中国西部下盾螨属一新种（蜱螨亚纲：厉螨科）

本文记述厉螨科Ｌａｅｌａｐｉｄａｅ Ｂｅｒｌｅｓｅ，１８９２下盾螨属Ｈｙｐｏａｓｐｉｓ ．Ｇ．Ｃａｎｅｓｔｒｉｎｉ，１８８５一新种，命我为青海下盾螨Ｈｙｐｏａｓｐｉｓ ｑｉｎｇｈａｉｅｎｓｉｓ ｓｐ．ｎｏｖ。模式标本采自青海省同德县巴水乡的长尾仓鼠Ｃｒｉｃｅｔｕｌｕｓ ｌｏｎｇｉｃａｕｄａｔｕｓ体，新种与鼠下盾螨Ｈｙｐｏａｓｐｉｓ ｓｃｒｅｃｉｓ Ｌｉ，Ｚｈｅｎｇ ｅｔ Ｙａｎｇ，１９９６接近，但     

旱獭青厉螨雄性的描述（蜱螨亚纲：厉螨科）

旱獭青厉螨雄性的描述（蜱螨亚纲：厉螨科）李明立李超杨锡正（青海省地方病防治研究所，西宁市８１１６０２）关键词旱獭青厉螨；雄性旱獭青厉螨Ｑｉｎｇｈａｉｌａｅｌａｐｓｍａｒｍｏｔａｅ是由顾以铭、杨锡正于１９８４年根据描述其雌性个体的新种（动物分类学报９（...     

茅舍血厉螨侵袭人体皮肤1例

本文报道了1例由茅舍血厉螨侵袭人体引起的螨性皮炎病例。形态学鉴定发现,检获的螨虫为茅舍血厉螨后若螨。该螨侵袭人体后导致患者皮肤局部出现丘疹、水疱等不同程度皮炎。对患者使用15%炉甘石洗剂和消炎止痒药治疗皮炎的同时进行居室杀螨,治疗1周后患者痊愈。居室床垫表面检获的茅舍血厉螨可侵袭人体致革螨性皮炎,应重视居室螨虫防治。     

中卫血厉螨雄性记述（蜱螨亚纲：厉螨科）

首次对中卫血厉螨Haemolaelaps zhongweiensis Bai,Chen et Wang 1987雄性进行记述。标本采自银川市月牙湖鼠疫监测点金龟（Geotrupes sp.）体上,保存于军事医学科学院微生物流行病研究所昆虫标本馆。     

耶氏厉螨及其在我国不同地区形态的差异（蜱螨亚纲：厉螨科）

耶氏厉螨;;巴氏厉螨;;极厉螨;;敏捷厉螨;;同物异名     

青海省革螨名录

螨类为一庞大的节肢动物类群 ,广泛寄生于啮齿类、食虫类等宿主动物体表及巢穴 ,部分类群是重要医学昆虫 ,传播多种自然疫源性疾病 ,如流行性出血热、鼠疫、地方性斑疹伤寒、森林脑炎等 ,是预防医学、医学昆虫学重点研究对象之一。我省对螨类的研究起步较晚 ,2 0世纪 80年代初结合鼠疫自然疫源地调查和监测工作进行了较为系统的区系研究 ,迄今已知革螨 89种 ,隶属 11科 2 6属。为方便医学昆虫工作者查找资料 ,现将青海省革螨名录报道如下。1　厉螨科 L aelapidae Berlese,18921.1　厉螨属 L aelaps Koch,1836 :(1)金氏厉螨 L.chini Wang …     

中国西部青厉螨属一新种（蜱螨亚纲：厉螨科）

本记述青厉螨属Ｑｉｎｇｈａｉｌａｅｌａｐｓ一新种，青海青厉螨。新种背板不完全覆盖躯体背部，腹表皮毛约３８对，生殖腹板与肛板间距超过肛板和肛板长易与近缘种顾氏青厉螨Ｑｉｎｇｈａｉｌａｅｌａｐｓ ｇｕｉ Ｂａｉ，１９９２相区别，标本采自青海省同德县北巴滩的熊蜂Ｂｏｍｂｕｓ ｓｐ．体，模式标本保存在青海省地方病防治研究所。     

新疆东部地区蜱螨区系及医学意义

通过对新疆东部地区蜱类和医螨的广泛调查，目前已发现蜱７属１２种；革螨６科１３属２７种；恙螨４属５种。革螨中的长毛美绥螨Ａｍｅｒｏｓｅｉｕｓ ｌｏｎｇｉｓｅｔｏｓｕｓ Ｙｅ ｅｔ Ｍａ，１９９３系近期发表的新种，阴阳血厉螨Ｈａｅｍｏｌａｅｌａｐｓ ａｎｄｒｏｇｙｎｕｓ Ｂｒｅｇｅｔｏｖａ，１９５２是国内新纪录，宽胸真厉螨Ｅｕｌａｅｌａｐｓ ｗｉｄｅｓｔｅｒｎａｌｉｓ Ｐｉａｏ ｅｔ Ｍａ，１９８０是     

美绥螨属两新种记述（蜱螨亚纲：美绥螨科）

本文报道美绥螨属两新种,命名为粗毛美绥螨,新种Ameroseius crassisetosus sp.nov.和长毛美绥螨,新种Ameroseius longisetosus sp.nov。前者采自新疆尼勒克的小林姬鼠,后者采自新疆奇台及和布克赛尔等地的黄兔尾鼠和短尾仓鼠。     

新疆博尔塔拉及伊犁地区革螨恙螨区系

本文以1990年对新疆西北部的博尔塔拉和伊犁地区进行革螨、恙螨区系调查所获结果为基础,结合既往标本资料对该地区革螨恙螨的种属组成、宿主关系及地理分布等作一记述。到目前为止,调查区内共发现革螨17属35种,其中纤细下盾螨Hypoaspis gracilis Meledjaeva,1963、沙氏阳厉螨Androlaelaps sardous Berlese,1911、海氏肺厉螨Pneumolaelaps hyatti Evans et Till,1966和毛寄螨parasitus setosus Oudemans et Voigts,1904是国内新纪录。采获恙螨7属11种,其中钳齿螨属Cheladonta一种是新疆地区首次发现的属种。对干旱荒漠鼠种寄生恙螨的一些特性进行了初步讨论。     

New instrument for transvenous cardiac biopsy

A new forceps is described for obtaining serial endomyocardial biopsy specimens from the human heart. The instrument is introduced percutaneously into the right internal jugular vein and used to obtain tissue from the apex of the right ventricle. Eighty-five biopsy procedures have been performed in 19 patients after cardiac transplantation. The tchnique used was 100 percent successful in obtaining endomyocardial biopsy specimens, and there were no significant complications. A biopsy procedure may be performed within 5 minutes. Serial percutneous transvenous endomyocardial biopsies are now routinely performed with this instrument in new heart transplant recipients and in patients with primary cardiomyopathy.     

"New" treatment for chronic intractable congestive heart failure.

The use of hexamethonium injected intravenously in successive 2.5 mg. doses resulted in alpha-adrenergic sympathetic nerve blocking and associated peripheral vasodilation with dramatic improvement of the symptoms and signs in patients with marked chronic intractable CHF. The vasodilatating effect of the drug is simple to monitor at the bedside and serves as an effective, simple means to "bleed" the patient intravenously by decreasing systemic venous tone and reducing the wall stress in the vessels. This intravenous "bleeding" results in a shifting of excessive blood from the lungs and central systemic venous areas to the larger volume of the more peripheral systemic venous reservoirs. Rheoplethysmorgraphic recordings of digital blood flow in the fingertips of the patients revealed marked constriction of all vessels of the fingers during CHF. Hexamethonium dilated all these vessels and increased digital blood flow even though arterial blood pressure was reduced by the drug. Theoretic discussions of aspects of the mechanism of congestive heart failure of the two-pump system of the heart of man and the mechanical or hemodynamic advantages of the small veins over the larger centrally located veins tend to explain why the use of hexamethonium benefits the circulation by producing vendoilatation. These studies indicate the therapeutic usefulness of hexamethonium in the management of acute and chronic intractable CHF and provides physiologic and theoretic data to explain why the drug is effective.     

The pathogenesis of autoimmunity in New Zealand mice

In the past few years there has been an explosion of information in the area of NZ mouse research. This has reflected a general increase in knowledge and technology in cellular immunology and lymphocyte biology. Continuing research in many specific areas promises to expand this increasing data base. The numerous abnormalities of CMI in NZ mice chronicled in the past are currently being analyzed in terms of cell synergism rather than as simple effector mechanisms. Such analysis is indicating more selective rather than general impairment of CMI in aging NZ mice. Similarly, the allogeneic H-2 identical cytotoxic activity expressed by NZ mice and its relevance to autoimmune disease are being explored. In the area of suppressor functions in NZ mice, continued refinement and standardization of suppressor assays must be achieved before adequate interpretation of the present information can be made. Suppressor activity in aging NZ mice has been described as depressed, normal, and elevated. This is an area of acute interest. While the primary etiologic significance of T cell regulatory defects is currently being minimized, their possible secondary role in pathogenesis of autoimmune disease in NZ mice is being investigated.The recent discovery of a significant B cell abnormality present from birth in all SLE-prone murine strains has increased the probability that intrinsic B cell hyperactivity is a primary etiologic factor in murine autoimmunity. The hypersecretion of IgM and the increased incidence of B cell colony-forming units observed in NZ mice is being subjected to further genetic analysis in many laboratories. Similarly, the continued evaluation of the (CBA/N × NZB) hybrid promises to be enlightening, particularly the development of congeneic mice with the CBZ/N X chromosome on the NZB background. In addition, the development of H-2 congeneic NZB strains and immunoglobulin allotype congeneic NZB strains are proceeding. These strains will provide information on the possible roles of MHC and V_H regions in autoimmune pathogenesis. The NZB.ch congeneic strain has already proven to be of great value.The continued development and further characterization of congenitally immunologic mutant NZ mice has been and will continue to be useful in elucidating the mechanism of autoimmune development in these animals. While NZ mice express a variety of host-viral interactions, recent genetic analysis suggests that viral infection is not a primary etiologic agent in the autoimmune disease of NZ mice. A well-defined clinical entity and a range of abnormalities have been delineated in NZ mice. The present goal is to define the cellular basis of autoimmunity through continued immunologic and genetic analysis of this important animal model of human autoimmune disease.