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目的 研究急性脑梗塞患者血清白细胞介素6(IL-6) 的变化,进而探讨其临床意义。方法 采用放免法检测40 例急性脑梗塞患者血清IL-6 水平,并与20 例健康人作对照。结果 脑梗塞患者急性期血清IL- 6 水平明显高于恢复期(P<0-01) 。恢复期血清IL-6 稍高于正常水平,与正常对照组比较无显著性差异( P>0-05) 。急性期血清IL- 6 升高的程度与梗塞灶大小密切相关。结论 脑梗塞急性期血清IL-6 升高,恢复期已降至基本正常。脑缺血、缺氧及脑组织损伤引起的急性应激反应可能是脑梗塞急性期血清IL-6 升高的重要机制。IL-6 升高对缺血神经元可能具有保护作用。 相似文献
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肝硬化合并感染及慢性肝病患者血清白介素-8的变化 总被引:1,自引:0,他引:1
目的观察慢性病毒性肝炎、肝硬化及肝硬化合并自发性细菌性腹膜炎患者血清白介素-8(IL-8)的变化,探讨其与肝细胞损伤及感染的关系。方法采用EIISA法检测25例慢性病毒性肝炎,30例肝硬化及15例肝硬化合并自发性细菌性腹膜炎患者血清IL-8的变化。常规方法检测慢性肝病患者谷丙转氨酶(ALT)的变化。结果慢性病毒性肝炎及肝硬化患者血清IL-8均明显高于正常对照组(P<0.001和P<0.01)。肝硬化合并自发性细菌性腹膜炎患者IL-8分别高于慢性病毒性肝炎(P<0.05)和肝硬化无自发性腹膜炎者(P<0.01)。慢性肝病患者血清IL-8水平与ALT呈正相关(r=0.61,P<0.05)。结论IL-8可能参与了慢性肝病时肝细胞的免疫损伤,但IL-8与感染的关系更为密切。 相似文献
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类风湿性关节炎患者血清白细胞介素的检测 总被引:1,自引:0,他引:1
为了解类风湿性关节炎(RA)患者的免疫损伤机理,应用酶联免疫吸附法(ELISA)检测38例RA患者血清可溶性白细胞介素2受体(sIL-2R)和白细胞介素8(IL-8)含量,并检验其相关性。结果:(1)RA患者血清sIL-2R和IL-8水平均比正常对照组显著升高(P<0.01);血清sIL-2R浓度在类风湿因子(RF)阳性与阴性组中差异有显著性(P<0.01);(2)RF阳性患者血清sIL-2R与IL-8浓度呈正相关(r=0.74,P<0.05)。提示RA患者T细胞异常活化以及免疫调节功能紊乱。 相似文献
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血浆P—选择素和IL—8的测定在急性吸收窘迫综合征中的临床价值 总被引:1,自引:0,他引:1
目的:探讨血浆P-选择素和IL-8的测定在急性呼吸窘迫综合征(ARDS)中的临床价值。方法:分别采用放免法和酶联免疫吸附法检测25例ARDS患者血浆P-选择素和IL-8,同时行APACHEⅡ评分,结果:ARDS患者血浆P-选择素和IL-8明显高于正常对照组(P〈0.01),且随着APACHEⅡ评分增高,P-选择素也明显增高。两者呈正相关(P〈0.01),死亡患者血浆P-选择素,IL-8及APACH 相似文献
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系统性红斑狼疮及狼疮性肾炎患者血清SIL—2R,IL—6,IL—8怠 总被引:5,自引:0,他引:5
本研究应用ELISA双抗体夹心法对SLE患者血清SIL-2R、IL-6和IL-8水平进行了测定。结果表明:SLE患者血清SIL-2R、IL-6和IL-8水平显著高于健康人(P〈0.001),且血清IL-8与IL-6浓度呈显著性正相关(n=51,r=0.372,P〈0.001),提示SLE存在免疫细胞活化,异常增高的细胞因子引起免疫紊乱和促发免疫损伤,血清细胞因子的变化有助于监测狼疮活动;LN患者血 相似文献
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哮喘患者血T淋巴细胞和嗜酸细胞活性指标的检测及意义 总被引:2,自引:0,他引:2
目的 为探讨哮喘患者体内T淋巴细胞和Eo 的激活程度。方法 选择血清白细胞介素(IL)- 5、可溶性白细胞介素2 受体(SIL-2R) 、嗜酸细胞阳离子蛋白(ECP)作为其活性指标进行检测。结果 30 例哮喘患者中22 例血清IL-5 浓度在35 ng/L以上,正常对照组均在35 ng/L以下( P<0-05);SIL- 2R浓度哮喘组为268-5 ±73-7 U/ml 较正常人138-5±55-3 U/ml 为高(P<0-01);而ECP浓度哮喘组14-4±11-8 μg/L也高于正常人5-6 ±4-8μg/L( P< 0-01) ,且血清IL- 5、SIL- 2R 浓度与其血清ECP浓度均呈显著正相关(r= 0-53 、0-46,P< 0-05) 而与FEV1 % 呈显著负相关(r= -0-52、- 0-42,P<0-05)。结论 哮喘患者体内T淋巴细胞和Eo 激活;血清IL-5、SIL-2R和ECP浓度可作为反映哮喘气道炎症消长的指标。 相似文献
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探讨慢性阻塞性肺疾病(COPD)患者免疫功能的机制。采用双抗体夹心酶联免疫吸附法(ELISA)检测22例COPD患者及20例健康对照血清可溶白细胞介素-2受体(SIL-2R)。结果表明:COPD患者SIL-2R显著高于正常对照,且急性发作期又高于缓解期,差异具显著性(p<0.001);缓解期COPD患者血清SIL-2R高于健康对照,差异无显著性(0.1<P<0.2)。结果提示:血清SIL-2R增高可能是COPD患者免疫功能减退与容易发生肺部感染的原因之一。 相似文献
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目的:观察自由光量子疗法(UBIO)治疗脑梗死时血清白细胞介素-12(IL-12)的变化,探讨UBIO治疗脑梗死的作用机制。方法:UBIO组36例急性梗死患接受药物治疗和UBIO治疗,对照组32例急性脑梗死患单用药物治疗,分别测定治疗前、疗程后1d的血清IL-12含量。结果:UBIL组治疗后血清IL-12水平明显下降,两组比较差异有显性(P〈0.05),结论:UBIO疗法能显降低脑梗死患血清IL-12水平,具有抗炎作用,是其治疗脑梗死的作用机理之一。 相似文献
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目的:探讨白细胞抗原CD4,CD8与白细胞介素(IL-2,IL-6,IL-8)在血液透析中的变化及其意义,方法:动态观察急性血液透析,维持性血透析和氮质血症患者透析前后CD4/CD8,IL-2,IL-6和IL-8水平。结果CD4/CD8和IL-2的急性透析者升高(P均〈0.01),IL-2,IL-6和IL-4随代谢产物排泄而下降(P均〈0.01),结论:CD4/CD8参与急性透析和维持性透析,IL 相似文献
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目的:了解慢性肾功能不全患者不同病期血浆白细胞介素8(IL8)的含量及其临床意义。方法:用双抗夹心酶联免疫吸附法分别测定慢性肾功能不全患者不同病期血浆IL8含量,并与正常对照组比较。结果:慢性肾功能不全代偿期及氮质血症期患者血浆IL8略低于正常对照组,但差异无显著性(P均>0.05)。肾功能衰竭期患者血浆IL8含量显著低于正常对照组(P<0.01)。在慢性肾功能不全病程中,血浆IL8含量与血肌酐、尿素氮呈显著负相关(r=-0.58,P<0.01;r=-0.51,P<0.01)。结论:慢性肾功能不全患者肾功能衰竭期机体全身免疫功能低下,急性炎症反应减弱。 相似文献
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《Veterinary Nursing Journal》2013,28(2):41-68
While some patients with heart disease have only mild forms and may never develop clinical signs as a result, many animals develop forms of heart disease which are progressive and cause signs of heart failure. Dogs and cats with heart failure generally have a poor prognosis. However, rational treatment can help to relieve the clinical signs and improve the chances of survival. 相似文献
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A girl with severe mental retardation and conspicuous phenotype features is described. The chromosomal aberration consists of a partial trisomy 8q of the region 8q22----qter. Minor deletion of the terminal part of the region 8p23 must be presumed, resulting in partial monosomy of this region. Inversion of chromosome 8 was found in the father and his mother. 相似文献
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Ashley L. Fowlkes Cedric Brown Minal M. Amin John D. Roback Robert Downing Esau Nzaro Jonathan Mermin Wolfgang Hladik Sheila C. Dollard 《Transfusion》2009,49(10):2208-2213
BACKGROUND: Human herpesvirus 8 (HHV-8) is endemic in Uganda where seroprevalence is approximately 40%. In a previous study, Ugandan patients receiving blood transfusions had multiple serum specimens collected for 6 months after transfusion to monitor for HHV-8 infection. It was observed that several HHV-8–seronegative patients were unexpectedly HHV-8 seropositive after blood transfusion.
STUDY DESIGN AND METHODS: This study measured HHV-8 antibody in serially collected serum specimens from 542 patients who received transfusions and evaluated the risk of HHV-8 infection as a function of HHV-8 antibody levels in the donors.
RESULTS: HHV-8 antibody was observed in 52% of patients transfused with HHV-8–seropositive blood in amounts that corresponded with their donor's antibody titer and waned within 40 days. Higher levels of passive HHV-8 antibody in patients who received transfusions appeared to be associated with a lower risk of HHV-8 infection.
CONCLUSION: The source of transient antibody in patients who received transfusions was determined to be the transfused blood. Donors with higher HHV-8 antibody titers may have been less likely to have infectious virus in the blood. 相似文献
STUDY DESIGN AND METHODS: This study measured HHV-8 antibody in serially collected serum specimens from 542 patients who received transfusions and evaluated the risk of HHV-8 infection as a function of HHV-8 antibody levels in the donors.
RESULTS: HHV-8 antibody was observed in 52% of patients transfused with HHV-8–seropositive blood in amounts that corresponded with their donor's antibody titer and waned within 40 days. Higher levels of passive HHV-8 antibody in patients who received transfusions appeared to be associated with a lower risk of HHV-8 infection.
CONCLUSION: The source of transient antibody in patients who received transfusions was determined to be the transfused blood. Donors with higher HHV-8 antibody titers may have been less likely to have infectious virus in the blood. 相似文献