共查询到20条相似文献,搜索用时 78 毫秒
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随着医疗体制的改革和医疗保险制度的建立,社会药房的迅猛增加,"人世"步伐的加快,医药市场呈现出新的态势,给医院药房提出了新的课题:医院门诊药房如何面对市场竞争?在这种形式下,医院门诊药房面临着严竣的考验.医院门诊药房是医院面对病人的重要窗口之一,其服务质量的好坏直接影响医院的形象和声誉,为了适应新形式下医院所面临的社会变化,首先就要提高门诊药房的服务质量来留住自己的病人.本人根据自己的工作经验,提出几点建议. 相似文献
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目的药房是整个医院运行机制的重要组成部分,服务质量好坏直接影响医院的声誉,所以加强门诊药房的管理,全面提高药房窗口服务水平至关重要。 相似文献
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目的:分析医院门诊药房管理质量与服务质量,为患者合理用药,提高服务质量提供指导意见.方法:根据我院门诊药房服务流程、门诊药房管理质量、药房环境布局,提高药师处理问题的能力、门诊药房服务水平.结论:对门诊药房实行规范化、科学化和现代化管理,健全医院药房管理制度,提高人文素质及服务质量,能缓解医患矛盾,提高工作效率. 相似文献
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门诊药房是医院药房,面对病人的一个重要窗口。其服务质量的优劣,直接影响到医院的形象和声誉。对医院门诊药房药品差错的发生原因进行分析,将有利于广大药师提高服务质量,笔者结合自身在药房工作近20年的经历,对有关药品差错发生的特点、类型、根源及应采取的措施进行简要的分析,供药师们参考。 相似文献
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目的探索门诊药房规范化管理,提高药学服务质量。方法结合基层医院工作实际,并查阅相关文件规定,针对影响门诊药房质量的各个环节制订管理措施。结果通过软硬件建设,门诊药房服务质量全面提高,差错事故与纠纷明显减少。结论加强管理及科学规范的操作等措施的落实,是医院门诊药房服务质量提升的关键。 相似文献
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门诊药房是医院直接为病人服务的重要窗口,其服务质量直接关系到病人能否及时得到药物的救治和本医院的声誉。而县级医院门诊药房又有其自身所处地位的局限。其面临病员以基层农村居多,故其服务导向应以高效、快捷、便利及简明为基准,门诊药房的管理也应随机运作。我们通过多年的工作实践验证拟定县级医院门诊药房的管理模式,以供探讨。 相似文献
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潘鸿妹 《中国临床医药实用杂志》2005,(4):97-98
新时期医院门诊药房面对的新挑战,医院门诊药房的服务质量,不仅关系到病人的健康与生命,而且与医院的经济效益与社会效益相关。新时期医院门诊药房是规行医院管理网络中的重要部门,深化改革门诊药房的管理模式——从人力资源的优化,到工作职能转变,从管理体制的创新发展到新的信息技术的全面应用推广。以新方法、新思路、新模式,全新的姿态主动参与到市场竞争中去。 相似文献
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Hepatic responses to the administration of high doses of BHT to the rat: their relevance to hepatocarcinogenicity 总被引:4,自引:0,他引:4
C J Powell J C Connelly S M Jones P Grasso J W Bridges 《Food and chemical toxicology》1986,24(10-11):1131-1143
Although butylated hydroxytoluene (BHT) is non-mutagenic, at high doses it has recently been associated with an increased incidence of liver tumours in laboratory rodents. To establish whether chronic liver cell injury may be involved in the genesis of these tumours, BHT was administered to rats by orogastric gavage at doses of 0, 25, 250 or 500 mg/kg/day for up to 28 days and also at daily doses of 1000 and 1250 mg BHT/kg for up to 4 days (sublethal doses). The sublethal doses induced centrilobular necrosis within 48 hr, whereas administration of BHT for 7 or 28 days caused dose-related hepatomegaly and at the highest dose level induced progressive periportal hepatocyte necrosis. The periportal lesions were associated with proliferation of bile ducts, persistent fibrous and inflammatory cell reactions, hepatocyte hyperplasia and hepatocellular and nuclear hypertrophy. Biochemical changes consisted of dose-related induction of epoxide hydrolase, dose-related changes in the ratio of cytochrome P-450 isoenzymes and depression of glucose-6-phosphatase. Measurement of BHT demonstrated a dose-related accumulation in fat but not in the liver. Changes in hepatic activating and detoxifying enzyme profiles are implicated both in the mechanism of periportal hepatocyte damage and in the change of site of damage according to the dose and duration of the treatment. The persistent and active nature of the lesions in rats dosed with 500 mg BHT/kg for 28 days, combined with evidence of cell damage at doses equivalent to those associated with hepatic tumours (250 mg BHT/kg), suggests that chronic liver cell damage may be involved in their aetiology. In this and several other studies, there was no evidence that BHT causes liver damage at a dose level of 25 mg/kg/day. As this is several hundred times higher than the normal human intake, it is considered unlikely that BHT poses a threat to human health. 相似文献
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