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1.
外来务工人员低钾性周期性麻痹52例临床分析   总被引:2,自引:0,他引:2  
目的探讨外来务工人员低钾型周期性麻痹(HoPP)的特点。方法回顾性调查结合追踪随访对东莞市桥头医院2004-01-2005-12收治的52例外来务工人员HoPP的临床资料进行分析。结果52例低钾性周期性麻痹患者全部为外来务工人员,经治疗均痊愈。发病诱因主要为剧烈运动或劳累、情绪激动、焦虑。男性发病明显多于女性,男∶女为9∶1。患者以青年为主,占92.3%。伴有甲亢者占23%。结论外来务工人员发病诱因比较明显,劳累、心理状态、社会因素与这一特殊群体的HoPP发病有一定关系。  相似文献
2.
Summary Early ultrastructural changes are studied in rat gastrochemius muscle during experimental hypokalemia. Vacuolization of muscle fibers is shown to originate in the terminal cisterns of the triads. Important degenerative changes are present in peripheral axons and neuromuscular junctions.  相似文献
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Summary A patient is described, who had muscle necrosis occurring with hypokalemia and a factitious mineralocorticoid excess syndrome. The syndrome was due to the prolonged use of a nasal spray containing 9-alpha-fluoroprednisolone acetate, a steroid with potent mineralocorticoid activity. It is emphasized that intranasal administration of steroid compounds must be considered in the differential diagnosis of hypokalemia and related muscle disorders.
Zusammenfassung Es wird ein Patient beschrieben, der hypokaliaemische Muskelnekrosen, ausgelöst durch einen exogenen Überschuß von Mineralcorticoiden, aufweist. Die Ursache war der langdauernde Gebrauch eines 9-alpha-fluorprednisolon-Nasensprays, ein Steroid mit einer starken mineralcorticoiden Aktivität. Intranasal verabreichte Steroide müssen in der Differentialdiagnose von Hypokaliaemie und dadurch hervorgerufenen Muskelveränderungen in Betracht gezogen werden.
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The suppression of active Na+-K+ transport in rat skeletal muscle during hypokalemia was counteracted by bilateral electrolytic lesion of the ventromedial hypothalamic nucleus. This reversal effect was unaffected even after pancreatectomy or adrenalectomy. The anomalous electrolyte content in hypokalemic rat muscles was aggravated by lesion of the dorsomedial hypothalamic nucleus and of the anterior hypothalamus. The results indicate that the hypothalamus is involved in the regulation of the Na+-K+ transport system in skeletal muscle during hypokalemia.  相似文献
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Summary Since the serum potassium level is under beta2-adrenergic influence, we studied serum potassium values on admission in psychiatric patients. Data are reported on 683 patients from nine major diagnostic groups. Among these nine groups significant differences were found concerning the mean serum potassium level and incidence of hypokalemia. Significant differences existed between the alcohol withdrawal and attempted suicide groups compared to the dysthymic, bipolar manic, schizophrenic and nonschizophrenic psychosis groups. Like alcohol withdrawal, attempted suicide is assumed to be a hyper-adrenergic state. Although the relative contribution of factors like nutritional state, aldosterone, insulin, and beta2-receptor density or sensitivity is unclear, the catecholamine-potassium relationship deserves further study.An abstract of this study will be published as a letter to the editor in the J Clin Psychiatr  相似文献
7.
回顾性分析住院精神疾病患者夜间突发猝倒共20例。其中有低血钾7例,药物性巴金森综合征、癫痫小发作、意识朦胧发作和短暂性脑缺血发作各2例,体位性低血压、糖尿病低血糖、骨质疏松症、酒精中毒性共济失调及原因不明者各1例。误诊7例,其中6例后被确诊为低血钾。精神疾病患者夜间跌倒原因众多,应充分考虑到低血钾的可能,可考虑对住院病人定期检查血钾。  相似文献
8.
Kunze K 《Journal of neurology》2002,249(9):1150-1159
Summary. Metabolic encephalopathies, usually multifacto-rial in origin, may be important complications of many diseases of patients treated in a critical care unit. In many cases these complications arise from more than one cause. Neurological signs of metabolic encephalopathies, ancillary tests and differential diagnosis, etiology and pathophysiology are discussed. In this context major single causes for metabolic encephalopathies are referred to. Metabolic encephalopathies as diseases per se (e. g. Wernicke's encephalopathy) and encephalopathies as consequences of deteriorating known diseases (e. g. renal or hepatic diseases) and encephalopathies as complications in patients treated with other diseases in the ICU have to be differentiated. Encephalopathies are known to be the most common complication of a large group of diseases treated in the ICU; on the other hand, manifestation of metabolic encephalopathy can be taken as a warning of deterioration or beginning organ dysfunction. So it would be misleading so far to reduce the clinical concept of metabolic encephalopathies in ICH to septic encephalopathy only. Received: 29 April 2002, Accepted: 23 May 2002  相似文献
9.
Isolated weakness of the extensor muscles of the neck is a relatively rare condition, known as “dropped head syndrome” (DHS). This paper reports the case of a patient with DHS whose symptoms resolved rapidly on supplementation with potassium and discontinuation of licorice consumption. To the best of our knowledge, there are no reports in the literature of DHS due to licorice-induced hypokalemia.  相似文献
10.
Introduction: We evaluated the effect of induced hypothermia on biochemical parameters in patients with severe traumatic brain injury. Methods: We obtained hemoglobin, hematocrit, white blood count, lymphocyte count, platelet count, and serum concentrations of sodium, potassium, glucose, albumin, and C-reactive protein, and prothrombin time, hepaplastin test, activated partial thromboplastin time, antithrombin-III, α2PI, and nitrogen excretion on the day of admission, and on days 1, 3, 5, 7, 14, and 21 after the injury in 31 patients with severe head injury who were treated with hypothermia of 33°ranging from 48 to 72 hours. We selected 33 normothermic patients as a control group; these patients were selected from patients who had been treated before hypothermia was used as a treatment modality, by the same criteria for hypothermia therapy. We compared the biochemical markers and rectal temperature and intracranial pressure in the hypothermia group with those in the normothermia group. Outcome was assessed using the Glasgow Outcome Scale at 6 months after injury. Results: The demographic characteristics, severity, and outcome were similar in the hypothermia and normothermia group. Intracranial pressure was significantly decreased by hypothermia. Serum potassium concentration decreased significantly during hypothermia. White blood cell counts and C-reactive protein levels were higher after rewarming in the hypothermia group, and these were also higher in the patients with infectious complications, although the incidence of infectious complications did not differ between the hypothermia and normothermia groups. There were no statistically significant prolongations of activated partial thromboplastin time and no decline in prothrombin time with hypothermia. Platelet count, antithrombin-III, and α2PI did not differ significantly between the two groups. Conclusion: Hypothermia of 33° for 48–72 hours does not appear to increase the risk for coagulopathy and infections, although hypothermic patients exhibited significant increments in inflammatory markers such as C-reactive protein and white blood counts after rewarming.  相似文献
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