80岁以上患者白内障超声乳化吸除联合折叠式人工晶状体植入术临床观察

目的分析80岁以上患者白内障超声乳化吸除联合折叠式人工晶状体植入术的临床效果。方法78例(78只眼)白内障在表面麻醉下透明角膜10～12点钟做切口,在3点或9点钟位角膜缘内做0.3～0.6mm辅助切口,超声乳化吸除皮质及核,植入ACRYSOF折叠式人工晶状体。术后观察视力、切口、前房、人工晶状体位置、眼底。结果术后1天、7天、1个月和3个月矫正视力≥0.5者分别为43只眼(55.13%)、49只眼(62.82%)、56只眼(71.79%)和60只眼(76.92%)。结论对80岁以上高龄患者施行白内障超声乳化吸除联合折叠式人工晶状体植入术,做好术前检查和术中监护可安全进行手术,术后可获得较好的视力。     

高龄患者白内障超声乳化吸除联合人工晶状体植入术

目的:探讨高龄患者白内障超声乳化吸除联合人工晶状体植入手术的可行性和安全性。方法:对105例(142眼)高龄患者进行白内障超声乳化吸除联合人工晶状体植入术,以88例(142眼)低龄老年性白内障患者作对照组。结果:两组术后视力提高差异无显著性(P>0.05),手术并发症有明显差异(P<0.01),两组均无因全身疾病而中断手术者。结论:对高龄患者施行白内障超声乳化吸除联合人工晶状体植入术是相对安全的,只要有复明的希望,就应尽早手术治疗。     

高龄患者白内障超声乳化吸出术效果分析

目的讨论高龄患者白内障进行晶状体超声乳化吸出后房人工晶状体植入术的效果。方法对154例(159眼)80岁以上高龄患者的白内障实行反眉式巩膜隧道切口晶状体超声乳化吸出联合硬性人工晶状体植入。结果术后第1天裸眼视力≥0.5者79眼,占49.69%。术后1周视力≥0.5者101眼,占63.52%。术后3月视力≥0.5者118眼,占74.21%。术后3月视力0.1~0.4者29眼,占18.24%。术后3月视力≤0.1者12眼,占7.55%。结论高龄患者白内障超声乳化吸出后房人工晶状体植入术术后视力恢复快,但视力恢复程度与眼底疾病有关。     

高龄白内障患者超声乳化人工晶状体植入术疗效分析

目的:评价高龄白内障患者超声乳化人工晶状体植入术的临床疗效及安全性.方法:对184例(221眼)80～103岁老人行白内障超声乳化人工晶状体植入术,对术后视力、并发症对比分析.结果:术后1 wk脱盲率95.0%,脱残率77.8%.无严重局部及全身并发症发生.49眼术后1 wk视力≤0.3,其中眼底病变占41眼(83.7%).结论:高龄白内障患者行超声乳化人工晶状体植入术安全、有效.眼底病变是影响术后视力恢复的主要原因.     

白内障超声乳化折叠人工晶状体植入250例临床分析

目的:观察超声乳化白内障吸除折叠人工晶状体植入术的临床疗效及并发症。方法:对250例(254眼)进行超声乳化晶状体吸除一期折叠人工晶状体植入术。结果:术后1wk;1,3mo裸眼或矫正视力≥0.5者分别为82.4%、84.6%、89.2%。结论:超声乳化白内障吸除折叠人工晶状体植入术疗效满意、安全、快捷。手术成功关键取决于术者技术熟练程度。     

手术治疗糖尿病性白内障60眼临床分析

目的:分析小切口白内障囊外摘出人工晶状体植入术和超声乳化白内障吸除人工晶状体植入术治疗糖尿病性白内障的疗效及并发症。方法:选择2006-01/2010-01确诊为糖尿病性白内障患者52例60眼,Ⅱ～Ⅲ级核47眼,采用超声乳化白内障吸除后房型人工晶状体植入术;Ⅳ～Ⅴ级核13眼,采用小切口白内障囊外摘出后房型人工晶状体植入术。结果:糖尿病患者52例60眼白内障术后视力均有不同程度的提高,其中50眼(83%)术后1wk裸眼视力≥0.4。术中术后主要并发症经处理对术后视力无影响。结论:对糖尿病性白内障患者采取恰当的术式,施行小切口白内障囊外摘出人工晶状体植入术或超声乳化白内障吸除人工晶状体植入术疗效肯定。     

高龄白内障患者超声乳化术的护理

目的评价高龄白内障患者行超声乳化吸除联合人工晶状体植入术围手术期护理的效果。方法 2007年4月以来,对41例（41只眼）80岁以上的白内障患者行白内障超声乳化吸除联合人工晶状体植入。完善术前检查,注意监测术中全身情况,加强患者围手术期的护理及指导。结果 41例（41只眼）高龄白内障患者手术顺利,术后视力恢复较好,未出现严重的全身及眼部并发症,患者满意。结论高龄白内障患者施行超声乳化术,经认真手术准备,做好围手术期护理,可降低手术的风险,取得满意的效果。     

80岁以上老年人白内障超声乳化摘除及人工晶状体植入术疗效分析

目的探讨高龄老年人白内障超声乳化摘除及人工晶状体植入临床效果。方法对80岁以上白内障患者(1301只眼)的白内障超声乳化摘除及人工晶状体植入术进行分析。结果87.8%高龄老年人通过白内障超声乳化摘除及人工晶状体植入术,视力均有不同程度提高。结论随着表面麻醉下白内障超声乳化摘除及人工晶状体植入术开展,高龄不再是手术的禁忌。     

高度近视合并白内障患者植入Array多焦点人工晶状体的临床观察

目的:评价白内障超声乳化吸除联合Array SA40N多焦点人工晶状体植入术治疗高度近视合并白内障的临床疗效及其安全性。方法:对25例(31眼)高度近视合并白内障患者行超声乳化白内障吸除联合低度数Array多焦点人工晶状体植入术,观察术中和术后并发症、术后视力和屈光状态。结果:所有患者术中均无并发症发生。术后6mo,所有患者裸眼远视力≥0.5者25眼(81%);最佳矫正远视力≥0.5者28眼(90%)。裸眼近视力≥0.5者12眼(39%);最佳矫正近视力≥0.5者22眼(71%)。术后屈光度数偏差值≤±0.50D者为24眼(77%)。结论:超声乳化白内障吸除联合低度数ArraySA40N多焦点折叠式人工晶状体植入术,是治疗高度近视合并白内障患者安全、有效的手术方法。     

高度近视白内障超声乳化人工晶状体植入术27例

目的:探讨高度近视眼合并白内障行超声乳化白内障吸除人工晶状体植入术治疗的临床疗效。方法:27例(37眼)因白内障合并高度近视行超声乳化白内障吸除+人工晶状体植入术。记录术前眼轴长度和术后视力,屈光度数,观察手术并发症术后眼部情况。术后随访时间为6～24mo。结果:术前平均眼轴长度为28.3mm。术后最佳矫正视力≥0.2共35眼(95%),≥0.5者共33眼(89%)。术中无后囊膜破裂;术后角膜水肿2眼,术后一过性高眼压1眼,后发性白内障2眼;术后发现黄斑变性和眼底出血共4例,无视网膜和脉络膜脱离者。结论:超声乳化白内障吸除低度数折叠式人工晶状体植入术是治疗白内障合并高度近视眼安全、有效的方法。     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.