白内障患者的118只眼,随机分为2组:超声乳化白内障吸除联合折叠人工晶状体植入术手术组58只眼与改良隧道切口非超声乳化白内障摘除联合折叠人工晶状体植入术组60只眼。对比观察两种术式并发症及术后视力。结果:改良隧道切口非超声乳化白内障摘除联合折叠人工晶状体植入术与超声乳化白内障吸除术联合折叠人工晶状体植入术在白内障手术中的并发症、近远期术后视力比较,差异均无统计学意义,而改良隧道切口非超声乳化白内障摘除术的手术成本低于超声乳化白内障吸除术,且学习曲线短。结论:改良隧道切口非超声乳化白内障摘除联合折叠人工晶状体植入术适合在基层医院的白内障治疗中推广。眼科学报2007;23:247-251.     

表面麻醉下小切口非超声乳化白内障手术并发症探讨

目的 观察表麻下小切口非超声乳化白内障手术术中、术后并发症，对其进行分析并提出处理意见。方法 96例（125眼）白内障，表面麻醉下进行小切口非超声乳化白内障摘除并人工晶体植入术。结果 术中：虹膜脱出13眼，晶状体后囊破裂9眼。术后：角膜内皮条样浑浊29眼，片状浑浊10眼，前房纤维素样渗出7眼。结论 表麻下小切口非超声乳化白内障手术避免了球周、球后麻醉带来的一系列并发症。对晶状体核的处理较关键，亦是易产生并发症的步骤。     

高度近视小切口非超声乳化白内障摘除术的临床报告

目的:探讨高度近视小切口非超声乳化白内障摘除联合人工晶状体植入术的效果及体会。方法:对32例(40眼)高度近视并发白内障患者实施小切口非超声乳化白内障摘除联合人工晶状体植入术。结果:术后3d矫正视力达0.3以上者27眼(67.5%),术后1mo矫正视力达0.3以上者35眼(87.5%)。结论:小切口非超声乳化白内障摘除联合人工晶状体植入术适合于高度近视并发白内障患者,但应注意术前准备及术中操作技巧。     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.