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排序方式: 共有4439条查询结果,搜索用时 15 毫秒
91.
92.
Susanna Millanta Anna Lisa Furfaro Paolo Carlier Bruno Tasso Mariapaola Nitti Cinzia Domenicotti Patrizio Odetti Maria Adelaide Pronzato Nicola Traverso 《Experimental and molecular pathology》2013
Malondialdehyde (MDA), a major lipid peroxidation product, spontaneously binds to, and modifies proteins. In vivo, proteins are physiologically exposed to micromolar MDA concentrations for long periods. In order to mimic this process in vitro, protein modification is often performed by short exposure to millimolar MDA concentrations, also in order to generate antigenic structures for antibody production. 相似文献
93.
William R. Marchand James N. Lee Susanna Johnson Phillip Gale John Thatcher 《Journal of affective disorders》2013
Background
Objective methods of differentiating unipolar versus bipolar depression would enhance our ability to treat these disorders by providing more accurate diagnoses. One first step towards developing diagnostic methodology is determining whether brain function as assessed by functional MRI (fMRI) and functional connectivity analyses might differentiate the two disorders.Methods
Fourteen subjects with bipolar II depression and 26 subjects with recurrent unipolar depression were studied using fMRI and functional connectivity analyses.Results
The first key finding of this study was that functional connectivity of the right posterior cingulate cortex differentiates bipolar II and unipolar depression. Additionally, results suggest that functional connectivity of this region is associated with suicidal ideation and depression severity in unipolar but not bipolar II depression.Limitations
The primary limitation is the relatively small sample size, particularly for the correlational analyses.Conclusions
The functional connectivity of right posterior cingulate cortex may differential unipolar from bipolar II depression. Further, connectivity of this region may be associated with depression severity and suicide risk in unipolar but not bipolar depression. 相似文献94.
Navas-Sánchez Francisco J. Fernández-Pena Alberto Martín de Blas Daniel Alemán-Gómez Yasser Marcos-Vidal Luís Guzmán-de-Villoria Juan A. Fernández-García Pilar Romero Julia Catalina Irene Lillo Laura Muñoz-Blanco José L. Ordoñez-Ugalde Andrés Quintáns Beatriz Pardo Julio Sobrido María-Jesús Carmona Susanna Grandas Francisco Desco Manuel 《Journal of neurology》2021,268(7):2429-2440
Journal of Neurology - SPG4 is an autosomal dominant pure form of hereditary spastic paraplegia (HSP) caused by mutations in the SPAST gene. HSP is considered an upper motor neuron disorder... 相似文献
95.
Li Tiffany Mizrahi David Goldstein David Kiernan Matthew C. Park Susanna B. 《Neurological sciences》2021,42(10):4109-4121
Neurological Sciences - Chemotherapy-induced peripheral neurotoxicity (CIPN) is a major dose-limiting side effect of many anti-cancer agents, including taxanes, platinums, vinca alkaloids,... 相似文献
96.
97.
Riccardo Masetti Alessandra Tiri Anna Tignanelli Elena Turrini Alberto Argentiero Andrea Pession Susanna Esposito 《Autoimmunity reviews》2021,20(9):102882
In many autoimmune rheumatic diseases, there is an increased risk of cancer compared to the general population. The link between autoimmunity and cancer is dynamic and bidirectional. Recent advances in terms of knowledge of biology, epidemiology, and long-term outcomes for the autoimmune rheumatic diseases have revealed several new connections between these two entities. Data suggest that chronic inflammation from the rheumatic diseases or their therapies may contribute to the onset and promotion of cancer. Conversely, antitumor immune responses may become cross-reactive with self-tissues resulting in the development of autoimmunity. In this review, we discuss about the potential mechanisms that link autoimmune rheumatic diseases and cancer and the association of malignancies with common autoimmune disorders. The increased incidence of malignancy in autoimmune rheumatic diseases has been largely described, although the biology underpinning this relationship should be further investigated. The development of evidence-based cancer screening recommendations in patients with autoimmune rheumatic diseases is complex due to the heterogeneity of clinical rheumatic phenotypes, cancer sites at risk and exposure to anti-neoplastic and anti-rheumatic treatment. In order to lay the foundation of risk stratification and targeted cancer screening, larger longitudinal cohort studies that provide a more detailed framework of the links between cancer and autoimmunity are urgently needed. 相似文献
98.
Ccile Toly‐Ndour Stphanie Huguet‐Jacquot Agns Mailloux Hlne Delaby Giorgia Canellini Martin L. Olsson Agneta Wikman Joke M. Koelewijn Jean‐Marc Minon Tobias J. Legler Frederik B. Clausen Mark Lambert Helen Ryan Irena Bricl Sys Hasslund Agnieszka Orzinska Katarzyna Guz Malgorzata Uhrynowska Antonella Matteocci Nuria Nogues Eduardo Muniz‐Diaz Susanna Sainio Masja De Haas C. Ellen Van der Schoot 《ISBT科学丛刊》2021,16(1):106-118
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Surgical management of early and late ureteral complications after renal transplantation: techniques and outcomes 下载免费PDF全文