首页 | 本学科首页   官方微博 | 高级检索  
文章检索
  按 检索   检索词:      
出版年份:   被引次数:   他引次数: 提示:输入*表示无穷大
  收费全文   3篇
  免费   0篇
  国内免费   2篇
临床医学   2篇
内科学   1篇
综合类   1篇
药学   1篇
  2020年   1篇
  2008年   1篇
  2006年   1篇
  2005年   1篇
  2003年   1篇
排序方式: 共有5条查询结果,搜索用时 15 毫秒
1
1.
目的研究超重和肥胖者减重前后炎症指标的变化。方法288例40~60岁超重和肥胖者入选为期3个月减重干预,其中超重组96例,肥胖组94例;同期体质量指数(BMI)正常者98例设为对照组。观察并分析各组减重前后炎症指标血清肿瘤坏死因子-α(TNF-α)、白细胞介素-6(IL-6)及C-反应蛋白(CRP)水平变化。结果减重前血清CRP、TNF-α、IL-6水平,超重组、肥胖组较对照组明显升高(P〈0.01),肥胖组较超重组也明显升高(P〈0.05);各组间BMI比较差异有显著性(P〈0.01)。经过3个月的干预减重后,超重组和肥胖组除IL-6水平下降差异无统计学意义外(P〉0.05),其它各参数均有明显下降(P〈0.05)。超重组和肥胖组血清TNF-α和CRP水平明显降低,差异有显著性(P〈0.05)。结论中年人群中CRP水平与超重及肥胖相关联,减重可明显降低血清TNF-α和CRP水平。  相似文献   
2.
王宝西  王茂贵 《医学争鸣》2003,24(5):472-474
目的 :探讨单纯性肥胖症与营养不良患儿胃动力学状态 .方法 :以牛奶作为胃液体试验餐 ,采用实时超声测定液体胃排空 .结果 :单纯性肥胖症、营养不良患儿及健康儿液体试验餐 12 0min后胃内残留率分别为 (7.4± 4 .0 ) % ,(2 3.4±5 .6 ) %和 (14 .2± 10 .5 ) % (单纯性肥胖症与营养不良患儿相比 ,P <0 .0 5 ) ;其 10 0 %胃排空所需时间分别为 (14 7± 2 8) ,(176± 18)和 (16 9± 4 7)min(营养不良患儿较肥胖患儿和正常对照胃排空时间明显延长P <0 .0 5 ) ;试餐后 6 0min各组胃体蠕动频率和胃窦收缩频率 (次·min-1)分别为 4 .2± 0 .6和 4 .6± 0 .6 ;2 .5± 0 .3和 2 .7± 0 .4及 3.8± 0 .6和 3.7± 0 .9.单纯性肥胖症空腹胃动素水平 (4 4 3± 83)ng·L-1明显高于营养不良患儿 (2 19± 6 6 )ng·L-1,P <0 .0 5 .结论 :单纯性肥胖儿胃液体排空明显加快 ;营养不良患儿胃液体排空延迟  相似文献   
3.
Dexfenfluramine (DEX) and sibutramine (SIB) are effective antiobesity agents. Their effects on weight control and hormone profile have not been previously studied in diet-switched diet-induced obese (DIO) mice, in which treatment is initiated upon cessation of a low-fat diet and resumption of a high-fat diet. Furthermore, their effects on circulating ghrelin in obese humans or in animal models of obesity have not yet been reported. Male C57BI/6J DIO mice after 16 wk on a high-fat diet (HF, 60 kcal% fat) were switched to a low-fat diet (LF, 10 kcal% fat) for 50 d. HF diet resumed concurrently with treatment for 28 d with DEX 3 and 10 mg/kg, twice a day (BID); SIB 5 mg/kg BID; or vehicle. Rapid weight regain ensued in vehicle-treated DIO mice. DEX or SIB treatment significantly blunted the body weight gain. Caloric intake was decreased acutely by DEX or SIB vs vehicle during the first 2 d treatment, but returned to control after 5 d. At the end of study, epididymal fat weight and whole body fat mass determined by DEXA scan were decreased by DEX 10 mg/kg, and whole body lean mass decreased with DEX 3 mg/kg treatment. Circulating ghrelin on d 28 was increased with either DEX 3 or 10 mg/kg treatment, while growth hormone and insulin were decreased. Leptin was also decreased in the DEX 10 mg/kg group. SIB did not significantly affect fat mass, ghrelin, growth hormone, insulin, or leptin. Mice chronically fed LF diet maintained a lower caloric intake, gained less weight and fat mass than diet-switched mice, and had higher ghrelin and lower insulin and leptin. In summary, weight regain in diet-switched DIO mice is delayed with either DEX or SIB treatment. DEX treatment of diet-switched DIO mice decreased growth hormone, insulin, leptin, fat mass, lean mass, and increased ghrelin, while SIB only decreased body weight.  相似文献   
4.
目的:研究运动干预对高脂饲养的小鼠炎性细胞因子和脂肪因子的影响。方法:将SPF级C57BL/6小鼠41只(雄性20只,雌性21只)随机分成正常对照组(NC组,n=11,雄性5只,雌性6只)和高脂饮食组(HFD组, n=30,雄性15只,雌性15只),分别以普通饲料和高脂饲料喂养。9周后,将HFD小鼠25只随机分为肥胖运动组(OE组,n=13,雄性5只,雌性8只)与肥胖对照组(OC组,n=12,雄性5只,雌性7只)。OE组进行为期8周无负重游泳训练。8周后每组随机挑选8只(雄性4只,雌性4只)小鼠,用ELISA法测定其血清脂联素(APN)、网膜素、瘦素、内脂素、C-反应蛋白(CRP)、白细胞介素-6(IL-6)、肿瘤坏死因子α(TNF-α)水平。结果:①OE组与OC组小鼠体重均显著高于NC组(P0.01),OE组体重显著低于OC组(P0.05)。②与NC组比较,OC组小鼠血清瘦素、内脂素、CRP、IL-6、TNF-α水平均显著上升(P0.01);脂联素和网膜素有下降趋势,但差异不显著(P0.05)。与OC组相比,OE组小鼠血清脂联素(P0.05)、网膜素(P0.01)水平显著上升;内脂素水平下降(P0.05);瘦素、CRP、IL-6、TNF-α水平均显著下降(P0.01)。③小鼠血清脂联素与血清IL-6有微弱的负相关关系(P0.05);血清脂联素与网膜素有较弱的正相关关系(P0.05);小鼠血清网膜素与血清CRP有中等程度的负相关关系(P0.01);小鼠血清瘦素、内脂素均与血清IL-6、TNF-α有中等程度的正相关关系(P0.01)。结论:8周游泳运动干预可以通过刺激能抑制炎症因子的脂联素、网膜素的分泌及抑制促炎因子瘦素、内脂素的分泌有效减轻肥胖小鼠炎症反应。  相似文献   
5.
目的:探索复方蘑芋片对降血脂、体重的作用.方法:用蘑芋葡甘聚糖,佐以黄芪浸膏制成0.5 g/片和对照用淀粉片剂(0.5g/片),选200例志愿者认为肥胖和高血脂者,以双盲法分组各100例受试,试前后检测血脂、体重,服药1个月为一个疗程,共观察1~3个疗程.结果:复方蘑芋组在降低血脂方面对胆固醇(TC)及β-脂蛋白(β-LP)降低十分显著,对甘油三酯(TG)也有显著降低;在降低体重方面,有效率93%,对于习惯性便秘也有较好的疗效.结论:复方蘑芋片是一种理想的降血脂、减肥、通便的药品.  相似文献   
1
设为首页 | 免责声明 | 关于勤云 | 加入收藏

Copyright©北京勤云科技发展有限公司  京ICP备09084417号