良性阵发性位置性眩晕的临床特征分析

目的分析总结良性阵发性位置性眩晕(BPPV)的临床诊治经验。方法对131例BPPV患者进行详细的病史调查,回顾性分析131例良性阵发性位置性眩晕患者的临床资料,并追踪随访3个月。结果 131例患者中,男51例,女80例,年龄20～82岁,平均47.7岁,其中单侧后半规管BPPV患者75例,单侧水平半规管BPPV39例,单侧水平嵴顶结石BPPV4例,前半规管BPPV1例,混合型BPPV8例,主观性BPPV3例。其中,原发性BPPV103例,继发性BPPV28例。结论 BPPV患者有其自身的临床特征,通过对其临床特征的认识和把握,选择合适的治疗方法,指导临床实践。     

手法复位治疗老年良性阵发性位置性眩晕患者的效果及安全性

目的 探讨手法复位治疗老年良性阵发性位置性眩晕(BPPV)患者的疗效及安全性.方法 选取2017年11月至2019年2月间收治的120例BPPV患者,根据年龄分成老年组80例和中青年组40例,同时,将伴有基础疾病(高血压、糖尿病、心脏病、脑梗塞等)患者分为A组,不伴基础疾病者分为B组,其中,老年A组58例,老年B组22...     

梅尼埃病合并良性阵发性位置性眩晕患者的临床特征

目的：了解梅尼埃病（Meniere’s disease,MD）患者合并良性阵发性位置性眩晕（benign paroxysmal positional vertigo,BPPV）的发生率及临床特征。方法详细询问并记录116例确诊为 MD患者的体位相关性眩晕病史,对有体位性眩晕者行体位试验及前庭诱发肌源性电位（vestibular evoked myogenic potentials,VEMP）检查;对确诊合并BPPV的患者行手法复位治疗。结果116例 MD患者中4例为病史支持的BPPV,4例确诊合并BPPV,MD合并BPPV发生率为3．45％（4／116）。这8例患者BPPV眩晕均在MD发病之后出现;4例确诊BPPV患者中,2例BPPV患耳与 MD同侧,1例为非 MD 患耳,1例不确定;6例病史支持或确诊合并 BPPV 并可提供VEMP结果的 MD患者中眼性前庭诱发肌源性电位（oVEMP）异常5例,这5例异常者中4例与 MD同侧;MD合并BPPV患者手法复位均治愈。结论本组病例MD合并BPPV发生率为3．45％,详细询问眩晕病史结合体位试验及VEMP检测可有效筛选 MD合并BPPV的患者。     

多管受累的老年良性阵发性位置性眩晕的临床研究

目的探讨累及多个半规管的老年性良性阵发性位置性眩晕(Benign paroxysmal positional vertigo,BPPV)的临床特点和手法复位的治疗效果。方法回顾性分析2012年9月至2015年9月我院诊治的48例多管受累的老年良性阵发性位置性眩晕患者的临床资料并观察手法复位的疗效。结果多管受累的老年性BPPV患者占同期全部老年BPPV患者的21.7%(48/221)。Dix-Hallpike和(或)Roll试验中,两个或两个以上头位可诱发出相同或不同的眼震。对称性双侧半规管同时受累的BPPV患者17例(35.42%),其中16例患者双侧后半规管同时受累,1例患者双侧前半规管同时受累,3例(6.25%)患者诊断为后半规管并同侧前半规管BPPV,25例(52.08%)患者诊断为后半规管并同侧或对侧水平半规管BPPV,1例(2.08%)患者诊断为一侧前半规管合并同侧水平半规管BPPV,2例(4.17%)患者诊断为多个半规管同时受累。根据患者眼震持续时间其中43例(89.58%)诊断为管结石症,5例(10.42%)诊断为嵴顶结石症。全部患者根据受累半规管及耳石类型采用相应手法复位治疗,首次手法复位后有效率为68.75%,多次复位后总有效率为87.50%。结论多管受累的老年BPPV患者临床中比较常见,尤其以后半规管合并水平半规管的管结石症较为多见,手法复位治疗可取得较为满意的疗效,准确判断受累半规管及耳石类型是复位成功的关键。     

儿童青少年良性阵发性位置性眩晕研究进展

良性阵发性位置性眩晕（Benign Paroxysmal Positional Vertigo,BPPV）是一种体位变化诱发反复发作性眩晕为临床特点的常见外周前庭疾病,其发病率随年龄增长而增加,既往认为BPPV在儿童青少年眩晕中少见而未被引起足够重视。然而,随着对儿童青少年BPPV的不断深入认识以及儿童眩晕检测技术的飞速发展,BPPV在儿童青少年眩晕患者中的诊断率呈上升趋势,BPPV逐渐被认为是导致儿童青少年眩晕的一种常见疾病而愈发引起关注。研究表明儿童青少年BPPV在流行病学、临床发病特点、诊断及治疗等方面均呈现出有异于成人BPPV的独特特点,因此本文对儿童青少年BPPV的相关研究进展进行综述。     

青年与老年良性阵发性位置性眩晕患者临床特征及复发危险因素分析

目的比较青年与老年良性阵发性位置性眩晕患者的临床特点。方法研究对象为北京天坛医院神经内科门诊2010年1月~2014年10月期间接受诊疗的良性阵发性位置性眩晕患者。年龄18~45岁者归入青年组(98例),60岁者归入老年组(197例)。比较两组患者的临床特征,应用多因素Logistic回归分析影响其BPPV复发的危险因素。结果青年组男41例,女57例;老年组男73例,女124例,共纳入295例。两组病例性别比差异无统计学意义(P=0.487)。两组病例梅尼埃病、突发性耳聋、偏头痛、头外伤及以往手术史的病例分布差异均无统计学意义(P0.05),但老年组患者中合并高血压病、高脂血症、糖尿病、冠心病、动脉斑块和脑梗死的比例明显均高于青年组(P0.05)。复位治疗1个月后青年组和老年组BPPV复发率分别为19.3%和29.4%,其差异无统计学意义(P0.05),高血压、糖尿病和脑梗死为影响BPPV复发的独立危险因素。结论老年BPPV患者合并高血压病、高脂血症、糖尿病、冠心病、动脉斑块和脑梗死的比例明显均高于年轻患者,高血压、糖尿病和脑梗死为BPPV复发的独立危险因素。     

良性阵发性位置性眩晕诊治分析

目的回顾性分析我科诊治的良性阵发性位置性眩晕(BPPV)患者,分析发病因素及治疗方式,旨在协助临床医生提高精准诊治。方法以眩晕问卷形式收集2010年6月-2016年11月接诊1200例BPPV患者,分别采用Epley复位法、Barbecue复位法、Semont复位法及Brandt-Daroff习服法,药物与心理辅助治疗。分析发病原因、临床特征、治疗方式。结果 1200例BPPV患者中,女性多见,男女比例为1:2.2;60-70岁年龄段发病多;症状仅表现为体位发作性眩晕,无基础病和合并症者多;诊断分型后半规管BPPV1012例(84.33%)、水平半规管BPPV162(13.50%)、上半规管BPPV0例(0.00%)、多个半规管BPPV26例(2.17%);耳石复位是首选治疗方法,首次复位有效率为88%。结论详细收集病史及临床特征,综合评估做出正确诊断,给予个性化治疗是有效治疗BPPV的关键。     

前庭性偏头痛与良性阵发性位置性眩晕的鉴别和共病机制

前庭性偏头痛（vestibular migraine,VM）和良性阵发性位置性眩晕（benign paroxysmal positional vertigo,BPPV）是临床最常见的两类发作性眩晕疾病,临床症状有相似性。不典型的VM与BPPV难以鉴别,可以通过临床表现、前庭功能检查、影像检查及生物标志物等多方面进行区分。相似的诱发因素、内耳血管痉挛、炎症反应、遗传因素等可能会导致VM与BPPV共存。因此,需综合分析VM与BPPV的区别和联系,避免误诊、漏诊,从而为患者提供更加精准的诊疗方案。     

165例良性阵发性位置性眩晕患者管石复位治疗疗效观察

目的评估BPPV患者管石复位治疗短期和长期疗效。方法回顾分析了后半规管和上半规管BPPV患者165例，男53例、女112例，年龄范围18岁至84岁（平均53岁），分为2组，手法复位治疗（治疗组）125例，非手法复位（对照组）40例。结果治疗组中，后半规管BPPV患者81例，上半规管BPPV患者31例，双侧后半规管BPPV13例。65岁以上老年BPPV患者41例。治疗结果分析．治疗组首次治愈83人（66．4％），其中后半规管BPPV60人（74．1％），上半规管BPPV14人（45．2％），双侧后半规管BPPV9人（69．2％），老年BPPV32人（78．0％）。对照组16人（40％）在一周后症状消失。在4个月至41个月的随访中，治疗组和对照组治愈率分别为92％和82．5％。有30人（24％）在随访中复发。有2例病史超过40个月的患者，手法复位治疗无效而进行后半规管阻塞术。结论手法复位治疗BPPV患者是一种非常有效的方法，它能有效的缩短病程，减轻患者痛苦，其方法简单、易行且费用低，可以在临床广泛使用。     

突发性聋伴良性阵发性位置性眩晕患者的预后研究

目的：研究突发性聋（突聋）伴良性阵发性位置性眩晕（BPPV）患者的预后情况。方法：分析24例突聋伴BPPV患者的临床资料,并与同期125例突聋不伴BPPV患者进行比较。结果：突聋伴BPPV和突聋不伴BPPV患者治疗后3个月听力恢复的总有效率分别为41．67％和72．80％,差异有统计学意义（P〈O．05）。结论：突聋伴BPPV患者听力的预后比突聋不伴BPPV患者更差,BPPV是突聋患者听力预后不良的一个影响因素。     

Diagnosis and management of benign paroxysmal positional vertigo

Benign paroxysmal positional vertigo (BPPV) is a common disorder of the vestibular labyrinth which should be suspected in all patients with a history of vertigo during changes of head position. The BPPV appears to be caused by free-floating debris in the posterior semicircular canal. The diagnosis is confirmed by eliciting characteristic symptoms during the Dix-Hallpike test. Although the BPPV usually is a self-limited disorder treatment with a specific bedside maneuver is effective and can shorten the duration of symptoms.     

Recurrent benign paroxysmal positional vertigo in two DFNB16 siblings: A CARE case report

IntroductionDeletions or variants of the STRC gene coding for stereocilin cause congenital bilateral mild-to-moderate sensorineural hearing loss without vestibular disorder: DFNB16. Stereocilin is a protein present in vestibular kinocilia embedded in the otoconial membrane of the utricular macula. Benign paroxysmal positional vertigo (BPPV) is a rare form of vertigo in children. The present study reports recurrent positional vertigo in two DFNB16 siblings.ObservationTwo patients, 10 and 15 years old, presented with recurrent disabling positional vertigo episodes, triggered by turning over in bed, with a falling sensation. The diagnosis of right posterior canal BPPV was confirmed on Dix-Hallpike maneuvers in one of the patients. Variations in the response of ocular vestibular-evoked myogenic potentials were observed. Probable BPPV was diagnosed in the second patient. Their other two siblings did not have hearing loss or vertigo.ConclusionThe absence of stereocilin due to homozygous deletions of the STRC gene in DFNB16 patients can cause vestibular dysfunction, including BPPV.     

Benign paroxysmal positional vertigo after use of noise-canceling headphones

Benign paroxysmal positional vertigo (BPPV) is a common cause of vertigo. We describe a case of a woman presenting acutely with a severe episode of disabling positional vertigo. Although she had no known etiologic risk factors, this attack followed 12 hours of continuously wearing digital noise-canceling headphones. This is the first such reported association between BPPV and the use of this gadget. We also provide a short review of BPPV and speculate on the possible pathogenic mechanisms involved.     

Benign paroxysmal positional vertigo

Benign paroxysmal positional vertigo (BPPV) is characterized by positional vertigo (brief attacks of rotatory vertigo triggered by head position changes in the direction of gravity) and is the most common peripheral cause of vertigo. There are two types of BPPV pathophysiology: canalolithiasis and cupulolithiasis. In canalolithiasis, otoconial debris is detached from the otolithic membrane and floats freely within the endolymph of the canal. In cupulolithiasis, the otoconial debris released from the otolithic membrane settles on the cupula of the semicircular canal and the specific gravity of the cupula is increased. Consensus has been reached regarding three subtypes of BPPV: posterior-canal-type BPPV (canalolithiasis), lateral-canal-type BPPV (canalolithiasis) and lateral-canal-type BPPV (cupulolithiasis). In the interview-based medical examination of BPPV, questions regarding the characteristics of vertigo, triggered movement of vertigo, duration of vertigo and cochlear symptoms during vertigo attacks are important for the diagnosis of BPPV. The Dix–Hallpike test is a positioning nystagmus test used for diagnosis of posterior-canal-type BPPV. The head roll test is a positional nystagmus test used for diagnosis of lateral-canal-type BPPV. When the Dix–Hallpike test is repeated, positional nystagmus and the feeling of vertigo typically become weaker. This phenomenon is called BPPV fatigue. The effect of BPPV fatigue typically disappears within 30 min, at which point the Dix–Hallpike test again induces clear positional nystagmus even though BPPV fatigue had previously caused the positional nystagmus to disappear. For the treatment of BPPV, sequential head movements of patients can cause the otoconial debris in the semicircular canal to move to the utricle. This series of head movements is called the canalith repositioning procedure (CRP). The appropriate type of CRP depends on the semicircular canal in which the otoconial debris is located. The CRP for posterior-canal-type BPPV is called the Epley maneuver, and the CRP for lateral-canal-type BPPV is called the Gufoni maneuver. Including a time interval between each head position in the Epley maneuver reduces the immediate effect of the maneuver. This finding can inform the development of methods for reducing the effort exerted by doctors and the discomfort experienced by patients with posterior-canal-type BPPV during the Epley maneuver.     

Is drug consumption correlated with benign paroxysmal positional vertigo (BPPV) recurrence?

European Archives of Oto-Rhino-Laryngology - Benign paroxysmal positional vertigo (BPPV) is the most common cause of peripheral vertigo and its recurrence is fairly common. Several studies...     

Dizziness handicap and clinical characteristics of posterior and lateral canal BPPV

European Archives of Oto-Rhino-Laryngology - Benign paroxysmal positional vertigo (BPPV) is diagnosed and divided into subtypes based on positioning vertigo and nystagmus. Whether these subtypes...     

Benign positional paroxysmal vertigo in the general ear, nose, and throat clinic

Benign positional paroxysmal vertigo is a common disease which may be caused by abnormal movement of utricular debris in the posterior semicircular canal. It is diagnosed by the Dix-Hallpike positional maneuver eliciting vertigo and nystagmus. Treatment generally consists of physical exercises with the Epley or Semont maneuvers. We review 43 consecutive patients diagnosed as BPPV in the last year and treated with physical therapy (Epley maneuvers). Results were very good, with a cure rate of 88.37% (53.5% after a single maneuver). Physical therapy is an effective treatment for BPPV.     

Inkurabler und atypischer benigner paroxysmaler Lagerungsschwindel

Benign paroxysmal positional vertigo (BPPV) is the most frequent vestibular disorder and a most common cause of dizziness and vertigo. The modern canalolithiasis theory postulates the existence of free-floating dense otolithic particles in the endolymph of the posterior semicircular canal. The symptoms were exactly described by Dix and Hallpike; BPPV is normally a self-limited disease with spontaneous recovery. There is however a small number of patients who do not respond to any treatment or who present with otolithic vertigo which does not fit all Dix-Hallpike criteria (atypical BPPV). While BPPV in its typical appearance cannot be diagnosed with radiologic imaging, the authors succeeded in identifying structural changes in the vestibular organs of patients suffering from intractable or atypical BPPV using three-dimensional magnetic resonance imaging.     

Bilateral benign paroxysmal positional vertigo following a tooth implantation

Benign paroxysmal positional vertigo (BPPV) is a common cause of vertigo and may occur following recent head trauma. Bilateral involvement in BPPV is considered rare and has received little attention in the medical literature. We describe an unusual case of bilateral BPPV in a middle-aged woman that presented during a dental implantation, performed with the use of osteotomes. We discuss the diagnostic and therapeutic challenge of this entity.     

Intractable and atypical benign paroxysmal vertigo. Pathological results of high-resolution three-dimensional MR-tomography of the vestibular organ

Benign paroxysmal positional vertigo (BPPV) is the most frequent vestibular disorder and a most common cause of dizziness and vertigo. The modern canalolithiasis theory postulates the existence of free-floating dense otolithic particles in the endolymph of the posterior semicircular canal. The symptoms were exactly described by Dix and Hallpike; BPPV is normally a self-limited disease with spontaneous recovery. There is however a small number of patients who do not respond to any treatment or who present with otolithic vertigo which does not fit all Dix-Hallpike criteria (atypical BPPV). While BPPV in its typical appearance cannot be diagnosed with radiologic imaging, the authors succeeded in identifying structural changes in the vestibular organs of patients suffering from intractable or atypical BPPV using three-dimensional magnetic resonance imaging.