眼挫伤致近视临床分析

目的分析眼球挫伤致近视的因素。方法对20例眼球挫伤致近视的情况和治疗结果进行分析。结果经观察治疗后10例近视度数均逐渐减小，平均恢复时间约3周。结论眼挫伤致近视由多种因素引起，经治疗后大部分视力可恢复。     

挫伤性近视临床分析

目的 探讨挫伤性近视的损伤机制和治疗.方法 对72例(72只眼)眼挫伤性近视患者进行治疗前后的观察,并以散瞳剂应用与否分为对照组和观察组的视力变化进行比较分析.结果 全部病例经治疗后视力都有显著提高(P<0.01),应用散瞳剂的治疗组视力恢复明显比未用散瞳剂组好(P<0.05).两组的屈光度有明显减轻,观察组与对照组的屈光度变化无明显的差异.结论 由于挫伤程度和损伤性质不同,挫伤性近视的损伤机制各异,主要包括睫状肌痉挛,晶状体悬韧带断裂、睫状体水肿、眶内组织压迫等.散瞳剂应用对视力恢复有帮助.     

眼挫伤致一过性近视23例

0引言钝挫伤在眼外伤患者中比较常见,机械性钝力作用于眼球后,可以引起眼内各种组织结构的改变。挫伤后发生的近视就是其中常见改变之一,主要表现为以往较好的视力,伤后出现明显的视力下降。这种改变在临床工作中容易被忽视,因为只注意到伤后眼部的损害,而忽略了由此引发的屈光变化,这需要引起我们临床医师的注意。1临床资料挑选2007-05/2009-05眼挫伤后发生一过性近视的病例23例23眼。其中男19例,女4例。年龄19~52(平均30.2)岁。23眼伤前均无屈光不正病史,视力1.0(16眼),视力1.2(5眼),视力1.5(2眼)。其中拳击伤13例,踢伤5例,球类击伤3例,撞击伤2例。23眼均有眼睑皮     

外伤性一过性近视的临床观察

目的：观察外伤性一过性近视的临床特点和预后。方法：对20例（25眼）外伤性一过性近视患者的临床资料进行回顾性分析。结果：小瞳检影验光发现所有外伤眼均存在近视，其中84％的外伤眼表现为轻、中度近视。睫状肌麻痹剂散瞳后验光发现所有外伤眼的近视度数均显著减轻。经合适的凹透镜矫正后所有外伤眼视力均可达1．0。对症治疗后4周内所有外伤眼的视力均恢复至≥1．0。结论：外伤性一过性近视病程短，对药物治疗反应好，且预后佳。     

眼挫伤184例临床分析

眼外伤是视力损害的主要原因之一,是世界上引起失明的6种主要原因之一[1],现就我院建院以来住院治疗的,184例严重眼挫伤病例分析如下:临床资料:据可靠统计的病历记录,共收治各种眼病2314例,其中眼外伤446例,而眼挫伤184例,占同期住院病人总数的795%,占同期眼外伤数的4126%。男161例占8750%,女23例占1250%,男女之比为7∶1。右眼85例占4620%,左眼91例占4946%,双眼8例占435%。年龄最小10月,最大约68岁,其中21岁～40岁最多占5326%。(表1)。工人、农民141例占7663%,学生28例占1521%,干部6例占326%,小孩9例占489%。工人、农民最多,其次学生。致伤…     

挫伤性近视28例临床分析

目的 探讨眼挫伤后近视致使视力下降的原因及治疗方法.方法 采用回顾性方法对我科28例(28眼)挫伤性近视进行分析.结果 治疗后随访的28例中25例裸眼视力达1.0以上,3例矫正视力在1.0以上.结论 挫伤性近视是眼挫伤视力下降的原因之一,经治疗后视力可恢复正常.     

挫伤性近视的临床调查分析

目的探讨挫伤性近视的临床特点和预后。方法回顾分析32例(32只眼)挫伤性近视患者的临床资料。结果伤后7天验光,32例(32只眼)均出现不同程度的近视,主要为轻、中度近视。睫状肌麻痹剂能缓解这种近视。戴镜下电生理检查正厂。多数于数周或数月恢复。结论挫伤性近视预后良好,但不能忽视这种状态的存在。     

眼挫伤致葡萄膜炎临床分析

目的 探讨眼挫伤致葡萄膜炎的发病机制及治疗方法。方法 对22例24眼眼挫伤所致葡萄膜炎进行分析。视力0．5～0．8者10眼，0．1～0．3者11眼，小于0．1者3眼。全部患者用皮质类固醇、消炎痛及散瞳等治疗。结果 22例24眼(100％)症状消失，13眼(54．17％)体征消失，裸眼视力≥1．0者4眼(16．67％)，0．5～0．9者14眼(58．33％)，0．1～0．3者5眼(20．83％)，小于0．1者l眼(4．17％)。结论 眼挫伤致葡萄膜炎主要是由于伤后前列腺素增加和组织胺、5-羟色胺、缓激肽等生物活性介质及一些炎性因子被启动、激活、释放所致。伤后早期服部体征，及时诊断、合理使用皮质类固醇、消炎痛及散瞳治疗，效果明显。     

挫伤性近视临床特点及发生机制的探讨

目的探讨挫伤性近视的临床特点和发生机制。方法眼挫伤269例中确定23例(24眼)为挫伤性近视,临床观察12周,对其结果进行分析。结果临床观察眼挫伤269例,有23例(24眼)检查眼底及屈光间质已恢复正常,视野、眼A/B超及眼电生理检查均正常,但仍有不同程度的近视表现。3例(4眼)应用睫状肌麻痹剂能缓解这种近视,20例(20眼)不能缓解,迟至2~12周大部分恢复。结论挫伤性近视发生机制可能由于睫状体水肿、调节痉挛、晶状体厚度增加、前移、或角膜曲率增加所致。挫伤性近视预后良好,但不能忽视其存在。     

挫伤性近视及其治疗

眼球挫伤后常可导致挫伤性近视或使原有的近视度数增加和视网膜震荡,但视力的矫正及屈光检查易被忽略币造成漏诊和误诊。现将近年来住院治疗的16例报告如下。 临床资料 本文共16例,男15例,女1例;年龄最小16岁,最大40岁。右眼7例,左眼9例;木块击伤2例,拳头击伤10例,高压气体冲击     

外伤性近视的临床观察

目的:观察外伤性近视的临床特点和预后.方法:对30例外伤性近视患者的临床资料进行回顾性分析.结果:小瞳检影验光发现所有外伤眼均存在近视,表现为轻、中度近视.睫状肌麻痹剂敞瞳验光检查发现所有外伤眼的近视度数均有不同程度的减轻.用合适的凹透镜均可使外伤眼矫正视力达到1.0.经过治疗有29例外伤眼4wk内,1例外伤眼6wk,视力恢复至≥1.0.结论:外伤性近视:有钝挫伤史,病程短,经药物治疗后效果佳,恢复快,关键是要重视.     

不同检查方法在眼球钝挫伤中的价值分析

眼球钝挫伤的发生可导致眼部结构和功能发生持久性改变。眼球钝挫伤常用的检查方法主要包括B型超声、超声生物显微镜(UBM)、光学相干断层扫描(OCT)、眼底血管荧光造影(FFA)、偏振光激光扫描仪、眼底自发荧光(FAF)等,这些检查方法各有侧重。本文旨在对不同检查方法的优缺点进行综述,以期为眼球钝挫伤的临床诊治提供参考。     

睫状体挫伤的形态学研究

目的探讨严重眼球伤后睫状体各种损伤形态的特点。方法应用超声生物显微镜(ultrasoundbiomicroscopy,UBM)检查眼挫伤连续病例共196例(202)眼。结果109例(121眼,59.90%)有睫状体的损伤,包括:房角后退42例(42眼,20.79%);睫状体分离28例(30眼,14.85%);睫状体脉络膜脱离31例(31眼,15.35%);睫状体脉络膜上腔积血12例(12眼,5.94%);睫状体上皮脱离3例(3眼,1.49%);睫状体破裂3例(3眼,1.49%)。结论眼挫伤后睫状体的损伤多种多样,UBM检查能提供这些损伤的清楚图像,是正确地诊断这些损伤的无创伤性检查方法。     

Evaluation of retinal nerve fiber layer and ganglion cell complex thickness after ocular blunt trauma

Purpose

To evaluate circumpapillary retinal nerve fiber layer (cpRNFL) and ganglion cell complex (GCC) after ocular blunt trauma.

Methods

Best-corrected visual acuity (BCVA), cpRNFL and GCC were evaluated by RTVue-100 OCT in all consecutive patients with previous monocular blunt trauma seen between January 2012 and December 2012.

Results

Twenty-two patients (11 females, 11 males, mean age 43.9±14.2 years) were included in the study. Patients were seen after a mean of 8.42±13.3 (range, 0.08–55.3) years from ocular blunt trauma. BCVA was normal in 11 cases and was less than 0.4 LogMAR in all cases. In 9/22 patients (40.9%), cpRNFL and GCC were reduced, whereas in one case an isolated reduction of GCC with normal cpRNFL was present. In patients with reduction of cpRNFL and GCC, mean BCVA was 0.17±0.17 LogMAR. In 6/9 patients (66.6%) with cpRNFL and GCC reduction, BCVA was ≤0.1 LogMAR.

Conclusion

cpRNFL and GCC reduction may be present after ocular blunt trauma and may be associated with preserved visual acuity.     

眼挫伤实验模型及其影响因素评估

目的探讨眼挫伤动物模型的实用性。方法分别用铜球自由落体、高压水柱或气柱致伤兔眼,观察91只兔眼致伤情况及病理改变。结果只要达到一定致伤强度,不同性状致伤物均可使兔眼产生类似损伤,其致伤程度主要与致伤物质量（m）、致伤时即时速度（Vt）以及致伤物和眼球接触面积有关。致伤物质量越大、致伤时即时速度越快、接触面积越小,则致伤程度越严重,且易向眼后段延伸。在几种实验条件下,固态致伤物致伤以自由落体致伤方式较好,其所涉变量少,可控范围大,重复性好,易于操作和定量计算。在一定范围内液态或气态致伤物对眼前段影响大,而固态致伤物则对眼后段影响更明显。结论对眼后段致伤情况评估以自由落体模型为优,对眼前段致伤观察则可选择液态或气态致伤物。     

Pathogenesis of transient high myopia after blunt eye trauma

OBJECTIVE: To determine the pathogenesis of transient high myopia after blunt eye trauma. DESIGN: Two observational case reports and literature review. METHODS: Refraction was measured in two patients with an autorefractometer in the acute and convalescent stages after a blunt eye injury. The anterior chamber angle, the ciliary body, and the choroid were examined by ultrasound biomicroscopy (UBM) in the acute and convalescent stages. In one patient, the anterior chamber depth, lens thickness, and axial length were measured by A-scan ultrasonography in the acute and convalescent stages. MAIN OUTCOME MEASURES: Comparison of the refraction, anterior chamber depth, lens thickness, axial length, and the UBM-determined appearance of the choroid and ciliary body during the acute stage with the values during the convalescent stages. RESULTS: The first patient showed a myopic shift of -9.75 diopters (D) and an anterior chamber shallowing of 0.94 mm measured 3 days after trauma by an air bag inflation compared with the measurements at the convalescent phase. UBM showed an annular ciliochoroidal effusion with ciliary body edema, anterior rotation of the ciliary processes, and disappearance of the ciliary sulcus. Eleven days after the injury, these UBM findings normalized, and the myopia decreased to -0.75 D, 27 days after trauma. The second patient had a myopic shift of -8.9 D compared with the convalescent phase, immediately after blunt trauma by a firework. Seven days after the injury, UBM revealed a partial cyclodialysis in addition to findings similar to those in the first patient. Ten days after injury, a myopic shift (-4.75 D), anterior chamber shallowing (by 1.1 mm), and thickening of the crystalline lens (by 0.27 mm) were observed compared with the convalescent phase. Associated UBM findings confirmed the anterior shift of the lens-iris diaphragm. Seventeen days after trauma, the UBM findings, including the cyclodialysis, were normalized, and the myopia had decreased to -1.0 D. CONCLUSIONS: Transient high myopia after blunt trauma is caused by anatomic changes in the ciliary body and crystalline lens. The anterior shift of the lens-iris diaphragm caused by ciliochoroidal effusion with ciliary body edema and thickening of the crystalline lens from blunt eye trauma are involved in traumatic high myopia.