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N. V. Sanotskaya V. P. Mikhailov D. D. Matsievskii 《Bulletin of experimental biology and medicine》1990,110(6):1620-1624
Laboratory of Pathophysiology of Respiration and Bioengineering Laboratory, Research Institute of General Pathology and Pathological Physiology, Academy of Medical Sciences of the USSR. Department of Pathophysiology, Yaroslavl' Medical Institute. (Presented by Academician of the Academy of Medical Sciences of the USSR G. N. Kryzhanovskii.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 110, No. 12, pp. 582–586, December, 1990. 相似文献
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V. P. Mikhailov L. D. Smirnov G. V. Kurygin N. N. Zolotov 《Bulletin of experimental biology and medicine》1992,113(2):175-177
Department of Pathophysiology, Yaroslavl' State Medical Institute. All-Union Research Center for Safety of Biologically Active Substances, Kupavna, Moscow Region. (Presented by Academician of the Academy of Medical Sciences of the USSR A. G. Chuchalin.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 113, No. 2, pp. 139–141, February, 1992. 相似文献
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V. P. Mikhailov L. D. Smirnov N. N. Zolotov 《Bulletin of experimental biology and medicine》1990,109(6):753-755
Department of Pathophysiology, Yaroslavl' Medical Institute. Research Institute of Drug Technology and Safety, Kupavna, Moscow Region. (Presented by Academician of the Academy of Medical Sciences of the USSR A. G. Chuchalin.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol 109, No. 6, pp. 565–567, June, 1990. 相似文献
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Laboratory of General Pathology of the Nervous System, Research Institute of General Pathology and Pathological Physiology, Academy of Medical Sciences of the USSR, Moscow. Department of Pathological Physiology, Yaroslavl' Medical Institute. (Presented by Academician of the Academy of Medical Sciences of the USSR G. N. Kryzhanovskii.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 105, No. 2, pp. 152–154, February, 1988. 相似文献
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目的探讨神经源性肺水肿的病因、诊断及治疗方法。方法回顾分析7例重型脑损伤后继发神经源性肺水肿患者救治情况及综合数点经验。结果临床资料提示血中儿茶酚胺(CA)升高造成肺泡萎陷、肺泡腔及肺间隙水肿,是引起通气和换气功能障碍、出现呼吸困难及严重缺的主要原因。采用降低颅内压、及时气管切开、机控呼吸及应用血管活性物质等综合治疗措施,使4例患者获救。结论本病起病急、病情凶险、死亡率高,早期诊断及合理治疗可提高其治疗效果。 相似文献
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N. V. Sanotskaya V. V. Polikarpov D. D. Matsievskii 《Bulletin of experimental biology and medicine》1989,107(2):163-167
Laboratory of Pathophysiology of Respiration and Bioengineering Laboratory, Research Institute of General Pathology and Pathological Physiology, Academy of Medical Sciences of the USSR, Moscow. Department of Pathological Physiology, Yaroslavl' Medical Institute. (Presented by Academician of the Academy of Medical Sciences of the USSR B. I. Tkachenko.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 107, No. 2, pp. 157–161, February, 1989. 相似文献
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N. V. Sanotskaya V. P. Mikhailov D. D. Matsievskii 《Bulletin of experimental biology and medicine》1990,110(3):1186-1189
Laboratory of Pathophysiology of Respiration and Bioengineering Laboratory, Research Institute of General Pathology and Pathological Physiology, Academy of Medical Sciences of the USSR, Moscow. Department of Pathophysiology, Yaroslavl' Medical Institute. (Presented by Academician of the Academy of Medical Sciences of the USSR B. I. Tkachenko.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 110, No. 9, pp. 254–257 September, 1990. 相似文献
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S. B. Tkachenko N. V. Sanotskaya D. D. Matsievskii 《Bulletin of experimental biology and medicine》1990,109(2):147-150
Laboratory of Pathophysiology of Respiration and Bioengineering Laboratory, Research Institute of General Pathology and Pathological Physiology, Academy of Medical Sciences of the USSR, Moscow. (Presented by Academician of the Academy of Medical Sciences of the USSR B. B. Moroz.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 109, No. 2, pp. 126–128, February, 1990. 相似文献
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目的:探讨高原肺水肿的发病机理。方法:采用右心漂浮导管检测法, 对9例高原肺水肿患者及9例同海拔高原健康人的血流动力学指标进行了检测, 同时也观察了吸入纯氧对高原肺水肿患者血流动力学的影响。结果:高原肺水肿患者发病时, 肺动脉平均压、肺血管阻力、心脏指数均明显高于同海拔高度健康人, 而患者肺动脉楔压, 右心房压力同对照组相比, 未见显著差异;吸氧后, 高原肺水肿患者心率、肺动脉平均压力, 肺血管阻力及心脏指数均较吸氧前明显下降, 特别是肺动脉平均压及肺血管阻力下降尤为明显, 肺动脉平均压力在吸氧1min后即明显下降, 吸氧5min后, 下降至最低值, 但吸氧20min后仍未达对照组水平。结论:高原肺水肿是非心源性肺水肿, 肺动脉高压在其发病中起重要作用。 相似文献
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Beena K. Parikh Guruprasad P. Bhosale Veena R. Shah 《Indian Journal of Critical Care Medicine》2011,15(4):230-232
Non-cardiogenic pulmonary edema (NCPE) is a clinical syndrome characterized by simultaneous presence of severe hypoxemia, bilateral alveolar infiltrates on chest radiograph, without evidence of left atrial hypertension/congestive heart failure/fluid overload. The diagnosis of drugrelated NCPE relies upon documented exclusion of other causes of NCPE like gastric aspiration, sepsis, trauma, negative pressure pulmonary edema. We describe a 28year-old, 50 kg male with ASA risk III posted for laparoscopic renal transplantation, who developed NCPE after 4 hours of administration of rabbit anti-human thymocyte immunoglobulin (ATG). He was successfully treated with mechanical ventilatory support and adjuvant therapy. This report emphasizes that this fatal complication may occur with use of ATG. 相似文献
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A 37-year-old woman was admitted to Dong-A University Hospital for rapidly progressive congestive heart failure. Transthoracic echocardiography demonstrated a large mass with a stalk that appeared to be a myxoma on the posterior wall of the left atrium. However, the histological diagnosis was undifferentiated pleomorphic sarcoma. We report a case of primary undifferentiated pleomorphic sarcoma of the left atrium with acute pulmonary edema caused by mitral inflow obstruction. 相似文献
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To report a non-fatal case of reperfusion pulmonary edema (RPE) after the removal of a hepatocellular carcinoma embolus, which had caused an acute obstruction of the tricuspid valve and pulmonary vasculature during a hepatic lobectomy. Pulmonary embolism caused by hepatocellular carcinoma embolus is extremely rare, and, in the present case, it was associated with unusual clinical features. A 69-year-old ASA II woman with hepatocellular carcinoma was presented for an elective left hepatic lobectomy. During the surgery, the tumor embolus was dislodged from the interior of the lumen of the inferior vena cava (IVC), which then drifted into the tricuspid valve area and pulmonary vasculature. The patient showed the specific signs of acute pulmonary embolism, such as a reduction in end-tidal carbon dioxide, an increase in central venous pressure, and a decrease in arterial pressure. The patient exhibited the symptoms for about 10 minutes. After this period, however, cardiovascular variables became relatively stable, even during a mechanical obstruction due to cross-clamping the pulmonary artery for embolectomy. After several hours of pulmonary embolectomy, the patient experienced an episode of RPE. The ventilatory supports for the treatment of RPE were successful, and the patient recovered without any complications. The patient's case in the present study demonstrates that pulmonary embolism may occur as a result of a hepatocellular carcinoma extending into the IVC during operative management. The anesthesiologist should be careful of the possibilities of RPE after removal of the tumor embolus. 相似文献
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Zygomycosis is an uncommon but frequently fatal infection and occurs mostly in immunosuppressed hosts, whereas approximately 50% of zygomycosis occurs in diabetic patients. The current patient initially presented with persistent pulmonary edema secondary to renal failure. This was the last of four admissions within 1 year for this 68-year-old woman, for whom the chief complaints were shortness of breath and chest pain. Her past medical history included insulin-requiring type 2 diabetes and hypertension for 10 years, and chronic heart and renal failure. She was previously admitted to the hospital for what appeared to be pulmonary edema secondary to renal failure. In the last admission the patient developed pulmonary hemorrhage and metabolic acidosis. Transbronchial biopsy was performed, showing irregular fungal hyphae in the blood vessels, morphologically consistent with zygomycosis. Central nervous system computed tomography also revealed a large infarct in the cerebral hemisphere. The patient died on the seventh hospital day. At autopsy three organs were extensively involved by zygomycosis: (i) lungs were diffusely hemorrhagic with acute infarcts; (ii) pericardium had fibrotic inflammation; and (iii) the left cerebral hemisphere, cerebellum and pons had large hemorrhagic infarct by zygomycosis infection. Corticosteroid medication and hemodialysis triggered increasing hyperglycemia, metabolic acidosis and iron overload, which contributed to zygomycosis infection that subsequently spread to the heart and brain as a rare consequence. 相似文献
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I. A. Tarakanov S. A. Kuz'michev G. A. Semkina 《Bulletin of experimental biology and medicine》1992,114(1):926-928
Research Institute of General Pathology and Pathological Physiology, Russian Academy of Medical Sciences. Department of Human and Animal Physiology, M. V. Lomonosov Moscow State University (Presented by Academician of the Russian Academy of Medical Sciences G. N. Kryzhanovskii.) Translated from Byulleten' Éksperimental'noi Biologii i Meditsiny, Vol. 114, No. 7, pp. 18–19, July, 1992. 相似文献