肝内胆汁瘤一例

患者　女 ,32岁。上腹包块 1个月伴疼痛入院。查体 :剑突下偏右可扪及一包块约 10ｃｍ× 10ｃｍ ,边界不清 ,活动差。既往史 :1个半月前患者因车祸行肝破裂修补术 ,当时外院ＣＴ报告肝挫裂伤。临床拟诊 :炎性包块。ＣＴ扫描 :相当于肝裂处见一囊状低密度灶 ,推挤相邻肝左、右叶并向下延伸推挤胆囊、胰腺向后下方 ,囊壁光滑 ,囊内为均匀液体密度 ,大小约为 8.8ｃｍ× 8.1ｃｍ× 6 .1ｃｍ(图 1)。ＣＴ诊断 :肝内胆汁瘤。手术所见 :右肝脏面有一约 10ｃｍ× 8ｃｍ大小囊性包块 ,内容物为混浊有“渣”性胆汁 ,无明显管道与肝内或肠内相通 ,壁较…     

医源性肝包膜下胆汁瘤的临床及 CT 表现

目的：探讨医源性肝包膜下胆汁瘤(HSB)的临床及 CT 表现。方法收集本院资料齐全、经穿刺确诊的医源性 HSB 患者7例,检索文献报道的医源性 HSB 病例18例,对其临床及 CT 表现特点进行回顾性分析。结果25例医源性 HSB 中,发生于腹腔镜胆囊切除术后9例,经皮经肝胆道引流术后5例,经皮经肝胆道取石术后4例,开腹胆囊切除术后2例,其他手术5例。CT 表现为肝脏包膜下水样密度影,局限性或弥漫性分布,呈新月形或囊袋状,部分病例伴有肝脏体积缩小,肝表面受压凹陷。结论肝胆手术,特别是腹腔镜及介入手术后,CT 显示肝脏包膜下局限性或弥漫性积液,应考虑 HSB 可能,需及早穿刺确诊并置管引流。     

肝胆汁瘤的介入治疗

目的 探讨肝胆汁瘤的形成原因及介入治疗.方法 2005年至2010年南通市第一人民医院介入科共收治15例胆汁瘤患者,并行介入治疗,其中11例为TACE术后所致,1例外伤引起,3例外科手术引起.治疗方法是通过B超引导下穿刺置管引流,术后间歇应用敏感抗生素冲洗,通过引流液量的变化及影像学资料观察胆汁瘤愈合情况.结果 15例...     

肺动脉主干内粘液瘤一例

患者　女 ,19岁。半年内晕厥 4次 ,均为活动后发作伴胸闷、气急 ,持续约 1分钟后自行好转。查体 :肺动脉瓣区可闻及Ⅲ级ＳＭ杂音 ,粗糙 ,向四周传导 ,与体位无关 ;心电图 :右房、右室增大 ;超声心动图 :肺动脉内占位 ,不除外粘液瘤。ＭＲＩ检查 (图 1～ 3 ) :右室、右房增大 ,于肺动脉瓣平面起向上直达右肺动脉主干近段腔内见血管铸形异常信号 ,Ｔ1ＷＩ呈中等信号 ,Ｔ2 ＷＩ呈不均匀高信号 ,除内侧与肺动脉壁部分相贴外 ,其前、后及外侧尚可见血管流空影 ,肿块边缘不光整。ＭＲＩ诊断 :首先考虑肺动脉主干内肿瘤。手术所见 :右室前壁、肺动…     

肝内胆汁瘤超声显像四例报道

肝内胆汁瘤因病因不同可分为外伤性胆汁瘤、医源性胆汁瘤和自发性胆汁瘤,临床表现缺乏特征性,影像学检查是主要的诊断途径.我们在两年内接诊了4名最终分别诊断为外伤性、自发性和医源性胆汁瘤的患者,现按时间顺序报告各自的超声影像表现及介入性超声的诊治结果.     

肝癌介入治疗后胆汁瘤的形成与临床意义

目的 探讨肝癌介入治疗后肝内胆汁瘤形成的病理基础和临床意义。方法 对３５０例接受ＴＡＥ（经导管肝动脉化疗栓塞）和ＰＥＩ（经皮肝穿刺癌灶内无水酒精清射术）治疗的中晚期肝癌患者进行ＣＴ随访,对疑有胆汁瘤的１１例又行经皮穿刺胆汁瘤造影,１０例行?皮穿刺活检,１例接受外科手术切除,所有胆汁瘤均进行外引流或酒精消融治疗。结果 共发现１１例胆汁瘤（１１／３５０,３．１％）,且均呈囊状扩张（１１／１１）。其中４     

外伤后肝包膜下陈旧性血肿并发脓肿破裂2例

外伤后8～4月肝包膜下陈旧性血肿并发脓肿破裂致全腹膜炎，临床少见，现报告2例。     

肝癌TACE术后破裂并发肝脓肿及肺动脉碘化油栓塞1例

临床资料患者男,67岁.因体检发现肝脏占位1周就诊,既往有乙肝病史20年,不规律服用抗病毒药.入院后完善相关检查,Child-Puge A级,AFP＞1 200 u/ml,CT显示肝内巨大占位,达18 cm×17 cm,诊断为原发性肝癌.外科会诊后已无手术指征,遂行TACE.     

胆汁瘤α-氰基丙烯酸正丁酯封堵1例

临床资料患者男,75岁.“结肠癌术后26个月,胆汁瘤引流3个月来院复查”.患者于26个月前因升结肠癌肝转移行右半结肠切除术,术后肝转移瘤行肝动脉化疗栓塞(TACE)9次,微波消融(MWA)2次,其后发现胆汁瘤,大小11.5 cm×6.8 cm,多次CT复查大小无变化,1年后行胆汁瘤穿刺引流,引流出草绿色胆汁样液体约300 ml,其后每日引流量约100 ml,7 d后引流液浑浊,患者出现发烧症状,行引流液细菌培养,培养出金黄色葡萄球菌和阴沟肠杆菌,给予敏感抗生素治疗后出院.胆汁瘤外引流近3个月,囊腔消失,但引流量逐渐增多,每日约300 ml,遂决定行胆汁瘤栓塞.经引流管造影,见有2个瘘口与胆管相通.     

成功抢救下腔静脉癌栓支架植入后肺栓塞一例

患者男,65岁。因肝细胞肝癌第6次TACE术后半年余,复查MRI发现肝右叶病灶增大伴下腔静脉癌栓形成而入院。患者有乙型肝炎病史30余年,于2007年12月随访AFP升高,CT发现肝左右叶肿瘤,在我院行肝左叶切除术,病理示肝细胞肝癌,分化Ⅱ～Ⅲ级。术后于2008年1月至2009年3月共行6次TACE,手术顺利,术后恢复均可。2009年9月     

A case report of primary pulmonary artery intimal sarcoma

Primary pulmonary artery sarcoma is a rare tumor that mimics pulmonary embolism. Patients may present with cough, dyspnea, chest pain, and weight loss. The diagnosis is challenging. Herein, we report a case of 29-year-old female patient who had presented with dyspnea, fatigue for 2 weeks. Computed tomography pulmonary angiography scan suggests pulmonary embolism. We decided to perform surgical embolectomy. The histopathological results, however demonstrated primary pulmonary artery intimal sarcoma. The patient died 1-month post-surgery because of respiratory and circulatory failure.     

Post-traumatic intrahepatic pseudoaneurysm: A case of successful conservative treatment

Although post-traumatic intrahepatic pseudoaneurysms are rare in adults and even more uncommon in children, this serious complication can result in life-threatening bleeding. Most case studies have recommended surgical or endovascular interventions for preventing rupture or managing the bleeding from a ruptured intrahepatic pseudoaneurysm. Some studies also reported pseudoaneurysms could resolve without intervention and advised conservative therapy. In this case study, we describe a 19-month-old boy diagnosed with an intrahepatic pseudoaneurysm, upper gastrointestinal bleeding from the biliary tract, and hematoma infection. The patient received successful conservative treatment. After 36 days, the patient was discharged without signs of gastrointestinal bleeding and the steady shrinkage of the hematoma surrounding the pseudoaneurysm. The pseudoaneurysm and hematoma vanished at follow-up 18 months after the trauma. Conservative treatment may be considered a viable option, particularly for cases of post-traumatic intrahepatic pseudoaneurysms in children.     

Post-traumatic biloma intrahepatic a rare complication of closed abdominal trauma: A case report

Post-traumatic hepatic biloma is a rare complication of closed trauma of the abdomen. Generally, biloma occurs spontaneously or secondary to traumatic or iatrogenic injury to the biliary system. It can lead to significant morbidity and mortality if not diagnosed promptly and properly managed. A 4-year-old child was admitted to the emergency room with abdominal pain following a traffic accident. Clinical examinations suspected closed abdominal trauma without biological signs of icteric cholestasis. Abdominal CT scan performed within 24 hours showed a focus of hepatic laceration, and follow-up imaging at 5 days showed post-traumatic intrahepatic cystic formations. This case report aims to emphasize the importance of imaging including ultrasound, CT, MRI, MRI cholangiopancreatography (MRCP), or hepatobiliary cholescintigraphy to establish the diagnosis and guide the therapeutic gestures.     

应用冠脉技术成功治疗重度肺动脉瓣狭窄一例

我国于20世纪80年代中期开展经皮球囊肺动脉瓣成形术(PBPV)技术,现已成为治疗先天性肺动脉瓣狭窄(PS)的首选方法。但对于重度狭窄瓣口面积极小的婴幼儿,常规的球     

肝癌合并肝静脉-下腔静脉-右心房癌栓介入诊疗1例北大核心CSCD

1临床资料患者,男,50岁。因“双下肢肿痛11 d”入院。11 d前无明显诱因出现双下肢肿痛,为凹陷性水肿。当地医院腹部超声提示肝硬化、肝实性结节、肝右静脉至下腔静脉内栓子。既往乙肝病史20余年。实验室检查:ALT 240 U/L,AST 192U/L,Alb 38.8 g/L,肌酐132μmol/L,AFP 4.52 ng/mL,CA12564.7 U/mL,余无明显异常。     

Unusual case of cerebral embolism after initiation of selexipag for sarcoidosis-related pulmonary hypertension: a case report

Background: Pulmonary hypertension is a rare complication of sarcoidosis. The pathogenesis of sarcoidosis-related pulmonary hypertension is multifactorial, and patients with sarcoidosis-related pulmonary hypertension can have variable treatment responses and prognoses. While selexipag (Nippon Shinyaku / Kyoto / Japan) was recently approved in Japan for the treatment of pulmonary hypertension, the risk of cerebral infarction has not been clearly reported. Case report: A 63-year-old Asian female with a diagnosis of ocular and cutaneous sarcoidosis developed shortness of breath and was referred to our department to rule out cardiac sarcoidosis. Swan–Ganz catheterization was performed, and she was diagnosed with pulmonary arterial hypertension and started on selexipag. A few days after starting treatment, she presented with hemiplegia and was diagnosed with cardiogenic cerebral embolism by using magnetic resonance imaging. As there was no evidence of pre-existing intracardiac thrombosis, we suspected unusual cerebral embolism. Echocardiography revealed a deep venous thrombus and a bubble study revealed a right-left shunt through a patent foramen ovale. Conclusions: The initiation of selexipag improved pulmonary blood flow and caused cerebral embolism, which was an unusual and unexpected event. This report highlights the importance of confirming the presence of patent foramen ovale and a deep venous thrombus before starting treatment for pulmonary hypertension.     

冠状动脉-支气管动脉瘘一例

病史摘要 患者男,67岁.因反复发作胸闷、心悸6年加重5 d入院.患者6年前无明显诱因出现心悸、心动过速发作,伴胸闷.此后反复出现心悸发作,无明显胸痛,偶伴夜间阵发性呼吸困难,平时稍动则喘,不能登3楼.