嗜酸性粒细胞性胃肠炎伴腹水1例报道

嗜酸性粒细胞性胃肠炎(eosinophilic gastroenteritis,EG)是一种以胃肠道组织中嗜酸性粒细胞异常浸润为特征的罕见胃肠道疾病。随着医务工作者对该病的认识不断深入,该病的文献报道逐渐增多。到目前为止,国内已有100余例报道[1],但因其临床表现缺乏特异性,故该病误诊率仍较高。本文报道我院2013年接诊的1例EG伴腹水的病例。     

小儿嗜酸性粒细胞性胃肠炎4例临床及胃镜特点分析

嗜酸性粒细胞性胃肠炎(eosinophilic gastricoenteritis,EG),是一种少见的胃肠道疾病,以胃肠道某些部位的弥漫性或局限性嗜酸性粒胞浸润为特征.自1937年Kaijser首次报道以来,国外报道了EG患儿20余例[1],国内报道EG患儿不足10例[1-3].我科2010年1月至8月间收治了4例EG患儿,报道如下.     

嗜酸细胞性胃肠炎的临床特点

嗜酸细胞性胃肠炎亦称嗜酸性胃肠炎（eosinophilic gastroenteritis，EG）是一种少见疾病，以胃肠道的某些部位弥漫性或局限性嗜酸性粒细胞浸润为特征。临床表现为腹部痉挛性疼痛，可伴恶心、呕吐、发热等，部分患者有特殊食物过敏史。约80％的患者外周血嗜酸性粒细胞高达15％-70％。Kaijser于1937年首先报道，迄今世界文献报道约300余例。     

误诊为嗜酸性粒细胞性胃肠炎的Cronkhite-Canada综合征1例报道

Cronkhite-Canada综合征(Cronkhite-Canada syndrome, CCS)是一种罕见的非遗传性疾病,其主要特征表现为胃肠道多发息肉、脱发、指甲营养不良、色素沉着、体质量减轻、腹泻等。该病病因不明,自身免疫因素可能是重要的病理生理过程,诊断主要依据病史、体格检查、消化道的内镜表现和组织病理学检查。迄今为止,关于CCS的文献大多是病例报道,全球报道的CCS病例仅500余例。嗜酸性粒细胞性胃肠炎(eosinophilic gastroenteritis, EG)是一种少见的炎症性疾病,以胃肠道嗜酸性粒细胞(eosinophils, EOS)浸润为主要表现。本文报道1例被误诊为EG的CCS患者,以供临床借鉴。     

嗜酸细胞性胃肠炎四例并文献复习

嗜酸细胞性胃肠炎（eosinophilic gastroentefitis，EG）是以胃肠道组织中嗜酸细胞异常浸润为特征的胃肠道疾病，可累及整个消化道。由于该病较少见，临床表现呈多样性，所以往往被误诊。我们分析了本院4例EG患者，结合文献探讨该病的临床特点和诊治方法。     

嗜酸粒细胞性胃肠炎的临床分析

嗜酸粒细胞性胃肠炎（EG）亦称变应性胃肠炎,是一种少见的胃肠道疾病,但近几年日趋增多.现将1980至2005年我院消化内科诊治的15例EG病例报道如下.     

嗜酸性胃肠炎临床分析

目的探讨嗜酸性胃肠炎(Eosinophilic Gastroenteritis,EG)病因、诊断、误诊原因、治疗方法.方法对我院7例嗜酸性胃肠炎临床表现、实验室检查、特殊检查结果进行分析,结合文献报道41例患者的资料,分析EG发病诱因,临床特点、内镜特征和误诊原因及治疗预后.结果EG患者以腹痛为首发症状,主要症状为腹痛、恶心、呕吐、腹泻,内镜下胃肠粘膜呈充血、糜烂为主,误诊原因:1.嗜酸性胃肠炎为临床少见病;2.临床表现多样、无特异性;3.特殊检查不能有效的提示.肌型属罕见型.皮质激素治疗疗效好,作用迅速.结论EG临床表现多样性、无特异性;易误诊,肌型属罕见型,皮质激素治疗疗效好,作用迅速.     

10例嗜酸细胞性胃肠炎内镜及影像检查分析

嗜酸细胞性胃肠炎（eosinophilic gastroenteritis,EG）是一种少见疾病,且临床表现多样性,极易引起误诊。多数研究认为,内镜及影像表现对该病诊断无特异性,本文回顾分析我院2003年3月至2007年12月确诊为EG10例患者的内镜及影像检查结果,报道如下。     

嗜酸细胞性胃肠炎1例和文献回顾

嗜酸细胞性胃肠炎由Kaizser于1937年首先报道,迄今世界文献报道约300余例[1].可发生于各年龄段,成人多在30～50岁之间发病.临床表现无特异性,可表现为腹部痉挛性疼痛,伴恶心、呕吐、发热、腹水或特殊食物过敏史.     

内镜检查对嗜酸细胞性胃肠炎的诊断意义

嗜酸细胞性胃肠炎(eosinophilic gastroenteritis,EG)系指胃壁和(或)肠壁以嗜酸粒细胞(EO)浸润为特征的慢性疾病.临床表现具有多样性和非特异性,易误诊[1,2].本研究旨在探讨内镜及病理特点在EG的诊断价值,加深对该病的认识,提高诊断率.     

State of knowledge of helminth fauna of freshwater fishes of Poland

A total 89 fish and lamprey species has been recorded from Polish freshwater habitats. Twenty-seven of them (30.3%) have not been surveyed for parasitic helminthes. Some of the latter fishes are either rare or not easily accessible. Other live only in specific habitats in scattered localities. An important obstacle for studying parasite faunas of some fishes may be their status on an endangered species. Among the non-surveyed fishes, are those which have been relatively recently introduced to Poland or migrated there on their own. The present paper attempts to review all hitherto not studied helminthologically fish species, their habitats, localities and current protection status.     

高血压降压治疗目标的再认识

根据传统的高血压水平的定义,1993年WHO高血压治疗指南提出血压控制目标为<140/90mm Hg(1mm Hg=0.133kPa),但是并非所有患者都必须将血压降至同一水平,而应根据患者情况进行个体化治疗。Framingham进行的一项长达10～12年的心血管事件研究发现,第5年后,正常上限血压[收缩压(SBP     

Lack of correlation of degree of villous atrophy with severity of clinical presentation of coeliac disease

BACKGROUND AND AIM: Both the clinical presentation and the degree of mucosal damage in coeliac disease vary greatly. In view of conflicting information as to whether the mode of presentation correlates with the degree of villous atrophy, we reviewed a large cohort of patients with coeliac disease. PATIENTS AND METHODS: We correlated mode of presentation (classical, diarrhoea predominant or atypical/silent) with histology of duodenal biopsies and examined their trends over time. RESULTS: The cohort consisted of 499 adults, mean age 44.1 years, 68% females. The majority had silent coeliac disease (56%) and total villous atrophy (65%). There was no correlation of mode of presentation with the degree of villous atrophy (p=0.25). Sixty-eight percent of females and 58% of males had a severe villous atrophy (p=0.052). There was a significant trend over time for a greater proportion of patients presenting as atypical/silent coeliac disease and having partial villous atrophy, though the majority still had total villous atrophy. CONCLUSIONS: Among our patients the degree of villous atrophy in duodenal biopsies did not correlate with the mode of presentation, indicating that factors other than the degree of villous atrophy must account for diarrhoea in coeliac disease.