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Schizencephaly is a neuronal migration malformation characterized by a cleft traversing the cerebral hemisphere. The cleft is lined by heterotopic gray matter, and the lips of the cleft may be fused or separated. Often associated with anomalies of the septum pellucidum or corpus callosum, and polymicrogyria, schizencephaly has characteristic CT findings. Of 17 patients with this lesion detected by CT 2 had bilateral schizencephaly.
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A two-week bile duct ligation (BDL) in Sprague-Dawley (SD) rats raised the serum billirubin level and decreased the mean arterial blood pressure and renal cortical ATP contents compared with those in sham-operated (SO) rats (3.6 +/- 1.15 mg% vs 0.54 +/- 0.36, P less than 0.001; 69 +/- 24 mmHg vs 86 +/- 21, P less than 0.05; 3.72 +/- 0.86 x 10(-10) mol/mg tissue vs 7.27 +/- 0.18, P less than 0.05). No difference could be found in the medullary ATP contents (8.42 +/- 2.20 vs 8.70 +/- 2.80, P = NS). In SO rats, injection of endotoxin (0.7 mg/kg BW) and E. coli (3.1 x 10(5) bacteria/100 g BW) reduced cortical ATP content to 1.86 +/- 0.97 and 1.30 +/- 0.47 (P less than 0.001), and medullar ATP to 1.33 +/- 0.31 and 2.12 +/- 0.46 (P less than 0.001) respectively. In BDL rats, the same treatment led to further decrease in cortical ATP to 1.25 +/- 0.40 and 0.62 +/- 0.20, medullary ATP to 0.97 +/- 0.41 and 1.64 +/- 0.83 (P less than 0.001). Basal Na-K ATPase activity in BDL is the same compared with that in SO both in the cortex (2.85 +/- 2.2 mumol/mgpr/h vs 2.19 +/- 0.75; P = NS) and medulla (2.79 +/- 1.83 vs 3.05 +/- 1.38; P = NS).(ABSTRACT TRUNCATED AT 250 WORDS)
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3.
In this paper two cases of homozygous HbG Coushatta compounded with β+ thalassemia are reported. They were born in Zhejiang, and are sisters of Han nationality. The diagnoses were based on family pedigree. The abnormal Hb was shown to be passed peptide chain dissociation by electro- phoresis and structural analysis. Case Ii (the father) had β十 thalassemia, I2 (the mother) heterozygous HbG Coushatta and siblings II2 and Il5 had homozy gous HbG Coushatta with β+thalassemia Analysis by constant chromatosome of dominant genetic re- gularity could not account for these two cases. No abnormal band was found in Case I1. The authors were not able to determine whether this was due to the abnormality being too small to measure easily by present methods or sudden gene change. Both cases, although having anemia and splenomegaly were not severe. They worked and lived normally.  相似文献
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In order to further understand the pattern of activities of urethral tumor reappearance after cystectomy for cancer of with the condition and 10 the bladder, 900 patients cases undergoing cystoure threctomy were evaluated. Of 184 male patients having total cystectomy, 18(10%) subsequently developed urethral tumors, whereas of 570 patients undergoing partial cystectomy only 6 (1%) developed this kind of tumor. Of additional 10 patients on whom en bloc cystourethrcctomy was performed for concomitant presence of carcinoma in situ and suspected urethral abnormality, 2 were carcinoma in situ, 1 papdlary transitional cell carcinoma, and 3 marked epithelial atypia of the urethra. Histo- pathologically, of the 24 bladder specimens, 15 were G2, 9 G3, 14 T1-T2, and 10 T3-T4 according to their grading and staging. Of the 24 urethras removed, 8 had cancer at the urethral stub, 5 had multiple lesions over the entire urethra, and 11 with lesions at the anterior urethra, of which most had no tumor cells micros copically at the excised margin. These indicate that tumor recurrence in the retained urethra following cystectomy could be a reflection of tumor multicen- tricity, particularly correlative to the histology and locality of the primary vesical tumor. En bloc cystourethrectomy is only justified when the extent of the bladder lesion and its urethral involvement are evident.  相似文献
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