先天性脉络膜缺损并视网膜脱离术后光凝治疗观察

目的 探讨先天性脉络膜缺损及先天性脉络膜缺损合并视网膜脱离术后的光凝治疗疗效.方法 选择先天性脉络膜缺损合并视网膜脱离患者10例,在玻璃体切除术后沿脉络膜缺损边缘行光凝治疗,对侧眼在脉络膜缺损区边缘做防治性光凝治疗,光凝病例对侧眼中有3只眼在脉络膜缺损区域内发现裂孔,但尚未发生视网膜脱离.结果 实施光凝治疗20只眼,随访观察玻璃体切除术后的10只眼视网膜均在位,无视网膜脱离再次发生;对侧10眼缺损区边缘光凝斑反应良好,均未发现视网膜脱离的存在.结论 先天性脉络膜缺损以及先天性脉络膜缺损合并视网膜脱离术后光凝治疗是一种安全、有效、可行的方法.     

玻璃体切割术治疗先天性脉络膜缺损并发视网膜脱离

目的观察玻璃体切割术治疗先天性脉络膜缺损并发视网膜脱离的效果。方法对42例50眼先天性脉络膜缺损并发视网膜脱离的患者,行玻璃体切割术联合硅油填充,术中在高倍显微镜下用眼内导光查找视网膜裂孔。观察视网膜脱离的复位和视力恢复情况。结果随访12~36个月(平均18个月),有43眼视网膜脱离复位,总治愈率为86·00%.术后视力≥0.02者44眼,≥0.05者36眼。结论充分的玻璃体切割术合并封闭缺损区、硅油填充,是治疗先天性脉络膜缺损并发视网膜脱离的有效方法。     

合并脉络膜脱离的视网膜脱离的临床观察

目的:探讨合并脉络膜脱离的视网膜脱离手术治疗方法和手术时机的选择并观察疗效。方法:总结、分析2010-01/2012-01因视网膜脱离合并脉络膜脱离在我院住院的患者45例45眼,其中原发性裂孔源性视网膜脱离合并脉络膜脱离38例,复发性视网膜脱离合并脉络膜脱离7例。手术方法包括巩膜环扎、经巩膜穿刺口脉络膜上腔引流、玻璃体视网膜手术、眼内光凝、C3F8填充术或硅油填充术。结果:患眼45眼均行巩膜环扎术,均于手术中成功引流脉络膜上腔液体,6眼行C3F8填充,39眼行硅油填充术,45眼视网膜脉络膜全部复位,视网膜裂孔封闭。其中23眼术后视力≥0.1。结论:合并脉络膜脱离的视网膜脱离,采用巩膜环扎和玻璃体视网膜手术联合经巩膜穿刺口脉络膜上腔积液引流的联合手术方式有效。     

高度近视眼黄斑裂孔视网膜脱离的手术治疗

目的 探讨治疗高度近视眼黄斑裂孔视网膜脱离手术的方法。方法 对172只眼（169例）高度近视眼黄斑裂孔视网膜脱离行玻璃体切除联合眼内光凝黄斑裂孔、12％C3F8填充术。术后取面向下体位两周。结果 172只眼黄斑裂孔闭合、视网膜复位。1只眼在气液交换过程中出现脉络膜大出血而作硅油填充术。172只眼术后视力均有不同程度的提高。结论 玻璃体切除联合眼内光凝黄斑裂孔、12％C3F8填充术是治疗高度近视眼黄斑裂孔视网膜脱离和提高手术成功率的安全有效的方法。     

先天性脉络膜缺损合并视网膜脱离的临床特点

目的探讨先天性脉络膜缺损合并视网膜脱离的临床特点,寻找较好的手术方法,以提高疗效。方法对８例（８只眼,其中７只眼视盘在缺损区内）脉络膜缺损合并视网膜脱离患者,施行玻璃体视网膜手术,术中在高倍显微镜下观察视网膜裂孔、玻璃体后界膜、缺损区的情况,行相应处理,硅油填充。结果术后视网膜均获得解剖复位,视力均有提高。结论此类视网膜脱离可因位于缺损区内的视网膜裂孔造成,其裂孔位于缺损区内带状视网膜脱离的边缘处,无玻璃体后脱离、缺损区巩膜外膨出等特点。充分的玻璃体切除合并封闭缺损区、硅油填充,可望成功     

合并视网膜下增生的儿童孔源性视网膜脱离手术

目的观察巩膜扣带术和玻璃体切除术两种手术方式治疗先天异常因素所致儿童视网膜脱离的近期疗效。方法儿童48例（56眼）先天异常因素所致视网膜脱离行巩膜扣带术和玻璃体切除术48例（56眼）。结果（1）巩膜扣带术33眼：随访6个月以上一次手术视网膜平伏20眼（60．81％）;残留网膜下液7眼,无效6眼。（2）玻璃体切除手术23眼：随访6个月一次手术视网膜复位14眼（60．86％）;二次手术视网膜复位6眼,3眼视网膜浅脱离,未取硅油。从近期疗效看,两种手术方式的一次手术成功率和视功能恢复程度的差异无统计学意义（P〉0．05）。结论儿童先天异常因素所致视网膜脱离如增生不重可优先考虑巩膜扣带术,如网膜不能平伏,再行玻璃体切除手术,但远期疗效需待进一步观察。     

玻璃体切割手术治疗先天性脉络膜缺损合并视网膜脱离

脉络膜缺损是一种少见的先天性眼底病 ,由眼泡胚裂闭合障碍所致。由于脉络膜缺损处视网膜发育不良呈变性萎缩状态 ,易出现视网膜裂孔或因玻璃体条索牵引而致视网膜脱离。以往采用巩膜扣带术治疗这类视网膜脱离成功率低 ,我们采用玻璃体切割手术治疗这种复杂型视网膜脱离 9例 9只眼取得较好效果 ,报道如下。1　对象和方法先天性脉络膜缺损合并视网膜脱离患者 9例 9只眼 ,男 4例 ,女 5例 ;右眼 7只 ,左眼 2只。年龄 10～ 32岁 ,2 5岁以下者 8例 ,占 88.9%。病程 0 .5～ 3.0个月。术前患眼视力为光感～0 .15 ,视力在 0 .0 1以下 7只眼 ,占 77.8…     

玻璃体视网膜手术联合巩膜扣带术治疗复杂性视网膜脱离

使用玻璃体视网膜手术联合巩膜扣带术的方法，进行复杂性视网膜脱离的治疗，达到提高治愈成功率的目的。（１）进行常规的巩膜扣带术，（２）进行睫状体平部三切口的闭合式玻璃体切除手术，包括膜剥离、气体、硅油眼内填充等技术。随访１－１６月，２９眼中，２２眼达到解剖复位，成功率７５．８％；其中，１５眼行硅油眼内填充，１２眼视网膜复位，成功率８０％，闭合式玻璃体手术的开展，使复杂性视网膜脱离的治疗成为可能，其解剖     

玻璃体视网膜手术联合脉络膜上腔引流术治疗视网膜脱离合并脉络膜脱离

目的:探讨视网膜脱离合并脉络膜脱离的手术治疗方法、手术时机并观察疗效。方法:总结、分析2000-02/2005-02间因视网膜脱离合并脉络膜脱离在我院接受手术治疗的连续患者共36例36眼,其中原发性孔源性视网膜脱离合并脉络膜脱离30例,复发性视网膜脱离合并脉络膜脱离6例。手术方法包括巩膜外环扎、经巩膜穿刺口脉络膜上腔引流、玻璃体视网膜手术、眼内光凝、硅油或C3F8填充术等。结果:36只患眼于术中成功引流脉络膜上腔液体。硅油填充30眼、C3F8填充6眼,视网膜脉络膜全部复位、视网膜裂孔封闭。36眼术后视力为手动至0.3,其中26例术后视力≥0.1。结论:玻璃体视网膜显微手术联合经巩膜穿刺口脉络膜上腔积液引流术是处理脉络膜脱离合并视网膜脱离的有效手术方法。     

玻璃体视网膜手术治疗脉络膜缺损合并视网膜脱离

玻璃体视网膜手术治疗脉络膜缺损合并视网膜脱离王丽丽朱赛林朱忠桥关键词无脉络膜／外科手术视网膜脱离／外科手术玻璃体切除术先天性脉络膜缺损是由于胎儿时期胚胎裂闭合不全所致。它常伴有许多眼部组织发育不良。本病伴视网膜脱离占４０％［１］。用传统的巩膜扣带术治...     

Strabisme Alternant - Etude clinique - traitement

The author defines motor and sensory alternation: the term alternation should not be used in isolation, it should always be accompanied by the name of the parameter concerned. Sensory alternation is always found together with motor alternation but the reverse is not true.The examining criteria for a diagnosis of sensory alternation are given, sensory alternation must not be confused with alternating inhibition. Working from clinical observations of cases of motor alternating strabismus, the author selects 2 types of binocular sensory relations which allow one to differentiate between:- cases of primary alternating strabismus- cases of secondary alternating strabismusThese forms will develop in different ways; in both cases a cure is possible providing that the right treatment is prescribed and once prescribed carefully followed, etc. It is always a case of serious forms of strabismus whose developmental period is spread over several years.According to the authors, the frequency of cases of true primary strabismus is from 1–3%, the frequency of cases of secondary alternating strabismus varies according to the type of therapy practised on cases of monocular strabismus with amblyopia. These latter will become cases of alternating strabismus under the influence of certain types of therapy carried out over several years (penalization, rocking, alternated occlusion, etc...).Experimental data on kittens confirm clinical data; kittens placed in abnormal environments during the sensitive period will show modification in the distribution of cortical cells and the absence of binocular cells (either because the excitation of the two eyes was not simultaneous, or not identical: artificial strabismus, occlusion, opaque glasses). This disturbances become irreversible after a certain period of exposure (a function of age, length of exposure, etc...).It is thus necessary to bear in mind: 1) the iatrogenic risks of certain orthoptic treatments, 2) the necessity for a binocular form of treatment as soon as possible, as once a certain stage is passed, cortical plasticity diminishes and the elaboration of normal binocular relations becomes impossible.

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Effects of Adenosine Agonists on Intraocular Pressure and Aqueous Humor Dynamics in Cynomolgus Monkeys

The effects of single or multiple topical doses of the relatively selective A₁adenosine receptor agonists (R)-phenylisopropyladenosine (R-PIA) and N⁶-cyclohexyladenosine (CHA) on intraocular pressure (IOP), aqueous humor flow (AHF) and outflow facility were investigated in ocular normotensive cynomolgus monkeys. IOP and AHF were determined, under ketamine anesthesia, by Goldmann applanation tonometry and fluorophotometry, respectively. Total outflow facility was determined by anterior chamber perfusion under pentobarbital anesthesia. A single unilateral topical application of R-PIA (20–250 μg) or CHA (20–500 μg) produced ocular hypertension (maximum rise=4.9 or 3.5 mmHg) within 30 min, followed by ocular hypotension (maximum fall=2.1 or 3.6 mmHg) from 2–6 hr. The relatively selective adenosine A₂antagonist 3,7-dimethyl-1-propargylxanthine (DMPX, 320 μg) inhibited the early hypertension, without influencing the hypotension. Neither 100 μg R-PIA nor 500 μg CHA clearly altered AHF. Total outflow facility was increased by 71% 3 hr after 100 μg R-PIA. In conclusion, the early ocular hypertension produced by topical adenosine agonists in cynomolgus monkeys is associated with the activation of adenosine A₂receptors, while the subsequent hypotension appears to be mediated by adenosine A₁receptors and results primarily from increased outflow facility.