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川崎病发病机制研究新进展
引用本文:何跃娥,项如莲,张园海,陈其.川崎病发病机制研究新进展[J].医学综述,2007,13(14):1103-1105.
作者姓名:何跃娥  项如莲  张园海  陈其
作者单位:温州医学院附属儿童医院小儿心内科,浙江,温州,325000
摘    要:川崎病的特征为广泛的中小血管炎症,以心血管系统的损害最为严重,可形成冠状动脉扩张和冠状动脉瘤。但目前川崎病及其冠状动脉损害的发病机制仍未明确,多种因子如一氧化氮、血管内皮生长因子、基质金属蛋白酶及遗传因素等可能参与其发病,引起广泛关注。

关 键 词:川崎病  冠状动脉损害  病因
文章编号:1006-2084(2007)14-0103-03
收稿时间:2006-10-28
修稿时间:2006-10-282007-06-30

Research Study about Pathogenesis of Kawasaki Disease
HE Yue'e,XIANG Ru-lian,ZHANG Yuan-hai,CHEN Qi.Research Study about Pathogenesis of Kawasaki Disease[J].Medical Recapitulate,2007,13(14):1103-1105.
Authors:HE Yue'e  XIANG Ru-lian  ZHANG Yuan-hai  CHEN Qi
Abstract:Kawasaki disease(KD) is featured by wide polyangitis,especially that of cardiovascular system is most severe,which can form coronary arteriectasis and coronary aneurysm.But the mechanism of kewasaki disease and coronary artery injury are still unclear.Many factors including NO,VEGF,MMPs and genetic factor perhaps participate in their pathogenesis,which is attracting more attention.
Keywords:Kawasaki disease  Coronary artery lesion  Etiology
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