Treatment of multidirectionalshoulder instability in the athlete

Multidirectional instability in the athlete refers to symptomatic laxity of the shoulder in more than one direction.This is in contrast to unidirectional instability, which involves subluxation or dislocation in only one direction. The diagnosis and treatment of this disorder can be challenging owing to the unusual demands athletes place on their shoulders to be effective in their sport. The laxity required for overhand throwing, gymnastics, swimming, volleyball and tennis — while increased compared with that required for activities of daily living or with that of the opposite shoulder — must be symptomatic to be pathologic. Formerly, it was thought that generalized ligamentous laxity was a requirement for multidirectional instability, but now it is realized many athletes have multidirectional instability in the setting of otherwise normal ligamentous laxity. Nonoperative treatment is frequently successful in these athletes. When conservative management is unsuccessful, the capsular shift procedure has demonstrated good results in allowing these athletes to return to their sport. Arthroscopic and thermal capsulorrhaphy also offer other options for surgical treatment of this problem and will be briefly discussed.     

Treatment of glenohumeral instabilityin the pediatric athlete

Shoulder instability in the skeletally immature athlete is an uncommon but challenging clinical problem. Theclassification of shoulder instability in the pediatric athlete is similar to that in the adult athlete, but may also include the voluntary and habitual dislocator. The natural history of such instability depends on the classification of instability type. Traumatic anterior dislocation has an exceedingly high recurrence, and posterior dislocation is less well known. A natural history of voluntary instability is also uncertain. As in the adult athlete, clinical evaluation is made by history and physical exam. An understanding of the bony development of the glenohumeral joint is crucial in interpreting radiographs of the skeletally immature athlete with glenohumeral instability. The treatment of glenohumeral instability will depend on the classification of the instability type. Surgical treatment, when indicated, should emphasize an anatomic approach aimed at correcting the underlying glenohumeral pathology. This article is intended to provide an overview of the approach to diagnosis and treatment of glenohumeral instability in the pediatric athlete.     

Traumatic anterior shoulder instability in the athlete

Anterior glenohumeral dislocation is common among athletes and may progress to recurrent instability. The pathoanatomy of instability and specific needs of each individual should be considered to prevent unnecessary absence from sport. Traditionally, primary dislocations have been managed with immobilization followed by rehabilitation exercises and a return to sporting activity. However, arthroscopic stabilization and external rotation bracing are increasingly used to prevent recurrent instability. In addition to the typical capsulolabral disruptions seen following a primary dislocation, patients with recurrent instability often have coexistent osseous injury to the humeral head and glenoid. In patients without significant bone loss, open soft‐tissue stabilizations have long been considered the ‘gold standard treatment’ for recurrent instability, but with advances in technology, arthroscopic procedures have gained popularity. However, enthusiasm for arthroscopic repair has not been supported with evidence, and there is currently no consensus for treatment. In patients with greater bone loss, soft‐tissue stabilization alone is insufficient to treat recurrent instability and open repair or bone augmentation should be considered. We explore the recent advances in epidemiology, classification, pathoanatomy and clinical assessment of young athletes with anterior shoulder instability, and compare the relative merits and outcomes of the different forms of treatment.     

Monopolar electrothermal arthroscopy for treatment of shoulder instability in the athlete

Arthroscopic stabilization of the shoulder has gained considerable interest as a treatment alternative for shoulder instability in athletes. Basic science and clinical studies are helping to define the ideal patient population, surgical techniques, and rehabilitation protocols that will enhance our surgical results and maximize patient satisfaction. We describe here our surgical program, basic science foundation, and early clinical results.[/]ab     

Atraumatic shoulder instability Discussion of classification and results after capsular imbrication

This study consists of 27 shoulders in 24 patients whose atraumatic shoulder instability was treated with the imbrication procedure described by Rockwood. Follow-up after 2 years or more showed the following results: 37% excellent, 30% good, 15% fair and 18% poor (Rowe score). No statistically significant differences in the results were observed between the dominant and non-dominant arm, nor in post-operative return to sports activities. Those with voluntary instability had more laxity at follow-up and tended to have poorer results. Those with unidirectional laxity had only good or excellent results and showed no post-operative laxity. We found no other reliable indication of the results of surgery. The difficulties of classifying shoulder instability by such factors as trauma, laxity and direction of instability are discussed. Overall, we consider capsular imbrication to be a good treatment for involuntary atraumatic shoulder instability.     

Dynamic MR imaging and stress testing in glenohumeral instability: comparison with normal shoulders and clinical/surgical findings

Our objectives were to test the hypotheses that: 1) during shoulder motion, glenohumeral alignment differs between asymptomatic shoulders and those with symptomatic instability; 2) during magnetic resonance (MR)-monitored physical exam or stress testing, glenohumeral alignment differs between asymptomatic shoulders and those with instability; and 3) glenohumeral translation during MR stress testing correlates with findings of shoulder instability by clinical exam and exam under anesthesia (EUA). Using an open-configuration 0.5 T MR imaging (MRI) system, we studied symptomatic shoulders in 11 subjects and compared them to their contralateral asymptomatic shoulders. Each shoulder was studied during abduction/adduction and internal/external rotation to determine the humeral head position on the glenoid. An examiner also performed the MR stress test on each shoulder by applying manual force on the humeral head during imaging. All shoulders were assigned an instability grade from the MR stress test, and this grade was correlated with: 1) clinical exam grade assigned during preoperative assessment by an orthopedic surgeon and 2) intraoperative instability grade by EUA immediately preceding arthroscopy. With dynamic abduction and internal/external rotation, the humeral head remained centered on the glenoid in 9 of 11 shoulders, but in two subjects there were dramatic demonstrations of subluxation. With stress testing, a trend toward more joint laxity was demonstrated in symptomatic than in asymptomatic joints (P = 0.11). MR grading of instability correlated directly with clinical grading in six cases and underestimated the degree of instability relative to clinical exam in the other cases. MR instability grading systematically underestimated instability compared with EUA in 7 of the 10 cases that underwent surgical repair. We concluded that dynamic MR evaluation of glenohumeral alignment did not demonstrate abnormalities in symptomatic shoulders in 8 of 10 patients, whereas 2 patients showed dramatic findings of subluxation. Manual stress testing during dynamic MR examination showed a strong correlation with clinical instability grading. Dynamic shoulder MR examination during stress testing could, with further validation, become a useful adjunct to shoulder instability evaluations. J. Magn. Reson. Imaging 2001;13:748-756.     

Postero-medial elbow problems in the adult athlete

The ligamentous, osseous, musculotendinous, and neural structures at the postero-medial side of the elbow are at risk for various injuries in overhead athletes. The combination of valgus and extension overload during overhead activities results in tensile forces along the medial stabilising structures, with compression on the lateral compartment and shear stress posteriorly. The combination of tensile forces medially and shear forces posteriorly can result in ulnar collateral ligament (UCL) tears, flexor-pronator mass injuries, neuritis of the ulnar nerve, posterior impingement, and olecranon stress fractures. Most symptomatic conditions of the overhead athlete can be treated conservatively initially. In cases where conservative treatment is unsuccessful surgical intervention is indicated. Recent advances in arthroscopic surgical techniques and ligamentous reconstruction ensure that the prognosis for return to pre-injury level is good.     

Evaluation and Treatment of Shoulder Instability

Abstract

The shoulder joint has a wide range of motion as a result of a complex interplay of soft tissue and bone structures. It is also the most frequently dislocated joint in the body. Shoulder dislocations are generally classified as traumatic and nontraumatic. There are many specific causes, each of which necessitate individualized treatment modalities. Accurate diagnosis requires a careful history and physical examination. Arthroscopic surgery and advances in imaging have expanded our understanding of anatomy and pathology relevant to shoulder instability and its treatment. Surgery is the treatment of choice for recurrent traumatic instability. Surgery may also be indicated in some first-time traumatic dislocations in young contact athletes, whereas rehabilitation is the initial treatment of choice in older patients with initial instability and in those with nontraumatic dislocations. Results of arthroscopic capsulolabral repair now equal those of open capsulolabral repair and have become the surgical treatment of choice for most patients. However, in cases of recurrent instability and significant bone deficiency of either the glenoid or humeral head, open bone reconstructive procedures are often necessary to ensure successful outcomes.     

关节镜下盂唇修补合并改良Remplissage术治疗伴Hill-Sachs损伤肩关节前方不稳的临床对照研究

目的:研究盂唇修补合并改良Remplissage手术治疗伴肱骨头中小型Hill-Sachs骨性缺损的创伤性复发性肩关节前方不稳的疗效。方法:选取2006年至2010年经影像学检查确诊为伴肱骨头中小型Hill-Sachs损伤的创伤性复发性肩关节前方不稳患者共42例行回顾性随访研究。所有患者均由同一名医生施行关节镜下前方稳定术。根据是否加用改良Remplissage术式分为A、B两组。A组26例,在2006年至2009年行关节镜下单纯盂唇修补术。B组16例,在2009年至2010年行关节镜下盂唇修补术加改良Remplissage术,采用双线锚钉将后方关节囊(非冈下肌腱)填充于肱骨头缺损处。两组患者术后采用相同方法进行康复训练。采用牛津肩关节不稳评分(OSIS)和ROWE评分进行疗效评估、对比术前和术后3个月、6个月、9个月及12个月时肩关节活动度。结果:所有患者均获得随访,A组随访平均(28.0±5.6)个月(20～38个月);术前、术后OSIS评分分别为(37.0±4.2)分(27～43分)和(18.0±3.3)分(12～25分),ROWE评分分别为(20.2±12.2)分(5～40分)和(83.8±7.3)分(70～95分);术后再脱位患者1例,由再次创伤造成,半脱位患者5例。B组随访平均(19.6±3.8)个月(14～27个月);术前、术后OSIS评分分别为(37.9±4.9)分(29～44分)和(13.4±2.1)分(12～20分),ROWE评分分别为(18.4±8.3)分(5～30分)和(95.3±5.3)分(80～100分);术后无再脱位患者。对两组患者术后肩关节活动度分别测量的结果显示,两组患者术后中立位外旋活动度恢复趋势无明显差异。Kaplan-Meier生存分析显示,两组患者术后不稳复发率差异有统计学意义(P=0.043)。结论:关节镜下盂唇修补合并改良Remplissage手术是治疗伴肱骨头中小型Hill-Sachs损伤的创伤性复发性肩关节前方不稳的有效方法,可显著提高肩关节稳定性,并对术后肩关节活动度无明显影响。     

The relationship of superior labralanteroposterior (SLAP) lesions and pseudolaxity to shoulder injuries in the overhead athlete

Understanding the effect of superior labral lesions on the function of the shoulder is essential to successfullytreating the overhead athlete. Recognizing the pseudolaxity owing to superior labral anteroposterior (SLAP) lesions and the pathological "peel-back" sign is critical in evaluating the injured shoulder in general and repairing the SLAP lesion in particular. The mechanical characteristics of suture anchors are more favorable than tacks in resisting the pathological forces responsible for the peel-back mechanism. The higher success rate of arthroscopic suture anchor repair of SLAP lesions in comparison with open capsulolabral reconstruction suggests that SLAP lesions are the usual cause of the "dead arm" syndrome. In our experience, arthroscopic repair of SLAP lesions can return the overhead athlete to their preoperative level of function in the vast majority of cases (87% return to preoperative level for two or more seasons).     

Arthroscopic treatment of posterior and multidirectional instability

The author can answer the three fundamental questions which were posed in the beginning of this chapter as follows: Atraumatic posterior and multidirectional laxity is attributable to capsular ligamentous laxity and can be asymptomatic initially. Over the time, repetitive subluxation of the humeral head exerts excessive rim-loading on the posteroinferior glenoid labrum which can develop into retroversion of the glenoid labrum and eventually leading to labral tears. In this stage, a patient develops shoulder pain during daily and sports activities. Besides increased translation, the diagnosis should be based on the symptoms reproduced by the jerk and Kim tests. The jerk test is a hallmark for predicting the prognosis of nonoperative treatment in posteroinferior instability. Shoulders with a painful jerk test have posteroinferior labral lesion and are unresponsive to nonoperative treatment. In these patients, early surgery may be indicated. Any successful surgical procedure should correct both the capsular laxity and the retroversion of the posteroinferior glenoid labrum. Simple capsular plication or an inferior capsular shift is insufficient for correcting the two major pathologies. Arthroscopic capsulolabroplasty restores capsular tension as well as labral height.     

Arthroscopic management of posterior shoulder instability

The etiology of posterior shoulder instability is multifactorial. Similarly, the surgical treatment of posterior shoulder instability requires more than one management technique. During the past 7 years we have used an anatomic specific approach to posterior shoulder instability, relying on physical examination and diagnostic arthroscopy to determine the correct repair technique. This study reports our results with 61 consecutive patients with refractory posterior shoulder instability requiring surgical correction. In each instance, the specific pathologic entity causing instability was corrected in an anatomic specific approach to the disorder. All patients were re-evaluated 1 to 6 years postoperatively (mean, 34 months). Fifty-five of 61 patients maintained stable shoulders, indicating a 90% success rate with this approach to posterior instability. We would recommend this approach in the management of posterior shoulder instability refractory to rehabilitative treatment.     

Shoulder injuries in tennis players

The mechanism of the overhead action in throwing sports has been studied extensively. This motion is unnatural and highly dynamic, often exceeding the physiological limits of the joint. Owing to overload of various anatomical structures, the shoulder is susceptible to injury. Optimal shoulder function requires good kinetic chain function, optimal stability, and coordination of the scapula in the overhead action. A well balanced action of the rotator cuff muscles and capsular structures is necessary to obtain a stable centre of rotation during the overhead action. This review concerns shoulder injuries, related to the overhead motion in tennis players, which can be explained by the same mechanism as thrower's shoulder.