超选择性肾动脉栓塞治疗先天性肾动静脉畸形的护理

总结5例经股动脉超选择性肾动脉栓塞治疗先天性肾动静脉畸形的护理。治疗前做好心理护理,完善准备,治疗后严密观察尿液颜色及生命体征的变化,警惕并发症的发生,加强体位护理、穿刺侧下肢护理及对症护理,重视出院指导。5例患者栓塞治疗后血尿即刻好转,未发现护理并发症,治愈出院。     

脑动静脉畸形的血管内栓塞治疗

目的：研究血管内栓塞治疗脑动静脉畸形（ｃＡＶＭ）的临床效果。方法：在数字减影全脑血管造影（ＤＳＡ）监视下,经股动脉插管,采用Ｍａｇｉｃ１．５￣１．８Ｆ微导管,α－氰基丙烯酸正丁酯（ＮＢＣＡ）栓寒剂对２１例脑ＡＶＭ患者行血管内栓塞。结果：完全栓塞达解剖治愈４例,脑ＡＶＭ团栓塞达９０％以上６例,７０％￣９０％９例,７０％以下２例,术后出现头晕、７头痛３例,２例栓寒后又行手术切除,３例栓塞后行γ－刀治疗     

儿童动静脉畸形病变的血管内治疗

目的 了解儿童颅内动静脉畸形（ＡＶＭ）病变的血管内治疗特点。方法 回顾分析２１例儿童动静脉畸形的临床表现、血管构筑学特点及治疗。结果 手术切除７例,１例不全切除后多次出血,继发颅内感染;单纯栓塞８例,栓塞加ｒ－刀立体放射治疗５例,均治愈。结论 儿童动静脉畸形的临床表现和血管构筑学具有一定的特点,尽早行血管内栓塞治疗或栓塞后合并ｒ－刀治疗效果满意。     

血管内栓塞治疗脑动静脉畸形

目的 :探讨血管内栓塞治疗脑动静脉畸形 (AVM)的临床效果及安全性。方法 :应用血管内栓塞治疗脑AVM 42例 ,栓塞剂为二氰基丙烯酸正丁酯 (NBCA)。结果 :16例完全闭塞 ,90 %～ 10 0 %闭塞 8例 ,70 %～ 90 %闭塞 11例 ,5 0 %～ 70 %闭塞 6例 ,<5 0 %闭塞 1例。并发症有导管断裂滞留畸形团 3例 ,NBCA意外泄露 1例。结论 :血管内栓塞治疗脑AVM是一种安全、可靠、有效的治疗手段。     

肾内动静脉畸形的介入栓塞治疗

本文报告5例肾内动静脉瘘畸形（AVM）反复间歇性无痛性血尿被误诊为“肾肿瘤、膀胱肿瘤、肾结核、紫癜性肾病”,经肾动脉造影确定为AVM。5例超选择将导管尖端置于病变供血动脉近端,分别给予明胶海绵、真丝线段进行栓塞。于出血栓后当天停止,血尿消失,取得临床介入治疗止血的满意效果,无严重并发症发生。     

脑动静脉畸形的血管内栓塞治疗

[目的]探讨脑动静脉畸形的血管内栓塞治疗价值.[方法]回顾性分析72例脑动静脉畸形的血管内栓塞治疗资料.[结果]72例栓塞治疗的技术成功率为100%,共行159次栓塞,每例栓塞次数为1～5次,平均1.9次.单纯真丝线段栓塞51例,真丝线段和NBCA联合栓塞21例.栓塞效果:畸形血管团完全消失者15例, 畸形血管团减少75%以上者26例,畸形血管团减少在50%～75%之间者20例,畸形血管团缩小不足50%者11例.栓塞后行X线刀放射治疗者26例,手术切除21例.所有病例术中及术后均无严重并发症发生.[结论]经超选择性插管后行栓塞治疗是脑动静脉畸形安全有效的治疗手段.     

先天性肾动静脉畸形一例报告并文献复习

[目的]提高对先天性肾动静脉畸形的认识.[方法]收治先天性肾动静脉畸形女性患者1例,结合文献复习讨论先天性肾动静脉畸形诊治要点.[结果]患者临床表现为肉眼血尿.增强CT示右肾前部靠近集合系统的高密度血管影,CT血管成像(CTA)示右肾中下部簇状血管团.磁共振血管成像(MRA)示右肾下部异常血管团.数字减影血管造影(DSA)证实右肾中下部动静脉畸形,肾静脉早期显影.予以介入栓塞治疗.随访12个月,患者临床症状消失无复发.[结论]肾动静脉畸形临床少见,肉眼血尿为主要临床表现之一,DSA是诊断金标准,介入栓塞为当前主要的治疗方法.     

血管内栓塞对颅内动静脉畸形的治疗

脑动静脉畸形(AVM)是颅内出血及癫痫的常见原因之一,其致残率及致死率均较高,我院1997- 0 2～2 0 0 3- 0 3共收治颅内动静脉畸形(AVM) 9例,经血管内NBCA胶栓塞治疗颅内AVM 7例(其中真丝线段 NBCA胶栓塞1例) ,钨丝弹簧圈栓塞治疗2例(其中真丝线段 钨丝弹簧栓栓塞1例) ,疗效较好,总结如下。1　对象和方法1.1　对象　本组男7例,女2例,年龄11～5 4岁,平均2 6岁。首发症状为蛛网膜下腔出血者6例,有头痛史5例,癫痫发作4例,合并脑内血肿2例,伴有肢体运动障碍者4例。全部患者均行CT、MRI或MRA及DSA检查。AVM病变均位于大脑半球(左侧6例…     

血管内Onyx栓塞脑动静脉畸形的护理

目的 探讨Onyx介入治疗的护理方法.方法 回顾14例行Onyx介入治疗脑动静脉畸形患者的护理方法.结果 术后无远期并发症,3～6 d出院.结论 以控制性低血压为核心的整体护理是保证栓塞效果的重要环节.     

血管内栓塞治疗脑动静脉畸形369例

【目的】评价应用α- 氰基丙烯酸正丁酯(NBCA)血管内栓塞治疗脑动静脉畸形(AVMs)的效果。【方法】经股动脉插管用微导管技术超选择注入病灶NBCA栓塞治疗脑AVMs,共369例。男159例,女210例,年龄4~75岁,平均32. 4岁。首发症状头痛头晕246例,癫痫发作48例,意识障碍(昏迷) 36例,肢体麻木16例,肢体无力(偏瘫)23例。AVM的直径1. 5~8cm,其中病变大于6cm者141例, 3 ~6cm者194例,小于3cm者34例。单支供血动脉58例, 2支108例, 3支以上203例。栓塞加手术切除巨大型脑AVMs27例。【结果】完全栓塞病变74例, 90%以上104例, 70% ~90% 129例, 50% ~70% 51例,栓塞<50%者11例。【结论】NBCA应作为栓塞治疗脑AVMs的首选材料。根据脑AVM的部位、大小以及供血动脉数,酌情选择血管内栓塞、手术切除、栓塞加手术切除、γ-刀或栓塞加γ-刀治疗。     

Cluster headache syndrome associated with middle cerebral artery arteriovenous malformation

Cluster headache (CH) is an idiopathic cephalagic syndrome, although several pathological processes have been described in association with this syndrome. We report two cases of cluster headache in hospitalized patients with middle cerebral artery dependent arteriovenous malformation (AVM). After surgical removal of the AVM the headache completely resolved, suggesting that complementary studies and treatment of the underlying actinology may be indicated for secondary forms of cluster headache.     

颌骨中心性动静脉畸形栓塞与手术治疗的临床研究

目的 总结动脉血管介入栓塞结合手术治疗颌骨动静脉畸形的临床疗效。方法 对10例颌骨中心性动静脉畸形血管永久栓塞。其中8例栓塞后行颌骨病灶刮治。结果 8例栓塞加手术治疗的患者，随访24个月无复发，2例单纯栓塞治疗的病例，随防36个月无复发。结论 颌骨动静脉畸形栓塞加手术治疗是一种新的安伞有效的治疗。     

MRA对脑动静脉畸形的诊断分析

目的:探讨磁共振血管造影(MRA)在脑动静脉畸形(AVM)中的诊断价值。方法:选取26例经DSA确诊的AVM患者,与术前脑血管MRA检查结果进行比较。结果:MRA与DSA诊断病灶大小、静脉支数上,结果比较有差异(P<0.05),MRA与DSA在诊断动脉支数上结果比较无差异(P>0.05)。结论:MRA影像技术能够清晰显示脑动脉畸形的畸形血管巢位置、供血动脉及引流静脉,为临床治疗方法的确定提供了充分的基础,值得在临床上推广使用。     

头皮巨大动静脉畸形1例

患者女,24岁,因头皮肿物进行性增大24年入院.入院查体:生命体征平稳,右侧头顶部可见一约13 cm×10 cm肿物,略高出皮肤,色红,毛发分布正常,肿物质软,有波动感,触之无压痛,内可触及血管搏动,颅神经检查未见异常.64排螺旋CT头颈部血管成像示:右侧颞顶枕部皮下见异常密度区,平扫呈高低混杂密度,增强扫描后其内可见条状或蚯蚓状纡曲强化的血管影,有粗大的供血动脉(右侧颈外动脉)及引流静脉(右侧颈外静脉);双侧颈内动脉、椎动脉、大脑中动脉、大脑前动脉、大脑后动脉及其分支动脉未见明显异常改变.     

Uterine arteriovenous malformation

Uterine arteriovenous vascular malformations (UAVM) are uncommon vascular diseases, occurring during reproductive age. Patients affected by UAMVs usually present with recurrent pregnancy loss or menorrhagia. Initial evaluation of UAVMs is made with ultrasonography (US) and US-Doppler. Magnetic resonance is used when a UAMV is suspected at US. Treatment can be surgical (hysterectomy or surgical removal of AVM), or with selective uterine arterial embolization. We report a case of UAMV, from its clinical signs to diagnostic confirmation and subsequent treatment.     

CTA诊断先天性颈内静脉、颈内外动脉瘘合并后颈部血管畸形1例

患者,女,28岁.头晕,时有视物不清.活动后头晕加重,查体:后颈部闻及持续吹风杂音.彩色多普勒血流显像异常,疑左侧颈内动脉闭塞,常规CT平扫颈部及颅底无异常,CTA血管重建MIP显示左侧颈内静脉明显增粗,密度增高,其后方见大量纡曲,畸形血管密度非常高,正常颈部大血管被其掩盖,显示不清.     

De novo intracranial arteriovenous malformation development after endovascular treatment for a pial arteriovenous fistula in capillary malformation-arteriovenous malformation syndrome

Capillary malformation-arteriovenous malformation (CM-AVM) syndrome is a newly described entity characterized by autosomal dominantly inherited multifocal capillary malformations caused by RASA1 mutations (CM-AVM1) or EPHB4 mutations (CM-AVM2). Concurrent high-flow vascular anomalies in the brain are often present in the form of intracranial AVM or arteriovenous fistula (AVF). These high-flow lesions are often identified at or soon after birth because of the characteristic unique capillary malformations or a systemic disorder due to a high-flow shunt, such as respiratory distress or heart failure. However, de novo intracranial AVMs have not been reported in patients with CM-AVM syndrome. Herein, we report the case of a six-year-old boy with CM-AVM1 who had been treated for an intracranial pial arteriovenous fistula approximately five years previously, in whom a de novo intracranial AVM was identified on a follow-up angiographic study. To the best of our knowledge, this report is the first to document a de novo intracranial AVM in a patient with CM-AVM. We recommend careful neuroimaging follow-up even if initial neuroimaging screening is negative because of the risk of de novo AVM development.     

Migraine with aura-like syndrome due to arteriovenous malformation. The clinical value of transcranial Doppler in early diagnosis

Arteriovenous malformations are an acknowledged cause of migraine that can long constitute the only clinical manifestation before bleeding. We describe two cases of patients suffering from symptoms like migraine with aura in whom arteriovenous malformations were detected by transcranial Doppler examination. We suggest that a screening of migraine patients to prevent bleeding from a possible underlying unruptured arteriovenous malformation could be performed by using transcranial Doppler, a non-invasive and low cost examination.