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1.
目的 评价Nd:YAG激光对不同充填材料与牙本质之间边缘微渗漏的影响。方法 选用人离体前磨牙36颗,制备V类洞,随机分为激光组和对照组,激光组使用80 rnJ、10 Hz脉冲Nd:YAG激光照射后,分别用全酸蚀粘接树脂、自酸蚀粘接树脂和玻璃离子充填洞型,对照组不使用激光,同样用如前所述的3种材料充填洞型。将所有标本浸泡在2%亚甲基蓝溶液中24 h后,用金刚砂片从颊舌向通过充填物中央纵向切开牙体,显微镜下观察龈壁染料渗透的深度。采用SAS8.0软件对数据进行统计学处理。结果 经Nd:YAG激光照射后尽管3种材料与牙本质之间微渗漏程度均有降低趋势,但只有玻璃离子组激光照射后较未照射组微渗漏降低具有统计学意义(P<0.05)。结论 脉冲Nd:YAG激光照射牙本质壁,在一定程度上可能会减少玻璃离子充填材料与洞壁的微渗漏。  相似文献   

2.
目的:研究固化光源光照方向及角度对复合树脂固化后微渗漏的影响。方法:选择63颗离体前磨牙,在颊面牙颈部以釉牙骨质界为中心制备圆形V类洞,常规酸蚀粘结,充填复合树脂,将所有实验牙以光源不同朝向随机分为A组:光源方向朝向龈壁;B组:光源方向朝向牙合壁;C组:光源方向朝向近中壁。再将A、B、C每组按固化光源与充填材料表面所成的角度不同随机分1组:成30°光照;2组:成60°光照;3组:成90°光照。所有实验牙均光照40s。分别浸泡于0.1%罗丹明B荧光染色剂中24h。然后经修复体中央,沿牙体长轴沿颊舌向纵向剖成两半,在激光扫描共聚焦显微镜(Laser Scanning Confocal Microscopy,LSCM)下分别观察检测微渗漏大小。结果:龈壁处的微渗漏情况:在光源的不同朝向下,不同的光照角度对龈壁处微渗漏的影响均具有统计学意义,光照角度均为90°时微渗漏值最小(P〈0.05);牙合壁处的微渗漏情况:当光源朝向牙合壁及近中壁照射时,牙合壁处3组之间微渗漏有明显差异(P〈0.05),光照角度90°时微渗漏最小;当光源朝向龈壁时,牙合壁处微渗漏之间没有明显的差异。结论:固化光源的光照方朝向及光照角度对微渗漏有影响。  相似文献   

3.
Nd:YAG激光对Dyract复合体充填微渗漏的影响   总被引:2,自引:0,他引:2  
目的 观察Nd :YAG激光照射洞缘牙釉质对复合体Dyract充填微渗漏的影响。方法 新鲜拔除的完整离体牙 5 0颗 ,随机分为实验组 ( 2 6颗 )和对照组 ( 2 4颗 )。在样本牙的颊舌面制备V类洞 ,对照组常规完成Dyract充填 ,实验组用Nd :YAG激光照射洞缘牙釉质后再行Dyract充填。所有样本经冷热循环处理后 ,再进行微渗漏实验。用体视显微镜和SEM观察并拍照记录。结果 激光光蚀组充填体边缘微渗漏比对照组明显低 (P <0 .0 5 )。微渗漏实验与扫描电镜观察结果一致。结论 Nd :YAG激光的光蚀作用有助于增强复合体Dyract与牙体组织的结合 ,改善边缘封闭 ,减少微渗漏 ,有利于充填成功  相似文献   

4.
目的:评价Nd:YAG激光对不同水门汀与牙本质之间边缘微渗漏的影响。方法:选用人离体磨牙48颗,间接法制作树脂嵌体,随机分为激光组和对照组,激光组使用80 mJ、10 Hz脉冲Nd:YAG激光照射后,分别用6种水门汀粘结树脂嵌体。全部试件放在37℃生理盐水中7 d,并冷热循环300次后,放置于0.5%的品红溶液中染色24 h,沿嵌体长轴纵剖后在根管显微镜下观察微渗漏情况,采用SPSS 17.0软件包对数据进行统计学处理。结果:6组牙本质经Nd:YAG激光照射后微渗漏程度均有降低趋势,且前2种水门汀有统计学差异(P<0.05)。另外,树脂水门汀与牙本质之间的微渗漏程度比水基水门汀低。结论:80 mJ、10 Hz脉冲Nd:YAG激光照射牙本质壁,可提高洞壁的密合度,减少微渗漏。  相似文献   

5.
目的应用离体牙比较复合树脂与Er:YAG激光及传统牙钻制备窝洞洞壁间微渗漏的差异。方法将30颗前磨牙随机分为3组,每组10颗牙,制备Ⅴ类洞:Ⅰ组牙钻预备加酸蚀处理;Ⅱ组单纯激光预备组、Ⅲ组激光预备加酸蚀处理组,经冷热循环染色后,在体视显微镜下记录微渗漏程度并应用非参数统计方法分析数据。结果各组内颈壁的染料渗透情况均比壁明显(P〈0.05);在壁,单纯激光预备组产生的微渗漏最严重(P〈0.05),激光预备加酸蚀处理组与牙钻预备加酸蚀处理组微渗漏无统计学差异;在颈壁,三组充填体边缘微渗漏情况无统计学差异。结论Er:YAG激光可代替牙钻备洞,结合酸蚀处理与充填体有良好的密合度。  相似文献   

6.
陈燕  杨明华  李晓玲  杨洁 《口腔医学研究》2011,27(11):1005-1007
目的:评价Er:YAG激光备洞对树脂充填材料边缘微渗漏的影响。方法:选择48颗年轻前磨牙随机分成4组,每组12颗牙,用Er:YAG激光或高速牙钻制备ⅴ类洞。对照组:牙钻备洞+35%磷酸酸蚀;实验1组:激光备洞+35%磷酸酸蚀;实验2组:激光备洞+自酸蚀粘接;实验3组:单纯激光备洞。树脂充填,经冷热循环试验后染色24h,体视显微镜下观察微渗漏,统计学处理数据。结果:龈壁的染料渗透,4组间无统计学差异;壁的染料渗透,以实验3组较明显,与对照组、实验1、2组有统计学差异(P〈0.05);各组内壁的染料渗透均比龈壁轻,但没有统计学差异。结论:Er:YAG激光备洞,35%磷酸酸蚀或自酸蚀粘接处理可减少洞壁与树脂充填边缘的微渗漏。  相似文献   

7.
目的研究脉冲Nd:YAG激光照射牙本质后对光固化树脂充填体边缘微渗漏的影响,以探讨脉冲Nd-YAG激光在龋病治疗中的临床应用意义。方法选用正畸要求拔除的正常双尖牙30颗,随机分为A。B,C三组,每组10颗,用高速裂钻备洞(洞长、宽为3mm;洞深为2mm),A组为对照组,常规酸蚀树脂充填;B,C两组分别用50mJ,10Hz和100mJ,10Hz的脉冲Nd:YAG激光能量照射窝洞牙本质壁30s后树脂充填,分别用体视显微镜和扫描电镜观察充填体边缘和洞壁结合情况。结果.体视显微镜下观察:A组的牙本质洞壁与光固化树脂充填体间染料渗入较多,着色较深;B、C组的牙本质洞壁与光固化树脂充填体间染料渗入较少,着色较浅。扫描电镜下观察:B、C组的牙本质洞壁与光固化树脂充填体间裂隙明显小于未经激光处理的对照组。结论脉冲Nd:YAG激光处理牙本质后进行光固化复合树脂充填,可以减少充填体微渗漏,有利于充填的成功.  相似文献   

8.
本文目的是研究用牙本质粘接剂和窝沟封闭剂封闭复合树脂修复的Ⅴ类洞边缘对微漏现象的影响。材料和方法选择30只拔除的无龋前磨牙,在颊面的釉牙骨质界处制洞,洞深1.5mm,近远中径4mm,(牙合)龈壁距2mm。酸蚀(牙合)壁30秒钟,用ScotchbondDual Cure(3M Dental Products Div)处理,颈  相似文献   

9.
目的 研究Er∶YAG激光备洞及酸蚀处理对复合树脂充填体微渗漏的影响.方法 将40颗离体前磨牙随机平均分为5组制备洞型:A组,高速牙钻预备加酸蚀;B组,Er∶YAG激光预备;C组,Er∶YAG激光预备加酸蚀;D组,高速牙钻预备加Er∶YAG激光蚀刻;E组,高速牙钻预备加Er∶YAG激光蚀刻后酸蚀.所有样本经复合树脂充填后交替放置于5℃与55℃水中各1 min,间隔45 s,共2000个周期进行冷热循环,然后用0.2%亚甲基蓝染色后颊舌向劈开,体视显微镜下观察剖面,记录其微渗漏情况,并进行统计学分析.另选6颗离体前磨牙,随机分为3组,按上述A、B、C三组方法制备,扫描电镜下观察其牙本质界面的结构.结果 在(牙合)壁与龈壁,B组染料渗入严重,到达洞底,微渗漏最大,与其他各组相比有统计学差异(P<0.05),A、C、D、E组染料渗入表浅,微渗漏程度之间没有统计学差异(P>0.05).扫描电镜下,A组牙本质表面较平整,无玷污层存在,牙本质小管口开放.B组牙本质表面不平整如鳞片状,无玷污层,牙本质小管口开放,直径小于酸蚀组.C组牙本质小管口开放,无玷污层存在,管周牙本质脱矿明显.结论 单纯Er∶YAG激光预备比传统牙钻制备结合酸蚀处理洞型更易发生微渗漏,若Er∶YAG激光预备结合酸蚀剂处理可以使微渗漏程度减小,Er∶YAG激光蚀刻可以达到和酸蚀剂类似的效果.  相似文献   

10.
目的:比较在自酸蚀黏结剂的应用条件下,Er,Cr:YSGG激光制备与传统牙钻制备离体牙牙颈部洞对光固化复合树脂边缘微渗漏的影响。方法:将20个因正畸拔除的新鲜完整无龋损、无隐裂、无充填物的前磨牙随机分为两组(n=10),分别使用牙钻和Er,Cr:YSGG激光制备牙颈部洞。窝洞制备后均匀涂覆FL-BOND,用TPH复合树脂进行分层充填,打磨、抛光,37℃生理盐水中存放7 d后,进行500次温度循环实验(5±2)℃—(55±2)℃。将上述所有样本置0.5%碱性品红液中室温浸泡24 h后,沿牙体长轴通过修复体正中纵行剖开,采用染料渗入法和扫描电镜方法观察充填体微渗漏情况,运用SPSS 11.0软件包进行统计学处理。结果:牙钻制备组与激光组相比,无论牙合壁还是龈壁的染料渗入评分和边缘微缝隙宽度均无显著性差异(P>0.05)。结论:在自酸蚀黏结剂的应用条件下,Er,Cr:YSGG激光制洞不能显著减少光固化复合树脂边缘微渗漏的发生。  相似文献   

11.
A model describing the relationship between self-reported quality of restorative dentistry and dentist characteristics for 119 Montana general dentists is presented. The best predictors formed a significant model explaining 22% of the variance of the quality measure. Results are contrasted with a previous estimation of the model for 102 Washington general practitioners. Evidence for the external validity of the model is presented.  相似文献   

12.
The reduction of hydrazones is generally suggested to proceed through a reductive cleavage of the nitrogen–nitrogen bond followed by a reduction of the carbon–nitrogen bond. This sequence of reduction processes is here supported for fluorenone (V) and benzophenone (VI) hydrazones as well as by a comparison of the reduction of fluorenone and benzophenone hydrazonium ions (I,III) with corresponding imines (II,IV). Another proof of the presence of imines as intermediates is the splitting of four-electron waves of hydrazones V and VI and hydrazonium ions I and VIII into two waves at pH < 2. This has been interpreted as due to differences in slopes dE1/2/dpH and pKa-values of protonated hydrazine derivatives on one side and corresponding imines on the other. In this pH-range imines formed in reductions of VI and VIII are reduced in a single two-electron wave, those of I and V in two one-electron steps. Fluorenone imine (II) is sufficiently stable to allow recording of time-independent current–voltage curves between pH 6 and 11. In this pH-range the imine (II) is reduced in two one-electron steps. Benzophenone imine (IV) has been found stable between pH 4.6 and 12. At pH 4.6–8 the reduction of the imine IV takes place in a single two-electron step, at pH 8–12 in two one-electron steps. Final proof of the initial cleavage of the N–N bond is presented by comparison with the reduction of nitrones.  相似文献   

13.
目的:研究、比较不同剂型玻璃离子水门汀的溶解性和表面微观形态改变,为临床使用提供依据.方法:将3M树脂加强型玻璃离子水门汀(水粉剂型)、GC玻璃离子水门汀(水粉剂型)及GC玻璃离子水门汀(双糊剂型)分别在人工唾液中浸泡30 d,冷热循环15000次,烘干测重,比较前后质量变化,计算溶解率,并用扫描电镜观察表面微观改变.结果:不同剂型的玻璃离子水门汀溶解率由高到低分别为3M树脂加强型玻璃离子水门汀(水粉剂型)、GC玻璃离子水门汀(水粉剂型)、GC玻璃离子水门汀(双糊剂型).3种玻璃离子水门汀经浸泡溶解后,SEM扫描表面微观形态可观察到GE玻璃离子水门汀(双糊剂型)表面形态改变较少,其他2组玻璃离子水门汀表面微观改变较多.结论:双糊剂型玻璃离子水门汀理化性能及溶解率均低于传统水粉剂型,是未来临床修复治疗的的良好选择.  相似文献   

14.
The present paper on the design of clinical trials of periodontal therapy first addresses the issue of the etiology of periodontal disease. It is suggested that most if not all forms of destructive periodontal disease are caused by microorganisms and that there are different forms of disease with different microbial etiologies. The progressive nature of destructive periodontal disease is subsequently discussed and it is emphasized that, in a given patient, periodontal sites which show signs of inflammation and attachment loss may not over a period of several months and years show further sign of attachment loss. The present methods of assessing periodontal disease do not allow us to discriminate between potentially active and inactive sites in untreated patients. The significance and variability of indicators of periodontal disease such as bleeding on probing, probing pocket depth and probing attachment level measurements are discussed. The errors inherent in the various measurements are analyzed and suggestions are presented describing how alterations in any of the above parameters could be identified and presented in a clinical trial. Of concern for the statistical analysis of clinical data of periodontal disease is the definition of the "experimental unit". For a number of years, the "experimental unit" in periodontal trials was the patient. It is clear, however, that different sites within the same individual show different patterns of disease progression and lesion morphology and often respond differently to periodontal therapy. Statistical analyses must consequently be designed which recognize differences in site-to-site infection and lesion morphology within a common host. Until such analyses are available, the investigator should be wary of pooling data within the same individual, since such pooling may obscure meaningful alternatives which may take place in individual periodontal sites. Some goals of periodontal therapy are subsequently identified. 4 goals are discussed more in detail, namely: to establish conditions which will allow the patient to maintain a dentition without further breakdown of the periodontium; to reduce pocket depth to establish an anatomy in the dentogingival region which with proper maintainance care will prevent the re-establishment of the subgingival infection; to gain attachment as a result of treatment; to assess the effect of a certain chemotherapeutic agent on periodontal disease.  相似文献   

15.
ObjectiveLeukoplakia is the most common potentially malignant disorder preceding oral cancer. Chemiluminescence has been developed as an adjunct to conventional examination for the diagnosis of these potentially malignant disorders. This study was conducted to assess the efficacy of chemiluminescence in the diagnosis of leukoplakia and to compare the results with histopathological examination.Study designA total of 50 patients with leukoplakia were included from the outpatients attending the Department of Oral Medicine and Radiology, Dental Hospital, Bengaluru, Karnataka, India. These patients were subjected to conventional oral examination followed by chemiluminescent examination with Vizilite (Zila, Fort Collins, CO, USA) and biopsy for histopathological confirmation.ResultsThe sensitivity, specificity, positive predictive value, and negative predictive value of chemiluminescence were 93.75%, 55.56%, 78.95%, and 83.3%, respectively. The overall accuracy of chemiluminescence was 80%. A statistically significant association was observed between histopathology results and chemiluminescence results.ConclusionAlthough it is an easy, safe, minimal time consuming, and noninvasive technique, it has only adjunctive utility and it does not replace biopsy for the diagnosis of leukoplakia.  相似文献   

16.
颌骨动静脉畸形的栓塞治疗   总被引:9,自引:0,他引:9  
目的:总结直接穿刺结合经血管内介入栓塞治疗颌骨动静脉静脉畸形的经验。方法:收治凳骨动静脉畸形患者6例,均进行了介入栓塞治疗。采用的栓塞材料为附凝血棉纤毛的螺圈,聚乙烯醇泡沫微粒和二氰基丙烯酸对丁酯。数字减影颈动脉造影在PHILIPSV300下完成。结果6例颌骨动静脉畸形患者中4,例急性出血得到了快速、有效控制,1例慢性渗血的右下 骨动静脉畸形患者,介入栓塞治疗,拔除松动的右下凳第一磨牙,有效地控制了出血,另1例伴局部软组织搏动性膨隆的上凳骨动静脉畸形患者,介入治疗后膨隆的搏动性得到明显改善,栓塞治疗后分别随访3-24个月,均未发现有口腔内渗血或出血。随访的X线片上,病灶区可见新骨形成。结论:局部穿刺结合经血管内介入栓塞治疗颌骨动静畸形是一种安全、有效的治疗方法。  相似文献   

17.
目的研究正畸患者曲面体层片上的切牙影像失真发生情况,并分析其原因。 方法从中山大学附属口腔医院放射科影像数据库中选取500例正畸患者的曲面体层片和头影测量侧位片,所有曲面体层片均采用咬合杆投照,分别从切牙牙体影像放大、缩小、牙根变短、根尖模糊等评价指标分析上下颌切牙影像失真的发生情况,在头影测量侧位片上测量中切牙根尖-对颌切牙切缘的距离,探讨切牙影像失真发生的原因。采用SPSS 19.0统计软件对所得数据进行统计学检验。 结果500例患者中,切牙牙体影像正常者共417例,切牙牙体影像失真者共83例,影像失真发生率16.6%,其中切牙牙体影像放大17例、牙体影像缩小0例、牙根变短30例,牙根影像变短伴模糊36例。影像失真患者的根尖-切缘距离大于影像正常的患者,差异有统计学意义(F = 5 187.18,P = 0);影像失真患者的覆盖值大于影像正常的患者,差异有统计学意义(F>477,P = 0)。 结论严重牙颌面畸形如反 、深覆盖是导致曲面体层片的切牙影像失真的主要原因之一。  相似文献   

18.
目的测量正常青年Monson球面半径。方法选择60名(男30名,女30名)正常青年制取全口印模,应用立体摄影成像的原理与方法对Monson球面半径进行测量和统计学处理。结果Monson球面的半径平均为10.173 cm,大于理论值10.160 cm,差异有显著性(P<0.01);男、女性球面半径差异无显著性。结论本实验所得到的数据可作为全口义齿修复中记录颌位关系的一个参量。  相似文献   

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鼻测量法的进展   总被引:1,自引:1,他引:0  
唇裂术后继发畸形是指唇裂修复术后,仍遗留或继发于手术操作和生长发育变化而表现出来的一类畸形[1]。包括唇畸形、鼻畸形和颌骨畸形。其修复较原发性唇裂修复更复杂,更灵活多变。而导致其修复复杂性的一个重要原因即是局部组织结构复杂变异和缺乏可靠的三维测量手段[2],鼻畸形  相似文献   

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