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High glucose concentration abrogates sevoflurane post‐conditioning cardioprotection by advancing mitochondrial fission but dynamin‐related protein 1 inhibitor restores these effects
Authors:J. Yu  Y. Maimaitili  P. Xie  J. J. Wu  J. Wang  Y. N. Yang  H. P. Ma  H. Zheng
Affiliation:1. Department of Anaesthesiology, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China;2. Department of Cardiology, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China
Abstract:
Keywords:high glucose  hypoxia/reoxygenation injury  mitochondrial division inhibitor  mitochondrial fusion and fission  sevoflurane post‐conditioning
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