Nrf2 promotes the development of fibrosis and tumorigenesis in mice with defective hepatic autophagy期刊界 All Journals 搜尽天下杂志传播学术成果专业期刊搜索期刊信息化学术搜索

Nrf2 promotes the development of fibrosis and tumorigenesis in mice with defective hepatic autophagy

Authors:	Hong-Min Ni Benjamin L. Woolbright Jessica Williams Bryan Copple Wei Cui James P. Luyendyk Hartmut Jaeschke Wen-Xing Ding

Affiliation:	¹ Department of Pharmacology, Toxicology and Therapeutics, The University of Kansas Medical Center, Kansas City, KS 66160, United States;² Department of Pathology & Laboratory Medicine, The University of Kansas Medical Center, Kansas City, KS 66160, United States;³ Department of Pathobiology and Diagnostic Investigation, Michigan State University, East Lansing, MI 48824, United States

Abstract:

Keywords:	Alb, albumin ALT, alanine aminotransferase α-SMA, α-smooth muscle actin CTGF, connective tissue growth factor CK 19, cytokeratin 19 ECM, extracellular matrix EM, electron microscopy FLIP, FLICE-like protein (FLIP) protein GCLC, glutamate-cysteine ligase catalytic subunit GCLM, glutamate-cysteine ligase modifier subunit HCC, hepatocellular carcinoma HSC, hepatic stellate cells ICAM-1, Intercellular adhesion molecule 1 IL-6, interleukin 6 KC, C-X-C motif ligand 1 Keap1, Kelch-like ECH-associated protein 1 KO, knockout LC3, microtubule light chain 3 LD, lipid droplets Ly6B, lymphocyte antigen B superfamily MIP1α, macrophage inflammatory protein 1α NQO1, NAD(P)H quinone oxidoreductase Nrf2, nuclear factor (erythroid-derived 2)-like 2 PCNA, proliferating cell nuclear antigen ROS, reactive oxygen species TGF-β1, transforming growth factor beta 1 TNF-α, tumor necrosis factor alpha WT, wild type
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