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1.
掌腱膜挛缩症10例报告   总被引:1,自引:0,他引:1  
掌腱膜挛缩症是由于掌腱膜的增生和挛缩引起的远端手掌和手指的屈曲畸形,又称Dupuytren挛缩犤1犦。此病在欧洲白种人中很常见,但在亚非人种中罕见。本院自1995年以来共收治10例。资料与方法1.一般资料:本组10例,男9例,女1例;年龄52~69岁,平均58.8岁。病程1~5年,平均3.7年。病变部位:小指7例,环指5例,中指1例。其中3例有两指以上发病。2.临床表现:手掌皮下结节3例,纵形纤维条索6例,手指曲屈挛缩3例,均无手指麻木。所有病例均无手掌外伤史及家族史。3.治疗方法:本组10例均采用手术…  相似文献   

2.
指掌侧推进皮瓣修复手指屈曲挛缩畸形   总被引:1,自引:0,他引:1  
自2003年1月始应用指掌侧推进皮瓣法矫正手指屈曲挛缩畸形5例8指,获得良好治疗效果。 1一般资料 本组5例8指,男3例5指,女2例3指。年龄9~55岁。示指2指,中指1指,环指3指,小指2指。创伤或感染挛缩3例5指,烧伤瘢痕挛缩1例2指,掌腱膜挛缩症1例1指。病程最短1年,最长9年。  相似文献   

3.
目的 探讨掌腹腱膜挛缩症治疗的临床经验。方法 1980年以来,我院共治疗掌腱膜挛缩症22例,全部采用掌腱膜部分切除术。结果 19例病人获得满意的治疗效果,应用Tubiana’s法评价,手术后优良率为86.4%,效果良好。结论 本手术成功的关键在于:仔细分离皮瓣,避免皮肤坏死;避免血管神经束损伤;彻底切除挛缩腹膜,避免术后复发;彻底止血,防止血肿形成和感染;术后早期进行功能锻炼。  相似文献   

4.
掌腱膜挛缩症13例报告   总被引:4,自引:1,他引:3  
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掌腱膜挛缩症是以掌腱膜增生、收缩引起手指屈曲挛缩为特征的一种系统性进行性疾病,病因不清,治疗现多倾向于局限性切除即部分切除术为最常用。自1994年8月~2005年6月,我们采用掌腱膜部分切除术治疗掌腱膜挛缩症17例,疗效满意,报道如下。  相似文献   

6.
掌腱膜挛缩症的手术治疗   总被引:1,自引:2,他引:1  
掌腱膜挛缩症是指掌筋膜及手指筋膜广泛的纤维变性 ,以皮下结节和纤维束带为特征 ;导致手指继发性屈曲挛缩畸形 ,并伴皮下脂肪变薄 ,皮肤与病变处粘连凹陷为特征的疾病[1] 。1993年以来 ,我院共收治 17例 ,均采用手术治疗 ,获得了满意的效果。一、资料与方法1.一般资料 :本组共 17例 18指 ,男 16例 ,女 1例 ;年龄43~ 65岁 ,平均 5 3岁。右手 12例 ,左手 5例。病变分型 :(1)轻度 :手指掌指关节、近侧指间关节、远侧指间关节屈曲挛缩度数的总和 <45° ;本组共 9指。 (2 )中度 :手指DP、PIP、DIP屈曲挛缩度数的总和为 45°~ 90° ;本…  相似文献   

7.
掌腱膜挛缩症的治疗进展   总被引:2,自引:1,他引:1  
掌腱膜挛缩症(Dupuytren'sDisease)流行于北欧及其他欧美地区,是以掌腱膜增生、收缩引起手指屈曲挛缩为特征的一种病症。早在1833年GuillaumeDupuytren在巴黎就作过一个专题报告。170多年来对于该病的发病原因的探究一直是医学界关心的热门课题。近年来,杂志上有不少专题报告介绍利用分子生物学、细胞生物学遗传学技术进行掌腱膜挛缩症的基础研究,并取得了一定的进展。HandClinics在1999年还出一期专刊,特此综述供同道们参考。1掌筋膜综合体的解剖与病理1.1解剖学研究Rayan用手术显微镜和小型关节镜对10具尸体手的掌腱膜作详尽…  相似文献   

8.
1997年10月-2005年7月,我科对11例(17指)掌腱膜挛缩症的患者进行了手术治疗,效果满意。  相似文献   

9.
掌腱膜挛缩症的诊断与治疗   总被引:4,自引:1,他引:4  
掌腱膜挛缩是一种进行性增殖组织纤维变性病,好发于老年人,主要以掌腱膜增厚,收缩,手指屈曲挛缩为特征的手部畸形,多伴有手掌皮下脂肪变薄,皮肤凹陷并与掌腱膜粘连。本病以欧美国家多见,国内发病率较低,但近年发病率呈上升趋势,可能与社会人口中老年人比例增加,生活条件改善等原因有关[1-3]。1资料与方法本组13例,年龄30~73岁,男女之比为10:3,病程1个月~2年,其中12人为体力劳动者,1人为非体力劳动者。患病手指:中指2指,环指10指,小指8指。双侧3例,单侧10例,其中左侧者4例,右侧者6例。根据黄硕麟分型标准:Ⅱ型1例,Ⅲ3例,Ⅳ9例。有明确家族…  相似文献   

10.
指背皮瓣旋转治疗严重手指屈曲挛缩畸形12例车遵军,张秀敏,郑元涛我们自1992年1月~1994年12月对12例27个手指屈曲畸形患者,利用指背皮瓣旋转进行修复,疗效满意。现报告如下。1临床资料本组12例中男9例,女3例;年龄2~65岁(平均23.5岁...  相似文献   

11.
The boutonnière deformity in Dupuytren's disease has been found to be due to fibrous contraction of the transverse retinacular ligament which anteriorly displaces the fibres of the extensor apparatus. Complete ablation of this ligament and liberation of the extensor apparatus allows easy extension of the finger when operating on severe cases of Dupuytren's contracture.  相似文献   

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PURPOSE: To report a congenital anomaly of the middle finger. METHOD: Nine patients (16 digits) are reported with congenital flexion deformity of the metacarpophalangeal (MCP) joint of the middle finger. Three patients (4 digits) had isolated deformities to the middle finger and in 6 the deformity was part of congenital ulnar drift (CUD) of the hand. Three patients had Freeman-Sheldon syndrome, 2 had nonsyndromic CUD, and 1 had arthrogryposis multiplex congenita. In CUD patients the middle finger had substantially greater flexion deformity of the MCP joint in comparison with other digits. Seven patients were treated surgically and 2 were treated nonsurgically. Five of the surgical patients had bilateral middle finger involvement. RESULTS: During surgery on 12 digits sagittal band hypoplasia of varying degrees was encountered in all patients and in all patients the extensor tendon of the middle finger was underdeveloped and often ulnarly displaced. Longitudinal imbrication of the remnants of the extensor tendon and centralizing the tendon if necessary by radial sagittal band reefing improved MCP joint flexion deformity. CONCLUSIONS: Congenital middle finger-in-palm deformity in our patients was caused by sagittal band and extensor tendon hypoplasia.  相似文献   

17.
A previously undescribed lesion of Dupuytren's disease is presented. An oblique cord coursed parallel to the oblique retinacular ligament of Landsmeer, but inserted proximal to the proximal interphalangeal joint, tethering the central slip and radial lateral band across the intervening transverse retinacular ligament. Contraction of this cord caused a rigid swan-neck deformity. Excision of the cord resulted in complete resolution of the deformity and a full range of motion in the affected digit.  相似文献   

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A 10-month-old child presented with a lack of extension at the distal interphalangeal joint. Despite the absence of trauma, a provisional diagnosis of mallet finger led to treatment using a short dorsal splint. Four months later the flexion had increased and included the proximal interphalangeal joint. Palpation revealed a palmar cord on the lateral aspect of the finger. Surgery disclosed a typical Dupuytren cord and the histology supported this diagnosis. There was no known family history of the disease. There was no sign of recurrence 27 months after surgery in this case of Dupuytren's disease that was present at birth.  相似文献   

20.
We present a prospective study, with 3-year follow-up, of the role and outcome of fasciectomy plus sequential surgical release of structures of the proximal interphalangeal joint in Dupuytren's contracture of the little finger. Our treatment programme involves fasciectomy for all patients followed by sequential release of the accessory collateral ligament and volar plate as necessary. Of the 19 fingers in the study, eight achieved a full correction by fasciectomy alone, and in these cases there was a fixed flexion deformity of 6 degrees at 3 months and 8 degrees at 3 years. The remaining 11 fingers (initial mean deformity 70 degrees flexion) were left with a fixed flexion deformity of 42 degrees after fasciectomy which reduced to 7 degrees with capsulo-ligamentous release. This increased to 26 degrees at 3 months but then remained relatively stable, increasing only to 29 degrees at 3 years. In our experience sequential proximal interphalangeal joint release has led to consistently good results with few complications in the correction of severe Dupuytren's disease of the little finger.  相似文献   

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