肺炎性肌纤维母细胞瘤临床病理分析

<正>肺炎性肌纤维母细胞瘤(inflammatory myofibroblastic tumor,IMT)是一种少见的肺部肿瘤。本文分析了我科在2012~2013年间遇见的2例肺IMT的临床及病理特征,结合文献,作如下报告。病例资料病例1患者男57岁,一周前无诱因下反复发作咳嗽,为刺激性干咳,无咳痰、咯血,无胸闷及呼吸困难,有右侧背部发胀及乏力,无畏寒发热,无盗汗,无胸痛,在当地医院就诊胸部CT示右上肺占位。     

肺炎性肌纤维母细胞瘤二例

炎性肌纤维母细胞瘤(inflammatory myofibroblastic tumor,IMT),曾被视为良性肿瘤样病变,2002年WHO制定的软组织肿瘤分类中将其归为"纤维母细胞肿瘤"或"肌纤维母细胞肿瘤".IMT的发病原因不清,可表现为良性病变,也可出现局部侵犯或远处转移.现报道2例肺IMT病例的临床特征,以提高对该病的认识.     

肺炎性肌纤维母细胞瘤临床病理分析

肺炎性肌纤维母细胞瘤(inflammatory myofibroblastic tumor,IMT)临床少见,占所有肺部肿瘤的0.04%～1%[1]。临床上易误诊为其他肺肿瘤,本文报告1例IMT并结合文献进行复习。一、病例报告1.临床资料:患者男性,57岁,因咳嗽一周于2012年7月25日入院。患者一周来无诱因下反复发作咳嗽,为刺激性干咳,无咳痰,无咯血,外院检查CT示右肺上叶后段片状阴影,大小约17 mm×23 mm,后段支气管局部截断,病灶内见空泡     

乳癌术后肝脏炎性肌纤维母细胞瘤1例

<正>炎性肌纤维母细胞瘤(inflammatory myofibroblastic tumor,IMT)是一种发病率较低的间叶性肿瘤^[1],可发生于多种组织或器官,最早发现于肺组织中,随后在胃、脾脏、唾液腺、中枢神经系统、皮肤等均有发现^[2-5],WHO将IMT定义为由梭形肌纤维母细胞伴慢性炎症细胞尤其是浆细胞浸润的特殊病变。而发生于肝脏的IMT较为少见^[6-8],称为肝脏炎性肌纤维母细胞瘤(hepatic inflammatory myofibroblastic tumor,HIMT)。我院收治乳癌术后肝脏炎性肌纤维母细胞瘤1例,现报道如下。     

老年性肺血管瘤的外科临床特点：附6例报告

肺血管瘤是一种发生在肺实质的肿瘤,以往报道在临床上老年人十分少见,术前较难准确诊断,多诊断为良性肺肿瘤,亦易误诊为肺部恶性肿瘤,而最后确诊依靠病理。     

肺部炎性肌纤维母细胞瘤误诊6例临床与影像学分析

<正>炎性肌纤维母细胞瘤(IMT)是一种较为少见的好发于肺组织的肿瘤,无症状或缺乏特征性临床表现,影像学表现无特异性,极易发生误诊,收集近3年间在我院肺部炎性肌纤维母细胞瘤资料,回顾性分析其影像学表现,以此提高对本病的了解和认识。现报告如下。临床资料一.本组6例,4例女性,2例男性,年龄在49-71岁,平均年龄59岁。2例患者无症状,体检时发现肺部病灶,3例短期内咳嗽,痰中带血,1例反复咯     

肾盂炎性肌纤维母细胞瘤合并HIV感染1例

<正>炎性肌纤维母细胞瘤（Inflammatory myofibroblastoma,IMT）是以肌纤维母细胞增生为主,炎症细胞浸润为特点的少见的一种交界性肿瘤。多见于肺部,较少发生在泌尿系系统。本文报道1例HIV感染者合并有肾盂IMT。1病例资料患者女,48岁。2020年8月确诊HIV感染。     

颈动脉超声对动脉粥样硬化性疾病的研究进展

颈动脉超声是一种安全、有效、非侵入性评价临床及亚临床动脉粥样硬化的一种检测手段。大量研究证实颈动脉内膜 中膜厚度 (IMT)与冠状动脉粥样硬化的出现密切相关 ,而且IMT增厚是冠心病事件、脑卒中和短暂性脑缺血的独立危险因素。有经验实验室精细的颈动脉超声检查及IMT测值能作为动脉粥样硬化性疾病特别是心血管病事件的替代指标。     

结节性甲状腺肿合并肺上皮样血管内皮瘤误诊为甲状腺癌一例

临床资料 结节性甲状腺肿好发于女性，发病率很高，临床上往往与甲状腺癌鉴别困难。肺上皮样血管内皮瘤（pulmonary epithelioidhemangioendotheliom，PEH）是一种罕见的肺内低度恶性血管源性肿瘤，     

肺畸胎瘤1例

肺畸胎瘤是原发于肺内而无纵隔畸胎瘤的肺部肿瘤,它是临床上一种罕见的肺部肿瘤。我院胸外科从1994年~2004年在肺切除术中发现1例右肺畸胎瘤。患者男,52岁,因咳嗽、咳痰、发热、胸痛1月入院。查体:体温37.9℃,右中肺叩诊呈浊音,双肺未闻及干、湿罗音。X线示右肺上叶片团状影。C     

State of knowledge of helminth fauna of freshwater fishes of Poland

A total 89 fish and lamprey species has been recorded from Polish freshwater habitats. Twenty-seven of them (30.3%) have not been surveyed for parasitic helminthes. Some of the latter fishes are either rare or not easily accessible. Other live only in specific habitats in scattered localities. An important obstacle for studying parasite faunas of some fishes may be their status on an endangered species. Among the non-surveyed fishes, are those which have been relatively recently introduced to Poland or migrated there on their own. The present paper attempts to review all hitherto not studied helminthologically fish species, their habitats, localities and current protection status.     

高血压降压治疗目标的再认识

根据传统的高血压水平的定义,1993年WHO高血压治疗指南提出血压控制目标为<140/90mm Hg(1mm Hg=0.133kPa),但是并非所有患者都必须将血压降至同一水平,而应根据患者情况进行个体化治疗。Framingham进行的一项长达10～12年的心血管事件研究发现,第5年后,正常上限血压[收缩压(SBP     

Lack of correlation of degree of villous atrophy with severity of clinical presentation of coeliac disease

BACKGROUND AND AIM: Both the clinical presentation and the degree of mucosal damage in coeliac disease vary greatly. In view of conflicting information as to whether the mode of presentation correlates with the degree of villous atrophy, we reviewed a large cohort of patients with coeliac disease. PATIENTS AND METHODS: We correlated mode of presentation (classical, diarrhoea predominant or atypical/silent) with histology of duodenal biopsies and examined their trends over time. RESULTS: The cohort consisted of 499 adults, mean age 44.1 years, 68% females. The majority had silent coeliac disease (56%) and total villous atrophy (65%). There was no correlation of mode of presentation with the degree of villous atrophy (p=0.25). Sixty-eight percent of females and 58% of males had a severe villous atrophy (p=0.052). There was a significant trend over time for a greater proportion of patients presenting as atypical/silent coeliac disease and having partial villous atrophy, though the majority still had total villous atrophy. CONCLUSIONS: Among our patients the degree of villous atrophy in duodenal biopsies did not correlate with the mode of presentation, indicating that factors other than the degree of villous atrophy must account for diarrhoea in coeliac disease.