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急性心肌梗死患者红细胞的变形能力与其三磷酸腺苷酶活性关系的研究 总被引:1,自引:0,他引:1
本文观察了54例急性心肌梗死(AMI)患者发病3天内红细胞变形能力(ED)和红细胞三磷酸腺苷酶(ATPase)活性及红细胞内离子浓度的变化,并分析其相互关系。结果显示,AMI患者红细胞滤过指数(IF)校对照组明显增高(P<0.001)。红细胞Na+-K+-ATPase、Ca2+-Mg2+-ATPase活性和红细胞K+、Mg2+浓度明显降低,而红细胞Na+、Ca2+浓度明显增高,与对照组相比差异有显著性意义(P<0.001)。AMI患者红细胞IF与红细胞Na+-K+-ATPase、Ca2+-Mg2+-ATPase活性、红细胞K+、Mg2+浓度呈负相关(P<0.001),与红细胞Na+、Ca2+浓度呈正相关(P<0.001)。这提示AMI患者ED降低与红细胞ATPase活性和K+、Mg2+浓度降低及Ca2+、Na+浓度增高有关。 相似文献
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血压持续偏高青少年红细胞内离子浓度及膜ATP酶活性的变化观察 总被引:4,自引:0,他引:4
我们测定了38例血压持续偏高青少年红细胞膜ATP酶活性和红细胞内阳离子浓度。结果表明:Na^+-K^+-ATPase和Ca^2+-Mg^2+-ATPase活性明显低于血压正常青少年。红细胞钠、钙含量,红细胞内游离钙水平均明显高于血压正常者。提示:青少年期血压持续偏高个体已存在细胞内钙、钙离子水平的改变,Na^+0K^+-ATPase和Ca^2+-Mg^2+ATPase活性降低是血压持续偏高的重要环 相似文献
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本实验通过对模拟克山病偏食饲料中补充一定量的钙和硒来观察Wistar大鼠胰岛素(Ins)。结果表明,病区良组血钙水平最低,仅及常规食(stock)组一半;血清硒(Se),α-VE水平低于Stock组;Ins减少,血清过氧化脂质水平明显高于Stock组,血清GSH-Px活笥低,有显著差异。补钙后钙水平升高,Se、α-VE水平变化不大;Ins显著升高。补Se/VE组Se、α-VE水平明显高于EM组。联 相似文献
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慢性肾功能衰竭患者红细胞变形能力与红细胞ATP酶活性及离子浓度关系的研究 总被引:1,自引:0,他引:1
检测65例CRF患者ED和红细胞ATP酶活性及离子浓度的变化。结果显示:CRF患者红细胞滤过指数(IF)和Ca2+,Na+明显高于对照组(P<0.001),而Na+-K+-ATP酶、Ca2+-Mg2+-ATP酶活性和K+,Mg2+明显低于对照组(P<0.001)。CRF患者IF与Na+-K+-ATP酶、Ca2+-Mg2+-ATP酶活性和K+、Mg2+浓度呈负相关,与Ca2+、Na+浓度呈正相关。提示CRF患者ED降低与红细胞ATP酶活性降低和离子浓度异常有关。 相似文献
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冠心病患者红细胞ATP酶,超氧化物歧化酶,钠,钾,钙,镁离子和血浆脂?… 总被引:4,自引:0,他引:4
目的 探讨冠心病患者红细胞ATP酶、超氧化物歧化酶、钠、钾、钙、镁离子和血浆脂质过氧化物的变化。方法 测定38例冠心病患者和42例正常人红细胞超氧化物歧化酶(SOD)、Na^+,K^+-ATP酶、Ca^2+-ATP酶活性及Na^+、k^+、Ca^2+、Mg^2+含量和血浆脂质过氧化物(LPO)含量。统计方法采用t检验和直线相关分析。结果 冠心病患者红细胞SOD、Na^+,k^+-ATP酶、Ca^2 相似文献
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检测65例CRF患者ED和红细胞ATP酶活性离子浓度的变化。结果显示;CRF患者红细胞滤过指数和Ca^2+,Na^+明显高于对照组,而Na^+、K^+-ATP酶Ca^2+-Mg^2+-ATP酶活性和K^+、Mg^2+明显低于对照组。CRF患者IF与Na^+-K^+-ATP酶、Ca^2+-Mg^2+-ATP酶活性和K^+、Mg^2+浓度呈负相关,与Ca^2+、Na^+浓度呈相关。 相似文献
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NIDDM病人红细胞变形能力与红细胞ATP酶活性和红细胞内离子浓度关系的研究 总被引:1,自引:0,他引:1
本文检测了42例NIDDM病人红细胞变形能力(ED)和红细胞ATP酶活性、红细胞内离子浓度的变化。结果显示NIDDM病人红细胞滤过指数(IF)较对照组明显增高(P<0.001);红细胞Na+-K+-ATP酶和Ca2+-ATP酶活性较对照组明显降低(P<0.01),Mg2+-ATP酶活性变化不明显;红细胞内Na+、Ca2+浓度较对照组明显增高(P<0.01),而Mg2+浓度较对照组明显降低(P<0.01)。有血管病变者这些变化较无血管病变者更明显。NIDDM病人红细胞IF与Na+-K+-ATP酶、Ca2+-ATP酶活性呈负相关(r=-0.468,-0.458,P<0.001),与红细胞内Na+、Ca2+浓度呈正相关(r=-0.473,0.466,P<0.D01),与Mg2+浓度呈负相关(r=-0.436,P<0.01)。 相似文献
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原发性高血压患者红细胞内CA^2+,Mg^2+水平及细胞膜ATP酶活性 总被引:5,自引:0,他引:5
对正常人及原发性高血压患者细胞内、外Ca2+、Mg2+浓度和细胞膜ATP酶活性之间的关系进行评价。方法检测32名正常人(NT),55例原发性高血压病(HT)患者血浆和细胞内Ca2+、Mg2+浓度(原子吸收法),细胞膜ATP酶活性(比色法)。结果HT组血浆Ca2+浓度明显低于NT组,红细胞内Ca2+浓度明显高于NT组,红细胞膜Na+-K+-ATP酶及Ca2+-ATP酶活性明显低于NT组。血浆Mg2+和红细胞内Mg2+浓度与NT组无明显差别。结论原发性高血压患者细胞膜Na+-K+-ATP酶和Ca2+-ATP酶活性明显减低,血浆Ca2+水平明显减低,红细胞内Ca2+超载。反映膜缺陷和离子运输系统失常与原发性高血压发病有关。而细胞内、外Mg2+水平变化与高血压之间的关系须进一步研究。 相似文献
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本实验通过对模拟克山病偏食饲料中补充一定量的钙和硒来观察Wistar大鼠胰岛素(Ins)的变化。结果表明,病区粮组血钙水平最低,仅及常规食(stock)组一半;血清硒(Se),α-VE水平低于Stock组;Ins减少;血清过氧化脂质水平明显高于Stock组;血清GSH-Px活性低,有显著差异。补钙后钙水平升高,Se、α-VE水平变化不大;Ins显著升高。补Se/VE组Se、α-VE水平明显高于EM组。联合补Se/VE和Ca组Se、α-VE水平高于单纯补Se/VE组,有显著差异,Ins升高。补Se/VE、Ca各组可使过氧化脂质水平下降,有显著差异。综合分析结果表明,补Ca各组与EM组比较差异显著,表明膳食钙量对胰岛素影响极大。 相似文献
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钙硒对模拟克山病偏食喂养大鼠生长激素分泌的影响 总被引:5,自引:0,他引:5
模拟克山病偏食饲料及在这种饲料中补充钙和硒对大鼠生长和生长激素的影响。结果表明,这种克山病偏食饲料中除低硒外低钙特别突出,所饲大鼠血清钙水平及全血GSH-Px活性、血清总蛋白、生长激素明显低于常规食组。在克山病偏食饲料中单纯补钙或硒可明显改善大鼠生长状态及生长激素增多。表明钙和硒在病区粮偏食喂养大鼠所致生长迟缓方面及生长激素的释放中起重要作用。补充钙同时补充硒效果更好。 相似文献
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以克山病病区玉米和黄豆为主,组成偏食低钙及补钙饲料喂养大鼠8周,结果病区粮所饲大鼠生长缓慢,血清总钙含量、肾脏Ⅰ型T_45′—脱碘酶(ID—I)活性、血清T_3浓度及全血和肾脏谷胱甘肽过氧化物酶(GSH—Px)活力显著降低,血清和肾脏脂质过氧化物(LPO)浓度增加。补钙使上述指标显著改善,恢复至常规饲料组水平。提示膳食低钙本身即可抑制ID—I活性,使甲状腺激素(TH)代谢发生障碍,机体抗氧化能力减退。膳食低钙可能参与了克山病发病中的TH代谢障碍过程。 相似文献
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用分别补充100ppm VE 的大骨节病区低硒粮配制的低硒饲料和加饲料饲硒养并繁殖大鼠,组织学上未发现母代或子代大鼠有肝坏死和骨骼肌病变。在母代大鼠中,低硒组的血硒含量明显地低于加硒组(P<0.01),而血清 VE 含量在低硒与加硒组间几乎相同。提示低硒不影响母代大鼠的血清 VE 含量。在子代大鼠中,各组动物血硒含量的变化与其母代基本相同;而低硒组的血清 VE 含量明显地高于加硒组(P<0.05),并且血清 VE 含量与血硒含量之间呈负相关关系(r=-0.658,P<0.05)。结果表明饲料低硒能使子代低硒大鼠血清 VE 含量增加。 相似文献
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Platelet and erythrocyte Mg2+, Ca2+, Na+, K+ and cell membrane adenosine triphosphatase activity in essential hypertension in blacks. 总被引:2,自引:0,他引:2
OBJECTIVE: To assess the relationship between intracellular Mg2+, Ca2+, Na+ and K+ and cell membrane adenosine triphosphatase (ATPase) activity in normotensive and hypertensive blacks. DESIGN: Intracellular cations and cell membrane ATPase activity were studied in black patients with untreated essential hypertension and age-, weight- and height-matched normotensive controls. Platelet, erythrocyte and serum Mg2+, Ca2+, Na+ and K+ levels as well as platelet and erythrocyte membrane Na+,K(+)-ATPase, Ca(2+)-ATPase and Mg(2+)-ATPase activities were measured in all subjects. METHODS: Intracellular Na+ and K+ were measured by flame photometry and Mg+ and Ca+ by atomic absorption spectrophotometry. Cell membrane ATPase activity was determined by a colorimetric method. RESULTS: The hypertensive group consistently demonstrated depressed activity of each ATPase studied, with significantly lower serum Mg2+, serum K+, erythrocyte Mg2+ and platelet Mg2+ levels compared with the normotensive group. Platelet Na+ and Ca2+ and erythrocyte Ca2+ were significantly elevated in the hypertensive group. In the hypertensive group, mean arterial pressure (MAP) was inversely correlated with platelet and erythrocyte membrane Na+,K(+)-ATPase, Ca(2+)-ATPase and Mg(2+)-ATPase. Serum Mg2+, serum Ca2+ and platelet Mg2+ were negatively correlated with MAP in the hypertensive group whilst erythrocyte and platelet Ca2+ were positively correlated. In the normotensive group, platelet Mg2+ and MAP were negatively, and erythrocyte Ca2+ and MAP, positively correlated. CONCLUSIONS: Black patients with essential hypertension have widespread depression of cell membrane Na+,K(+)-ATPase, Ca(2+)-ATPase and Mg(2+)-ATPase activities with serum and intracellular Mg2+ depletion and cytosolic Na+ and Ca2+ overload, which may reflect an underlying membrane abnormality in essential hypertension. These cellular abnormalities may be related to the defective transport mechanisms that in turn may be aggravated by Mg2+ depletion. 相似文献
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目的 研究低硒(Se)低维生素E(VE)能否诱导大鼠肝细胞凋亡及cAMP和细胞内Ca^2+所起的作用。方法 以天然的和人工半合成的低Se低VE饲料喂养大鼠17周,采用流式细胞术检测肝细胞凋亡,采用放免法检测cAMP含量,Fura-2负载荧光分光光度法测定细胞内Ca^2+含量。结果 与补Se和VE组大鼠相比,低Se低VE组大鼠肝细胞凋亡显著增加;cAMP含量明显升高;细胞内Ca^2+含量明显升高。结 相似文献
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本研究对 39例轻、中度高血压病 (EH)患者 ,通过皂素溶血定磷法测定红细胞ATPase活力 ,原子吸收火焰法测定红细胞离子含量 ,Fura 2 /AM荧光探针测定红细胞 [Ca2 ]i含量 ,并以正常人作对照 ,旨在探讨部分EH的发病机制。结果显示 ,EH组红细胞Ca2 ATPase活力明显降低 ,P <0 .0 1,而Na K ATPase无显著性差异 ,两者之间呈正相关 (r =0 .46 ) ;红细胞Na 和 [Ca2 ] i含量 ,均显著增高 (P <0 .0 1和 0 .0 0 1) ,而细胞K 含量明显降低 ,以致Na /K 比值增大 (均P <0 .0 1)。研究表明 ,EH患者红细胞高Na ,高 [Ca2 ] i及低K ,是膜缺陷及阳离子转运异常的标志或结果 ,是部分EH患者发病的病理基础和分子生物学基础。红细胞Na 泵活力正常 ,Ca2 泵活力抑制 ,可为EH的一种临床类型 相似文献
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ATPase and calcium binding activities were studied in sarcolemmal membranes from hearts of male rats fed either a control or 2% cholesterol diet for different time periods. Studies with isolated membrane revealed a significant increase in Na+-K+ ATPase activity, sialic acid content and ATP-independent calcium binding capacity in the presence of 1.25 mM CaCl2 in the 6 week cholesterol fed group. By 12 weeks, Na+-K+ ATPase, Mg2+-ATPase and Ca2+-ATPase activities as well as ATP-independent calcium binding in the presence of 0.05 mM CaCl2 were increased in membranes from cholesterol fed rats. A significant increase (P less than 0.05) in the sarcolemmal cholesterol/phospholipid molar ratio, which is an indicator of a decrease in membrane fluidity, was also noted in the 12 week cholesterol fed group. Concanavalin A, which is believed to decrease membrane fluidity, stimulated both Mg2+ and Ca2+-dependent ATPase activities and increased ATP-independent calcium binding in control sarcolemmal preparations and these changes resembled those observed in the sarcolemma from cholesterol fed rats. Since concanavalin A did not alter the activity of Na+-K+ ATPase, it appears that some of the observed differences in sarcolemmal activities upon cholesterol feeding did not correlate well with changes in membrane order. At 24 weeks, there was a generalized depression in the sarcolemmal ATPase activities of the cholesterol group; both Mg2+ ATPase and Ca2+ ATPase were significantly less than in control.(ABSTRACT TRUNCATED AT 250 WORDS) 相似文献
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TU Wei-Feng LIN Gui-Fang SHEN Jian-Fan XU Jian-Guo 《World journal of gastroenterology : WJG》1998,4(5)
AIM To observe the changes in erythrocyte membrane ATPases and plasma lipid peroxides (LPO) patients with in abdominal surgery under intravenous procaine-balanced anesthesia.METHODS By determining the ATPase activities of erythrocyte membrane, effects of upper abdominal surgery under intravenous procaine-balanced anesthesia on the function of erythrocytes were observed in 15 patients undergoing cholecystectomy and gastrectomy (5 males and 10 females, aged 45.9±10.20 years and weighed 60.60kg±11.93kg). All patients were free from severe renal, hepatic, pulmonary, cardiac, metabolic and endocrinological diseases and acute infection for at least 2 weeks before surgery. Patients receiving any drug known to affect carbohydrate metabolism prior to anesthesia were excluded from the study.RESULTS Erythrocyte membrane Na+, K+-ATPase, Mg2+-ATPase, Ca2+, Mg2+-ATPase activities were not significantly changed 60min-90min after incision as compared with 30min before anesthesia, but were decreased markedly 10min and 24 hours after completion of operation (P<0.01). Plasma lipid peroxides (LPO) were increased significantly 24 hours after surgery (P<0.01) following an initially marked but transient reduction. Plasma LPO changes were not correlated with erythrocyte membrane ATPase activities, r=-0.0396, -0.0097 and 0.4383, respectively (P>0.05).CONCLUSION Abdominal surgical trauma under intravenous procaine-balanced anesthesia may be associated with the decreased ATPase activities of erythrocyte membrane and increased LPO in plasma. 相似文献
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INTRODUCTIONRedbloodcelsareresponsibleforthetransportationofoxygenandcarbondioxideintoandoutoftisuesandorgansofthebody.Itssha... 相似文献