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Expansion of CREB''s DNA recognition specificity by Tax results from interaction with Ala-Ala-Arg at positions 282-284 near the conserved DNA-binding domain of CREB. 总被引:17,自引:1,他引:17
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N Adya L J Zhao W Huang I Boros C Z Giam 《Proceedings of the National Academy of Sciences of the United States of America》1994,91(12):5642-5646
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Hepatitis B virus transactivator protein, HBx, associates with the components of TFIIH and stimulates the DNA helicase activity of TFIIH. 总被引:10,自引:0,他引:10
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I Qadri J W Conaway R C Conaway J Schaack A Siddiqui 《Proceedings of the National Academy of Sciences of the United States of America》1996,93(20):10578-10583
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The trans-activator tax of human T-cell leukemia virus type 1 (HTLV-1) interacts with cAMP-responsive element (CRE) binding and CRE modulator proteins that bind to the 21-base-pair enhancer of HTLV-1. 总被引:31,自引:1,他引:31
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T Suzuki J I Fujisawa M Toita M Yoshida 《Proceedings of the National Academy of Sciences of the United States of America》1993,90(2):610-614
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Human T-cell lymphotropic virus type I (HTLV-I) transcriptional activator, Tax, enhances CREB binding to HTLV-I 21-base-pair repeats by protein-protein interaction. 总被引:40,自引:3,他引:40
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L J Zhao C Z Giam 《Proceedings of the National Academy of Sciences of the United States of America》1992,89(15):7070-7074
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Functional antagonism between the retinoic acid receptor and the viral transactivator BZLF1 is mediated by protein-protein interactions. 总被引:3,自引:1,他引:3
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E Pfitzner P Becker A Rolke R Schüle 《Proceedings of the National Academy of Sciences of the United States of America》1995,92(26):12265-12269
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Identification of a PU.1-IRF4 protein interaction surface predicted by chemical exchange line broadening
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McKercher SR Lombardo CR Bobkov A Jia X Assa-Munt N 《Proceedings of the National Academy of Sciences of the United States of America》2003,100(2):511-516
Relaxation values reflecting residue-specific line broadening revealed amino acids in the DNA-binding domain of PU.1 on a surface potentially involved in protein-protein interactions. Mutation of these amino acids did not cause protein unfolding but destabilized PU.1-DNA binding. Addition of IFN response factor 4 to form the ternary complex recovered binding stability. Fluorescence quenching experiments proved that this surface of PU.1 interacts with IFN response factor 4 during binding. Our results provide evidence that residues that display increased conformational exchange can be used to predict areas of protein-protein interactions. 相似文献
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