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Escitalopram alters gene expression and HPA axis reactivity in rats following chronic overexpression of corticotropin-releasing factor from the central amygdala
Authors:Elizabeth I Flandreau  Chase H Bourke  Kerry J Ressler  Wylie W Vale  Charles B Nemeroff  Michael J Owens
Institution:1. Psychiatry Department, University of California San Diego, 9500 Gilman Drive MC 0804, La Jolla, CA 92093-0804, United States;2. Laboratory of Neuropsychopharmacology, Department of Psychiatry and Behavioral Sciences, Emory University, Woodruff Memorial Research Building, Suite 4000, 101 Woodruff Circle, Atlanta, GA 30322, United States;3. Department of Psychiatry and Behavioral Sciences, Yerkes Research Center, Emory University, 954 Gatewood Dr, Atlanta, GA 30329, United States;4. Peptide Biology Laboratory, Salk Institute for Biological Studies, 10010 North Torrey Pines Rd., La Jolla, CA 92037, United States;5. Department of Psychiatry & Behavioral Sciences, University of Miami Miller School of Medicine, Clinical Research Building, 1120 NW 14th Street, Room 1455 (D-21), Miami, FL 33136, United States
Abstract:
Keywords:Corticotropin-releasing factor  Amygdala  Escitalopram  Anxiety  Hypothalamic-pituitary-adrenal (HPA) axis
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