| 脂联素研究进展 |
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| 引用本文： | 自加吉,邓刚.脂联素研究进展[J].四川解剖学杂志,2010,18(2):27-31. |
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| 作者姓名： | 自加吉 邓刚 |
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| 作者单位： | 大理学院基础医学院病理学教研室,大理,671000 |
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| 摘 要： | 肥胖及与肥胖相关联的胰岛素抵抗最终可导致代谢综合征。脂肪组织参与能量代谢的调节，并作为一个重要的内分泌器官分泌大量的脂肪因子。脂联素是其中的一种胰岛素增敏因子。研究发现，肥胖相关的脂联素下调是引起肥胖、导致胰岛素抵抗和糖尿病的重要机制。
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| 关 键 词： | 脂联素 脂联素受体 胰岛素抵抗 糖尿病 |
Review of Adiponectin |
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| Zi Jiaji,Deng Gang.Review of Adiponectin[J].Sichuan Journal of Anatomy,2010,18(2):27-31. |
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| Authors: | Zi Jiaji Deng Gang |
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| Institution: | Zi Jiaji Deng Gang (School of Basic Medical Science, Dali University, Dali 671000, China) |
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| Abstract: | Metabolic syndrome is thought to result form obesity and obesity-linked insulin resistance. Adipose tissue participate in the regulation of energy homeostasis as an important endocrine organ that secretes a number of biologically active ＂adipokines＂. Adiponectin serves as an insulin-sensitizing adipokine. In fact, obsety-linked down-regulation of adiponeetin is a significant mechanism whereby obesity could cause insulin resistance and diabetes. |
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| Keywords: | Adiponectin Adiponectin reeeptor Insulin resistanee Diabetes |
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