(1) Department of Internal Medicine, University of Michigan Health System, Ann Arbor, Michigan, USA;(2) Department of Epidemiology, University of Michigan School of Public Health, Ann Arbor, Michigan, USA
Abstract:
Background
A sizeable number of HIV-infected patients receiving HAART do not maintain prolonged virologic suppression. We evaluated long-term HIV viral load (VL) responses to HAART as a risk factor for AIDS events (AE) that is independent of CD4 responses.