共查询到20条相似文献,搜索用时 31 毫秒
1.
《Journal of stroke and cerebrovascular diseases》2020,29(11):105212
IntroductionEncephalopathy is a common complication of coronavirus disease 2019. Although the encephalopathy is idiopathic in many cases, there are several published reports of patients with posterior reversible encephalopathy syndrome in the setting of coronavirus disease 2019.ObjectiveTo describe the diverse presentations, risk factors, and outcomes of posterior reversible encephalopathy syndrome in patients with coronavirus disease 2019.MethodsWe assessed patients with coronavirus disease 2019 and a diagnosis of posterior reversible encephalopathy syndrome at our institution from April 1 to June 24, 2020. We performed a literature search to capture all known published cases of posterior reversible encephalopathy syndrome in patients with coronavirus disease 2019.ResultsThere were 2 cases of posterior reversible encephalopathy syndrome in the setting of coronavirus 2019 at our institution during a 3-month period. One patient was treated with anakinra, an interleukin-1 inhibitor that may disrupt endothelial function. The second patient had an underlying human immunodeficiency virus infection. We found 13 total cases in our literature search, which reported modest blood pressure fluctuations and a range of risk factors for posterior reversible encephalopathy syndrome. One patient was treated with tocilizumab, an interleukin-6 inhibitor that may have effects on endothelial function. All patients had an improvement in their neurological symptoms. Interval imaging, when available, showed radiographic improvement of brain lesions.ConclusionsRisk factors for posterior reversible encephalopathy syndrome in patients with coronavirus disease 2019 may include underlying infection or immunomodulatory agents with endothelial effects in conjunction with modest blood pressure fluctuations. We found that the neurological prognosis for posterior reversible encephalopathy syndrome in the setting of coronavirus disease 2019 infection is favorable. Recognition of posterior reversible encephalopathy syndrome in this patient population is critical for prognostication and initiation of treatment, which may include cessation of potential offending agents and tight blood pressure control. 相似文献
2.
Khayat-Khoei Mahsa Bhattacharyya Shamik Katz Joshua Harrison Daniel Tauhid Shahamat Bruso Penny Houtchens Maria K. Edwards Keith R. Bakshi Rohit 《Journal of neurology》2022,269(3):1093-1106
Journal of Neurology - The availability of vaccines against the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), provides hope towards mitigation of the coronavirus disease 2019... 相似文献
3.
Rettenmaier Leigh A. Abdel-Wahed Lama Abdelmotilib Hisham Conway Kyle S. Narayanan Nandakumar Groth Christopher L. 《Journal of neurovirology》2022,28(1):172-176
Journal of NeuroVirology - Acute necrotizing encephalopathy (ANE) is a rare complication of coronavirus disease 2019 (COVID-19) secondary to severe acute respiratory syndrome coronavirus 2... 相似文献
4.
Morassi Mauro Bagatto Daniele Cobelli Milena D’Agostini Serena Gigli Gian Luigi Bnà Claudio Vogrig Alberto 《Journal of neurology》2020,267(8):2185-2192
Journal of Neurology - Italy is one of the most affected countries by the coronavirus disease 2019 (COVID-19). The responsible pathogen is named severe acute respiratory syndrome coronavirus... 相似文献
5.
Aslan Cynthia Nikfarjam Sepideh Asadzadeh Mohammad Jafari Reza 《Journal of neurovirology》2021,27(2):217-227
Journal of NeuroVirology - The novel coronavirus disease 2019 (COVID-19) caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has instigated a global pandemic as a formidable... 相似文献
6.
Emekli Ahmed Serkan Parlak Asuman Göcen Nejla Yılmaz Kürtüncü Murat 《Neurological sciences》2021,42(10):3995-4002
Neurological Sciences - The coronavirus disease 2019 (COVID-19), caused by the novel severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), continues to spread rapidly all over the world.... 相似文献
7.
Saqib Amin 《International Journal of Mental Health Promotion, The》2020,22(1):29-38
Coronaviruses are a category of associated viruses that trigger disease
in mammals and birds. Human coronaviruses have been identified including
severe acute respiratory syndrome-related coronavirus (SARS-CoV) in 2003,
human coronavirus NL63 (HCoV NL63) in 2004, human coronavirus HKU1
(HKU1) in 2005, Middle East respiratory syndrome-related coronavirus (MERSCoV) in 2012, and severe acute respiratory syndrome-related coronavirus-2
(SARS-CoV-2) in December, 2019. This study aims to examine whether social
media at residing/admittance in quarantine ward (due to corona virus pandemic
disease) affects psychological health or not? We asked questions from 250
quarantined patients infected from coronavirus (restricted to quarantine ward)
about their psychological health who have accessed of different channels of
social media. This study concludes that social media has caused of various
psychological symptoms in quarantined patients (due to coronavirus) in form of
anxiety, depression, negative well-being, lack of self-control, general health and
vitality. This study suggests that there is an urgency to provide awareness/right
information to each infected individual and their family members to create
peaceful society and social rest. Thus, productive use of social media is
recommended while minimizing its negative impact. 相似文献
8.
We describe a man whose first manifestations of Creutzfeldt-Jakob disease occurred in tandem with symptomatic onset of coronavirus disease 2019 (COVID-19). Drawing from recent data on prion disease pathogenesis and immune responses to SARS-CoV-2, we hypothesize that the cascade of systemic inflammatory mediators in response to the virus accelerated the pathogenesis of our patient’s prion disease. This hypothesis introduces the potential relationship between immune responses to the novel coronavirus and the hastening of preclinical or manifest neurodegenerative disorders. The global prevalence of both COVID-19 and neurodegenerative disorders adds urgency to the study of this potential relationship. 相似文献
9.
The etiology of Parkinson's disease remains unknown, and a search for environmental agents continues. In 1985, Fishman induced infection of the basal ganglia by a coronavirus in mice. Although coronavirus is recognized primarily as a respiratory pathogen in humans, its affinity for the basal ganglia led us to investigate its possible role in human Parkinson's disease. The cerebrospinal fluid of normal controls (CTL) (n = 18), and patients with Parkinson's disease (PD (n = 20) and other neurological disease (OND) (n = 29) was analyzed in a blinded manner by enzyme-linked immunosorbent assay [measurements in optical density (OD) units] for antibody response to four coronavirus antigens: mouse hepatitis virus JHM (J) and A59 (A), and human coronavirus 229E (E) and OC43 (O). When compared with CTL, PD patients had an elevated (p less than 0.05) mean OD response to J (0.0856 vs. 0.0207) and A (0.1722 vs. 0.0636). Response (p greater than 0.05) to O (0.0839 vs. 0.0071) was greater than that to E (0.1261 vs. 0.0743). When compared to OND, PD patients had an elevated mean OD response to J (0.0856 vs. 0.0267, p less than 0.05). Responses (p greater than 0.05) to A (0.1722 vs. 0.0929) and O (0.0839 vs. 0.0446) were greater than that to E (0.1261 vs. 0.0946). These results suggest that there may be an association between coronavirus and PD. 相似文献
10.
Although health anxiety and corresponding safety behaviors can facilitate disease transmission avoidance, they can be maladaptive in excess, including during the coronavirus pandemic. Disgust proneness (i.e., tendency to experience and be sensitive to disgust) is one factor that may predict elevated coronavirus anxiety and safety behaviors during the pandemic, given the role of disgust in avoiding disease transmission. The present study examined the relations between pre-pandemic disgust proneness and coronavirus anxiety and safety behaviors in community adults who completed a 2016 study and were re-contacted on 4/1/2020 (N = 360). Interactions between pre-pandemic disgust proneness and current perceived stress were tested to examine a diathesis-stress model of the role of disgust proneness in anxiety response to the pandemic. Increased pre-pandemic disgust proneness predicted increased coronavirus anxiety and safety behaviors, controlling for number of COVID-19 cases by state. Consistent with a diathesis-stress model, current perceived stress moderated this effect, such that highest coronavirus anxiety and safety behaviors were reported by those with high disgust proneness and high stress. Trait disgust proneness may be a vulnerability factor for anxiety responses to the coronavirus pandemic, particularly among individuals experiencing high stress. Assessing disgust proneness and current stress may facilitate targeted anxiety intervention during the pandemic. 相似文献
11.
Karadas Omer Ozturk Bilgin Sonkaya Ali Rıza Duzgun Ulkuhan Shafiyev Javid Eskin Mehmet Burak Bostan Tehlil Ozon Akcay Ovunc 《Neurological sciences》2022,43(4):2277-2283
Neurological Sciences - Coronavirus disease (COVID-19) is an infectious disease caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). The disease was declared a pandemic on... 相似文献
12.
Neurotoxicity Research - As a severe and highly contagious infectious disease, coronavirus disease 2019 (COVID-19) has caused a global pandemic. Several case reports have demonstrated that the... 相似文献
13.
Ghosh Ritwik Roy Dipayan Sengupta Samya Benito-León Julián 《Journal of neurovirology》2020,26(6):964-966
Journal of NeuroVirology - Albeit primarily a disease of respiratory tract, the 2019 coronavirus infectious disease (COVID-19) has been found to have causal association with a plethora of... 相似文献
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15.
Siepmann Timo Sedghi Annahita Barlinn Jessica de With Katja Mirow Lutz Wolz Martin Gruenewald Thomas Helbig Sina Schroettner Percy Winzer Simon von Bonin Simone Moustafa Haidar Pallesen Lars-Peder Rosengarten Bernhard Schubert Joerg Gueldner Andreas Spieth Peter Koch Thea Bornstein Stefan Reichmann Heinz Puetz Volker Barlinn Kristian 《Journal of neurology》2021,268(3):773-784
Journal of Neurology - To determine whether a history of cerebrovascular disease (CVD) increases risk of severe coronavirus disease 2019 (COVID-19). In a retrospective multicenter study, we... 相似文献
16.
Pierre J. Talbot Jean-Sbastien Paquette Cristina Ciurli Jack P. Antel France Ouellet 《Annals of neurology》1996,39(2):233-240
Multiple sclerosis (MS) is an inflammatory demyelinating neurological disease in which autoreactive T lymphocytes sensitized to myelin components of the central nervous system are postulated to contribute to pathogenesis. The possible relevance of molecular mimicry between a human coronavirus and the myelin basic protein component of myelin in the generation of this autoimmune reaction was evaluated. Myelin basic protein– and virus-reactive T-cell lines were established from 16 MS patients and 14 healthy donors and shown to be mostly CD4+. In contrast to healthy donors, several T-cell lines isolated from MS patients showed cross-reactivity between myelin and coronavirus antigens. Overall, 29% of T-cell lines from MS patients (10 donors) but only 1.3% of T-cell lines from normal control subjects (2 donors) showed an HLA-DR-restricted cross-reactive pattern of antigen activation after in vitro selection with either myelin basic protein or human coronavirus strain 229E antigens. Moreover, reciprocal reactivities were only observed in MS patients (4 donors). This establishes molecular mimicry between a common viral pathogen, such as this human coronavirus, and myelin as a possible immunopathological mechanism in MS and is consistent with the possible involvement of more than one infectious pathogen as an environmental trigger of disease. 相似文献
17.
The coronavirus disease 2019 pandemic represents an enduring transformation in health care and education with the advancement of smart universities, telehealth, adaptive research protocols, personalized medicine, and self-controlled or artificial intelligence-controlled learning. These changes, of course, also cover mental health and long-term realignment of coronavirus disease 2019 survivors. Fatigue or anxiety, as the most prominent psychiatric “long coronavirus disease 2019” symptoms, need a theory-based and empirically-sound procedure that would help us grasp the complexity of the condition in research and treatment. Considering the systemic character of the condition, such strategies have to take the whole individual and their sociocultural context into consideration. Still, at the moment, attempts to build an integrative framework for providing meaning and understanding for the patients of how to cope with anxiety when they are confronted with empirically reduced parameters (e.g., severe acute respiratory syndrome coronavirus type 2) or biomarkers (e.g., the FK506 binding protein 5) are rare. In this context, multidisciplinary efforts are necessary. We therefore join in a plea for an establishment of ‘translational medical humanities’ that would allow a more straightforward intervention of humanities (e.g., the importance of the therapist variable, continuity, the social environment, etc) into the disciplinary, medial, political, and popular cultural debates around health, health-care provision, research (e.g., computer scientists for simulation studies), and wellbeing. 相似文献
18.
Vinores SA Wang Y Vinores MA Derevjanik NL Shi A Klein DA Detrick B Hooks JJ 《Journal of neuroimmunology》2001,119(2):175-182
Intraocular coronavirus inoculation results in a biphasic retinal disease in susceptible mice (BALB/c) characterized by an acute inflammatory response, followed by retinal degeneration associated with autoimmune reactivity. Resistant mice (CD-1), when similarly inoculated, only develop the early phase of the disease. Blood-retinal barrier (BRB) breakdown occurs in the early phase in both strains, coincident with the onset of inflammation. As the inflammation subsides, the extent of retinal vascular leakage is decreased, indicating that BRB breakdown in experimental coronavirus retinopathy (ECOR) is primarily due to inflammation rather than to retinal cell destruction. Vascular endothelial growth factor (VEGF) is upregulated only in susceptible mice during the secondary (retinal degeneration) phase. 相似文献
19.
Corrêa Diogo Goulart Hygino da Cruz Jr. Luiz Celso Lopes Fernanda Cristina Rueda Rangel Cláudio de Carvalho de Araújo Henriques Tessarollo Anna Luiza Coelho Karla Cristina Godeiro Brandão Rafael Zandonadi Novis Ricardo Augusto Faro Novis Shenia Sbardellotto Colnaghi Silveira Rodrigo Carvalho Viviane T. Nascimento Osvaldo J. M. 《Journal of neurovirology》2021,27(1):171-177
Journal of NeuroVirology - The complete features of the neurological complications of coronavirus disease 2019 (COVID-19) still need to be elucidated, including associated cranial nerve... 相似文献
20.
Wang Haili Lu Juan Zhao Xia Qin Rongyin Song Kangping Xu Yao Zhang Jun Chen Yingzhu 《Neurological sciences》2021,42(12):4913-4920
Neurological Sciences - Advanced age correlates with higher morbidity and mortality among patients affected with the novel coronavirus disease 2019 (COVID-19). Because systemic inflammation and... 相似文献