Neutrophil chloramines: missing links between innate and acquired immunity |
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| Affiliation: | 1. Unidad de Gestión Clínica de Cirugía Pediátrica, Hospital Universitario Reina Sofía, Córdoba, España;2. Facultad de Medicina Universidad de Córdoba, Córdoba España;3. Unidad de Gestión Clínica de Cirugía Pediátrica, Hospital Universitario Reina Sofía, Córdoba, España;1. Clinical Department of Internal Disease, Dermatology, and Allergology in Zabrze, Medical University of Silesia, Katowice, Poland;2. Outpatient Clinic of Allergy, Swietochlowice, Poland;3. Clinical Department of Laryngology in Zabrze, Medical University of Silesia, Katowice, Poland;1. Division of Allergy and Immunology, Department of Internal Medicine, University of South Florida Morsani College of Medicine and James A. Haley Veterans'' Affairs Hospital, Tampa, Fla;2. Section of Allergy and Clinical Immunology, Imperial College London - National Heart and Lung Institute, Royal Brompton Hospital, London, United Kingdom;1. Department of Anaesthesiology and Intensive Care Medicine, University Hospital Schleswig-Holstein, Campus Kiel, Germany;2. Department of Anaesthesiology, Intensive Care Medicine and Pain Therapy, University Hospital Frankfurt, Frankfurt am Main, Germany;3. Department of Anaesthesiology, University Hospital RTWH Aachen, Germany;1. Department of Urology, Technical University of Munich, Munich, Germany;2. Clinic for Urology, Pediatric Urology and Andrology, Justus Liebig University, Giessen, Germany |
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| Abstract: | ![]()
Neutrophils are the major cellular component of the acute inflammatory response. By contrast, macrophages are the major cellular component in most chronic immunological responses, and act as key regulators of the specific acquired response. Here, Janusz Marcinkiewicz examines recent data indicating that chloramines, the neutrophil-specific products of the myeloperoxidase-hydrogen-peroxide-halide system, may provide a bridge between the afferent branches of the innate and acquired immune response. |
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