超声乳化联合经后囊硅油取出及人工晶状体植入术

目的 探讨超声乳化联合经后囊硅油取出和人工晶状体植入术治疗硅油填充眼白内障的效果.方法 超声乳化联合经后囊硅油取出和人工晶状体植入术治疗硅油眼合并白内障96例（96眼）.超声乳化吸出术后行后囊环形撕囊,经上方角膜缘切口进入后囊撕囊口取出硅油,植入人工晶状体.结果 术后随访3～6月,所有患者裸眼及矫正视力均较术前提高,人工晶状体居中.3例早期角膜水肿,3～5 d消失,2例视网膜再脱离行再次视网膜复位手术;5例高眼压联合用药后控制正常.结论 对硅油填充眼白内障,行超声乳化联合经后囊硅油取出和人工晶状体植入术,可减少手术次数,并降低手术风险.     

硅油取出联合白内障摘出人工晶状体植入

目的 探讨硅油填充眼硅油取出联合白内障摘出人工晶状体植入的临床效果.方法 对1999年以来我科14例（14眼）硅油充填眼合并白内障者行硅油取出联合白内障囊外摘出人工晶状体植入,术后随访3～12月.结果 术后视力提高11眼,不变2眼,降低1眼.结论 对眼部状况较好的硅油填充眼,在硅油取出同时行白内障囊外摘出人工晶状体植入可有较好的视力效果.     

硅油填充眼的白内障超声乳化联合硅油取出术

目的：探讨玻璃体视网膜手术联合硅油眼内填充术后并发性白内障行白内障超声乳化和/或人工晶体植入联合硅油取出术的临床效果。方法：对22例22眼硅油填充术后并发性白内障进行白内障超声乳化联合硅油取出手术,其中9例通过巩膜隧道切口植入硬性人工晶体,3例通过角膜切口植入折叠人工晶体,10例未植入人工晶体。结果：22例手术中后囊膜保持完整,除10例因高度近视、视网膜条件很差或再发视网膜脱离而未植入人工晶体外,蓁均顺利植入人工晶体。术后5例发生角膜水肿,均在术后3～7天内消退。硅油取出顺利。3例术中发现限局性网脱,行视网膜复位后,2例C2F6气体填充后2周再发网脱,1例再次硅油填充视网膜保持复位。视力除3例再发网脱外,其余均达到玻璃体手术后最佳视力。结论：硅油填充眼合并白内障行白内障超声乳化联合硅油取出手术不仅安全、有效、而且可减少病人多次手术的痛苦。     

硅油填充眼并发性白内障的手术疗效观察

目的：观察玻璃体切割术后硅油填充眼并发白内障行超声乳化联合硅油取出及人工晶状体植入术的临床效果及安全性。方法：对32例32眼硅油填充术后并发白内障患者行超声乳化联合经睫状体平坦部切口取硅油及折叠式人工晶状体植入术。术后随访3～18（平均9）mo。结果：术后最佳矫正视力：光感～数指/眼前者3眼,0.02～0.1者10眼,〉0.1～0.2者12眼,≥0.3者7眼。所有患者均无视网膜脱离、角膜内皮失代偿、人工晶状体移位或硅油残留等。结论：白内障行超声乳化联合硅油取出及折叠式人工晶状体植入术治疗硅油填充眼并发性白内障安全、有效。     

硅油填充术后并发性白内障超声乳化人工晶状体植入术联合硅油取出的临床观察

目的:观察硅油填充术后并发性白内障行超声乳化人工晶状体植入术联合硅油取出的治疗效果。方法:回顾性分析了40例(41只眼)硅油填充术后并发白内障行超声乳化联合硅油取出及人工晶状体植入的病例资料,并随访5~18个月,分析其视力恢复情况及术中、术后并发症。结果:除3例因硅油取出术后视网膜脱离外,其余患者均有不同程度的视力增加。术中主要并发症为后囊膜破裂,术后的主要并发症为视网膜脱离。结论:硅油填充术后并发性白内障行超声乳化人工晶状体植入术联合硅油取出术是一种安全有效的方法。     

硅油填充眼并发白内障的手术治疗

目的探讨小切口非超乳白内障摘出,经睫状体平坦部硅油取出联合人工晶状体植入治疗硅油填充眼并发白内障的疗效。方法对16例（16眼）进行小切口非超乳的白内障摘出,经睫状体平坦部硅油取出,人工晶状体植入联合手术。术后随访1年,观察术后视力、视网膜情况及并发症。结果术后随访56．25％（9／16）视力提高明显,1例出现视网膜脱离,所有患者无人工晶状体移位等特殊并发症。晶状体前后囊有不同程度浑浊,中央部因无囊组织而透明。结论该联合手术治疗硅油眼并发白内障安全有效的方法,同时降低医疗成本,减少患者多次手术造成的痛苦; 严格掌握手术适应证、选择合适病例和具备娴熟技巧是手术成功的关键。     

硅油充填眼行白内障超声乳化人工晶状体植入联合硅油取出术

目的 探讨玻璃体切割术后硅油充填眼经透明角膜切口行白内障超声乳化摘除、硅油取出、人工晶状体植入联合手术(1组)以及经睫状体平坦部置灌注管行白内障超声乳化摘除、硅油取出、人工晶状体植人(2组)两种手术方式的临床疗效观察。方法 对2000年1月～2003年4月间玻璃体切割术后硅油充填眼白内障患者78例(78只眼)经透明角膜行联合手术。对49例(49只眼)经睫状体平坦部行联合手术。观察眼底视网膜复位良好，行人工晶状体植入术。结果 1组病例有中52只眼最佳矫正视力达0.1～0.6(66．67％)；19只眼视力为0.02～0.1(24．36％)；7只眼视力为光感～指数(8．97％，包括5只眼视网膜脱离复发者)。2组病例中31只眼最佳矫正视力达0.1～0.6(65．3％)；11只眼视力为0.02～0.1(22．45％)；7只眼视力为光感～指数(14．3％，包括2只眼视网膜脱离复发者)。2组病例中有1只眼出现睫状体平坦部切口渗血，所有病例均无持续性角膜内皮失代偿、硅油泡残留、人工晶状体移位等并发症发生。结论 硅油眼行白内障超声乳化、硅油取出、人工晶状体植入术对于玻璃体切除手术后硅油充填眼并发白内障是一种安全、有效的方法。经透明角膜切口行此手术简化了手术步骤，减少了手术并发症的发生。     

硅油填充眼并发性白内障手术探讨

目的探讨硅油充填眼术后并发性白内障行超声乳化联合硅油取出及人工晶状体植入术的临床效果。方法对26例（26眼）硅油填充术后并发性白内障进行超声乳化经睫状体平坦部切口取硅油联合折叠式人工晶状体植入术。术后随访4-15个月（平均8个月）。结果手术后最佳矫正视力：光感-眼前数指者2眼,0.02-0.1者5眼,〉0.1-0.2者11眼,≥0.3者8眼。1眼因视网膜脱离复发,经再次手术硅油充填后视网膜复位。所有患者均无角膜内皮失代偿、人工晶状体移位或硅油残留等。结论硅油填充眼合并白内障行超声乳化联合硅油取出及折叠式人工晶状体植入术能有效提高视力,减少手术次数,提高手术安全性。     

硅油取出、白内障超声乳化摘除联合人工晶状体植入术临床观察

目的探讨玻璃体切除硅油填充术后,硅油取出联合白内障超声乳化摘除联合人工晶状体植入的手术方法和临床效果。方法采用标准三通道睫状体平坦部巩膜切口取出硅油联合白内障超声乳化摘除及人工晶状体植入术,对13例(13只眼)硅油填充眼行三联手术治疗,术后随访超过3月。结果三联手术顺利,最佳矫正视力0.04~0.1者3例,0.1~0.5者9例;术后因再次发生视网膜脱离需再次手术治疗者1例;并发症主要有角膜水肿、黄斑囊样水肿、复发性视网膜脱离及后发性白内障。结论硅油取出联合白内障超声乳化摘除及人工晶状体植入是安全有效的方法,既可以减少手术次数、减轻手术损伤,同时还可以减轻患者的经济负担,能有效提高患者的视力。     

硅油取出联合白内障超声乳化及人工晶状体植入术疗效观察

目的探讨硅油充填眼术后并发性白内障行白内障超声乳化及人工晶体植入联合硅油取出术的临床效果。方法对19例(19只眼)硅油填充术后并发性白内障患者进行经睫状体扁平部切口取硅油联合白内障超声乳化及人工晶状体植入术。结果术后随访3-12个月(平均7个月)。手术后最佳矫正视力光感～眼前指数2只眼,0.02～0.1者4只眼,0.1～0.2者8只眼,0.2以上5只眼。1眼因视网膜脱离复发,经再次手术硅油充填后视网膜复位。1眼术后玻璃体出血,经药物治疗后出血吸收。所有患者均无角膜内皮失代偿、人工晶体移位、硅油残留等。结论硅油取出联合白内障超声乳化及人工晶状体植入术是安全、有效的。严把联合手术的适应症及具备相当的手术技巧是手术成功的关键所在。     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.